384 research outputs found
OCRL1 engages with the F-BAR protein pacsin 2 to promote biogenesis of membrane-trafficking intermediates
- Publication venue
- 'American Society for Cell Biology (ASCB)'
- Publication date
- 23/10/2015
- Field of study
Mutation of the inositol 5-phosphatase OCRL1 causes Lowe syndrome and Dent-2 disease. Loss of OCRL1 function perturbs several cellular processes, including membrane traffic, but the underlying mechanisms remain poorly defined. Here we show that OCRL1 is part of the membrane-trafficking machinery operating at the trans-Golgi network (TGN)/endosome interface. OCRL1 interacts via IPIP27A with the F-BAR protein pacsin 2. OCRL1 and IPIP27A localize to mannose 6-phosphate receptor (MPR)–containing trafficking intermediates, and loss of either protein leads to defective MPR carrier biogenesis at the TGN and endosomes. OCRL1 5-phosphatase activity, which is membrane curvature sensitive, is stimulated by IPIP27A-mediated engagement of OCRL1 with pacsin 2 and promotes scission of MPR-containing carriers. Our data indicate a role for OCRL1, via IPIP27A, in regulating the formation of pacsin 2–dependent trafficking intermediates and reveal a mechanism for coupling PtdIns(4,5)P2 hydrolysis with carrier biogenesis on endomembranes
Changes in the expression of the type 2 diabetes-associated gene VPS13C in the β cell are associated with glucose intolerance in humans and mice
- Author
- Publication venue
- 'American Physiological Society'
- Publication date
- 01/01/2016
- Field of study
Single nucleotide polymorphisms (SNPs) close to the VPS13C, C2CD4A and C2CD4B genes on chromosome 15q are associated with impaired fasting glucose and increased risk of type 2 diabetes. eQTL analysis revealed an association between possession of risk (C) alleles at a previously implicated causal SNP, rs7163757, and lowered VPS13C and C2CD4A levels in islets from female (n = 40, P < 0.041) but not from male subjects. Explored using promoter-reporter assays in β-cells and other cell lines, the risk variant at rs7163757 lowered enhancer activity. Mice deleted for Vps13c selectively in the β-cell were generated by crossing animals bearing a floxed allele at exon 1 to mice expressing Cre recombinase under Ins1 promoter control (Ins1Cre). Whereas Vps13cfl/fl:Ins1Cre (βVps13cKO) mice displayed normal weight gain compared with control littermates, deletion of Vps13c had little effect on glucose tolerance. Pancreatic histology revealed no significant change in β-cell mass in KO mice vs. controls, and glucose-stimulated insulin secretion from isolated islets was not altered in vitro between control and βVps13cKO mice. However, a tendency was observed in female null mice for lower insulin levels and β-cell function (HOMA-B) in vivo. Furthermore, glucose-stimulated increases in intracellular free Ca2+ were significantly increased in islets from female KO mice, suggesting impaired Ca2+ sensitivity of the secretory machinery. The present data thus provide evidence for a limited role for changes in VPS13C expression in conferring altered disease risk at this locus, particularly in females, and suggest that C2CD4A may also be involved
An Overview of the Management of Flexor Tendon Injuries
- Author
- Al-Qattan MM
- Aoki M
- Bainbridge LC
- Banes AJ
- Barrie KA
- Barrie KA
- Barrie KA
- Barrie KA
- Bassett CAL
- Benjamin M
- Boyer MI
- Bruner JM
- Cao Y
- Cassell OC
- Chang J
- Chesney A
- D Jordan
- Diao E
- Diao E
- Dovan TT
- Elliot D
- Fujita M
- Gordon L
- Grobbelaar AO
- Hall RF
- Hatanaka H
- Hatanaka H
- Hatanaka H
- Hess GP
- Hooley CJ
- Hotokezaka S
- Hunter JM
- Kleinert HE
- Kleinert HE
- Komanduri M
- Kusano N
- Kvist M
- Kvist M
- Kwai B
- Kwai B
- Langlais F
- Lawrence TM
- Lee EW
- Lee H
- Lim BH
- Lin GT
- Lin GT
- Lotz JC
- Lui PP
- M Griffin
- M Saleh
- Manske PR
- Manske PR
- Manske PR
- Mashadi ZB
- Mehta V
- Mehta V
- Michna H
- Moutiet F
- Movin T
- Nakamura N
- Nakamura N
- O’Brien M
- Pennington DG
- Pruitt DL
- Reddy GK
- Riccio M
- Rust RA
- S Hindocha
- Sanders DW
- Savage R
- Savage R
- Shaieb MD
- Silfverskiöld KL
- Silfverskiöld KL
- Small JO
- Strickland JW
- Strickland JW
- Su BW
- Tang JB
- Tang JB
- Tang JB
- Taras JS
- Taras JS
- Ting J
- Ting J
- Tipton CM
- Tonkin MA
- Trail IA
- Viinikainen A
- W Khan
- Wada A
- Wade PJ
- Wade PJ
- Wade PJ
- Wang CJ
- Winters SC
- Wolfman NM
- Zhao C
- Publication venue
- Bentham Open
- Publication date
- 01/01/2012
- Field of study
Flexor tendon injuries still remain a challenging condition to manage to ensure optimal outcome for the patient. Since the first flexor tendon repair was described by Kirchmayr in 1917, several approaches to flexor tendon injury have enabled successful repairs rates of 70-90%. Primary surgical repair results in better functional outcome compared to secondary repair or tendon graft surgery. Flexor tendon injury repair has been extensively researched and the literature demonstrates successful repair requires minimal gapping at the repair site or interference with tendon vascularity, secure suture knots, smooth junction of tendon end and having sufficient strength for healing. However, the exact surgical approach to achieve success being currently used among surgeons is still controversial. Therefore, this review aims to discuss the results of studies demonstrating the current knowledge regarding the optimal approach for flexor tendon repair. Post-operative rehabilitation for flexor tendon surgery is another area, which has caused extensive debate in hand surgery. The trend to more active mobilisation protocols seems to be favoured but further study in this area is needed to find the protocol, which achieves function and gliding but avoids rupture of the tendons. Lastly despite success following surgery complications commonly still occur post surgery, including adhesion formation, tendon rupture and stiffness of the joints. Therefore, this review aims to discuss the appropriate management of these difficulties post surgery. New techniques in management of flexor tendon will also be discussed including external laser devices, addition of growth factors and cytokines
dOCRL maintains immune cell quiescence in Drosophila by regulating endosomal traffic
- Author
- A Faucherre
- A Guha
- A Legido
- A Maminska
- A Witsell
- AA Lazareva
- AK Satoh
- AK Shia
- AM Allmendinger
- Avital A. Rodal
- B Lemaitre
- B Zhou
- Benjamin H. Rosenfeld
- BV Shravage
- BZ Shilo
- C Cauvin
- C Savarin
- CJ Noakes
- CJ Zettervall
- D Dambournet
- D Stein
- DA Kimbrell
- E Beurel
- E Kurucz
- E Owusu-Ansah
- F Oltrabella
- G Emery
- G Guglielmi
- GD Gupta
- GR Hammond
- H Yang
- HR Huang
- IB Ramirez
- IP Nezis
- J Clasadonte
- J Manaka
- J Zhang
- JC Hombria
- JM Abrams
- JQ Ni
- JR van Weering
- JW Wang
- K Ben El Kadhi
- K Ketel
- K Koles
- K Makhijani
- Katherine S. Lehmann
- KJ Venken
- KS Erdmann
- KS Gold
- L Sun
- M Crozatier
- M Erta
- M Mavrakis
- M Vicinanza
- M Yamamoto-Hino
- MA De Matteis
- ME Haberland
- MG De Leo
- MJ Williams
- MM Davis
- MR Schmid
- N Luo
- N Matova
- NC Franc
- Norbert Perrimon
- P Zhang
- PG Billcliff
- R Choudhury
- R Markus
- R Markus
- R Nandez
- R Wubbolts
- RJ Khadilkar
- S Codeluppi
- S Dunst
- S Jean
- S Minakhina
- S Petraki
- S Takats
- SA Sinenko
- Sarah A. Biber
- Sean T. Sweeney
- SF Soukup
- SH Jung
- SP Bothwell
- Stephanie R. Heimler
- Steven J. Del Signore
- SW Brubaker
- T Lin
- Tania L. Eskin
- TM Khuong
- V Honti
- V Honti
- VI Korolchuk
- W Mobius
- Y Shen
- ZB Mehta
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/10/2017
- Field of study
Lowe Syndrome is a developmental disorder characterized by eye, kidney, and neurological pathologies, and is caused by mutations in the phosphatidylinositol-5-phosphatase OCRL. OCRL plays diverse roles in endocytic and endolysosomal trafficking, cytokinesis, and ciliogenesis, but it is unclear which of these cellular functions underlie specific patient symptoms. Here, we show that mutation of Drosophila OCRL causes cell-autonomous activation of hemocytes, which are macrophage-like cells of the innate immune system. Among many cell biological defects that we identified in docrl mutant hemocytes, we pinpointed the cause of innate immune cell activation to reduced Rab11-dependent recycling traffic and concomitantly increased Rab7-dependent late endosome traffic. Loss of docrl amplifies multiple immune-relevant signals, including Toll, Jun kinase, and STAT, and leads to Rab11-sensitive mis-sorting and excessive secretion of the Toll ligand Spåtzle. Thus, docrl regulation of endosomal traffic maintains hemocytes in a poised, but quiescent state, suggesting mechanisms by which endosomal misregulation of signaling may contribute to symptoms of Lowe syndrome
Search for gravitational waves from Scorpius X-1 in the second Advanced LIGO observing run with an improved hidden Markov model
- Author
- Abbott Bp
- Abbott R
- Abbott Td
- Abraham S
- Acernese F
- Ackley K
- Adams C
- Adhikari Rx
- Adya Vb
- Affeldt C
- Agathos M
- Agatsuma K
- Aggarwal N
- Aguiar Od
- Aiello L
- Ain A
- Ajith P
- Allen G
- Allocca A
- Aloy Ma
- Altin Pa
- Amato A
- Anand S
- Ananyeva A
- Anderson Sb
- Anderson Wg
- Angelova Sv
- Antier S
- Appert S
- Arai K
- Araya Mc
- Areeda Js
- Arene M
- Arnaud N
- Aronson Sm
- Ascenzi S
- Ashton G
- Aston Sm
- Astone P
- Aubin F
- Aufmuth P
- AultONeal K
- Austin C
- Avendano V
- Avila-Alvarez A
- Babak S
- Bacon P
- Badaracco F
- Bader Mkm
- Bae S
- Baird J
- Baker Pt
- Baldaccini F
- Ballardin G
- Ballmer Sw
- Bals A
- Banagiri S
- Barayoga Jc
- Barbieri C
- Barclay Se
- Barish Bc
- Barker D
- Barkett K
- Barnum S
- Barone F
- Barr B
- Barsotti L
- Barsuglia M
- Barta D
- Bartlett J
- Bartos I
- Bassiri R
- Basti A
- Bawaj M
- Bayley Jc
- Bazzan M
- Becsy B
- Bejger M
- Belahcene I
- Bell As
- Beniwal D
- Benjamin Mg
- Berger Bk
- Bergmann G
- Bernuzzi S
- Berry Cpl
- Bersanetti D
- Bertolini A
- Betzwieser J
- Bhandare R
- Bidler J
- Biggs E
- Bilenko Ia
- Bilgili Sa
- Billingsley G
- Birney R
- Birnholtz O
- Biscans S
- Bischi M
- Biscoveanu S
- Bisht A
- Bitossi M
- Bizouard Ma
- Blackburn Jk
- Blackman J
- Blair Cd
- Blair Dg
- Blair Rm
- Bloemen S
- Bobba F
- Bode N
- Boer M
- Boetzel Y
- Bogaert G
- Bondu F
- Bonnand R
- Booker P
- Boom Ba
- Bork R
- Boschi V
- Bose S
- Bossilkov V
- Bosveld J
- Bouffanais Y
- Bozzi A
- Bradaschia C
- Brady Pr
- Bramley A
- Branchesi M
- Brau Je
- Breschi M
- Briant T
- Briggs Jh
- Brighenti F
- Brillet A
- Brinkmann M
- Brockill P
- Brooks Af
- Brooks J
- Brown Dd
- Brunett S
- Buikema A
- Bulik T
- Bulten Hj
- Buonanno A
- Buskulic D
- Bustillo Jc
- Buy C
- Byer Rl
- Cabero M
- Cadonati L
- Cagnoli G
- Cahillane C
- Callister Ta
- Calloni E
- Camp Jb
- Campbell Wa
- Cannon Kc
- Cao H
- Cao J
- Carapella G
- Carbognani F
- Caride S
- Carney Mf
- Carullo G
- Casentini C
- Castro Jmg
- Caudill S
- Cavaglia M
- Cavalier F
- Cavalieri R
- Cella G
- Cerda-Duran P
- Cesarini E
- Chaibi O
- Chakravarti K
- Chamberlin Sj
- Chan M
- Chao S
- Charlton P
- Chase Ea
- Chassande-Mottin E
- Chatterjee D
- Chaturvedi M
- Cheeseboro Bd
- Chen Hy
- Chen X
- Chen Y
- Cheng Hp
- Cheong Ck
- Chia Hy
- Chiadini F
- Chincarini A
- Chiummo A
- Cho G
- Cho Hs
- Cho M
- Christensen N
- Chu Q
- Chua S
- Chung Kw
- Chung S
- Ciani G
- Cieslar M
- Ciobanu Aa
- Ciolfi R
- Cipriano F
- Cirone A
- Clara F
- Clark Ja
- Clearwater P
- Cleva F
- Coccia E
- Cohadon Pf
- Cohen D
- Colleoni M
- Collette Cg
- Collins C
- Colpi M
- Cominsky Lr
- Constancio M
- Conti L
- Cooper Sj
- Corban P
- Corbitt Tr
- Cordero-Carrion I
- Corezzi S
- Corley Kr
- Cornish N
- Corre D
- Corsi A
- Cortese S
- Costa Ca
- Costa Cfd
- Cotesta R
- Coughlin Mw
- Coughlin Sb
- Coulon Jp
- Countryman St
- Couvares P
- Covas Pb
- Cowan Ee
- Coward Dm
- Cowart Mj
- Coyne Dc
- Coyne R
- Creighton Jde
- Creighton Td
- Cripe J
- Croquette M
- Crowder Sg
- Cullen Tj
- Cumming A
- Cunningham L
- Cuoco E
- D'Angelo B
- D'Antonio S
- Dal Canton T
- Dalya G
- Danilishin Sl
- Danzmann K
- Dasgupta A
- Datrier Leh
- Dattilo V
- Dave I
- Davier M
- Davis D
- Daw Ej
- De Laurentis M
- De Pietri R
- De Rosa R
- De Rossi C
- de Varona O
- DeBra D
- Deenadayalan M
- Degallaix J
- Del Pozzo W
- Deleglise S
- DeMarchi Lm
- Demos N
- Dent T
- DeSalvo R
- Dhurandhar S
- Di Fiore L
- Di Giorgio C
- Di Giovanni F
- Di Giovanni M
- Di Girolamo T
- Di Lieto A
- Di Pace S
- Di Palma I
- Di Renzo F
- Diaz Jc
- Diaz Mc
- Dietrich T
- DiFronzo C
- Ding B
- Divakarla Ak
- Dmitriev A
- Doctor Z
- Donovan F
- Dooley Kl
- Doravari S
- Dorrington I
- Downes Tp
- Drago M
- Driggers Jc
- Du Z
- Ducoin Jg
- Dupej P
- Durante O
- Dwyer Se
- Easter Pj
- Eddolls G
- Edo Tb
- Effler A
- Ehrens P
- Eichholz J
- Eikenberry Ss
- Eisenmann M
- Eisenstein Ra
- Errico L
- Essick Rc
- Estelles H
- Estevez D
- Etienne Zb
- Etzel T
- Evans M
- Evans Tm
- Fafone V
- Fairhurst S
- Fan X
- Farinon S
- Farr B
- Farr Wm
- Fauchon-Jones Ej
- Favata M
- Fays M
- Fazio M
- Fee C
- Feicht J
- Fejer Mm
- Feng F
- Fernandez-Galiana A
- Ferrante I
- Ferreira Ec
- Ferreira Ta
- Fidecaro F
- Fiori I
- Fiorucci D
- Fishbach M
- Fisher Rp
- Fishner Jm
- Fittipaldi R
- Fitz-Axen M
- Fiumara V
- Flaminio R
- Fletcher M
- Floden E
- Flynn E
- Fong H
- Font Ja
- Forsyth Pwf
- Fournier Jd
- Frasca S
- Frasconi F
- Frei Z
- Freise A
- Frey R
- Frey V
- Fritschel P
- Frolov Vv
- Fronze G
- Fulda P
- Fyffe M
- Gabbard Ha
- Gadre Bu
- Gaebel Sm
- Gair Jr
- Gammaitoni L
- Gaonkar Sg
- Garcia-Quiros C
- Garufi F
- Gateley B
- Gaudio S
- Gaur G
- Gayathri V
- Gemme G
- Genin E
- Gennai A
- George D
- George J
- Gergely L
- Ghonge S
- Ghosh A
- Ghosh A
- Ghosh S
- Giacomazzo B
- Giaime Ja
- Giardina Kd
- Gibson Dr
- Gill K
- Glover L
- Gniesmer J
- Godwin P
- Goetz E
- Goetz R
- Goncharov B
- Gonzalez G
- Gopakumar A
- Gossan Se
- Gosselin M
- Gouaty R
- Grace B
- Grado A
- Granata M
- Grant A
- Gras S
- Grassia P
- Gray C
- Gray R
- Greco G
- Green Ac
- Green R
- Gretarsson Em
- Grimaldi A
- Grimm Sj
- Groot P
- Grote H
- Grunewald S
- Gruning P
- Guidi Gm
- Gulati Hk
- Guo Y
- Gupta A
- Gupta A
- Gupta P
- Gustafson Ek
- Gustafson R
- Haegel L
- Halim O
- Hall Br
- Hall Ed
- Hamilton Ez
- Hammond G
- Haney M
- Hanke Mm
- Hanks J
- Hanna C
- Hannuksela Oa
- Hansen Tj
- Hanson J
- Harder T
- Hardwick T
- Haris K
- Harms J
- Harry Gm
- Harry Iw
- Hasskew Rk
- Haster Cj
- Haughian K
- Hayes Fj
- Healy J
- Heidmann A
- Heintze Mc
- Heitmann H
- Hellman F
- Hello P
- Hemming G
- Hendry M
- Heng Is
- Hennig J
- Hernandez Im
- Heurs M
- Hild S
- Hinderer T
- Hochheim S
- Hofman D
- Holgado Am
- Holland Na
- Holt K
- Holz De
- Hopkins P
- Horst C
- Hough J
- Howell Ej
- Hoy Cg
- Huang Y
- Hubner Mt
- Huerta Ea
- Huet D
- Hughey B
- Hui V
- Husa S
- Huttner Sh
- Huynh-Dinh T
- Idzkowski B
- Iess A
- Inchauspe H
- Ingram C
- Inta R
- Intini G
- Irwin B
- Isa Hn
- Isac Jm
- Isi M
- Iyer Br
- Jacqmin T
- Jadhav Sj
- Jani K
- Janthalur Nn
- Jaranowski P
- Jariwala D
- Jenkins Ac
- Jiang J
- Johnson Ds
- Jones Aw
- Jones Di
- Jones Jd
- Jones R
- Jonker Rjg
- Ju L
- Junker J
- Kalaghatgi Cv
- Kalogera V
- Kamai B
- Kandhasamy S
- Kang G
- Kanner Jb
- Kapadia Sj
- Karki S
- Kashyap R
- Kasprzack M
- Katsanevas S
- Katsavounidis E
- Katzman W
- Kaufer S
- Kawabe K
- Keerthana Nv
- Kefelian F
- Keitel D
- Kennedy R
- Key Js
- Khalili Fy
- Khan I
- Khan S
- Khazanov Ea
- Khetan N
- Khursheed M
- Kijbunchoo N
- Kim C
- Kim Jc
- Kim K
- Kim W
- Kim Ws
- Kim Ym
- Kimball C
- King Pj
- Kinley-Hanlon M
- Kirchhoff R
- Kissel Js
- Kleybolte L
- Klika Jh
- Klimenko S
- Knowles Td
- Koch P
- Koehlenbeck Sm
- Koekoek G
- Koley S
- Kondrashov V
- Kontos A
- Koper N
- Korobko M
- Korth Wz
- Kovalam M
- Kozak Db
- Kramer C
- Kringel V
- Krishnendu N
- Krolak A
- Krupinski N
- Kuehn G
- Kumar A
- Kumar P
- Kumar R
- Kumar R
- Kuo L
- Kutynia A
- Kwang S
- Lackey Bd
- Laghi D
- Lai Kh
- Lam Tl
- Landry M
- Lane Bb
- Lang Rn
- Lange J
- Lantz B
- Lanza Rk
- Lartaux-Vollard A
- Lasky Pd
- Laxen M
- Lazzarini A
- Lazzaro C
- Leaci P
- Leavey S
- Lecoeuche K
- Lee Ch
- Lee Hk
- Lee Hm
- Lee Hw
- Lee J
- Lee K
- Lehmann J
- Lenon Ak
- Leroy N
- Letendre N
- Levin Y
- Li A
- Li J
- Li Kjl
- Li Tgf
- Li X
- Lin F
- Linde F
- Linker Sd
- Littenberg Tb
- Liu J
- Liu X
- Llorens-Monteagudo M
- Lo Rkl
- London Lt
- Longo A
- Lorenzini M
- Loriette V
- Lormand M
- Losurdo G
- Lough Jd
- Lousto Co
- Lovelace G
- Lower Me
- Luck H
- Lumaca D
- Lundgren Ap
- Lynch R
- Ma Y
- Macas R
- Macfoy S
- MacInnis M
- Macleod Dm
- Macquet A
- Magana-Sandoval F
- Magee Rm
- Majorana E
- Maksimovic I
- Malik A
- Man N
- Mandic V
- Mangano V
- Mansell Gl
- Manske M
- Mantovani M
- Mapelli M
- Marchesoni F
- Marion F
- Marka S
- Marka Z
- Markakis C
- Markosyan As
- Markowitz A
- Maros E
- Marquina A
- Marsat S
- Martelli F
- Martin Iw
- Martin Rm
- Martinez V
- Martynov Dv
- Masalehdan H
- Mason K
- Massera E
- Masserot A
- Massinger Tj
- Masso-Reid M
- Mastrogiovanni S
- Matas A
- Matichard F
- Matone L
- Mavalvala N
- McCann Jj
- McCarthy R
- McClelland De
- McCormick S
- McCuller L
- McGuire Sc
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- Meacher D
- Meadors Gd
- Mehmet M
- Mehta Ak
- Meidam J
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- Merzougui M
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- Meylahn F
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- Middleton H
- Milano L
- Miller L
- Millhouse M
- Mills Jc
- Milovich-Goff Mc
- Minazzoli O
- Minenkov Y
- Mishkin A
- Mishra C
- Mistry T
- Mitra S
- Mitrofanov Vp
- Mitselmakher G
- Mittleman R
- Mo G
- Moffa D
- Mogushi K
- Mohapatra Srp
- Molina-Ruiz M
- Mondin M
- Montani M
- Moore Cj
- Moraru D
- Morawski F
- Moreno G
- Morisaki S
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- Muciaccia F
- Mukherjee A
- Mukherjee D
- Mukherjee S
- Mukherjee S
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- Nardecchia I
- Naticchioni L
- Nayak Rk
- Neil Bf
- Neilson J
- Nelemans G
- Nelson Tjn
- Nery M
- Neunzert A
- Nevin L
- Ng Ky
- Ng S
- Nguyen C
- Nguyen P
- Nichols D
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- Nissanke S
- Nocera F
- North C
- Nuttall Lk
- O'Brien Bd
- O'Reilly B
- O'Shaughnessy R
- Obergaulinger M
- Oberling J
- Oganesyan G
- Ogin Gh
- Oh Jj
- Oh Sh
- Ohme F
- Ohta H
- Okada Ma
- Oliver M
- Oppermann P
- Oram Rj
- Ormiston Rg
- Ortega Lf
- Ossokine S
- Ottaway Dj
- Overmier H
- Owen Bj
- Pace Ae
- Pagano G
- Page Ma
- Pagliaroli G
- Pai A
- Pai Sa
- Palamos Jr
- Palashov O
- Palomba C
- Pan H
- Panda Pk
- Pang Pth
- Pankow C
- Pannarale F
- Pant Bc
- Paoletti F
- Paoli A
- Parida A
- Parker W
- Pascucci D
- Pasqualetti A
- Passaquieti R
- Passuello D
- Patil M
- Patricelli B
- Payne E
- Pearlstone Bl
- Pechsiri Tc
- Pedersen Aj
- Pedraza M
- Pedurand R
- Pele A
- Penn S
- Perego A
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- Zimmerman Ab
- Zucker Me
- Zweizig J
- Publication venue
- College Park, MD : American Physical Society
- Publication date
- 01/01/2019
- Field of study
We present results from a semicoherent search for continuous gravitational waves from the low-mass x-ray binary Scorpius X-1, using a hidden Markov model (HMM) to track spin wandering. This search improves on previous HMM-based searches of LIGO data by using an improved frequency domain matched filter, the J-statistic, and by analyzing data from Advanced LIGO's second observing run. In the frequency range searched, from 60 to 650 Hz, we find no evidence of gravitational radiation. At 194.6 Hz, the most sensitive search frequency, we report an upper limit on gravitational wave strain (at 95% confidence) of h095%=3.47×10-25 when marginalizing over source inclination angle. This is the most sensitive search for Scorpius X-1, to date, that is specifically designed to be robust in the presence of spin wandering. © 2019 American Physical Society
Search for gravitational waves from Scorpius X-1 in the second Advanced LIGO observing run with an improved hidden Markov model
- Author
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- Zimmerman Ab
- Zucker Me
- Zweizig J
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2019
- Field of study
We present results from a semicoherent search for continuous gravitational waves from the low-mass x-ray binary Scorpius X-1, using a hidden Markov model (HMM) to track spin wandering. This search improves on previous HMM-based searches of LIGO data by using an improved frequency domain matched filter, the J-statistic, and by analyzing data from Advanced LIGO’s second observing run. In the frequency range searched, from 60 to 650 Hz, we find no evidence of gravitational radiation. At 194.6 Hz, the most sensitive search frequency, we report an upper limit on gravitational wave strain (at 95% confidence) of h95%0=3.47×10−25 when marginalizing over source inclination angle. This is the most sensitive search for Scorpius X-1, to date, that is specifically designed to be robust in the presence of spin wandering
Cohort Profile: Post-Hospitalisation COVID-19 (PHOSP-COVID) study
- Author
- Abel K
- Adamali H
- Adeloye D
- Adeyemi O
- Adrego R
- Ahmad S
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- Zawia A
- Zeidan L
- Zhao B
- Zheng B
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- Publication venue
- Oxford University Press (OUP)
- Publication date
- 18/12/2023
- Field of study
Measurement of the tt¯tt¯ production cross section in pp collisions at √s=13 TeV with the ATLAS detector
- Author
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- Abbott B
- Abbott DC
- Abed Abud A
- Abeling K
- Abhayasinghe DK
- Abidi SH
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- Zemaityte G
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- Zhemchugov A
- Zheng Z
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- Zieminska D
- Zimine NI
- Zimmermann S
- Ziolkowski M
- Zoccoli A
- Zoch K
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 16/11/2021
- Field of study
A measurement of four-top-quark production using proton-proton collision data at a centre-of-mass energy of 13 TeV collected by the ATLAS detector at the Large Hadron Collider corresponding to an integrated luminosity of 139 fb−1 is presented. Events are selected if they contain a single lepton (electron or muon) or an opposite-sign lepton pair, in association with multiple jets. The events are categorised according to the number of jets and how likely these are to contain b-hadrons. A multivariate technique is then used to discriminate between signal and background events. The measured four-top-quark production cross section is found to be 26+17−15 fb, with a corresponding observed (expected) significance of 1.9 (1.0) standard deviations over the background-only hypothesis. The result is combined with the previous measurement performed by the ATLAS Collaboration in the multilepton final state. The combined four-top-quark production cross section is measured to be 24+7−6 fb, with a corresponding observed (expected) signal significance of 4.7 (2.6) standard deviations over the background-only predictions. It is consistent within 2.0 standard deviations with the Standard Model expectation of 12.0 ± 2.4 fb
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
- Author
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- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Adam Bourdarios C
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- Zhemchugov A
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- Zhong D
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- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Measurements of W+W−+ ≥ 1 jet production cross-sections in pp collisions at s = 13 TeV with the ATLAS detector
- Author
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- Zhemchugov A
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- Zhulanov V
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- Zimine NI
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- Zinonos Z
- Ziolkowski M
- Zoccoli A
- Zoch K
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/06/2021
- Field of study
Fiducial and differential cross-section measurements of W+W− production in
association with at least one hadronic jet are presented. These measurements are sensitive
to the properties of electroweak-boson self-interactions and provide a test of perturbative
quantum chromodynamics and the electroweak theory. The analysis is performed using
proton-proton collision data collected at p
s = 13TeV with the ATLAS experiment, corresponding
to an integrated luminosity of 139 fb−1. Events are selected with exactly one oppositely
charged electron-muon pair and at least one hadronic jet with a transverse momentum
of pT > 30 GeV and a pseudorapidity of |�| < 4.5. After subtracting the background
contributions and correcting for detector effects, the jet-inclusive W+W−+ � 1 jet fiducial
cross-section and W+W−+ jets differential cross-sections with respect to several kinematic
variables are measured. These measurements include leptonic quantities, such as the lepton
transverse momenta and the transverse mass of the W+W− system, as well as jet-related
observables such as the leading jet transverse momentum and the jet multiplicity. Limits on
anomalous triple-gauge-boson couplings are obtained in a phase space where interference
between the Standard Model amplitude and the anomalous amplitude is enhanced
- …