131 research outputs found
Organizational downsizing: Constraining, cloning, learning
While downsizing rages through the U.S. economy, there is a great deal of uncertainty about its bottom-line effects. This uncertainty raises questions about why corporations have been so eager to engage in downsizing. In this article, we propose an answer to these questions. Three social forces, which we call constraining, cloning and learning, frequently provide a major impetus for downsizing. We describe these forces, and point out conditions that lead to the adoption of downsizing without due regard for its mixed consequences. We suggest methods to improve executives\u27 downsizing decision routines ... methods that should enhance the chances of achieving intended benefits
Density waves and density fluctuations in granular flow
We simulate the granular flow in a narrow pipe with a lattice-gas automaton
model. We find that the density in the system is characterized by two features.
One is that spontaneous density waves propagate through the system with
well-defined shapes and velocities. The other is that density waves are so
distributed to make the power spectra of density fluctuations as
noise. Three important parameters make these features observable and they are
energy dissipation, average density and the rougness of the pipe walls.Comment: Latex (with ps files appended
Static Friction Phenomena in Granular Materials: Coulomb Law vs. Particle Geometry
The static as well as the dynamic behaviour of granular material are
determined by dynamic {\it and} static friction. There are well known methods
to include static friction in molecular dynamics simulations using scarcely
understood forces. We propose an Ansatz based on the geometrical shape of
nonspherical particles which does not involve an explicit expression for static
friction. It is shown that the simulations based on this model are close to
experimental results.Comment: 11 pages, Revtex, HLRZ-33/9
Velocity and density profiles of granular flow in channels using lattice gas automaton
We have performed two-dimensional lattice-gas-automaton simulations of
granular flow between two parallel planes. We find that the velocity profiles
have non-parabolic distributions while simultaneously the density profiles are
non-uniform. Under non-slip boundary conditions, deviation of velocity profiles
from the parabolic form of newtonian fluids is found to be characterized solely
by ratio of maximal velocity at the center to the average velocity, though the
ratio depends on the model parameters in a complex manner. We also find that
the maximal velocity () at the center is a linear function of the
driving force (g) as with non-zero in
contrast with newtonian fluids. Regarding density profiles, we observe that
densities near the boundaries are higher than those in the center. The width of
higher densities (above the average density) relative to the channel width is a
decreasing function of a variable which scales with the driving force (g),
energy dissipation parameter () and the width of the system (L) as
with exponents and . A phenomenological theory based on a scaling argument is presented to
interpret these findings.Comment: Latex, 15 figures, to appear in PR
Path integral Monte Carlo simulation of the second layer of helium-4 adsorbed on graphite
We have developed a path integral Monte Carlo method for simulating helium
films and apply it to the second layer of helium adsorbed on graphite. We use
helium-helium and helium-graphite interactions that are found from potentials
which realistically describe the interatomic interactions. The Monte Carlo
sampling is over both particle positions and permutations of particle labels.
From the particle configurations and static structure factor calculations, we
find that this layer possesses, in order of increasing density, a superfluid
liquid phase, a sqrt(7) x sqrt(7) commensurate solid phase that is registered
with respect to the first layer, and an incommensurate solid phases. By
applying the Maxwell construction to the dependence of the low-temperature
total energy on the coverage, we are able to identify coexistence regions
between the phases. From these, we deduce an effectively zero-temperature phase
diagram. Our phase boundaries are in agreement with heat capacity and torsional
oscillator measurements, and demonstrate that the experimentally observed
disruption of the superfluid phase is caused by the growth of the commensurate
phase. We further observe that the superfluid phase has a transition
temperature consistent with the two-dimensional value. Promotion to the third
layer occurs for densities above 0.212 atom/A^2, in good agreement with
experiment. Finally, we calculate the specific heat for each phase and obtain
peaks at temperatures in general agreement with experiment.Comment: 14 double-column pages, 10 figures, revtex. Accepted for publication
in Phys. Rev. B. 3 figures added, some text revisions, 6 figures remove
The Allelic Landscape of Human Blood Cell Trait Variation and Links to Common Complex Disease
Many common variants have been associated with hematological traits, but identification of causal genes and pathways has proven challenging. We performed a genome-wide association analysis in the UK Biobank and INTERVAL studies, testing 29.5 million genetic variants for association with 36 red cell, white cell, and platelet properties in 173,480 European-ancestry participants. This effort yielded hundreds of low frequency (<5%) and rare (<1%) variants with a strong impact on blood cell phenotypes. Our data highlight general properties of the allelic architecture of complex traits, including the proportion of the heritable component of each blood trait explained by the polygenic signal across different genome regulatory domains. Finally, through Mendelian randomization, we provide evidence of shared genetic pathways linking blood cell indices with complex pathologies, including autoimmune diseases, schizophrenia, and coronary heart disease and evidence suggesting previously reported population associations between blood cell indices and cardiovascular disease may be non-causal.We thank members of the Cambridge BioResource Scientific Advisory Board and Management Committee for their support of our study and the National Institute for Health Research Cambridge Biomedical Research Centre for funding. K.D. is funded as a HSST trainee by NHS Health Education England. M.F. is funded from the BLUEPRINT Grant Code HEALTH-F5-2011-282510 and the BHF Cambridge Centre of Excellence [RE/13/6/30180]. J.R.S. is funded by a MRC CASE Industrial studentship, co-funded by Pfizer. J.D. is a British Heart Foundation Professor, European Research Council Senior Investigator, and National Institute for Health Research (NIHR) Senior Investigator. S.M., S.T, M.H, K.M. and L.D. are supported by the NIHR BioResource-Rare Diseases, which is funded by NIHR. Research in the Ouwehand laboratory is supported by program grants from the NIHR to W.H.O., the European Commission (HEALTH-F2-2012-279233), the British Heart Foundation (BHF) to W.J.A. and D.R. under numbers RP-PG-0310-1002 and RG/09/12/28096 and Bristol Myers-Squibb; the laboratory also receives funding from NHSBT. W.H.O is a NIHR Senior Investigator. The INTERVAL academic coordinating centre receives core support from the UK Medical Research Council (G0800270), the BHF (SP/09/002), the NIHR and Cambridge Biomedical Research Centre, as well as grants from the European Research Council (268834), the European Commission Framework Programme 7 (HEALTH-F2-2012-279233), Merck and Pfizer. DJR and DA were supported by the NIHR Programme ‘Erythropoiesis in Health and Disease’ (Ref. NIHR-RP-PG-0310-1004). N.S. is supported by the Wellcome Trust (Grant Codes WT098051 and WT091310), the EU FP7 (EPIGENESYS Grant Code 257082 and BLUEPRINT Grant Code HEALTH-F5-2011-282510). The INTERVAL study is funded by NHSBT and has been supported by the NIHR-BTRU in Donor Health and Genomics at the University of Cambridge in partnership with NHSBT. The views expressed are those of the authors and not necessarily those of the NHS, the NIHR, the Department of Health of England or NHSBT. D.G. is supported by a “la Caixa”-Severo Ochoa pre-doctoral fellowship
Visuospatial Integration: Paleoanthropological and Archaeological Perspectives
The visuospatial system integrates inner and outer functional processes, organizing spatial, temporal, and social interactions between the brain, body, and environment. These processes involve sensorimotor networks like the eye–hand circuit, which is especially important to primates, given their reliance on vision and touch as primary sensory modalities and the use of the hands in social and environmental interactions. At the same time, visuospatial cognition is intimately connected with memory, self-awareness, and simulation capacity. In the present article, we review issues associated with investigating visuospatial integration in extinct human groups through the use of anatomical and behavioral data gleaned from the paleontological and archaeological records. In modern humans, paleoneurological analyses have demonstrated noticeable and unique morphological changes in the parietal cortex, a region crucial to visuospatial management. Archaeological data provides information on hand–tool interaction, the spatial behavior of past populations, and their interaction with the environment. Visuospatial integration may represent a critical bridge between extended cognition, self-awareness, and social perception. As such, visuospatial functions are relevant to the hypothesis that human evolution is characterized by changes in brain–body–environment interactions and relations, which enhance integration between internal and external cognitive components through neural plasticity and the development of a specialized embodiment capacity. We therefore advocate the investigation of visuospatial functions in past populations through the paleoneurological study of anatomical elements and archaeological analysis of visuospatial behaviors
Institutional Forces In Accounting Information Systems Choice
The selection and implementation of accounting information systems (AIS) has been portrayed by the past literature to follow models of rational behavior, such as the system resource model and the goal model. The analysis in this paper develops the argument that these models cannot fully explain AIS choices with regard to which systems are implemented and which objectives and goals are pursued. Institutional models, developed by theorists in the sociology of organizations, can provide a broader evaluative framework within which AIS choice can be explained. The paper presents the mechanisms of coercive isomorphism, mimetic isomorphism and normative isomorphism through which institutional influences can affect AIS choice. These mechanisms have been developed by institutional theorists to explain the movement towards the implementation of increasingly similar institutionalized procedures and practices across organizations. External dependencies, uncertainty in performance standards, and interaction patterns during the system selection process are identified also as conditions that could moderate the strength of institutional influences on AIS choice. Implications for account professionals are drawn and recommendations for future research are made.
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