Geographical variation in food plant selection of Eupithecia gelidata Möschler, 1860 (Lepidoptera, Geometridae)

The relict bog populations of Eupithecia gelidata Möschl. in South Bohemia are obligatorily associated with Ledum palustre L., which is here the dominant shrub, all Salix species being absent. In the subarctic and boreal populations of E. gelidata, altemative food plants are willows (Salix spp.). Under laboratory conditions, larvae of the Bohemian populations can accomplish their development feeding on Salix, but Ledum is clearly preferred. The larvae living on Ledum and Salix are usually differently coloured. Thus, in spite of the different food plants and coloration of the larvae, the species seems to be taxonomically homogeneous

Effect of Boron and Cross-Section Thickness on Microstructure and Mechanical Properties of Ductile Iron

Eeffect of Boron addition on the microstructure and mechanical properties of ductile iron, GJS-500-7 grade was studied. Three cast batches with the Boron content of 10, 49 and 131ppm were cast in a casting geometry containing plates with thicknesses of 7, 15, 30, 50 and 75mm. Microstructure analysis, tensile test, and hardness test were performed on the samples which were machined from the casting plates. Addition of 49 ppm Boron decreased pearlite fraction by an average of 34±6% in all the cast plates. However, minor changes were observed in the pearlite fraction by increasing Boron from 49 to 131 ppm. Variation in the plate thickness did not affect the pearlite fraction. The 0.2% offset yield and ultimate tensile strength was decreased by an average of 11±1% and 18±2%, respectively. Addition of 49 ppm Boron decreased Brinell hardness by 16±1%, while 11±2% reduction was obtained by addition of 131ppm Boron

Search for direct photons in p+Pb and p+C collisions at sqrt(sNN) = 17.4 GeV

Upper limits on direct photon production were determined as a function of the transverse momentum for 0.7 < pT <= 3.2 GeV/c with the WA98 experiment in p+C and p+Pb collisions at sqrt(sNN) = 17.4 GeV. The results are compared to direct photon measurements in Pb+Pb collisions at sqrt(sNN) = 17.3 GeV by WA98. Implications for a possible thermal direct photon contribution are discussed.Comment: 4 pages, 3 figures, to appear in the proceedings of the Quark Matter 2008 conference, Jaipur, India, 4-10 Feb 200

Secondary insults following traumatic brain injury enhance complement activation in the human brain and release of the tissue damage marker S100B

To access publisher full text version of this article. Please click on the hyperlink in Additional Links field.OBJECT: Complement activation has been suggested to play a role in the development of secondary injuries following traumatic brain injury (TBI). The present study was initiated in order to analyze complement activation in relation to the primary brain injury and to secondary insults, frequently occurring following TBI. METHODS: Twenty patients suffering from severe TBI (Glasgow coma score ≤ 8) were included in the study. The "membrane attack complex," C5b9, which is the cytolytic end product of the complement system was analyzed in cerebrospinal fluid (CSF). The degree of brain tissue damage was assessed using the release of S100B and neuron-specific enolase (NSE) to the CSF and blood. The blood-brain barrier was assessed using the CSF/serum quotient of albumin (Q (A)). RESULTS: Following impact, initial peaks (0-48 h) of C5b9, S100B, and NSE with a concomitant loss of integrity of the blood-brain barrier were observed. Secondary insults at the intensive care unit were monitored. Severe secondary insults were paralleled by a more pronounced complement activation (C5b9 in CSF) as well as increased levels of S100B (measured in CSF), but not with NSE. CONCLUSION: This human study indicates that complement activation in the brain is triggered not only by the impact of trauma per se but also by the amount of secondary insults that frequently occur at the scene of accident as well as during treatment in the neurointensive care unit. Complement activation and in particular the end product C5b9 may in turn contribute to additional secondary brain injuries by its membrane destructive properties