Distribution of <i>FCN2</i> genotypes and alleles among visceral leishmaniasis cases and healthy controls.

<p>Note: CI, confidence interval; OR, odds ratio; NS, not significant; NA, not applicable. Percentage may not add up to 100 due to rounding errors</p><p>^# Adjusted <i>P</i> values for age, gender and ethnicity</p><p>Distribution of <i>FCN2</i> genotypes and alleles among visceral leishmaniasis cases and healthy controls.</p

Association of functional <i>FCN2</i> haplotypes and visceral leishmaniasis.

<p>Note: CI, confidence interval; OR, odds ratio; NS, not significant; NA, not applicable. Percentage may not add up to 100 due to rounding errors</p><p>^#Adjusted <i>P</i> values for age, gender and ethnicity</p><p>Association of functional <i>FCN2</i> haplotypes and visceral leishmaniasis.</p

Distribution of ficolin-2 serum levels with +6359C>T variant in controls.

<p>Box-plots illustrate medians with 25 and 75 percentiles with whiskers to 10 and 90 percentiles. Ficolin-2 serum levels were measured and separated based on different genotypes of <i>FCN2</i> variant +6359C>T. <i>P =</i> 0.03 illustrated in the figure is calculated by Kruskal-Wallis rank sum test.</p

Mutations in the <i>S</i> gene in occult hepatitis B and HBsAg positive patients.

<p>Mutations in the <i>S</i> gene in occult hepatitis B and HBsAg positive patients.</p

Reconstructed phylogenetic tree of <i>preC/C</i> region in the HBV genome.

<p>Phylogenetic analysis inferred from distance analysis (Kimura 2 parameters model) and neighbor-joining reconstruction from <i>preC/C</i> region of OBI sample sequences showing that the HBV sequences mostly clustered in the HBV genotype E branch. HBV sequences are referred to as “number”, i.e., “017”. The HBV sequences were compared to HBV reference sequences gathering the 8 HBV genotypes (NCBI-Genbank accession numbers are denoted). The numbers at the nodes indicate bootstrapping values in percentage of 1000 replicates.</p

Reconstructed phylogenetic tree of <i>preS/S</i> region of the HBV genome.

<p>Phylogenetic analysis inferred from distance analysis (Kimura 2 parameters model) and neighbor-joining reconstruction from <i>preS/S</i> region of OBI sample sequences showing that the HBV sequences clustered in the HBV genotype E branch. HBV sequences are referred to as “number”, i.e., “016”. The HBV sequences were compared to HBV reference sequences gathering the 8 HBV genotypes (NCBI-Genbank accession numbers are denoted). The numbers at the nodes indicate bootstrapping values in percentage of 1000 replicates.</p

Reconstructed phylogenetic tree of <i>preC/C</i> region in the HBV genome.

<p>Phylogenetic analysis inferred from distance analysis (Kimura 2 parameters model) and neighbor-joining reconstruction from <i>preC/C</i> region of OBI sample sequences showing that the HBV sequences mostly clustered in the HBV genotype E branch. HBV sequences are referred to as “number”, i.e., “017”. The HBV sequences were compared to HBV reference sequences gathering the 8 HBV genotypes (NCBI-Genbank accession numbers are denoted). The numbers at the nodes indicate bootstrapping values in percentage of 1000 replicates.</p

Mutations in the RT domain of the <i>P</i> gene in occult hepatitis B and HBsAg positive patients.

<p>Mutations in the RT domain of the <i>P</i> gene in occult hepatitis B and HBsAg positive patients.</p

Mutations in the <i>S</i> gene in occult hepatitis B and HBsAg positive patients.

<p>Mutations in the <i>S</i> gene in occult hepatitis B and HBsAg positive patients.</p

Baseline characteristics of occult hepatitis B and HBsAg positive patients.

<p>Baseline characteristics of occult hepatitis B and HBsAg positive patients.</p