Glutathione S Transferases M1 and T1 Genetic Variants are Associated with Coronary Artery Ectasia

29th Turkish Cardiology Congress of the Turkish-Society-of-Cardiology (TSC) with International Participation -- OCT 26-29, 2013 -- Antalya, TURKEYWOS: 000329858400026…Turkish Soc Cardio

Evaluation of the Apolipoprotein B R3500Q Gene Mutation in Nonvalvular Atrial Fibrillation with Ischemic Stroke in Turkish Population

29th Turkish Cardiology Congress of the Turkish-Society-of-Cardiology (TSC) with International Participation -- OCT 26-29, 2013 -- Antalya, TURKEYWOS: 000329858400355…Turkish Soc Cardio

Plasma lipoprotein(a) levels in patients with slow coronary flow

WOS: 000327688400001PubMed ID: 24570746Introduction: Slow coronary flow (SCF) is a microvascular disorder characterized by delayed opacification of coronary vessels with normal coronary angiogram. It may be due to endothelial dysfunction and diffuse atherosclerosis. Lipoprotein(a) [Lp(a)] is related to cardiovascular events. Plasma Lp(a) levels have not been studied previously in SCF patients. Aim: We investigated plasma Lp(a) and fibrinogen levels, and their relation to coronary flow rate in patients with SCE Material and methods: This cross-sectional study included 50 patients with SCF and 30 age- and sex-matched controls who had normal coronary arteries and normal flow. Coronary flow rates of patients and controls were counted with the thrombolysis in myocardial infarction (TIMI) frame count. Plasma Lp(a) and fibrinogen levels were measured in SCF patients and controls, with routine biochemical tests. Results: There were no significant differences between the two groups with respect to plasma Lp(a) (21 mg/dl vs. 14 mg/dl, p = 0.11) and fibrinogen (278 mg/dl vs. 291 mg/dl, p = 0.48) levels. The TIMI frame count was not correlated with plasma Lp(a) (r = 0.13, p = 0.25) or fibrinogen (r = -0.14, p = 0.28) levels. Conclusions: Our results show that there is no significant association between SCF and Lp(a) and fibrinogen levels

APOLIPOPROTEIN E GENE POLYMORPHISM IN SLOW CORONARY FLOW

WOS: 000317946500304

EVALUATION OF LYMPHOTOXIN-ALPHA C804A GENE POLYMORPHISMS IN PATIENTS WITH CORONARY HEART DISEASE

WOS: 000317946500309

ENDOTHELIAL NITRIC OXIDE SYNTHASE GLU298ASP (G894T) GENE POLYMORPHISM IN PATIENTS WITH CORONARY ARTERY DISEASE

WOS: 000317946500020

THE RELATIONSHIP BETWEEN ENDOTHELIAL NITRIC OXIDE SYNTHASE GENE POLYMORPHISM (T-786 C) AND ISOLE CORONARY ARTERY ECTASIA

WOS: 000317946500068

T-786 C POLYMORPHISM OF THE ENDOTHELIAL NITRIC OXIDE SYNTHASE GENE IS ASSOCIATED WITH CORONARY ARTERY DISEASE

WOS: 000317946500302

Angiotensin-Converting Enzyme Insertion/Deletion (I/D) Polymorphism Associated with Atrial Fibrilation in Turkish Population

29th Turkish Cardiology Congress of the Turkish-Society-of-Cardiology (TSC) with International Participation -- OCT 26-29, 2013 -- Antalya, TURKEYADALI, MEHMET KORAY/0000-0002-0054-6252WOS: 000329858400347Background Atrial fibrillation (AF) is the most commonly observed arrhythmia in clinical practice and associated with increased cardiovascular morbidity and mortality. The renin-angiotensin system may play a role in the pathogenesis of atrial fibrillation. Increased angiotensin-converting enzyme (ACE) expression in the atrial tissue of patients with AF has suggested the involvement of the RAS in AF. Some initial studies indicated an association between an angiotensin-converting enzyme insertion/deletion (ACE I/D) polymorphism and AF, however, the results have been inconsistent. We aimed to investigate relationship between AF and polymorphism of ACE I/D in Turkish patients.Turkish Soc Cardio

The Relationship between Endothelial Nitric Oxide Synthase Gene Polymorphism (G894T) and Isole Coronary Artery Ectasia

29th Turkish Cardiology Congress of the Turkish-Society-of-Cardiology (TSC) with International Participation -- OCT 26-29, 2013 -- Antalya, TURKEYWOS: 000329858400493…Turkish Soc Cardio