Are the HI deficient galaxies on the outskirts of Virgo recent arrivals?

The presence on the Virgo cluster outskirts of spiral galaxies with gas deficiencies as strong as those of the inner galaxies stripped by the intracluster medium has led us to explore the possibility that some of these peripheral objects are not newcomers. A dynamical model for the collapse and rebound of spherical shells under the point mass and radial flow approximations has been developed to account for the amplitude of the motions in the Virgo I cluster (VIC) region. According to our analysis, it is not unfeasible that galaxies far from the cluster, including those in a gas-deficient group well to its background, went through its core a few Gyr ago. The implications would be: (1) that the majority of the HI-deficient spirals in the VIC region might have been deprived of their neutral hydrogen by interactions with the hot intracluster medium; and (2) that objects spending a long time outside the cluster cores might keep the gas deficient status without altering their morphology.Comment: Accepted for publication in ApJ. 4 pages, 3 figures. Uses emulateapj

Hamiltonian and physical Hilbert space in polymer quantum mechanics

In this paper, a version of polymer quantum mechanics, which is inspired by loop quantum gravity, is considered and shown to be equivalent, in a precise sense, to the standard, experimentally tested, Schroedinger quantum mechanics. The kinematical cornerstone of our framework is the so called polymer representation of the Heisenberg-Weyl (H-W) algebra, which is the starting point of the construction. The dynamics is constructed as a continuum limit of effective theories characterized by a scale, and requires a renormalization of the inner product. The result is a physical Hilbert space in which the continuum Hamiltonian can be represented and that is unitarily equivalent to the Schroedinger representation of quantum mechanics. As a concrete implementation of our formalism, the simple harmonic oscillator is fully developed.Comment: 19 pages, 2 figures. Comments and references added. Version to be published in CQ

Rapid and MR-Independent IK1 activation by aldosterone during ischemia-reperfusion

In ST elevation myocardial infarction (STEMI) context, clinical studies have shown the deleterious effect of high aldosterone levels on ventricular arrhythmia occurrence and cardiac mortality. Previous in vitro reports showed that during ischemia-reperfusion, aldosterone modulates K+ currents involved in the holding of the resting membrane potential (RMP). The aim of this study was to assess the electrophysiological impact of aldosterone on IK1 current during myocardial ischemia-reperfusion. We used an in vitro model of “border zone” using right rabbit ventricle and standard microelectrode technique followed by cell-attached recordings from freshly isolated rabbit ventricular cardiomyocytes. In microelectrode experiments, aldosterone (10 and 100 nmol/L, n=7 respectively) increased the action potential duration (APD) dispersion at 90% between ischemic and normoxic zones (from 95±4ms to 116±6 ms and 127±5 ms respectively, P<0.05) and reperfusion-induced sustained premature ventricular contractions occurrence (from 2/12 to 5/7 preparations, P<0.05). Conversely, potassium canrenoate 100 nmol/L and RU 28318 1 μmol/l alone did not affect AP parameters and premature ventricular contractions occurrence (except Vmax which was decreased by potassium canrenoate during simulated-ischemia). Furthermore, aldosterone induced a RMP hyperpolarization, evoking an implication of a K+ current involved in the holding of the RMP. Cell-attached recordings showed that aldosterone 10 nmol/L quickly activated (within 6.2±0.4 min) a 30 pS K+-selective current, inward rectifier, with pharmacological and biophysical properties consistent with the IK1 current (NPo =1.9±0.4 in control vs NPo=3.0±0.4, n=10, P<0.05). These deleterious effects persisted in presence of RU 28318, a specific MR antagonist, and were successfully prevented by potassium canrenoate, a non specific MR antagonist, in both microelectrode and patch-clamp recordings, thus indicating a MR-independent IK1 activation. In this ischemia-reperfusion context, aldosterone induced rapid and MR-independent deleterious effects including an arrhythmia substrate (increased APD90 dispersion) and triggered activities (increased premature ventricular contractions occurrence on reperfusion) possibly related to direct IK1 activation

Astrometry at OAFA. Earth rotation

A brief report on one of the astrometric activities (Earth Rotation) carried out at the "Félix Aguilar" Astronomical Observatory since its opening up to the present is given. I deals with: the importance of this research: the OAFA collaboration to the International Services, BIH and IPMS; the application of new techniques: VLBI, LLR and LSR, the new International Service, IERS, and its Reference System; and a brief account on the main tasks carried out and to be developed at the OAFA.Asociación Argentina de Astronomí

Magnetic field fluctuations in the shocked umbral chromosphere

Several studies have reported magnetic field fluctuations associated with umbral shock waves. We aim to study the properties and origin of magnetic field fluctuations in the umbral chromosphere. Temporal series of spectropolarimetric observations were acquired with the GREGOR telescope. The chromospheric and photospheric conditions were derived from simultaneous inversions of the He I 10830 \AA\ triplet and the Si I 10827 \AA\ line using HAZEL2. The oscillations are interpreted using wavelet analysis and context information from UV observations acquired with SDO/AIA and IRIS. The chromospheric magnetic field shows strong fluctuations in the sunspot umbra, with peak field strengths up to 2900 G. Magnetic field and velocity umbral oscillations exhibit a strong coherence, with the magnetic field lagging the shock fronts detected in the velocity fluctuations. This points to a common origin of the fluctuations in both parameters, whereas the analysis of the phase shift between photospheric and chromospheric velocity is consistent with upwards wave propagation. These results suggest that the strong inferred magnetic field fluctuations are caused by changes in the response height of the He I 10830 \AA\ line to the magnetic field, which is sensitive to high photospheric layers after the shock fronts. The coronal activity seen in EUV data could possibly have some impact on the inferred fluctuations, but it is not the main driver of the magnetic field oscillations since they are found before EUV events take place. Chromospheric magnetic field fluctuations measured with the He I 10830 \AA\ triplet arise due to variations in the opacity of the line. After shocks produced by slow magnetoacoustic waves, the response of the line to the magnetic field can be shifted down to the upper photosphere. This is seen as remarkably large fluctuations in the line of sight magnetic field strength.Comment: Accepted for publication in A&A. Abstract abridged due to arXiv's 1920 character limi

Numerical model characterization of the sound transmission mechanism in the tympanic membrane from a high-speed digital holographic experiment in transient regime

A methodology for the development of a finite element numerical model of the tympanic membrane (TM) based on experiments carried out in the time domain on a cadaveric human temporal bone is presented. Using a high-speed digital holographic (HDH) system, acoustically-induced transient displacements of the TM surface are obtained. The procedure is capable to generate and validate the finite element model of the TM by numerical and experimental data correlation. Reverse engineering approach is used to iden- tify key material parameters that define the mechanical response of the TM. Finally, modal numerical simulations of the specimen are performed. Results show the feasibility of the methodology to obtain an accurate model of a specific specimen and to help interpret its behaviour with additional numerical simulations.This work has been funded by the University of Malaga/CBUA, the FEDER program grant number UMA18-FEDERJA-214 and by the National Institutes of Health (NIH), National Institute on Deafness and Other Communication Disorders (NIDCD), grant number R01, DC016079. This research has also been partially supported by Research Grant Jose Castillejo number CAS19/00125 from Spain Government. Funding for open access charge: Universidad de Málaga / CBU