65 research outputs found
Chaos synchronization in the multi-feedback Ikeda model
We investigate synchronization between two unidirectionally coupled chaotic
multi-feedback Ikeda systems and find both the existence and stability
conditions for anticipating, lag, and complete synchronizations.Generalization
of the approach to a wide class of nonlinear systems is also presented.Comment: 5 pages. submitte
ΠΠ·Π°ΠΈΠΌΠΎΡΠ²ΡΠ·Ρ Π±ΠΈΠΎΡ ΠΈΠΌΠΈΡΠ΅ΡΠΊΠΈΡ ΠΏΠΎΠΊΠ°Π·Π°ΡΠ΅Π»Π΅ΠΉ ΠΈ Π°Π²ΠΈΠ΄Π½ΠΎΡΡΠΈ Π°Π½ΡΠΈΡΠ΅Π» Ρ Π±ΠΎΠ»ΡΠ½ΡΡ ΡΠ°Ρ Π°ΡΠ½ΡΠΌ Π΄ΠΈΠ°Π±Π΅ΡΠΎΠΌ ΡΠΈΠΏΠ° 2
We evaluated some biochemical and immune parameters, such as IgA, IgM, IgG, CIC, phagocytic index, T-lymphocytes, as well as avidity of antibodies in type 2 diabetic patients.Avidity, high- and low-avid antibody ratio was determined by test-systems for express-diagnostics of clinical and preclinical forms of immunologic deficiency. All patients (n = 47), depending on the level of glycemia and disease duration were divided into 3 groups: in the stage of compensation, subcompensation and decompensation. Control group included 17 healthy subjects.Value of high-avid antibodies in diabetes mellitus patients decreased in 75, 57 and 26% respectively, whereas in healthy subjects it is 89%. Conversely, increased amount of low-avid antibodies was observed, respectively 25, 43, and 74% (11% in control).These parameters prove antibody-folding disorders in diabetes mellitus and show that, change of metabolic parameters affects antibody avidity: G class high-avid antibody level decreases and shows weak protective function. This indicates functional disorders in B-system immunity.ΠΡΠΎΠ²Π΅Π΄Π΅Π½ΠΎ ΠΈΡΡΠ»Π΅Π΄ΠΎΠ²Π°Π½ΠΈΠ΅ Π½Π΅ΠΊΠΎΡΠΎΡΡΡ
Π±ΠΈΠΎΡ
ΠΈΠΌΠΈΡΠ΅ΡΠΊΠΈΡ
ΠΈ ΠΈΠΌΠΌΡΠ½Π½ΡΡ
ΠΏΠΎΠΊΠ°Π·Π°ΡΠ΅Π»Π΅ΠΉ (ΠΈΠΌΠΌΡΠ½ΠΎΠ³Π»ΠΎΠ±ΡΠ»ΠΈΠ½Ρ A, M, G, ΡΠΈΡΠΊΡΠ»ΠΈΡΡΡΡΠΈΠ΅ ΠΈΠΌΠΌΡΠ½Π½ΡΠ΅ ΠΊΠΎΠΌΠΏΠ»Π΅ΠΊΡΡ, ΡΠ°Π³ΠΎΡΠΈΡΠ°ΡΠ½ΠΎΠ΅ ΡΠΈΡΠ»ΠΎ, Π’-Π»ΠΈΠΌΡΠΎΡΠΈΡΡ, Π°Π²ΠΈΠ΄Π½ΠΎΡΡΡ Π°Π½ΡΠΈΡΠ΅Π»). ΠΠ²ΠΈΠ΄ΠΈΡΠ΅Ρ ΠΈ ΡΠΎΠΎΡΠ½ΠΎΡΠ΅Π½ΠΈΠ΅ Π²ΡΡΠΎΠΊΠΎ- ΠΈ Π½ΠΈΠ·ΠΊΠΎΠ°Π²ΠΈΠ΄Π½ΡΡ
Π½ΠΎΡΠΌΠ°Π»ΡΠ½ΡΡ
Π°Π½ΡΠΈΡΠ΅Π» Π² ΡΡΠ²ΠΎΡΠΎΡΠΊΠ΅ ΠΊΡΠΎΠ²ΠΈ ΠΎΠΏΡΠ΅Π΄Π΅Π»ΡΠ»ΠΈ Ρ ΠΏΠΎΠΌΠΎΡΡΡ ΡΠ΅ΡΡ-ΡΠΈΡΡΠ΅ΠΌ Π΄Π»Ρ ΡΠΊΡΠΏΡΠ΅ΡΡ-Π΄ΠΈΠ°Π³Π½ΠΎΡΡΠΈΠΊΠΈ Π΄ΠΎΠΊΠ»ΠΈΠ½ΠΈΡΠ΅ΡΠΊΠΈΡ
ΠΈ ΠΊΠ»ΠΈΠ½ΠΈΡΠ΅ΡΠΊΠΈ Π²ΡΡΠ°ΠΆΠ΅Π½Π½ΡΡ
ΡΠΎΡΠΌ ΠΈΠΌΠΌΡΠ½ΠΎΠ»ΠΎΠ³ΠΈΡΠ΅ΡΠΊΠΎΠΉ Π½Π΅Π΄ΠΎΡΡΠ°ΡΠΎΡΠ½ΠΎΡΡΠΈ. ΠΡΠ΅ Π±ΠΎΠ»ΡΠ½ΡΠ΅ (47 ΡΠ΅Π»ΠΎΠ²Π΅ΠΊ) Π² Π·Π°Π²ΠΈΡΠΈΠΌΠΎΡΡΠΈ ΠΎΡ ΡΡΠΎΠ²Π½Ρ Π³Π»ΠΈΠΊΠ΅ΠΌΠΈΠΈ ΠΈ ΠΏΡΠΎΠ΄ΠΎΠ»ΠΆΠΈΡΠ΅Π»ΡΠ½ΠΎΡΡΠΈ Π·Π°Π±ΠΎΠ»Π΅Π²Π°Π½ΠΈΡ Π±ΡΠ»ΠΈ ΡΠ°Π·Π΄Π΅Π»Π΅Π½Ρ Π½Π° ΡΡΠΈ Π³ΡΡΠΏΠΏΡ: Π½Π°Ρ
ΠΎΠ΄ΡΡΠΈΠ΅ΡΡ Π² ΡΡΠ°Π΄ΠΈΠΈ ΠΊΠΎΠΌΠΏΠ΅Π½ΡΠ°ΡΠΈΠΈ, ΡΡΠ±ΠΊΠΎΠΌΠΏΠ΅Π½ΡΠ°ΡΠΈΠΈ ΠΈ Π΄Π΅ΠΊΠΎΠΌΠΏΠ΅Π½ΡΠ°ΡΠΈΠΈ. Π ΠΊΠ°ΡΠ΅ΡΡΠ²Π΅ ΠΊΠΎΠ½ΡΡΠΎΠ»Ρ ΡΠ»ΡΠΆΠΈΠ»ΠΈ 17 Π·Π΄ΠΎΡΠΎΠ²ΡΡ
Π΄ΠΎΠ½ΠΎΡΠΎΠ². Π£ Π±ΠΎΠ»ΡΠ½ΡΡ
ΡΠ°Ρ
Π°ΡΠ½ΡΠΌ Π΄ΠΈΠ°Π±Π΅ΡΠΎΠΌ (Π‘Π) Π½Π°Π±Π»ΡΠ΄Π°Π»ΠΎΡΡ ΡΠ½ΠΈΠΆΠ΅Π½ΠΈΠ΅ ΡΠΎΠ΄Π΅ΡΠΆΠ°Π½ΠΈΡ Π²ΡΡΠΎΠΊΠΎΠ°Π²ΠΈΠ΄Π½ΡΡ
Π°Π½ΡΠΈΡΠ΅Π» Π΄ΠΎ 75, 57 ΠΈ 26% ΡΠΎΠΎΡΠ²Π΅ΡΡΡΠ²Π΅Π½Π½ΠΎ, ΡΠΎΠ³Π΄Π° ΠΊΠ°ΠΊ Ρ Π·Π΄ΠΎΡΠΎΠ²ΡΡ
Π΄ΠΎΠ½ΠΎΡΠΎΠ² ΡΡΠΎΡ ΠΏΠΎΠΊΠ°Π·Π°ΡΠ΅Π»Ρ Π±ΡΠ» ΡΠ°Π²Π΅Π½ 89%. ΠΠ°ΠΎΠ±ΠΎΡΠΎΡ, ΡΠΎΠ΄Π΅ΡΠΆΠ°Π½ΠΈΠ΅ Π½ΠΈΠ·ΠΊΠΎΠ°Π²ΠΈΠ΄Π½ΡΡ
Π°Π½ΡΠΈΡΠ΅Π» Π½Π°ΡΠ°ΡΡΠ°Π΅Ρ ΡΠΎΠΎΡΠ²Π΅ΡΡΡΠ²Π΅Π½Π½ΠΎ Π΄ΠΎ 25, 43, 74% (Π² ΠΊΠΎΠ½ΡΡΠΎΠ»Π΅ 11%). Π£ Π±ΠΎΠ»ΡΠ½ΡΡ
Π‘Π ΠΈΠ·ΠΌΠ΅Π½Π΅Π½ΠΈΡ ΠΌΠ΅ΡΠ°Π±ΠΎΠ»ΠΈΡΠ΅ΡΠΊΠΈΡ
ΠΏΠΎΠΊΠ°Π·Π°ΡΠ΅Π»Π΅ΠΉ ΠΎΡΡΠΈΡΠ°ΡΠ΅Π»ΡΠ½ΠΎ Π²Π»ΠΈΡΡΡ Π½Π° Π°Π²ΠΈΠ΄Π½ΠΎΡΡΡ Π°Π½ΡΠΈΡΠ΅Π»: ΠΏΡΠΎΠΈΡΡ
ΠΎΠ΄ΠΈΡ ΡΠ΅Π·ΠΊΠΎΠ΅ ΡΠ½ΠΈΠΆΠ΅Π½ΠΈΠ΅ Π²ΡΡΠΎΠΊΠΎΠ°Π²ΠΈΠ΄Π½ΡΡ
Π°Π½ΡΠΈΡΠ΅Π» ΠΊΠ»Π°ΡΡΠ° G Ρ Π°Π½ΠΎΠΌΠ°Π»ΡΠ½ΠΎΠΉ ΠΊΠΎΠ½ΡΠΎΡΠΌΠ°ΡΠΈΠ΅ΠΉ ΠΈ Π½ΠΈΠ·ΠΊΠΎΠΉ ΡΡΠ½ΠΊΡΠΈΠΎΠ½Π°Π»ΡΠ½ΠΎΠΉ ΠΈ ΠΏΡΠΎΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΡΠΌΠΈ, ΡΡΠΎ ΡΠ²ΠΈΠ΄Π΅ΡΠ΅Π»ΡΡΡΠ²ΡΠ΅Ρ ΠΎ Π½Π°ΡΡΡΠ΅Π½ΠΈΠΈ ΠΏΡΠΎΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΠΉ ΡΠΈΡΡΠ΅ΠΌΡ ΡΡΠ½ΠΊΡΠΈΠΈ Π-ΡΠΈΡΡΠ΅ΠΌΡ ΠΈΠΌΠΌΡΠ½ΠΈΡΠ΅ΡΠ°
Association between breastfeeding, host genetic factors, and calicivirus gastroenteritis in a Nicaraguan birth cohort
Background Norovirus and sapovirus are important causes of childhood acute gastroenteritis (AGE). Breastfeeding prevents AGE generally; however, it is unknown if breastfeeding prevents AGE caused specifically by norovirus and sapovirus. Methods We investigated the association between breastfeeding and norovirus or sapovirus AGE episodes in a birth cohort. Weekly data on breastfeeding and AGE episodes were captured during the first year of life. Stools were collected from children with AGE and tested by RT-qPCR for norovirus and sapovirus. Time-dependent Cox models estimated associations between weekly breastfeeding and time to first norovirus or sapovirus AGE. Findings From June 2017 to July 2018, 444 newborns were enrolled in the study. In the first year of life, 69 and 34 children experienced a norovirus and a sapovirus episode, respectively. Exclusive breastfeeding lasted a median of 2 weeks, and any breastfeeding lasted a median of 43 weeks. Breastfeeding in the last week did not prevent norovirus (HR: 1.09, 95% CI: 0.62, 1.92) or sapovirus (HR: 1.00, 95% CI: 0.82, 1.21) AGE in a given week, adjusting for household sanitation, consumption of high-risk foods, and motherβs and childβs histo-blood group phenotypes. Maternal secretor-positive phenotype was protective against norovirus AGE, whereas childβs secretor-positive phenotype was a risk factor for norovirus AGE. Interpretation Exclusive breastfeeding in this population was short-lived, and no conclusions could be drawn about its potential to prevent norovirus or sapovirus AGE. Non-exclusive breastfeeding did not prevent norovirus or sapovirus AGE in the first year of life. However, maternal secretor-positive phenotype was associated with a reduced hazard of norovirus AGE
ΠΠ΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½Π°Ρ Π°ΠΊΡΠΈΠ²Π½ΡΡΡΡ ΡΠΈΡΠΎΠ²Π°ΡΠΊΠΈ ΠΊΡΠΎΠ²Ρ Ρ ΡΡΠ²Π΅Π½Ρ Π΅Π½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΡ ΡΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΡΡ Ρ Π΄ΡΡΠ΅ΠΉ Π· Π³ΠΎΡΡΡΠΈΠΌ ΡΡΠ΅ΡΠΈΡ ΠΈΠΎΠ·ΠΎΠΌ, Π²ΠΈΠΊΠ»ΠΈΠΊΠ°Π½ΠΈΠΌ ΡΠ½ΡΠ΅ΡΠΎΡΠΎΠΊΡΠΈΠ³Π΅Π½Π½ΡΠΌΠΈ Escherichia coli
Objective.Β To identify the features of a betalactamase activity in the blood serum and the levels of endogenous intoxication in children with acute ehsheri hioza caused by enterotoxigenic strains of Escherichia coli.Patients and methods.Β A total of 28 healthy children in the age from 1 to 7 years and 210 children with acute ehsherihioza of moderate and severe course were under observation. Determination of betalactamase activity in the blood serum and its quantitative evaluation was performed with the use of Β«BioLaktamΒ» testsystem. The study of endogenous intoxication was performed by the way of determining of the level of intermediate molecules of average mass in the blood plasma.Results.Β In healthy and most children with ehsherihioza in the blood serum were found the factors with betalactamase activity, the level of which is progres sively decreased with increasing severity of the disease and an increase in the circulation of the contents of the intermediate molecules of average mass.Conclusions.Β In patients was marked the increase in the level of endogenous intoxication. It is characterized by excessive accumulation of the intermediate molecules in the circulation of average mass. The increase in the level of the intermediate molecules of average mass was accompanied by a decrease in the average betalactamase activity of blood serum.Key words:Β betalactamase activity of blood serum, acute ehsherihioza, Escherichia coli, endogenous intoxication.Π¦Π΅Π»Ρ:Β Π²ΡΡΠ²ΠΈΡΡ ΠΎΡΠΎΠ±Π΅Π½Π½ΠΎΡΡΠΈ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΠΈ ΡΡΠ²ΠΎΡΠΎΡΠΊΠΈ ΠΊΡΠΎΠ²ΠΈ ΠΈ ΡΡΠΎΠ²Π½Ρ ΡΠ½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΠΉ ΠΈΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΠΈΠΈ Ρ Π΄Π΅ΡΠ΅ΠΉ Ρ ΠΎΡΡΡΡΠΌ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·ΠΎΠΌ, Π²ΡΠ·Π²Π°Π½Π½ΡΠΌ ΡΠ½ΡΠ΅ΡΠΎΡΠΎΠΊΡΠΈΠ³Π΅Π½Π½ΡΠΌΠΈ ΡΡΠ°ΠΌΠΌΠ°ΠΌΠΈ Escherichia coli.ΠΠ°ΡΠΈΠ΅Π½ΡΡ ΠΈ ΠΌΠ΅ΡΠΎΠ΄Ρ.Β ΠΠΎΠ΄ Π½Π°Π±Π»ΡΠ΄Π΅Π½ΠΈΠ΅ΠΌ Π½Π°Ρ
ΠΎΠ΄ΠΈΠ»ΠΈΡΡ 28 ΠΏΡΠ°ΠΊΡΠΈΡΠ΅ΡΠΊΠΈ Π·Π΄ΠΎΡΠΎΠ²ΡΡ
Π΄Π΅ΡΠ΅ΠΉ Π² Π²ΠΎΠ·ΡΠ°ΡΡΠ΅ ΠΎΡ 1 Π΄ΠΎ 7 Π»Π΅Ρ ΠΈ 210 Π΄Π΅ΡΠ΅ΠΉ Ρ ΠΎΡΡΡΡΠΌ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·ΠΎΠΌ ΡΡΠ΅Π΄Π½Π΅ΡΡΠΆΠ΅Π»ΠΎΠ³ΠΎ ΠΈ ΡΡΠΆΠ΅Π»ΠΎΠ³ΠΎ ΡΠ΅ΡΠ΅Π½ΠΈΡ. ΠΠΏΡΠ΅Π΄Π΅Π»Π΅Π½ΠΈΠ΅ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΠΈ Π² ΡΡΠ²ΠΎΡΠΎΡΠΊΠ΅ ΠΊΡΠΎΠ²ΠΈ ΠΈ Π΅Π΅ ΠΊΠΎΠ»ΠΈΡΠ΅ΡΡΠ²Π΅Π½Π½ΡΡ ΠΎΡΠ΅Π½ΠΊΡ ΠΏΡΠΎΠ²ΠΎΠ΄ΠΈΠ»ΠΈ Ρ ΠΈΡΠΏΠΎΠ»ΡΠ·ΠΎΠ²Π°Π½ΠΈΠ΅ΠΌ ΡΠ΅ΡΡ-ΡΠΈΡΡΠ΅ΠΌΡ Β«ΠΠΈΠΎΠΠ°ΠΊΡΠ°ΠΌΒ». ΠΡΡΠ»Π΅Π΄ΠΎΠ²Π°Π½ΠΈΠ΅ ΡΠ½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΠΉ ΠΈΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΠΈΠΈ ΠΏΡΠΎΠ²ΠΎΠ΄ΠΈΠ»ΠΈ ΠΏΡΡΠ΅ΠΌ ΠΎΠΏΡΠ΅Π΄Π΅Π»Π΅Π½ΠΈΡ ΡΡΠΎΠ²Π½Ρ ΠΏΡΠΎΠΌΠ΅ΠΆΡΡΠΎΡΠ½ΡΡ
ΠΌΠΎΠ»Π΅ΠΊΡΠ» ΡΡΠ΅Π΄Π½Π΅ΠΉ ΠΌΠ°ΡΡΡ Π² ΠΏΠ»Π°Π·ΠΌΠ΅ ΠΊΡΠΎΠ²ΠΈ.Π Π΅Π·ΡΠ»ΡΡΠ°ΡΡ.Β Π£ Π·Π΄ΠΎΡΠΎΠ²ΡΡ
ΠΈ Π±ΠΎΠ»ΡΡΠΈΠ½ΡΡΠ²Π° Π΄Π΅ΡΠ΅ΠΉ Ρ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·ΠΎΠΌ Π² ΡΡΠ²ΠΎΡΠΎΡΠΊΠ΅ ΠΊΡΠΎΠ²ΠΈ ΠΎΠ±Π½Π°ΡΡΠΆΠ΅Π½Ρ ΡΠ°ΠΊΡΠΎΡΡ, ΠΎΠ±Π»Π°Π΄Π°ΡΡΠΈΠ΅ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΡΡ, ΡΡΠΎΠ²Π΅Π½Ρ ΠΊΠΎΡΠΎΡΠΎΠΉ ΠΏΡΠΎΠ³ΡΠ΅ΡΡΠΈΠ²Π½ΠΎ ΡΠ½ΠΈΠΆΠ°Π»ΡΡ Ρ Π²ΠΎΠ·ΡΠ°ΡΡΠ°Π½ΠΈΠ΅ΠΌ ΡΡΠΆΠ΅ΡΡΠΈ ΡΠ΅ΡΠ΅Π½ΠΈΡΒ Π·Π°Π±ΠΎΠ»Π΅Π²Π°Π½ΠΈΡ ΠΈ ΡΠ²Π΅Π»ΠΈΡΠ΅Π½ΠΈΠ΅ΠΌ Π² ΡΠΈΡΠΊΡΠ»ΡΡΠΈΠΈ ΡΠΎΠ΄Π΅ΡΠΆΠ°Π½ΠΈΡ ΠΏΡΠΎΠΌΠ΅ΠΆΡΡΠΎΡΠ½ΡΡ
ΠΌΠΎΠ»Π΅ΠΊΡΠ» ΡΡΠ΅Π΄Π½Π΅ΠΉ ΠΌΠ°ΡΡΡ.ΠΡΠ²ΠΎΠ΄Ρ.Β Π£ Π±ΠΎΠ»ΡΠ½ΡΡ
ΠΎΡΠΌΠ΅ΡΠ΅Π½ΠΎ Π²ΠΎΠ·ΡΠ°ΡΡΠ°Π½ΠΈΠ΅ ΡΡΠΎΠ²Π½Ρ ΡΠ½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΠΉ ΠΈΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΠΈΠΈ, Ρ
Π°ΡΠ°ΠΊΡΠ΅ΡΠΈΠ·ΡΡΡΠ΅Π΅ΡΡ ΠΈΠ·Π±ΡΡΠΎΡΠ½ΡΠΌ Π½Π°ΠΊΠΎΠΏΠ»Π΅Π½ΠΈΠ΅ΠΌ Π² ΡΠΈΡΠΊΡΠ»ΡΡΠΈΠΈ ΠΏΡΠΎΠΌΠ΅ΠΆΡΡΠΎΡΠ½ΡΡ
ΠΌΠΎΠ»Π΅ΠΊΡΠ» ΡΡΠ΅Π΄Π½Π΅ΠΉ ΠΌΠ°ΡΡΡ. ΠΠΎΠ·ΡΠ°ΡΡΠ°Π½ΠΈΠ΅ ΡΡΠΎΠ²Π½Ρ ΠΠΠ‘Π ΡΠΎΠΏΡΠΎΠ²ΠΎΠΆΠ΄Π°Π»ΠΎΡΡ ΡΠ½ΠΈΠΆΠ΅Π½ΠΈΠ΅ΠΌ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΠΈ ΡΡΠ²ΠΎΡΠΎΡΠΊΠΈ ΠΊΡΠΎΠ²ΠΈ.ΠΠ»ΡΡΠ΅Π²ΡΠ΅ ΡΠ»ΠΎΠ²Π°:Β Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½Π°Ρ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΡ ΡΡΠ²ΠΎΡΠΎΡΠΊΠΈ ΠΊΡΠΎΠ²ΠΈ, ΠΎΡΡΡΡΠΉ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·, Escherichia coli, ΡΠ½Π΄ΠΎΠ³Π΅Π½Π½Π°Ρ ΠΈΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΠΈΡ.ΠΠ΅ΡΠ°:Β Π²ΠΈΡΠ²ΠΈΡΠΈ ΠΎΡΠΎΠ±Π»ΠΈΠ²ΠΎΡΡΡ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΡ ΡΠΈΡΠΎΠ²Π°ΡΠΊΠΈ ΠΊΡΠΎΠ²Ρ Ρ ΡΡΠ²Π½Ρ Π΅Π½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΡ ΡΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΡΡ Ρ Π΄ΡΡΠ΅ΠΉ Π· Π³ΠΎΡΡΡΠΈΠΌ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·ΠΎΠΌ, Π²ΠΈΠΊΠ»ΠΈΠΊΠ°Π½ΠΈΠΌ ΡΠ½ΡΠ΅ΡΠΎΡΠΎΠΊΡΠΈΠ³Π΅Π½Π½ΡΠΌΠΈ ΡΡΠ°ΠΌΠ°ΠΌΠΈ Escherichia coli.ΠΠ°ΡΡΡΠ½ΡΠΈ Ρ ΠΌΠ΅ΡΠΎΠ΄ΠΈ.Β ΠΡΠ΄ Π½Π°Π³Π»ΡΠ΄ΠΎΠΌ Π·Π½Π°Ρ
ΠΎΠ΄ΠΈΠ»ΠΈΡΡ 28 ΠΏΡΠ°ΠΊΡΠΈΡΠ½ΠΎ Π·Π΄ΠΎΡΠΎΠ²ΠΈΡ
Π΄ΡΡΠ΅ΠΉ Ρ Π²ΡΡΡ Π²ΡΠ΄ 1 Π΄ΠΎ 7 ΡΠΎΠΊΡΠ² Ρ 210 Π΄ΡΡΠ΅ΠΉ Π· Π³ΠΎΡΡΡΠΈΠΌ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·ΠΎΠΌ ΡΡΠ΅Π΄Π½Π΅ΡΡΠΆΠ΅Π»ΠΎΠ³ΠΎ Ρ Π²Π°ΠΆΠΊΠΎΡ ΡΠ΅ΡΡΡ. ΠΠΈΠ·Π½Π°ΡΠ΅Π½Π½Ρ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΡ Π² ΡΠΈΡΠΎΠ²Π°ΡΡΡ ΠΊΡΠΎΠ²Ρ Ρ ΡΡ ΠΊΡΠ»ΡΠΊΡΡΠ½Ρ ΠΎΡΡΠ½ΠΊΡ ΠΏΡΠΎΠ²ΠΎΠ΄ΠΈΠ»ΠΈ Π· Π²ΠΈΠΊΠΎΡΠΈΡΡΠ°Π½Π½ΡΠΌ ΡΠ΅ΡΡ-ΡΠΈΡΡΠ΅ΠΌΠΈ "ΠΡΠΎΠ»Π°ΠΊΡΠ°ΠΌ". ΠΠΎΡΠ»ΡΠ΄ΠΆΠ΅Π½Π½Ρ Π΅Π½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΡ ΡΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΡΡ ΠΏΡΠΎΠ²ΠΎΠ΄ΠΈΠ»ΠΈ ΡΠ»ΡΡ
ΠΎΠΌ Π²ΠΈΠ·Π½Π°ΡΠ΅Π½Π½Ρ ΡΡΠ²Π½Ρ ΠΏΡΠΎΠΌΡΠΆΠ½ΠΈΡ
ΠΌΠΎΠ»Π΅ΠΊΡΠ» ΡΠ΅ΡΠ΅Π΄Π½ΡΠΎΡ ΠΌΠ°ΡΠΈ Π² ΠΏΠ»Π°Π·ΠΌΡ ΠΊΡΠΎΠ²Ρ.Π Π΅Π·ΡΠ»ΡΡΠ°ΡΠΈ.Β Π£ Π·Π΄ΠΎΡΠΎΠ²ΠΈΡ
Ρ Π±ΡΠ»ΡΡΡΡΡΡ Π΄ΡΡΠ΅ΠΉ Π· ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·ΠΎΠΌ Π² ΡΠΈΡΠΎΠ²Π°ΡΡΡ ΠΊΡΠΎΠ²Ρ Π²ΠΈΡΠ²Π»Π΅Π½Π° ΡΠΈΠ½Π½ΠΈΠΊΠΈ, ΡΠΎ ΠΌΠ°ΡΡΡ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΠΉ Π°ΠΊΡΠΈΠ²Π½ΡΡΡΡ, ΡΡΠ²Π΅Π½Ρ ΡΠΊΠΎΡ ΠΏΡΠΎΠ³ΡΠ΅ΡΠΈΠ²Π½ΠΎ Π·Π½ΠΈΠΆΡΠ²Π°Π²ΡΡ ΡΠ· Π·ΡΠΎΡΡΠ°Π½Π½ΡΠΌ ΡΡΠΆΠΊΠΎΡΡΡ ΠΏΠ΅ΡΠ΅Π±ΡΠ³Ρ Π·Π°Ρ
Π²ΠΎΡΡΠ²Π°Π½Π½Ρ Ρ Π·Π±ΡΠ»ΡΡΠ΅Π½Π½ΡΠΌ Π² ΡΠΈΡΠΊΡΠ»ΡΡΡΡ Π·ΠΌΡΡΡΡ ΠΏΡΠΎΠΌΡΠΆΠ½ΠΈΡ
ΠΌΠΎΠ»Π΅ΠΊΡΠ» ΡΠ΅ΡΠ΅Π΄Π½ΡΠΎΡ ΠΌΠ°ΡΠΈ.ΠΠΈΡΠ½ΠΎΠ²ΠΊΠΈ.Β Π£ Ρ
Π²ΠΎΡΠΈΡ
Π²ΡΠ΄ΠΌΡΡΠ΅Π½ΠΎ Π·ΡΠΎΡΡΠ°Π½Π½Ρ ΡΡΠ²Π½Ρ Π΅Π½Π΄ΠΎΠ³Π΅Π½Π½ΠΎΡ ΡΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΡΡ, ΡΠΎ Ρ
Π°ΡΠ°ΠΊΡΠ΅ΡΠΈΠ·ΡΡΡΡΡΡ Π½Π°Π΄ΠΌΡΡΠ½ΠΈΠΌ Π½Π°ΠΊΠΎΠΏΠΈΡΠ΅Π½Π½ΡΠΌ Π² ΡΠΈΡΠΊΡΠ»ΡΡΡΡ ΠΏΡΠΎΠΌΡΠΆΠ½ΠΈΡ
ΠΌΠΎΠ»Π΅ΠΊΡΠ» ΡΠ΅ΡΠ΅Π΄Π½ΡΠΎΡ ΠΌΠ°ΡΠΈ. ΠΡΠΎΡΡΠ°Π½Π½Ρ ΡΡΠ²Π½Ρ ΠΠΠ‘Π ΡΡΠΏΡΠΎΠ²ΠΎΠ΄ΠΆΡΠ²Π°Π»ΠΎΡΡ Π·Π½ΠΈΠΆΠ΅Π½Π½ΡΠΌ Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½ΠΎΡ Π°ΠΊΡΠΈΠ²Π½ΠΎΡΡΡ ΡΠΈΡΠΎΠ²Π°ΡΠΊΠΈ ΠΊΡΠΎΠ²Ρ.ΠΠ»ΡΡΠΎΠ²Ρ ΡΠ»ΠΎΠ²Π°:Β Π±Π΅ΡΠ°-Π»Π°ΠΊΡΠ°ΠΌΠ°Π·Π½Π° Π°ΠΊΡΠΈΠ²Π½ΡΡΡΡ ΡΠΈΡΠΎΠ²Π°ΡΠΊΠΈ ΠΊΡΠΎΠ²Ρ, Π³ΠΎΡΡΡΠΈΠΉ ΡΡΠ΅ΡΠΈΡ
ΠΈΠΎΠ·, Escherichia coli, Π΅Π½Π΄ΠΎΠ³Π΅Π½Π½Π° ΡΠ½ΡΠΎΠΊΡΠΈΠΊΠ°ΡΡΡ
ENDOCRINE DISEASES IN PRACTICE OF A FAMILY DOCTOR AND DENTIST
<p>This article consideres an important role of collaboration between a family doctor and a dentist in the prevention of oral manifestations of type 2 diabetes mellitus. Diabetes mellitus is currently a very wide spread endocrine disease. It has many oral manifestations. Very often, diabetes mellitus is diagnosed accidentally; patients may not pay attention to the symptoms, especially oral signs until they become more serious. Diabetes mellitus is usually accompanied by dental diseases, so the dentist and family doctor need to know all the possible initial oral manifestations of this terrible disease in order to make a timely diagnosis [1]. Currently, the prevention and treatment of diabetes mellitus requires the Ρonjoint efforts of a family doctor and a dentist [2].</p>
METHODS OF ORTHOPEDIC TREATMENT OF PATIENTS WITH DISORDERS OF THE TEMPOROMANDIBULAR JOINT
<p>Untimely provision of orthopedic care to the population in the presence of defects in the dentition leads to the emergence of complex clinical situations caused by the development of dentoalveolar deformities. This aspect predetermines the severity in the diagnosis and treatment of patients with dentition defects.</p>
RM-059 The effect of adjuvant chemotherapy following extended gastrectomy for Siewert's type II adenocarcinoma of the esophagogastric junction
THE ROLE OF A FAMILY DOCTOR AND DENTIST IN THE PREVENTION OF LUPUS ERYTHEMATOSUS
<p>This article provides an overview of role of the family doctor and dentist in the prevention of chronic lupus erythematosus. One of the most important problems of modern medicine is diseases of the oral mucosa. Despite the fact that a disease such as lupus erythematosus is an autoimmune and rheumatic disease, it is also manifested in the oral cavity, which develops the prerequisites for cooperation between a family doctor and a dentist [1, 6-10] A characteristic feature of chronic lupus erythematosus is the diversity of first manifestations course and clinical manifestations. Typically, the disease begins with one or more symptoms. Clinical picture at the onset of the disease may differ strikingly from the "classical" descriptions of this disease, which often causes diagnostic difficulties not only for general practitioners, but also for rheumatologists [5]. According to 2010 data, the prevalence of the disease in the world it is 3.3β8.8 per 100, 000 children [1]. Lupus erythematosus affects mainly girls and young women, approximately 80% of patients with lupus erythematosus are women. Lupus erythematosus rarely begins in children under 5 years of age, an increase in incidence is observed from the age of 8β9 years, and the culmination occurs at the age of 14β25 years (average age of beginning is - 11β12 years) [1, 3, 4, 12]. Currently, family doctors and dentists are increasingly encountering this pathology, and therefore methods of its treatment are being improved, and much attention is paid to methods of preventing this disease [6, 7, 10, 11].</p>
Editorial for the special issue on photosynthesis and hydrogen energy research for sustainabilityβ2019
The relationship between biochemical parameters and antibody avidity in diabetes mellitus type 2 patients
We evaluated some biochemical and immune parameters, such as IgA, IgM, IgG, CIC, phagocytic index, T-lymphocytes, as well as avidity of antibodies in type 2 diabetic patients.Avidity, high- and low-avid antibody ratio was determined by test-systems for express-diagnostics of clinical and preclinical forms of immunologic deficiency. All patients (n = 47), depending on the level of glycemia and disease duration were divided into 3 groups: in the stage of compensation, subcompensation and decompensation. Control group included 17 healthy subjects.Value of high-avid antibodies in diabetes mellitus patients decreased in 75, 57 and 26% respectively, whereas in healthy subjects it is 89%. Conversely, increased amount of low-avid antibodies was observed, respectively 25, 43, and 74% (11% in control).These parameters prove antibody-folding disorders in diabetes mellitus and show that, change of metabolic parameters affects antibody avidity: G class high-avid antibody level decreases and shows weak protective function. This indicates functional disorders in B-system immunity
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