245 research outputs found
Interference cancellation for non-orthogonal multiple access used in future wireless mobile networks
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Sulindac targets nuclear β-catenin accumulation and Wnt signalling in adenomas of patients with familial adenomatous polyposis and in human colorectal cancer cell lines
- Author
- C Hirota
- E Kopp
- E M J Boon
- EM Boon
- F M Giardiello
- FM Giardiello
- FM Giardiello
- FM Giardiello
- G Hawcroft
- G J A Offerhaus
- G Winde
- GA Piazza
- J J Keller
- JJ Keller
- JJ Keller
- K Schmeiser
- KP Nugent
- KW Kinzler
- L Picariello
- M Bienz
- M Cruz-Correa
- M Shtutman
- MF McEntee
- ML Smith
- MM Entius
- MT Spagnesi
- PJ Pasricha
- R Hanif
- R van der Neut
- RN DuBois
- S Dihlmann
- S T Pals
- SJ Baek
- SJ Shiff
- T A M Wormhoudt
- TC He
- WA Brown
- WJ Thompson
- X Zhang
- Publication venue
- Nature Publishing Group
- Publication date
- 01/01/2004
- Field of study
Nonsteroidal anti-inflammatory drugs (NSAIDs) have chemopreventive potential against colorectal carcinomas (CRCs). Inhibition of cyclooxygenase (COX)-2 underlies part of this effect, although COX-2-independent mechanisms may also exist. Nonsteroidal anti-inflammatory drugs appear to inhibit the initial stages of the adenoma-carcinoma sequence, suggesting a link to the APC/beta-catenin/TCF pathway (Wnt-signalling pathway). Therefore, the effect of the NSAID sulindac on nuclear (nonphosphorylated) beta-catenin and beta-catenin/TCF-mediated transcription was investigated. Nuclear #946;-catenin expression was assessed in pretreatment colorectal adenomas and in adenomas after treatment with sulindac from five patients with familial adenomatous polyposis (FAP). Also, the effect of sulindac sulphide on beta-catenin/TCF-mediated transcription was studied. Adenomas of FAP patients collected after treatment with sulindac for up to 6 months showed less nuclear beta-catenin expression compared to pretreatment adenomas of the same patients. Sulindac sulphide abrogated beta-catenin/TCF-mediated transcription in the CRC cell lines DLD1 and SW480, and decreased the levels of nonphosphorylated beta-catenin. As a result, the protein levels of the positively regulated TCF targets Met and cyclin D1 were downregulated after sulindac treatment. This study provides in vivo and in vitro evidence that nuclear beta-catenin localisation and beta-catenin/TCF-regulated transcription of target genes can be inhibited by sulindac. The inhibition of Wnt-signalling provides an explanation for the COX-2-independent mechanism of chemoprevention by NSAID
Uncertainty area-based interference mitigation for cognitive radio
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Measuring routine childhood vaccination coverage in 204 countries and territories, 1980-2019: a systematic analysis for the Global Burden of Disease Study 2020, Release 1
- Author
- Ab Khan M
- Abady GG
- Abbas KM
- Abbasi SW
- Abbastabar H
- Abd-Allah F
- Abdoli A
- Abolhassani H
- Abosetugn AE
- Adabi M
- Adamu AA
- Adetokunboh OO
- Adnani QES
- Advani SM
- Afzal S
- Aghamir SMK
- Ahinkorah BO
- Ahmad S
- Ahmad T
- Ahmadi S
- Ahmed H
- Ahmed MB
- Akalu Y
- Aklilu A
- Akunna CJ
- Al Hamad H
- Al Montasir A
- Al-Eyadhy A
- Alahdab F
- Albano L
- Alemayehu Y
- Alene KA
- Alhassan RK
- Ali L
- Aljunid SM
- Almustanyir S
- Altirkawi KA
- Alvis-Guzman N
- Amu H
- Andrei CL
- Andrei T
- Ansar A
- Ansari-Moghaddam A
- Antonazzo IC
- Antony B
- Arab-Zozani M
- Arabloo J
- Artanti KD
- Arulappan J
- Awan AT
- Awoke MA
- Ayza MA
- Azarian G
- Azzam AY
- Babar Z-U-D
- Bach XT
- Balakrishnan S
- Banach M
- Bante SA
- Barnighausen TW
- Barqawi HJ
- Barrow A
- Bassat Q
- Bayarmagnai N
- Bekuma TT
- Belay HG
- Belgaumi UI
- Bhagavathula AS
- Bhandari D
- Bhardwaj N
- Bhardwaj P
- Bhaskar S
- Bhattacharyya K
- Bibi S
- Bijani A
- Biondi A
- Boloor A
- Braithwaite D
- Buonsenso D
- Butt ZA
- Camargos P
- Carreras G
- Carvalho F
- Castaneda-Orjuela CA
- Chakinala RC
- Charan J
- Chatterjee S
- Chattu SK
- Chattu VK
- Chowdhury FR
- Christopher DJ
- Chu D-T
- Chung S-C
- Cortesi PA
- Costa VM
- Couto RAS
- Dadras O
- Dagnew AB
- Dagnew B
- Dai X
- Dandona L
- Dandona R
- Darshan BB
- De Neve J-W
- Defo BK
- Derseh BT
- Desai R
- Desta AA
- Dhamnetiya D
- Dhimal M
- Dhimal ML
- Dianatinasab M
- Diaz D
- Djalalinia S
- Dorostkar F
- Edem B
- Edinur HA
- Eftekharzadeh S
- El Sayed I
- El-Jaafary S
- Elhadi M
- Elsharkawy A
- Enany S
- Erkhembayar R
- Esezobor CI
- Eskandarieh S
- Ezeonwumelu IJ
- Ezzikouri S
- Fares J
- Faris PS
- Feleke BE
- Ferede TY
- Fernandes E
- Fernandes JC
- Ferrara P
- Filip I
- Fischer F
- Francis MR
- Fukumoto T
- Fullman N
- Gad MM
- Gaidhane S
- Galles NC
- Gallus S
- Garg T
- Geberemariyam BS
- Gebre T
- Gebregiorgis BG
- Gebremedhin KB
- Gebremichael B
- Gessner BD
- Ghadiri K
- Ghafourifard M
- Ghashghaee A
- Giang TV
- Gilani SA
- Glushkova EV
- Golechha M
- Gonfa KB
- Gopalani SV
- Goudarzi H
- Gubari MIM
- Guo Y
- Gupta VB
- Gupta VK
- Gutierrez RA
- Haeuser E
- Halwani R
- Hamidi S
- Hanif A
- Haque S
- Harapan H
- Hargono A
- Hashi A
- Hassan S
- Hassanein MH
- Hassanipour S
- Hassankhani H
- Hay S
- Hayat K
- Hegazy M
- Heidari G
- Hezam K
- Holla R
- Hoque ME
- Hosseini M
- Hosseinzadeh M
- Hostiuc M
- Househ M
- Hsieh VC-R
- Huang J
- Humayun A
- Hussain R
- Hussein NR
- Ibitoye SE
- Ilesanmi OS
- Ilic IM
- Ilic MD
- Inamdar S
- Iqbal U
- Irham LM
- Irvani SSN
- Islam SMS
- Ismail NE
- Itumalla R
- Jha RP
- Joukar F
- Kabir A
- Kabir Z
- Kalhor R
- Kamal Z
- Kamande SM
- Kan FP
- Kandel H
- Kandel SN
- Karch A
- Kassahun G
- Kassebaum NJ
- Katoto PD
- Kelkay B
- Kengne AP
- Khader YS
- Khajuria H
- Khalil IA
- Khan EA
- Khan G
- Khan J
- Khan M
- Khang Y-H
- Khoja AT
- Khubchandani J
- Kim GR
- Kim MS
- Kim YJ
- Kimokoti RW
- Kisa A
- Kisa S
- Korshunov VA
- Kosen S
- Kulkarni V
- Kumar A
- Kumar GA
- Kumar N
- Kwarteng A
- La Vecchia C
- Lami FH
- Landires I
- Lasrado S
- Lassi ZS
- Lee H
- Lee YY
- Levi M
- Lewycka S
- Li S
- Lim SS
- Linh GV
- Liu PY
- Liu X
- Lobo SW
- Lopukhov PD
- Lozano R
- Makki A
- Malik AA
- Mansour-Ghanaei F
- Mansournia MA
- Mantovani LG
- Marks A
- Martins-Melo FR
- Matthews PC
- Medina JRC
- Mendoza W
- Menezes RG
- Mengesha EW
- Meretoja TJ
- Mersha AG
- Mesregah MK
- Mestrovic T
- Miazgowski B
- Milne GJ
- Mirica A
- Mirrakhimov EM
- Mirzaei HR
- Misra S
- Mithra P
- Moghadaszadeh M
- Mohamed TA
- Mohammad KA
- Mohammad Y
- Mohammadi M
- Mohammadian-Hafshejani A
- Mohammed A
- Mohammed S
- Mohapatra A
- Mokdad AH
- Molla MD
- Molokhia M
- Monasta L
- Moni MA
- Moore CE
- Moradi G
- Moradzadeh R
- Moraga P
- Mosser JF
- Mueller UO
- Munro SB
- Murray CJL
- Naghavi M
- Naimzada MD
- Naveed M
- Nayak BP
- Negoi I
- Nguyen J
- Nguyen TH
- Nikbakhsh R
- Ningrum DNA
- Nixon MR
- Nnaji CA
- Noubiap JJ
- Nunez-Samudio V
- Nwatah VE
- Oancea B
- Ochir C
- Ogbo FA
- Olagunju AT
- Olakunde BO
- Onwujekwe OE
- Otstavnov N
- Otstavnov SS
- Owolabi MO
- Padubidri JR
- Pakshir K
- Park E-C
- Pathak M
- Paudel R
- Pawar S
- Pereira J
- Peres MFP
- Perianayagam A
- Pinheiro M
- Pirestani M
- Podder V
- Polibin R
- Pollok RCG
- Postma MJ
- Pottoo FH
- Rabiee M
- Rabiee N
- Radfar A
- Rafiei A
- Rahimi-Movaghar V
- Rahman M
- Rahmani AM
- Rahmawaty S
- Rajesh A
- Ramirez DFB
- Ramshaw RE
- Ranasinghe P
- Rao CR
- Rao SJ
- Rashid TA
- Rathi P
- Rawaf DL
- Rawaf S
- Razek MMAE
- Renzaho AMN
- Rezaei N
- Rezai MS
- Rios-Blancas M
- Rogowski ELB
- Rolfe S
- Ronfani L
- Rwegerera GM
- Saad AM
- Sabour S
- Saddik B
- Saeb MR
- Saeed U
- Sahebkar A
- Sahraian MA
- Salam N
- Salih YA
- Salimzadeh H
- Samaei M
- Samy AM
- Sanabria J
- Sanmarchi F
- Santric-Milicevic MM
- Sartorius B
- Sarveazad A
- Sathian B
- Saute RL
- Sawhney M
- Saxena D
- Saxena S
- Sbarra AN
- Schipp MF
- Seidu A-A
- Seylani A
- Shaikh MA
- Shamsizadeh M
- Shetty PH
- Shigematsu M
- Shin JI
- Sidemo NB
- Singh A
- Singh JA
- Sinha S
- Skryabin VY
- Skryabina AA
- Soheili A
- Tadesse EG
- Tamiru AT
- Tan K-K
- Tekalegn Y
- Temsah M-H
- Thakur B
- Thapar R
- Thavamani A
- Tobe-Gai R
- Tohidinik HR
- Tovani-Palone MR
- Traini E
- Tripathi M
- Tsegaye B
- Tsegaye GW
- Ullah A
- Ullah S
- Ullah S
- Unim B
- Updike RL
- Vacante M
- Velazquez DZ
- Vo B
- Vollmer S
- Waheed Y
- Winkler AS
- Wiysonge CS
- Yigit V
- Yirdaw BW
- Yon DK
- Yonemoto N
- Yu C
- Yuce D
- Yunusa I
- Zaki MES
- Zamani M
- Zamanian M
- Zewdie DT
- Zhang Z-J
- Zhong C
- Zumla A
- Publication venue
- ELSEVIER SCIENCE INC
- Publication date
- 07/08/2021
- Field of study
Background: Measuring routine childhood vaccination is crucial to inform global vaccine policies and programme implementation, and to track progress towards targets set by the Global Vaccine Action Plan (GVAP) and Immunization Agenda 2030. Robust estimates of routine vaccine coverage are needed to identify past successes and persistent vulnerabilities. Drawing from the Global Burden of Diseases, Injuries, and Risk Factors Study (GBD) 2020, Release 1, we did a systematic analysis of global, regional, and national vaccine coverage trends using a statistical framework, by vaccine and over time. //
Methods: For this analysis we collated 55 326 country-specific, cohort-specific, year-specific, vaccine-specific, and dose-specific observations of routine childhood vaccination coverage between 1980 and 2019. Using spatiotemporal Gaussian process regression, we produced location-specific and year-specific estimates of 11 routine childhood vaccine coverage indicators for 204 countries and territories from 1980 to 2019, adjusting for biases in country-reported data and reflecting reported stockouts and supply disruptions. We analysed global and regional trends in coverage and numbers of zero-dose children (defined as those who never received a diphtheria-tetanus-pertussis [DTP] vaccine dose), progress towards GVAP targets, and the relationship between vaccine coverage and sociodemographic development. //
Findings: By 2019, global coverage of third-dose DTP (DTP3; 81·6% [95% uncertainty interval 80·4–82·7]) more than doubled from levels estimated in 1980 (39·9% [37·5–42·1]), as did global coverage of the first-dose measles-containing vaccine (MCV1; from 38·5% [35·4–41·3] in 1980 to 83·6% [82·3–84·8] in 2019). Third-dose polio vaccine (Pol3) coverage also increased, from 42·6% (41·4–44·1) in 1980 to 79·8% (78·4–81·1) in 2019, and global coverage of newer vaccines increased rapidly between 2000 and 2019. The global number of zero-dose children fell by nearly 75% between 1980 and 2019, from 56·8 million (52·6–60·9) to 14·5 million (13·4–15·9). However, over the past decade, global vaccine coverage broadly plateaued; 94 countries and territories recorded decreasing DTP3 coverage since 2010. Only 11 countries and territories were estimated to have reached the national GVAP target of at least 90% coverage for all assessed vaccines in 2019. //
Interpretation: After achieving large gains in childhood vaccine coverage worldwide, in much of the world this progress was stalled or reversed from 2010 to 2019. These findings underscore the importance of revisiting routine immunisation strategies and programmatic approaches, recentring service delivery around equity and underserved populations. Strengthening vaccine data and monitoring systems is crucial to these pursuits, now and through to 2030, to ensure that all children have access to, and can benefit from, lifesaving vaccines
Canagliflozin and Renal Outcomes in Type 2 Diabetes and Nephropathy
- Author
- Abdallah J
- Abdul Ghani R
- Abdul Kadir K
- Abdullah R
- Abejuela Z
- Abouglila K
- Abraham O
- Abrahamsen I
- Abraira Munoz Ld
- Abramowitz M
- Abusnana S
- Accini Mendoza Jl
- Acosta I
- Agafyina A
- Agarwal R
- Agarwal Rajiv
- Aggarwal N
- Agra J
- Ahmed F
- Ahuad Guerrero Ra
- Aiello J
- Ainsworth P
- Aizenberg D
- Akiyama H
- Akright L
- Akyea-Djamson A
- Al-Karadsheh A
- Alamartine E
- Alappan R
- Albisu Jp
- Ali N
- Ali Z
- Alicic R
- Allison Dc
- Alvarez Garcia P
- Alvarisqueta A
- Alves da Costa Fa
- Amerena J
- Amigo A
- Amodeo C
- Andreeva V
- Andreotti Turatti La
- Angelescu Lm
- Angelova A
- Anghel V
- Antic S
- Aoki H
- Aquitania G
- Arauz-Pacheco C
- Arcos E
- Arfeen S
- Arif A
- Aronson R
- Arutchelvam V
- Arvind M
- Asano A
- Asmarats Mercadal L
- Atray N
- August P
- Avendano J
- Avram Ri
- Awad A
- Babikova J
- Badat A
- Bajaj H
- Baker J
- Bakris G
- Bakris George
- Barbarash O
- Barna O
- Barnard M
- Barnhill P
- Barranco E
- Barrera C
- Barrios C
- Bartaskova D
- Bartolacci I
- Barton P
- Barysheva O
- Beacom M
- Behara V
- Bellary S
- Belo D
- Belobradkova J
- Bentley-Lewis R
- Benusova O
- Berenguer R
- Berli Ma
- Bermudez L
- Bernardo M
- Bhushan Rs
- Bijata-Bronisz R
- Bilyk Sd
- Bin Shudim Ss
- Biscoveanu M
- Bompoint Severine
- Bonadonna R
- Bordonava A
- Borot S
- Botsyurko V
- Bowman-Stroud C
- Brandon D
- Breedt J
- Bregman R
- Brenner Barry M
- Brenner Bm
- Brusco O
- Buganova I
- Bull Scott
- Burst V
- Busch R
- Busegeanu Mm
- Calella P
- Camelo Sanches Fc
- Canaan Y
- Canani Lh
- Cannon Christopher P
- Cannon Cp
- Cantero Mc
- Capuano George
- Caringal C
- Cariou B
- Cartasegna Lr
- Castellino P
- Cercos E
- Cereto Castro F
- Ceriello A
- Chacra Ar
- Chae Dw
- Chang Ct
- Chang T
- Chapin L
- Charytan David M
- Charytan Dm
- Che Yusof Md
- Cheifetz A
- Chen E
- Chen N
- Chen Q
- Cherney D 3rd
- Chertow G
- Cheung Nw
- Chilito A
- Chiovato L
- Chizhov D
- Cho Ym
- Chouinard G
- Christensen T
- Christiano C
- Christofides E
- Chu Pei-Ling
- Chuang Lm
- Chuateco C
- Cif A
- Cigarran Guldris S
- Coffee L
- Cohen K
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- Cohen-Stein D
- Coloma Gc
- Colombo H
- Comia Rs
- Commendatore V
- Conway J
- Cook C
- Correa Rotter Jr
- Cosma D
- Cournoyer S
- Coyne D
- Crisan C
- Cruz Jb
- Csecsei G
- Csiky B
- Cuadrado J
- Culak J
- Cuneo Ca
- Cunha Borges Jl
- Cunha V\ueancio Sa
- Cusumano Am
- D'Avila D
- da Silva Franco Rj
- Daboul N
- Danos P
- Daroza G
- Darwish R
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- Davies E
- Davies M
- Davies S
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- De Brouwer K
- de Carvalho Contieri Fl
- De Cosmo S
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- De Serres S
- de Souza Portes E
- de Souza P
- de Zeeuw D
- de Zeeuw Dick
- Deak L
- Deboni Lm
- Deck K
- Deepak D
- Delos Santos L
- Demian Ld
- Derosa G
- Desouza C
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- Diaz Escalante T
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- Dimitrov S
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- Djordjevic M
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- Dran Rd
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- El Kossi M
- El-Shahawy M
- Elosegui Am
- English P
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- Ervin J
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- Faulmann G
- Fauvel Jp
- Ferariu Ie
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- Fernandez Fresnedo G
- Fernandez Mf
- Feuerstein J
- Filho Ff
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- Fraser D
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- Fretes Jo
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- Goldenberg R
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- Gomez Rangel Jd
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- Gonzalez Colaso Pdc
- Gonzalez Martinez Fj
- Gonzalez Orellana Ra
- Gonzalez A
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- Gopalakrishnan N
- Gorelick P
- Gorson D
- Gourdy P
- Govender R
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- Halmagyi I
- Hammoud J
- Hancu N
- Handelsman S
- Hanif W
- Hansraj A
- Harcsa E
- Harris R
- Hartman I
- Hawley C
- Heerspink Hiddo J L
- Heerspink Hjl
- Henein S
- Hermida S
- Hernandez Triana E
- Herrera Jimenez Le
- Hershon K
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- Higuera Jd
- Hillis G
- Hiser D
- Holman R
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- Hsieh Ch
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- Ignjatovic J
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- Keshavamurthy Cb
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- Khirmanov V
- Khitan Z
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- Klyver Mi
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- Koizumi M
- Kolansky D
- Kolesnyk I
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- Komisarenko I
- Kong Wy
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- Koundurdjiev A
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- Kumeda Y
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- Zateyshshikov D
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- Zhang H
- Zhang Hong
- Zhao M
- Zheng H
- Zinman B
- Zinman Bernard
- Zlova T
- Zsom M
- Zub L
- Zykova T
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2019
- Field of study
BACKGROUND Type 2 diabetes mellitus is the leading cause of kidney failure worldwide, but few effective long-term treatments are available. In cardiovascular trials of inhibitors of sodium–glucose cotransporter 2 (SGLT2), exploratory results have suggested that such drugs may improve renal outcomes in patients with type 2 diabetes. METHODS In this double-blind, randomized trial, we assigned patients with type 2 diabetes and albuminuric chronic kidney disease to receive canagliflozin, an oral SGLT2 inhibitor, at a dose of 100 mg daily or placebo. All the patients had an estimated glomerular filtration rate (GFR) of 30 to 300 to 5000) and were treated with renin–angiotensin system blockade. The primary outcome was a composite of end-stage kidney disease (dialysis, transplantation, or a sustained estimated GFR of <15 ml per minute per 1.73 m 2), a doubling of the serum creatinine level, or death from renal or cardiovascular causes. Prespecified secondary outcomes were tested hierarchically. RESULTS The trial was stopped early after a planned interim analysis on the recommendation of the data and safety monitoring committee. At that time, 4401 patients had undergone randomization, with a median follow-up of 2.62 years. The relative risk of the primary outcome was 30% lower in the canagliflozin group than in the placebo group, with event rates of 43.2 and 61.2 per 1000 patient-years, respectively (hazard ratio, 0.70; 95% confidence interval [CI], 0.59 to 0.82; P=0.00001). The relative risk of the renal-specific composite of end-stage kidney disease, a doubling of the creatinine level, or death from renal causes was lower by 34% (hazard ratio, 0.66; 95% CI, 0.53 to 0.81; P<0.001), and the relative risk of end-stage kidney disease was lower by 32% (hazard ratio, 0.68; 95% CI, 0.54 to 0.86; P=0.002). The canagliflozin group also had a lower risk of cardiovascular death, myocardial infarction, or stroke (hazard ratio, 0.80; 95% CI, 0.67 to 0.95; P=0.01) and hospitalization for heart failure (hazard ratio, 0.61; 95% CI, 0.47 to 0.80; P<0.001). There were no significant differences in rates of amputation or fracture. CONCLUSIONS In patients with type 2 diabetes and kidney disease, the risk of kidney failure and cardiovascular events was lower in the canagliflozin group than in the placebo group at a median follow-up of 2.62 years
The global burden of cancer attributable to risk factors, 2010-19: a systematic analysis for the Global Burden of Disease Study 2019
- Author
- Aali A
- Ab Khan M
- Abbas Q
- Abbasi B
- Abbasi-Kangevari M
- Abbasi-Kangevari Z
- Abbastabar H
- Abd-Elsalam S
- Abdelmasseh M
- Abdelwahab AA
- Abdoli G
- Abdulkader RS
- Abdulkadir HA
- AbdulmuhsinMohammad K
- Abedi A
- Abegaz KH
- Abidi H
- Aboagye RG
- Abolhassani H
- Absalan A
- Abtew YD
- Abu-Gharbieh E
- Achappa B
- Acheson AR
- Acuna JM
- Addison D
- Addo IY
- Adegboye OA
- Adesina MA
- Adnan M
- Adnani QES
- Advani SM
- Afrin S
- Afzal MS
- Aggarwal M
- Ahinkorah BO
- Ahmad AR
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- Ahmad S
- Ahmadi S
- Ahmed H
- Ahmed LA
- Ahmed MB
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- Ajami M
- Akalu GT
- Akbarzadeh-Khiavi M
- Aklilu A
- Akonde M
- Akunna CJ
- Al Hamad H
- Al Montasir A
- Al-Hanawi MK
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- Al-Raddadi RM
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- Al-Tammemi AB
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- Publication venue
- Publication date
- 20/08/2022
- Field of study
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
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- Fiorini L
- Fisher WC
- Fitschen T
- Fitzhugh PM
- Fleck I
- Fleischmann P
- Flick T
- Flores Castillo LR
- Flores Sanz De Acedo L
- Flores M
- Follega FM
- Fomin N
- Foo JH
- Forland BC
- Formica A
- Forti AC
- Fortin E
- Fortman AW
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- Francescato S
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- Fung KY
- Furtado De Simas Filho E
- Furukawa M
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- Gaudio G
- Gautam V
- Gauzzi P
- Gavrilenko IL
- Gavrilyuk A
- Gay C
- Gaycken G
- Gazis EN
- Geanta AA
- Gee CM
- Gemme C
- Genest MH
- Gentile S
- Gentry AD
- George S
- George WF
- Geralis T
- Gessinger-Befurt P
- Geyik ME
- Ghani M
- Ghneimat M
- Ghorbanian K
- Ghosal A
- Ghosh A
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- Giacobbe B
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- Giani T
- Giannetti P
- Giannini A
- Gibson SM
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- Gilbert AK
- Gilbert BJ
- Gillberg D
- Gilles G
- Gillwald NEK
- Ginabat L
- Gingrich DM
- Giordani MP
- Giraud PF
- Giugliarelli G
- Giugni D
- Giuli F
- Gkialas I
- Gladilin LK
- Glasman C
- Gledhill GR
- Glemža G
- Glisic M
- Gnesi I
- Go Y
- Goblirsch-Kolb M
- Gocke B
- Godin D
- Gokturk B
- Goldfarb S
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- Gololo MGD
- Golubkov D
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- Gonzalez Fernandez S
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- Gonzalvo Rodriguez GR
- González Andana RY
- González de la Hoz S
- Gonçalo R
- Goossens L
- Gorini B
- Gorini E
- Gorišek A
- Gosart TC
- Goshaw AT
- Gostkin MI
- Goswami S
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- Gross E
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- Guan W
- Gubbels C
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- Guescini F
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- Guilloton E
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- Kalaitzidou I
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- Karentzos E
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- Lakomiec IK
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- Landgraf U
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- Lang VS
- Langenberg RJ
- Langrekken OKB
- Lankford AJ
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- Lanza A
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- Laporte JF
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- Lasagni Manghi F
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- Zhemchugov A
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- Zhulanov V
- Zimine NI
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adam Bourdarios C
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
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- Agaras MN
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- Aggarwal A
- Agheorghiesei C
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- Akolkar NN
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- Albouy GL
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- Aleksa M
- Aleksandrov IN
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- Öncel ÖO
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- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Model demonstrates functional purpose of the nasal cycle
- Author
- AI Atanasov
- B Button
- B Button
- B Button
- B Drettner
- B Winther
- C Jiang
- D Elad
- D White
- David E White
- DE White
- DE White
- DE White
- DG Roblin
- DW Kennedy
- DXY Wu
- FM White
- H-J Cho
- HM Druce
- HP Lee
- I Hahn
- I Hörschler
- J Bartley
- J Hanif
- J Lindemann
- J Lindemann
- J Lindemann
- JF Douglas
- JH Widdicombe
- JH Widdicombe
- Jim Bartley
- K Keyhani
- K Wiesmiller
- L Trout
- LM Hanna
- LM Hanna
- M Hasegawa
- M Wolf
- MF Refojo
- MS Quraishi
- N Chhabra
- N Mygind
- N Warren
- P Cole
- R Bossi
- R Eccles
- R Fodil
- R Tarran
- R Tarran
- RA Cone
- RB Williams
- Roy J Nates
- S Naftali
- S Naftali
- SS Davis
- ST Ballard
- T Keck
- WF Whimster
- WM Boek
- X Guan-xia
- YS Cheng
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Recommended from our members
Prevalence, years lived with disability, and trends in anaemia burden by severity and cause, 1990-2021: findings from the Global Burden of Disease Study 2021
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- Zheng P
- Zoeckler LZ
- Zoladl M
- Publication venue
- Elsevier
- Publication date
- 29/08/2023
- Field of study
Background
Anaemia is a major health problem worldwide. Global estimates of anaemia burden are crucial for developing appropriate interventions to meet current international targets for disease mitigation. We describe the prevalence, years lived with disability, and trends of anaemia and its underlying causes in 204 countries and territories.
Methods
We estimated population-level distributions of haemoglobin concentration by age and sex for each location from 1990 to 2021. We then calculated anaemia burden by severity and associated years lived with disability (YLDs). With data on prevalence of the causes of anaemia and associated cause-specific shifts in haemoglobin concentrations, we modelled the proportion of anaemia attributed to 37 underlying causes for all locations, years, and demographics in the Global Burden of Disease Study 2021.
Findings
In 2021, the global prevalence of anaemia across all ages was 24·3% (95% uncertainty interval [UI] 23·9–24·7), corresponding to 1·92 billion (1·89–1·95) prevalent cases, compared with a prevalence of 28·2% (27·8–28·5) and 1·50 billion (1·48–1·52) prevalent cases in 1990. Large variations were observed in anaemia burden by age, sex, and geography, with children younger than 5 years, women, and countries in sub-Saharan Africa and south Asia being particularly affected. Anaemia caused 52·0 million (35·1–75·1) YLDs in 2021, and the YLD rate due to anaemia declined with increasing Socio-demographic Index. The most common causes of anaemia YLDs in 2021 were dietary iron deficiency (cause-specific anaemia YLD rate per 100 000 population: 422·4 [95% UI 286·1–612·9]), haemoglobinopathies and haemolytic anaemias (89·0 [58·2–123·7]), and other neglected tropical diseases (36·3 [24·4–52·8]), collectively accounting for 84·7% (84·1–85·2) of anaemia YLDs.
Interpretation
Anaemia remains a substantial global health challenge, with persistent disparities according to age, sex, and geography. Estimates of cause-specific anaemia burden can be used to design locally relevant health interventions aimed at improving anaemia management and prevention.
Funding
Bill & Melinda Gates Foundation
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