Comparison of Planted Loblolly and Slash Pine Performance in Southeast Texas

The performance of young (less than 10 yr) loblolly (Pinus taeda L.) and slash (Pinus elliottii Engelm.) pine trees was compared on planted sites in southeast Texas. Performance was compared for: total tree height; tree diameter; heigh to live tree crown; tree volume index,; incidence of fusiform rust (Cronartium quercuum [Berk.] Miyabe ex Shira if. sp. fusiforme); crookedness of stems; and survival rates. For these young trees, slash pine tended to perform better in southeast Texas than loblolly pine in total tree height, tree diameter, stem size, height to first live branch and stem straightness. However, loblolly pine was less susceptible to fusiform rust than slash pine, and its mortality rate was lower than slash pine. However, based on the performance of these young plantations, a recommendation the preferred pine species to plant in southeast Texas might be premature. South. J. Appl. For. 17(1): 26-31

Finite abundant semigroups in which the idempotents form a subsemigroup

We consider certain abundant semigroups in which the idempotents form a subsemigroup, and which we call bountiful semigroups. We find a simple criterion for a finite bountiful semigroup to be a member of the join of the pseudovarieties of finite groups and finite aperiodic semigroups

Pathogenic variants in USP7 cause a neurodevelopmental disorder with speech delays, altered behavior, and neurologic anomalies.

Purpose: Haploinsufficiency of USP7, located at chromosome 16p13.2, has recently been reported in seven individuals with neurodevelopmental phenotypes, including developmental delay/intellectual disability (DD/ID), autism spectrum disorder (ASD), seizures, and hypogonadism. Further, USP7 was identified to critically incorporate into the MAGEL2-USP7-TRIM27 (MUST), such that pathogenic variants in USP7 lead to altered endosomal Factin polymerization and dysregulated protein recycling.Methods: We report 16 newly identified individuals with heterozygous USP7 variants, identified by genome or exome sequencing or by chromosome microarray analysis. Clinical features were evaluated by review of medical records. Additional clinical information was obtained on the seven previously reported individuals to fully elucidate the phenotypic expression associated with USP7 haploinsufficiency.Results: The clinical manifestations of these 23 individuals suggest a syndrome characterized by DD/ID, hypotonia, eye anomalies, feeding difficulties, GERD, behavioral anomalies, and ASD, and more specific phenotypes of speech delays including a nonverbal phenotype and abnormal brain magnetic resonance image findings including white matter changes based on neuroradiologic examination.Conclusion: The consistency of clinical features among all individuals presented regardless of de novo USP7 variant type supports haploinsufficiency as a mechanism for pathogenesis and refines the clinical impact faced by affected individuals and caregivers