Limits for Rumor Spreading in Stochastic Populations

Biological systems can share and collectively process information to yield emergent effects, despite inherent noise in communication. While man-made systems often employ intricate structural solutions to overcome noise, the structure of many biological systems is more amorphous. It is not well understood how communication noise may affect the computational repertoire of such groups. To approach this question we consider the basic collective task of rumor spreading, in which information from few knowledgeable sources must reliably flow into the rest of the population. In order to study the effect of communication noise on the ability of groups that lack stable structures to efficiently solve this task, we consider a noisy version of the uniform PULL model. We prove a lower bound which implies that, in the presence of even moderate levels of noise that affect all facets of the communication, no scheme can significantly outperform the trivial one in which agents have to wait until directly interacting with the sources. Our results thus show an exponential separation between the uniform PUSH and PULL communication models in the presence of noise. Such separation may be interpreted as suggesting that, in order to achieve efficient rumor spreading, a system must exhibit either some degree of structural stability or, alternatively, some facet of the communication which is immune to noise. We corroborate our theoretical findings with a new analysis of experimental data regarding recruitment in Cataglyphis Niger desert ants

A process of rumor scotching on finite populations

Rumor spreading is a ubiquitous phenomenon in social and technological networks. Traditional models consider that the rumor is propagated by pairwise interactions between spreaders and ignorants. Spreaders can become stiflers only after contacting spreaders or stiflers. Here we propose a model that considers the traditional assumptions, but stiflers are active and try to scotch the rumor to the spreaders. An analytical treatment based on the theory of convergence of density dependent Markov chains is developed to analyze how the final proportion of ignorants behaves asymptotically in a finite homogeneously mixing population. We perform Monte Carlo simulations in random graphs and scale-free networks and verify that the results obtained for homogeneously mixing populations can be approximated for random graphs, but are not suitable for scale-free networks. Furthermore, regarding the process on a heterogeneous mixing population, we obtain a set of differential equations that describes the time evolution of the probability that an individual is in each state. Our model can be applied to study systems in which informed agents try to stop the rumor propagation. In addition, our results can be considered to develop optimal information dissemination strategies and approaches to control rumor propagation.Comment: 13 pages, 11 figure

Epidemic processes in complex networks

In recent years the research community has accumulated overwhelming evidence for the emergence of complex and heterogeneous connectivity patterns in a wide range of biological and sociotechnical systems. The complex properties of real-world networks have a profound impact on the behavior of equilibrium and nonequilibrium phenomena occurring in various systems, and the study of epidemic spreading is central to our understanding of the unfolding of dynamical processes in complex networks. The theoretical analysis of epidemic spreading in heterogeneous networks requires the development of novel analytical frameworks, and it has produced results of conceptual and practical relevance. A coherent and comprehensive review of the vast research activity concerning epidemic processes is presented, detailing the successful theoretical approaches as well as making their limits and assumptions clear. Physicists, mathematicians, epidemiologists, computer, and social scientists share a common interest in studying epidemic spreading and rely on similar models for the description of the diffusion of pathogens, knowledge, and innovation. For this reason, while focusing on the main results and the paradigmatic models in infectious disease modeling, the major results concerning generalized social contagion processes are also presented. Finally, the research activity at the forefront in the study of epidemic spreading in coevolving, coupled, and time-varying networks is reported.Comment: 62 pages, 15 figures, final versio

Identifying spatial invasion of pandemics on metapopulation networks via anatomizing arrival history

Spatial spread of infectious diseases among populations via the mobility of humans is highly stochastic and heterogeneous. Accurate forecast/mining of the spread process is often hard to be achieved by using statistical or mechanical models. Here we propose a new reverse problem, which aims to identify the stochastically spatial spread process itself from observable information regarding the arrival history of infectious cases in each subpopulation. We solved the problem by developing an efficient optimization algorithm based on dynamical programming, which comprises three procedures: i, anatomizing the whole spread process among all subpopulations into disjoint componential patches; ii, inferring the most probable invasion pathways underlying each patch via maximum likelihood estimation; iii, recovering the whole process by assembling the invasion pathways in each patch iteratively, without burdens in parameter calibrations and computer simulations. Based on the entropy theory, we introduced an identifiability measure to assess the difficulty level that an invasion pathway can be identified. Results on both artificial and empirical metapopulation networks show the robust performance in identifying actual invasion pathways driving pandemic spread.Comment: 14pages, 8 figures; Accepted by IEEE Transactions on Cybernetic