A Time-Evolving 3D Method Dedicated to the Reconstruction of Solar plumes and Results Using Extreme Ultra-Violet Data

An important issue in the tomographic reconstruction of the solar poles is the relatively rapid evolution of the polar plumes. We demonstrate that it is possible to take into account this temporal evolution in the reconstruction. The difficulty of this problem comes from the fact that we want a 4D reconstruction (three spatial dimensions plus time) while we only have 3D data (2D images plus time). To overcome this difficulty, we introduce a model that describes polar plumes as stationary objects whose intensity varies homogeneously with time. This assumption can be physically justified if one accepts the stability of the magnetic structure. This model leads to a bilinear inverse problem. We describe how to extend linear inversion methods to these kinds of problems. Studies of simulations show the reliability of our method. Results for SOHO/EIT data show that we are able to estimate the temporal evolution of polar plumes in order to improve the reconstruction of the solar poles from only one point of view. We expect further improvements from STEREO/EUVI data when the two probes will be separated by about 60 degrees

The Solar Radius in the EUV during the Cycle XXIII

Aims. To determine the solar transition region and coronal radius at EUV wavelengths and its time evolution during Solar Cycle XXIII. Methods. We use daily 30.4 and 17.1 nm images obtained by the Extreme Ultraviolet Imager (EIT) aboard the SoHO satellite and derive the solar radius by fitting a circle to the limb brightness ring. Results. The weighted mean of the temporal series gives (967''.56 +/- 0''.04) and (969''.54 +/- 0''.02) at 30.4 and 17.1 nm respectively. No significant correlation was found with the solar cycle at any of the two wavelengths. Conclusions. Since the temperature formation of the 30.4 nm line is between (60 - 80) 10^3 K (Transition Region), the obtained result is bigger than that derived from present atmospheric models. On the contrary this height is compatible with radio models.Comment: accepted for publication in Astronomy & Astrophysics minor changes introduced during review proces

Comprehensive Determination of the Hinode/EIS Roll Angle

We present a new coalignment method for the EUV Imaging Spectrometer (EIS) on board the Hinode spacecraft. In addition to the pointing offset and spacecraft jitter, this method determines the roll angle of the instrument, which has never been systematically measured, and is therefore usually not corrected. The optimal pointing for EIS is computed by maximizing the cross-correlations of the Fe XII 195.119 \r{A} line with images from the 193 \r{A} band of the Atmospheric Imaging Assembly (AIA) on board the Solar Dynamics Observatory (SDO). By coaligning 3336 rasters with high signal-to-noise ratio, we estimate the rotation angle between EIS and AIA and explore the distribution of its values. We report an average value of (-0.387 $\pm$ 0.007)\deg. We also provide a software implementation of this method that can be used to coalign any EIS raster.Comment: Accepted for publication in Solar Physics, 11 pages, 7 figure

The coronal monsoon : thermal nonequilibrium revealed by periodic coronal rain

P.A. has received funding from the UK Science and Technology Facilities Council (Consolidated Grant ST/K000950/1) and the European Union Horizon 2020 research and innovation programme (grant agreement No. 647214).We report on the discovery of periodic coronal rain in an off-limb sequence of Solar Dynamics Observatory/Atmospheric Imaging Assembly images. The showers are co-spatial and in phase with periodic (6.6 hr) intensity pulsations of coronal loops of the sort described by Auchère et al. and Froment et al. These new observations make possible a unified description of both phenomena. Coronal rain and periodic intensity pulsations of loops are two manifestations of the same physical process: evaporation/condensation cycles resulting from a state of thermal nonequilibrium. The fluctuations around coronal temperatures produce the intensity pulsations of loops, and rain falls along their legs if thermal runaway cools the periodic condensations down and below transition-region temperatures. This scenario is in line with the predictions of numerical models of quasi-steadily and footpoint heated loops. The presence of coronal rain—albeit non-periodic—in several other structures within the studied field of view implies that this type of heating is at play on a large scale.Publisher PDFPeer reviewe

Apn1 AP-endonuclease is essential for the repair of oxidatively damaged DNA bases in yeast frataxin-deficient cells

International audienceFrataxin deficiency results in mitochondrial dysfunction and oxidative stress and it is the cause of the hereditary neurodegenerative disease Friedreich ataxia (FA). Here, we present evidence that one of the pleiotrop-ic effects of oxidative stress in frataxin-deficient yeast cells (Dyfh1 mutant) is damage to nuclear DNA and that repair requires the Apn1 AP-endonuclease of the base excision repair pathway. Major phenotypes of Dyfh1 cells are respiratory deficit, disturbed iron homeostasis and sensitivity to oxidants. These phenotypes are weak or absent under anaerobiosis. We show here that exposure of anaerobically grown Dyfh1 cells to oxygen leads to down-regulation of antioxidant defenses, increase in reactive oxygen species, delay in G1-and S-phases of the cell cycle and damage to mitochondrial and nuclear DNA. Nuclear DNA lesions in Dyfh1 cells are primarily caused by oxidized bases and single-strand breaks that can be detected 15-30 min after oxygen exposition. The Apn1 enzyme is essential for the repair of the DNA lesions in Dyfh1 cells. Compared with Dyfh1, the double Dyfh1Dapn1 mutant shows growth impairment, increased mu-tagenesis and extreme sensitivity to H 2 O 2. On the contrary, overexpression of the APN1 gene in Dyfh1 cells decreases spontaneous and induced mutagenesis. Our results show that frataxin deficiency in yeast cells leads to increased DNA base oxidation and requirement of Apn1 for repair, suggesting that DNA damage and repair could be important features in FA disease progression