Simulación de la calidad de madera en los modelos de gestión forestal

The raw material properties of wood develop as the tree grows, laying down wood cells with specific properties, and forming the stem structure that is focal for timber quality. This development is influenced by genetic and environmental factors and forest management practices. It is desirable in growth and yield models intended for the economic assessment of management practices to include some indication of wood quality and how it is affected by genetics, environmental factors and silvicultural measures. This paper reviews approaches and models that allow us to consider the development of wood quality in combination with tree growth, and thus to include wood quality in the assessment of the value of the yield. We present such models as classified into three categories based on their complexity and information needs: quality indicators, static quality models, and dynamic quality models. We illustrate three advanced dynamic quality models and their applications with example case studies. These include empirical, hybrid, and mechanistic models applied to predictions of both sawn timber and fibre properties. Finally, we consider the current challenges for wood quality modelling in connection with growth models.Las propiedades de la madera se desarrollan cuando crece el arbol, estableciendose las células de la madera con propiedades específicas, y formandose la estructura del tronco que es central para la calidad de la madera. Esta desarrollo se ve influido por factores genéticos y ambientales y por las prácticas de gestión forestal. Es deseable incluir en los modelos de crecimiento y producción destinados a la evaluación económica de las prácticas de gestión una indicación de la calidad de la madera y cómo se ve afectada por factores genéticos y ambientales y por las medidas silvícolas. En el sector forestal, la comprensión del desarrollo y la variación de la calidad de la madera es importante en distintos programas de gestión y planificación que se pueden clasificar en (1) la gestión de las operaciones de cosecha, y (2) la planificación silvícola a largo plazo. Este artículo discute los principales enfoques y modelos que nos permiten estudiar estos dos problemas en términos cuantitativos. Comenzamos la revisión discutiendo el concepto de «modelo de calidad de madera», y luego clasificamos los enfoques sobre la base de su complejidad, el principio subyacente y el uso previsto. Mostramos tres modelos dinámicos avanzados de calidad y sus aplicaciones con estudios de casos. Estos incluyen modelos empíricos, híbridos y mecánicistas aplicadaos a las predicciones tanto de la madera aserrada como a las propiedades de la fibra. Por último, consideramos los retos actuales para el modelado de la calidad de la madera en relación con la gestión forestal

Simulation tools for decision support to adaptive forest management in Europe

Peer reviewe

Increased S-nitrosylation and proteasomal degradation of caspase-3 during infection contribute to the persistence of adherent invasive escherichia coli (AIEC) in immune cells

Adherent invasive Escherichia coli (AIEC) have been implicated as a causative agent of Crohn's disease (CD) due to their isolation from the intestines of CD sufferers and their ability to persist in macrophages inducing granulomas. The rapid intracellular multiplication of AIEC sets it apart from other enteric pathogens such as Salmonella Typhimurium which after limited replication induce programmed cell death (PCD). Understanding the response of infected cells to the increased AIEC bacterial load and associated metabolic stress may offer insights into AIEC pathogenesis and its association with CD. Here we show that AIEC persistence within macrophages and dendritic cells is facilitated by increased proteasomal degradation of caspase-3. In addition S-nitrosylation of pro- and active forms of caspase-3, which can inhibit the enzymes activity, is increased in AIEC infected macrophages. This S-nitrosylated caspase-3 was seen to accumulate upon inhibition of the proteasome indicating an additional role for S-nitrosylation in inducing caspase-3 degradation in a manner independent of ubiquitination. In addition to the autophagic genetic defects that are linked to CD, this delay in apoptosis mediated in AIEC infected cells through increased degradation of caspase-3, may be an essential factor in its prolonged persistence in CD patients