80 research outputs found
Trust and Reciprocity among International Groups : Experimental Evidence from Austria and Japan
This paper explores national identity in trust and reciprocity at the intra- and international levels by adopting a modified trust game played among groups from Austria and Japan, in which subjects play the roles of trustor and trustee consecutively without any information feedback. Intra- and international trust levels are identical across nationalities. Intranational reciprocity in Austria is greater than in Japan, but there is no significant difference in international reciprocity between the two countries. We also examine the relationship between trust and reciprocity and the relathionship between trust/reciprocity and expectations, demographic variables, the individualism index, and trust indices. International trust enhances international reciprocity in both countries, although this effect is weaker in Japan than in Austria. Expected returns enhance trust in both countries at the intra- and international levels. In Japan, expected transfers enhance international reciprocity less than they do intranational reciprocity. A larger number of females in the group reduce international trust. In Austria, this reduces international trust more than it does intranational trust.ISER discussion paperMay 2009, Revised August 201
Conditional cooperation on three continents
We show in a public goods experiment on three continents that conditional cooperation is a universal behavioral regularity. Yet, the number of conditional cooperators and the extent of conditional cooperation are much higher in the U.S.A. than anywhere else
Optically thick clumps: not the solution to the Wolf-Rayet wind momentum problem?
The hot star wind momentum problem η = Mν∞/(L/c)» much greater than 1 is revisited, and it is shown that the conventional belief, that it can be solved by a combination of clumping of the wind and multiple scattering of photons, is not self-consistent for optically thick clumps. Clumping does reduce the mass loss rate M, and hence the momentum supply, required to generate a specified radio emission measure epsilon, while multiple scattering increases the delivery of momentum from a specified stellar luminosity L. However, in the case of thick clumps, when combined the two effects act in opposition rather than in unison since clumping reduces multiple scattering. From basic geometric considerations, it is shown that this reduction in momentum delivery by clumping more than offsets the reduction in momentum required, for a specified ε. Thus the ratio of momentum deliverable to momentum required is maximal for a smooth wind and the momentum problem remains for the thick clump case. In the case of thin clumps, all of the benefit of clumping in reducing η lies in reducing M for a given ε so that extremely small filling factors f ≈ 10-4 are needed. It is also shown that clumping affects the inference of M from radio ε not only by changing the emission measure per unit mass but also by changing the radio optical depth unity radius Rrad, and hence the observed wind volume, at radio wavelengths. In fact, for free-free opacity proportional to αn2, contrary to intuition, Rrad increases with increasing clumpiness
A Population of X-ray Weak Quasars: PHL 1811 Analogs at High Redshift
We report the results from Chandra and XMM-Newton observations of a sample of
10 type 1 quasars selected to have unusual UV emission-line properties (weak
and blueshifted high-ionization lines; strong UV Fe emission) similar to those
of PHL 1811, a confirmed intrinsically X-ray weak quasar. These quasars were
identified by the Sloan Digital Sky Survey at high redshift (z~2.2); eight are
radio quiet while two are radio intermediate. All of the radio-quiet PHL 1811
analogs are notably X-ray weak by a mean factor of ~13. These sources lack
broad absorption lines and have blue UV/optical continua, suggesting they are
intrinsically X-ray weak. However, their average X-ray spectrum appears to be
harder than those of typical quasars, which may indicate the presence of heavy
intrinsic X-ray absorption. Our radio-quiet PHL 1811 analogs support a
connection between an X-ray weak spectral energy distribution and PHL 1811-like
UV emission lines; this connection provides an economical way to identify X-ray
weak type 1 quasars. The fraction of radio-quiet PHL 1811 analogs in the
radio-quiet quasar population is estimated to be < 1.2%. We have investigated
correlations between relative X-ray brightness and UV emission-line properties
for a sample combining radio-quiet PHL 1811 analogs, PHL 1811, and typical type
1 quasars. These correlation analyses suggest that PHL 1811 analogs may have
extreme wind-dominated broad emission-line regions. Observationally,
radio-quiet PHL 1811 analogs appear to be a subset (~30%) of radio-quiet
weak-line quasars. The existence of a subset of quasars in which
high-ionization "shielding gas" covers most of the BELR, but little more than
the BELR, could potentially unify the PHL 1811 analogs and WLQs. The two
radio-intermediate PHL 1811 analogs are X-ray bright. One of them appears to
have jet-dominated X-ray emission, while the nature of the other remains
unclear.Comment: ApJ accepted; 25 pages, 11 figures and 8 table
The Ensemble Photometric Variability of ~25000 Quasars in the Sloan Digital Sky Survey
Using a sample of over 25000 spectroscopically confirmed quasars from the
Sloan Digital Sky Survey, we show how quasar variability in the rest frame
optical/UV regime depends upon rest frame time lag, luminosity, rest
wavelength, redshift, the presence of radio and X-ray emission, and the
presence of broad absorption line systems. The time dependence of variability
(the structure function) is well-fit by a single power law on timescales from
days to years. There is an anti-correlation of variability amplitude with rest
wavelength, and quasars are systematically bluer when brighter at all
redshifts. There is a strong anti-correlation of variability with quasar
luminosity. There is also a significant positive correlation of variability
amplitude with redshift, indicating evolution of the quasar population or the
variability mechanism. We parameterize all of these relationships. Quasars with
RASS X-ray detections are significantly more variable (at optical/UV
wavelengths) than those without, and radio loud quasars are marginally more
variable than their radio weak counterparts. We find no significant difference
in the variability of quasars with and without broad absorption line troughs.
Models involving multiple discrete events or gravitational microlensing are
unlikely by themselves to account for the data. So-called accretion disk
instability models are promising, but more quantitative predictions are needed.Comment: 41 pages, 21 figures, AASTeX, Accepted for publication in Ap
Long-term Optical Variability of Radio-Selected Quasars from the FIRST Survey
We have obtained single-epoch optical photometry for 201 quasars, taken from
the FIRST Bright Quasar Survey, which span a wide range in radio loudness.
Comparison with the magnitudes of these objects on the POSS-I plates provides
by far the largest sample of long-term variability amplitudes for
radio-selected quasars yet produced. We find the quasars to be more variable in
the blue than in the red band, consistent with work on optically selected
samples. The previously noted trend of decreasing variability with increasing
optical luminosity applies only to radio-quiet objects. Furthermore, we do not
confirm a rise in variability amplitude with redshift, nor do we see any
dependence on radio flux or luminosity. The variability over a radio-optical
flux ratio range spanning a factor of 60,000 from radio-quiet to extreme
radio-loud objects is largely constant, although there is a suggestion of
greater variability in the extreme radio-loud objects. We demonstrate the
importance of Malmquist bias in variability studies, and develop a procedure to
correct for the bias in order to reveal the underlying variability properties
of the sample.Comment: 16 pages, 8 figures To be published in 2001 April 1 Astronomical
Journa
Autoimmune and autoinflammatory mechanisms in uveitis
The eye, as currently viewed, is neither immunologically ignorant nor sequestered from the systemic environment. The eye utilises distinct immunoregulatory mechanisms to preserve tissue and cellular function in the face of immune-mediated insult; clinically, inflammation following such an insult is termed uveitis. The intra-ocular inflammation in uveitis may be clinically obvious as a result of infection (e.g. toxoplasma, herpes), but in the main infection, if any, remains covert. We now recognise that healthy tissues including the retina have regulatory mechanisms imparted by control of myeloid cells through receptors (e.g. CD200R) and soluble inhibitory factors (e.g. alpha-MSH), regulation of the blood retinal barrier, and active immune surveillance. Once homoeostasis has been disrupted and inflammation ensues, the mechanisms to regulate inflammation, including T cell apoptosis, generation of Treg cells, and myeloid cell suppression in situ, are less successful. Why inflammation becomes persistent remains unknown, but extrapolating from animal models, possibilities include differential trafficking of T cells from the retina, residency of CD8(+) T cells, and alterations of myeloid cell phenotype and function. Translating lessons learned from animal models to humans has been helped by system biology approaches and informatics, which suggest that diseased animals and people share similar changes in T cell phenotypes and monocyte function to date. Together the data infer a possible cryptic infectious drive in uveitis that unlocks and drives persistent autoimmune responses, or promotes further innate immune responses. Thus there may be many mechanisms in common with those observed in autoinflammatory disorders
Rationale, design and methods for a community-based study of clustering and cumulative effects on chronic disease process and their effects on ageing: the Busselton healthy ageing study
Background: The global trend of increased life expectancy and increased prevalence of chronic and degenerative diseases will impact on health systems. To identify effective intervention and prevention strategies, greater understanding of the risk factors for and cumulative effects of chronic disease processes and their effects on function and quality of life is needed. The Busselton Healthy Ageing Study aims to enhance understanding of ageing by relating the clustering and interactions of common chronic conditions in adults to function. Longitudinal (3–5 yearly) follow-up is planned. Methods/design: Phase I (recruitment) is a cross-sectional community-based prospective cohort study involving up to 4,000 ‘Baby Boomers’ (born from 1946 to 1964) living in the Busselton Shire, Western Australia. The study protocol involves a detailed, self-administered health and risk factor questionnaire and a range of physical assessments including body composition and bone density measurements, cardiovascular profiling (blood pressure, ECG and brachial pulse wave velocity), retinal photography, tonometry, auto-refraction, spirometry and bronchodilator responsiveness, skin allergy prick tests, sleep apnoea screening, tympanometry and audiometry, grip strength, mobility, balance and leg extensor strength. Cognitive function and reserve, semantic memory, and pre-morbid intelligence are assessed. Participants provide a fasting blood sample for assessment of lipids, blood glucose, C-reactive protein and renal and liver function, and RNA, DNA and serum are stored. Clinically relevant results are provided to all participants. The prevalence of risk factors, symptoms and diagnosed illness will be calculated and the burden of illness will be estimated based on the observed relationships and clustering of symptoms and illness within individuals. Risk factors for combinations of illness will be compared with those for single illnesses and the relation of combinations of illness and symptoms to cognitive and physical function will be estimated. Discussion: This study will enable a thorough characterization of multiple disease processes and their risk factors within a community-based sample of individuals to determine their singular, interactive and cumulative effects on ageing. The project will provide novel cross-sectional data and establish a cohort that will be used for longitudinal analyses of the genetic, lifestyle and environmental factors that determine whether an individual ages well or with impairment
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