Image Quality In Partially Erased Denoptix® Storage Phosphor Plates

This study aimed at investigating the effect of the partial erasing of DenOptix® system storage phosphor plates on the image quality of digital radiographs. Standardized digital radiographs were acquired of a phantom mandible, using size 2 intraoral DenOptix® storage phosphor plates (n = 10). Subsequently, the active areas of the plates were placed in a viewing box with a constant light intensity of 1,700 lux for 130 seconds to achieve complete erasing (control plate), as well as for 0, 5, 10, 1S, 20, 25, 34, 66, and 98 seconds, to compose the experimental group of partially erased plates. The same exposure settings were repeated using the control and experimental plates, which were scanned at a resolution of 300 dpi. Five radiologists independently examined the pairs of digital radiographs obtained with the control and partially erased plates, in random order, and indicated the best image for oral diagnosis. Cochran-Mantel-Haenszel's chi-square test, at a significance level of 5%, was used to compare the percentages of superior quality images in each combination of control and partially erased plates, subjectively assessed. No significant differences were found between radiographic images acquired with control and partially erased plates, except for the combination of 0 second (30%) versus 130 seconds (70%), p = 0.0047. It can be concluded that, under adequate light intensity conditions, erasing intraoral DenOptix® storage phosphor plates may require time intervals of as little as 5 seconds.2217883Akdeniz, B.G., Gröndahl, H.G., Kose, T., Effect of delayed scanning of storage phosphor plates (2005) Oral Surg Oral Med Oral Pathol Oral Radiol Endod, 99 (5), pp. 603-607Akdeniz, B.G., Gröndahl, H.G., Degradation of storage phosphor images due to scanning delay (2006) Dentomaxillofac Radiol, 35 (2), pp. 74-77Almeida, S.M., Oliveira, A.E.F., Ferreira, R.I., Bóscolo, F.N., Image quality in digital radiographic systems (2003) Braz Dent J, 14 (2), pp. 136-141Borg, E., Attaelmanan, A., Gröndahl, H.G., Subjective image quality of solid-state and photostimulable phosphor systems for digital intra-oral radiography (2000) Dentomaxillofac Radiol, 29 (2), pp. 70-75Ferreira, R.I., Haiter-Neto, F., Tabchoury, C.P.M., Paiva, G.A.N., Bóscolo, F.N., Assessment of enamel demineralization using conventional, digital, and digitized radiography (2006) Braz Oral Res, 20 (2), pp. 114-119Wenzel, A., Gröndahl, H.G., Direct digital radiography in the dental office (1995) Int Dent J, 45 (1), pp. 27-34Oliveira, A.E., Almeida, S.M., Paganini, G.A., Haiter Neto, F., Bóscolo, F.N., Comparative study of two digjtal radiographic storage phosphor systems (2000) Braz Dent J, 11 (2), pp. 111-116Borg, E., Attaelmanan, A., Gröndahl, H.G., Image plate systems differ in physical performance (2000) Oral Surg Oral Med Oral Pathol Oral Radiol Endod, 89 (1), pp. 118-124Martins, M.G.B.Q., Haiter Neto, F., Whaites, E.J., Analysis of digital images acquired using different phosphor storage plates (PSPs) subjected to varying reading times and storage conditions (2003) Dentomaxillofac Radiol, 32 (3), pp. 186-190(1998) DenOptix digital imaging system - user manual and installation guide, , Gendex Dental X-Ray Division, Des Plaines: The Division;Menig, J.J., The DenOptix digital radiographic system (1999) J Clin Orthod, 33 (7), pp. 407-410Berkhout WE, Beuger DA, Sanderink GC, van der Stelt PF. The dynamic range of digital radiographic systems: dose reduction or risk of overexposure? Dentomaxillofac Radiol. 2004;33(1):1-SHildebolt, C.F., Couture, R.A., Whiting, B.R., Dental photostimulable phosphor radiography (2000) Dent Clin North Am, 44 (2), pp. 273-297Martins, M.G.B.Q., Whaites, E.J., Ambrosano, G.M.B., Haiter Neto, F., What happens if you delay scanning Digora phosphor storage plates (PSPs) for up to 4 hours? (2006) Dentomaxillofac Radiol, 35 (3), pp. 143-146Gröndahl, H.G., Wenzel, A., Borg, E., Tammisalo, E., An image plate system for digital intra-oral radiography (1996) Dent Update, 23 (8), pp. 334-33

Teleportation of a Zero-and One-photon Running Wave State by Projection Synthesis

We show how to teleport a running wave superposition of zero- and one-photon field state through the projection synthesis technique. The fidelity of the scheme is computed taking into account the noise introduced by dissipation and the efficiency of the detectors. These error sources have been introduced through a single general relationship between input and output operators.Comment: 11 pages, 1 figur

Phytoestrogen agathisflavone ameliorates neuroinflammation-induced by LPS and IL-1β and protects neurons in cocultures of glia/neurons

Inflammation and oxidative stress are common aspects of most neurodegenerative diseases in the central nervous system. In this context, microglia and astrocytes are central to mediating the balance between neuroprotective and neurodestructive mechanisms. Flavonoids have potent anti-inflammatory and antioxidant properties. Here, we have examined the anti-inflammatory and neuroprotective potential of the flavonoid agathisflavone (FAB), which is derived from the Brazilian plant Poincianella pyramidalis, in in vitro models of neuroinflammation. Cocultures of neurons/glial cells were exposed to lipopolysaccharide (LPS, 1 µg/mL) or interleukin (IL)-1β (10 ng/mL) for 24 h and treated with FAB (0.1 and 1 µM, 24 h). FAB displayed a significant neuroprotective effect, as measured by nitric oxide (NO) production, Fluoro-Jade B (FJ-B) staining, and immunocytochemistry (ICC) for the neuronal marker β-tubulin and the cell death marker caspase-3, preserving neuronal soma and increasing neurite outgrowth. FAB significantly decreased the LPS-induced microglial proliferation, identified by ICC for Iba-1/bromodeoxyuridine (BrdU) and CD68 (microglia M1 profile marker). In contrast, FAB had no apparent effect on astrocytes, as determined by ICC for glial fibrillary acidic protein (GFAP). Furthermore, FAB protected against the cytodestructive and proinflammatory effects of IL-1β, a key cytokine that is released by activated microglia and astrocytes, and ICC showed that combined treatment of FAB with α and β estrogen receptor antagonists did not affect NF-κB expression. In addition, qPCR analysis demonstrated that FAB decreased the expression of proinflammatory molecules TNF-α, IL-1β, and connexins CCL5 and CCL2, as well as increased the expression of the regulatory molecule IL-10. Together, these findings indicate that FAB has a significant neuroprotective and anti-inflammatory effect in vitro, which may be considered as an adjuvant for the treatment of neurodegenerative diseases

Combate à lagarta do cartucho Spodoptera frugiperda (J.E.Smith, 1797), em plantação de milho

A corn field work was carried out in Tietê, State of São Paulo, Brazil in order to control the fall armyworm. The treatments, with four repetitions, and the quantities of active ingredients, per hectare, were as follows: A - check; B - bendiocarb, 250 g; C - hydrazone BTS 54439, 250 g ; D - trichlorfon, 250 g ; E - pyrethroid biphenthrin, 30 g ; F - pyrethroid FMC 65318, 30 g ; G - thiodicarb, 200 g ; H - triazophos, 200 g. The best results were obtained with pyrethroid FMC 65318, thiodicarb and pyrethroid biphenthrin.Com a finalidade de estudar o eleito de alguns defensivos químicos sobre a "lagarta do cartucho do milho" foi instalado o presente ensaio no "Campo de Produção de Mudas Tietê", da Coordenadoria de Assistência Técnica Integral (CATI), em Tietê, SP. Foram utilizados os seguintes tratamentos: A - testemunha; B- bendiocarbe, 250 g C- hidrazona BTS 54439, 250 g; D - triclorfom, 250 g; E - piretróide bifentrina (FMC 54800), 30 g; F- piretróide (FMC - 65318), 30 g; G - tiodicarbe, 200 g; H - triazofós, 200 g. As quantidades são de ingrediente ativo por hectare. Os tratamentos tiveram quatro repetições, em blocos ao acaso, com parcelas de 150 m² (cada tratamento, 600 m² ). Aos dois e nove dias da aplicação, os piretróides (FMC - 54800 (bifentrina) e FMC 65318) e tiodicarbe foram promissores

The flavonoid agathisflavone modulates the microglial neuroinflammatory response and enhances remyelination

Myelin loss is the hallmark of the demyelinating disease multiple sclerosis (MS) and plays a significant role in multiple neurodegenerative diseases. A common factor in all neuropathologies is the central role of microglia, the intrinsic immune cells of the central nervous system (CNS). Microglia are activated in pathology and can have both pro- and anti-inflammatory functions. Here, we examined the effects of the flavonoid agathisflavone on microglia and remyelination in the cerebellar slice model following lysolecithin induced demyelination. Notably, agathisflavone enhances remyelination and alters microglial activation state, as determined by their morphology and cytokine profile. Furthermore, these effects of agathisflavone on remyelination and microglial activation were inhibited by blockade of estrogen receptor α. Thus, our results identify agathisflavone as a novel compound that may act via ER to regulate microglial activation and enhance remyelination and repair

Quality of parental emotional care and calculated risk for coronary heart disease

Objective: To evaluate associations between perceived quality of parental emotional care and calculated 10-year risk for coronary heart disease (CHD). Little is understood about the role of parental emotional care in contributing to the risk for CHD. Methods: The study sample was composed of 267 participants from the New England Family Study. Quality of parental emotional care was measured, using a validated short version of the Parental Bonding Instrument (PBI) as the average care scores for both parents (range = 0-12), with higher scores indicating greater care. Ten-year CHD risk was calculated, using the validated Framingham Risk Algorithm that incorporates the following prevalent CHD risk factors: age, sex, diabetes, smoking, total cholesterol, high-density lipoprotein cholesterol, and blood pressure. Multiple linear regression assessed associations of PBI with calculated CHD risk after adjusting for childhood socioeconomic status, depressive symptomatology, educational attainment, and body mass index. Results: Among females, a 1-unit increase in the parental emotional care score resulted in a 4.6% (p =.004) decrease in the 10-year CHD risk score, after adjusting for covariates. There was no association between parental emotional care score and calculated CHD risk score in males (p =.22). Conclusion: Quality of parental emotional care was inversely associated with calculated 10-year CHD risk in females, and not males. Although the gender differences need further investigation and these findings require replication, these results suggest that the early childhood psychosocial environment may confer risk for CHD in adulthood

Decoherence in trapped ions due to polarization of the residual background gas

We investigate the mechanism of damping and heating of trapped ions associated with the polarization of the residual background gas induced by the oscillating ions themselves. Reasoning by analogy with the physics of surface electrons in liquid helium, we demonstrate that the decay of Rabi oscillations observed in experiments on 9Be+ can be attributed to the polarization phenomena investigated here. The measured sensitivity of the damping of Rabi oscillations with respect to the vibrational quantum number of a trapped ion is also predicted in our polarization model.Comment: 26 pdf pages with 5 figures, http://www.df.ufscar.br/~quantum

Neuroimmunomodulatory and neuroprotective effects of the flavonoid apigenin in in vitro models of neuroinflammation associated with Alzheimer's disease

Neurodegenerative disorders (ND) are characterized by the progressive and irreversible loss of neurons. Alzheimer’s Disease (AD) is the most incident age-related ND, in which the presence of a chronic inflammatory compound seems to be related to its pathogenesis. Different stimuli in the central nervous system (CNS) can induce activation, proliferation, and changes in phenotype and glial function, which can be modulated by anti-inflammatory agents. Apigenin (4,5,7–trihydroxyflavone) is a flavonoid found in abundance in many fruits and vegetables, that has shown important effects upon controlling the inflammatory response. This study evaluated the neuroprotective and neuroimmunomodulatory potential of apigenin using in vitro models of neuroinflammation associated with AD. Co-cultures of neurons and glial cells were obtained from the cortex of newborn and embryonic Wistar rats. After 26 days in vitro, cultures were exposed to lipopolysaccharide (LPS; 1 μg/ml), or IL-1β (10 ng/ml) for 24 h, or to Aβ oligomers (500 nM) for 4 h, and then treated with apigenin (1 μM) for further 24 h. It was observed that the treatment with apigenin preserved neurons and astrocytes integrity, determined by Rosenfeld’s staining and immunocytochemistry for β-tubulin III and GFAP, respectively. Moreover, it was observed by Fluoro-Jade-B and caspase-3 immunostaining that apigenin was not neurotoxic and has a neuroprotective effect against inflammatory damage. Additionally, apigenin reduced microglial activation, characterized by inhibition of proliferation (BrdU+ cells) and modulation of microglia morphology (Iba-1 + cells), and decreased the expression of the M1 inflammatory marker CD68. Moreover, as determined by RT-qPCR, inflammatory stimuli induced by IL-1β increased the mRNA expression of IL-6, IL-1β, and CCL5, and decreased the mRNA expression of IL-10. Contrary, after treatment with apigenin in inflammatory stimuli (IL-1β or LPS) there was a modulation of the mRNA expression of inflammatory cytokines, and reduced expression of OX42, IL-6 and gp130. Moreover, apigenin alone and after an inflammatory stimulus with IL-1β also induced the increase in the expression of brain-derived neurotrophic factor (BDNF), an effect that may be associated with anti-inflammatory and neuroprotective effects. Together these data demonstrate that apigenin presents neuroprotective and anti-inflammatory effects in vitro and might represent an important neuroimmunomodulatory agent for the treatment of neurodegenerative conditions

Defensivos químicos no combate ao "ácaro rajado" Tetranychus urticae Koch, 1836, em algodão

A field test was carried out in Americana, State of São Paulo, Brazil, to evaluate the control of the "two spotted spider mite", the main pest on cotton in that area. The treatments were eitht with four repetitions (each repetition ocupied 150 m² and each treatment, 600 m²). The treatments and active ingredients per hectare were as follows: A) check; B) triazophos, 300 g; C) monocrotophos, 600 g; D) biphenthrin, 55 g; E) fenpropathrin, 150 g; F) biphenthrin, 60 g; G) binapacryl, 800 g; H) clofentezine, 750 g. Five counts of the mite were made: the first, three days before the spray and 01, 05, 08 and 13 days after spraying. The best results were obtained with biphenthrin and binapacryl (05 days); biphenthrin and clofentezine (08 days) and clofentezine (13 days).Com o objetivo de se conhecer a ação de vários produtos químicos contra o ácaro rajado, principal praga algodão em diversas áreas do Estado de São Paulo, foi instalado campo de experimentação em Americana. A aplicação foi feita em 17.02.1984, por meio de atomizador costal de 12 litros de capacidade, à razão de 200 litros de água por hectare. Tratamentos: oito, com quatro repetições, em delineamento experimental de blocos ao acaso. Cada parcela (canteiro) media 150 m² (portanto, 600 m² por tratamento). A- testemunha; B - triazofós, 300 g; C - monocrotofós, 600 g; D- bifentrina, 55 g; E - fempropatrina , 150 g; - bifentrina, 60 g; G - binapacril, 800 g; H- clofentezina, 750 g. As quantidades citadas acima são de ingre diente ativo por hectare. Os melhores foram a clofentezina e a bifentrina (Quadros II e III)