184 research outputs found
Experiments and simulations of MEMS thermal sensors for wall shear-stress measurements in aerodynamic control applications
- Author
- Bandyopadhyay P R
- Bellhouse B J
- Bellhouse B J
- Brown G L
- Bushnell D M
- Cain A Chandrasekaran V Nishida T Sheplak M
- Chih-Ming Ho
- Fukang Jiang
- Goldstein S
- Haritonidis J H
- Ho C-M
- Huang J B
- Hyman D
- Incropera F P
- James Lew
- Jiang F Tai Y-C Gupta B Goodman R Tung S Huang J B Ho C-M
- Jiang F Tai Y-C Huang J B Ho C-M
- Jiang F Tai Y-C Walsh K Tsao T Lee G B Ho C-M
- Kalvesten E
- Liu C Tai Y-C Huang J B Ho C-M
- Lofdahl L
- Ludwieg H
- Moin P Kim J Choi H
- Pan T
- Qiao Lin
- Sheplak M
- Tsao T Jiang F Miller R A Tai Y-C Gupta B Goodman R Tung S Ho C-M
- Wang X Q Han Z Jiang F Tsao T Lin Q Tai Y C Koosh V Goodman R Lew J Ho C M
- Xu Y Jiang F Lin Q Clendenen J Tung S Tai Y-C
- Xuan-Qi Wang
- Yong Xu
- Yu-Chong Tai
- Zhigang Han
- Publication venue
- 'IOP Publishing'
- Publication date
- Field of study
Simple Dip-Coating Process for the Synthesis of Small Diameter Single-Walled Carbon Nanotubes—Effect of Catalyst Composition and Catalyst Particle Size on Chirality and Diameter
- Author
- Arfsten N. J.
- Artur Malolepszy
- Bachilo S. M.
- Bachilo S. M.
- Baigl D.
- Balasubramanian K.
- Bindl D. J.
- Chattopadhyay D.
- Chen T. T.
- Cheuk W. Tai
- Chhowalla M.
- Choi Y. C.
- Ding F.
- Ding F.
- Ding L.
- Downard A. J.
- Dresselhaus M. S.
- Eres G.
- Florian Nitze
- Gledhill S.
- Groenvel P.
- Hamid R. Barzegar
- Harutyunyan A. R.
- Herrera J. E.
- Hersam M. C.
- Ho G. W.
- Hsiou Y. F.
- Huang H. J.
- Iijima S.
- Journet C.
- Kim S. N.
- Kim W. J.
- Krozel J. W.
- Krupke R.
- Kwok C. T. M.
- Leszek Stobinski
- Lin M.
- Liu B. B.
- Lolli G.
- Madeleine Ramstedt
- Maruyama S.
- Nitze F.
- Ortega-Cervantez G.
- Ortiz-Lopez J.
- Picher M.
- Qu L. T.
- Rao A. M.
- Reich S.
- Rinzler A. G.
- Rinzler A. G.
- Rizzato A. P.
- Rodriguez-Manzo J. A.
- Strano M. S.
- Strano M. S.
- Thess A.
- Thomas Wågberg
- Thostenson E. T.
- Tiva Sharifi
- Wang B.
- Wei Y. Y.
- Yoon Y. J.
- Zhao N. Q.
- Publication venue
- American Chemical Society
- Publication date
- Field of study
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
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- A. -G. Wu
- A. C. Ma
- A. K. Au
- Abdel-Aziz A. K.
- Abdelfatah S.
- Abdellatif M.
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- Abeliovich H.
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- Acevedo-Arozena A.
- Adamopoulos I. E.
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- Zerovnik E.
- Zhan L.
- Zhang B.
- Zhang D. D.
- Zhang H.
- Zhang H.
- Zhang H.
- Zhang H.
- Zhang H.
- Zhang H.
- Zhang H.
- Zhang H. -L.
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- Zhang J.
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- Zhang L.
- Zhang L. W.
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- Zhang P.
- Zhang S.
- Zhang W.
- Zhang X.
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- Zhang X.
- Zhang X.
- Zhang X. -W.
- Zhang X. D.
- Zhang Y.
- Zhang Y.
- Zhang Y.
- Zhang Y.
- Zhang Y.
- Zhang Y. -D.
- Zhang Y. -Y.
- Zhang Z.
- Zhang Z.
- Zhang Z.
- Zhang Z.
- Zhang Z.
- Zhang Z.
- Zhao H.
- Zhao L.
- Zhao S.
- Zhao T.
- Zhao X. -F.
- Zhao Y.
- Zhao Y.
- Zhao Y.
- Zhao Y.
- Zheng G.
- Zheng K.
- Zheng L.
- Zheng S.
- Zheng X. -L.
- Zheng Y.
- Zheng Z. -G.
- Zhivotovsky B.
- Zhong Q.
- Zhou A.
- Zhou B.
- Zhou C.
- Zhou G.
- Zhou H.
- Zhou H.
- Zhou H.
- Zhou J.
- Zhou J.
- Zhou J.
- Zhou J.
- Zhou K.
- Zhou R.
- Zhou X. -J.
- Zhou Y.
- Zhou Y.
- Zhou Y.
- Zhou Z.
- Zhou Z. -Y.
- Zhu B.
- Zhu C.
- Zhu G. -Q.
- Zhu H.
- Zhu H.
- Zhu H.
- Zhu W. -G.
- Zhu Y.
- Zhu Y.
- Zhuang H.
- Zhuang X.
- Zientara-Rytter K.
- Zimmermann C. M.
- Ziviani E.
- Zoladek T.
- Zong W. -X.
- Zorov D. B.
- Zorzano A.
- Zou W.
- Zou Z.
- Zou Z.
- Zuryn S.
- Zwerschke W.
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
Estimating PM 2.5 concentrations in Xi'an City using a generalized additive model with multi-source monitoring data
- Author
- A Notario
- A Saiz-Lopez
- APK Tai
- AW Strawa
- B Gao
- BL MR Boys
- BV Bhaskar
- C Liousse
- C Yang
- C Zhang
- CA Pope
- CJ Paciorek
- CQ LY Lin
- DA Chu
- DC Carslaw
- DE Abbey
- DK Deshmukh
- DP Edwards
- DYH Pui
- F Costabile
- F Yang
- G Lin
- G Wang
- GX Li
- Hong-Lei Yang
- HS Bian
- HS Kim
- J Schwartz
- J Tao
- J Tian
- J Wang
- J Wang
- JG Watson
- JH Seinfeld
- JJ Cao
- Jun-Huan Peng
- JY Xin
- K Zhang
- K Zhang
- KF Ho
- L Glasser
- L Tao
- LF Li
- LWA Chen
- M Khodeir
- M Schaap
- M Sorek-Hamer
- ML Bell
- N Kumar
- P Glantz
- P Gupta
- Qian Sun
- Qinghua Sun
- R Federal
- R Federal
- RA Moyeed
- RB Schlesinger
- RBA Koelemeijer
- RM Hoff
- S Wood
- SC Dogruparmak
- SS Lim
- W Song
- X Chi
- XF Hu
- Y Huang
- Y Liu
- Y Liu
- Y Liu
- Y Wang
- Yi-Rong Song
- YL Sun
- YM Guo
- Yong-Ze Song
- YP DJ Lu
- YS Wang
- Yuan Li
- Z Hu
- Z Li
- Z Ma
- Z Meng
- Z Sun
- Z Wang
- ZW Yan
- ZX Shen
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2015
- Field of study
© 2015 Song et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Particulate matter with an aerodynamic diameter <2.5 μm (PM2.5) represents a severe environmental problem and is of negative impact on human health. Xi'an City, with a population of 6.5 million, is among the highest concentrations of PM2.5 in China. In 2013, in total, there were 191 days in Xi'an City on which PM2.5 concentrations were greater than 100 μg/m3. Recently, a few studies have explored the potential causes of high PM2.5 concentration using remote sensing data such as the MODIS aerosol optical thickness (AOT) product. Linear regression is a commonly used method to find statistical relationships among PM2.5 concentrations and other pollutants, including CO, NO2, SO2, and O3, which can be indicative of emission sources. The relationships of these variables, however, are usually complicated and non-linear. Therefore, a generalized additive model (GAM) is used to estimate the statistical relationships between potential variables and PM2.5 concentrations. This model contains linear functions of SO2 and CO, univariate smoothing non-linear functions of NO2, O3, AOT and temperature, and bivariate smoothing non-linear functions of location and wind variables. The model can explain 69.50% of PM2.5 concentrations, with R2 = 0.691, which improves the result of a stepwise linear regression (R2 = 0.582) by 18.73%. The two most significant variables, CO concentration and AOT, represent 20.65% and 19.54% of the deviance, respectively, while the three other gas-phase concentrations, SO2, NO2, and O3 account for 10.88% of the total deviance. These results show that in Xi'an City, the traffic and other industrial emissions are the primary source of PM2.5. Temperature, location, and wind variables also non-linearly related with PM2.5
SPARC Overexpression Inhibits Cell Proliferation in Neuroblastoma and Is Partly Mediated by Tumor Suppressor Protein PTEN and AKT
- Author
- A Chlenski
- A Chlenski
- A Francki
- A Francki
- A Mettouchi
- A Tivnan
- A Yokomizo
- AD Bradshaw
- B Stiles
- BD Manning
- Bharathi Gorantla
- BJ Schiemann
- C Chetty
- C Chetty
- C Chetty
- C Kupprion
- CA Granville
- Christopher S. Gondi
- CJ Clark
- DE Discher
- E Healy
- F Teyssier
- G Iliakis
- G Poste
- G. S. Sailaja
- GK Yiu
- H Liu
- H Sage
- I Tonic
- IT Tai
- IT Tai
- J Briggs
- J Brognard
- J Li
- J Nie
- Jasti S. Rao
- Jeffrey D. Klopfenstein
- JT Dong
- Jun Li
- K Motamed
- K Motamed
- L Li
- L Wang
- L Weng
- LC Cantley
- LP Weng
- M Zhan
- MB Kastan
- MC Hollander
- ME Perry
- Meena Gujrati
- MG Vander Heiden
- MH Cardone
- MJ Tang
- N Dhanesuan
- N Said
- NG Bazan
- OL Podhajcer
- P Bhoopathi
- P Bhoopathi
- P Bhoopathi
- P Kurschat
- PA Janmey
- PA Puolakkainen
- PA Steck
- Praveen Bhoopathi
- Q Yan
- R Ho
- RA Brekken
- S Petersen
- SL Thomas
- SR Datta
- T Yeung
- TJ Jorgensen
- W Zhang
- WC Huang
- XY Yin
- Y Shiloh
- Y Yan
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Secreted protein acidic and rich in cysteine (SPARC) is also known as BM-40 or Osteonectin, a multi-functional protein modulating cell–cell and cell–matrix interactions. In cancer, SPARC is not only linked with a highly aggressive phenotype, but it also acts as a tumor suppressor. In the present study, we sought to characterize the function of SPARC and its role in sensitizing neuroblastoma cells to radio-therapy. SPARC overexpression in neuroblastoma cells inhibited cell proliferation in vitro. Additionally, SPARC overexpression significantly suppressed the activity of AKT and this suppression was accompanied by an increase in the tumor suppressor protein PTEN both in vitro and in vivo. Restoration of neuroblastoma cell radio-sensitivity was achieved by overexpression of SPARC in neuroblastoma cells in vitro and in vivo. To confirm the role of the AKT in proliferation inhibited by SPARC overexpression, we transfected neuroblastoma cells with a plasmid vector carrying myr-AKT. Myr-AKT overexpression reversed SPARC-mediated PTEN and increased proliferation of neuroblastoma cells in vitro. PTEN overexpression in parallel with SPARC siRNA resulted in decreased AKT phosphorylation and proliferation in vitro. Taken together, these results establish SPARC as an effector of AKT-PTEN-mediated inhibition of proliferation in neuroblastoma in vitro and in vivo
11-Oxoeicosatetraenoic Acid Is a Cyclooxygenase-2/15-Hydroxyprostaglandin Dehydrogenase-Derived Antiproliferative Eicosanoid
- Author
- Backlund M. G.
- Bishop-Bailey D.
- Blair I. A.
- Celis J. E.
- Chou W. L.
- Ciucci A.
- Dixon D. A.
- Fitzpatrick F. A.
- Forman B. M.
- Grant G. E.
- Grant G. E.
- Groeger A. L.
- Ho T. C.
- Holla V. R.
- Huang R. Y.
- Hughes D.
- Ian A. Blair
- Ide T.
- Jasbir S. Arora
- Lee S. H.
- Lee S. H.
- Lee S. H.
- Markowitz S. D.
- Matsumura F.
- Meijerman I.
- Mesaros C.
- Nathaniel W. Snyder
- Ouyang W.
- Ouyang W.
- O’Flaherty J. T.
- Pham H.
- Piva R.
- Rangachari P. K.
- Scher J. U.
- Shao J.
- Straus D. S.
- Suhong Zhang
- Sumit J. Shah
- Tai H. H.
- Thiel A.
- Tseng-Rogenski S.
- Waddington E.
- Waku T.
- Wang D.
- Wang D.
- Wei C.
- Xiaojing Liu
- Xin X.
- Publication venue
- American Chemical Society
- Publication date
- Field of study
Global patient outcomes after elective surgery: prospective cohort study in 27 low-, middle- and high-income countries.
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- Ab Majid MA
- Ab Rahman AS
- Ab Rahman R
- Abayasinghe C
- Abbott T
- Abdullah NH
- Abdur-Rahman L
- Abeloos J
- Abernethy C
- Abidin UN
- Abrunhosa A
- Absar M
- Adam S
- Adams D
- Adams G
- Adamson M
- Adekola O
- Adelaja Y
- Ademola A
- Adenekan A
- Adenike O
- Adeolu AA
- Adetoye A
- Adewale B
- Adigun T
- Adolfsson A
- Aflori L
- Africander C
- Afshari A
- Agarwal V
- Agodirin O
- Aguero Vera M
- Aguzzoli C
- Ahmad A
- Ahmad N
- Ahmad T
- Ahmad T
- Ahmad Y
- Ahmed A
- Ahmed J
- Ai Y
- Aignatoaie M
- Aimoniotou-Georgiou E
- Akanmu O
- Akerman N
- Akinwale M
- Akring I
- Al 'Amri K
- Al Anizi M
- Al hussaini S
- Al-Soudaine Y
- Aladin H
- Alagbe-Briggs O
- Alaman Laguarda G
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- Alcock D
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- Alexander H
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- Alherz F
- Ali H
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- Almeida W
- Alonso E
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- Alves MJ
- Amador JCB
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- Amendola CP
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- Anagnou A
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- Zhu X
- Zhu X
- Zhu Y
- Zhu Z
- Zhuang X
- Zilis G
- Zin NHM
- Zingerle N
- Zingg U
- Zoerner F
- Zonneveldt H
- Zou X
- Zoulamoglou M
- Zsisku L
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2016
- Field of study
BACKGROUND: As global initiatives increase patient access to surgical treatments, there remains a need to understand the adverse effects of surgery and define appropriate levels of perioperative care. METHODS: We designed a prospective international 7-day cohort study of outcomes following elective adult inpatient surgery in 27 countries. The primary outcome was in-hospital complications. Secondary outcomes were death following a complication (failure to rescue) and death in hospital. Process measures were admission to critical care immediately after surgery or to treat a complication and duration of hospital stay. A single definition of critical care was used for all countries. RESULTS: A total of 474 hospitals in 19 high-, 7 middle- and 1 low-income country were included in the primary analysis. Data included 44 814 patients with a median hospital stay of 4 (range 2-7) days. A total of 7508 patients (16.8%) developed one or more postoperative complication and 207 died (0.5%). The overall mortality among patients who developed complications was 2.8%. Mortality following complications ranged from 2.4% for pulmonary embolism to 43.9% for cardiac arrest. A total of 4360 (9.7%) patients were admitted to a critical care unit as routine immediately after surgery, of whom 2198 (50.4%) developed a complication, with 105 (2.4%) deaths. A total of 1233 patients (16.4%) were admitted to a critical care unit to treat complications, with 119 (9.7%) deaths. Despite lower baseline risk, outcomes were similar in low- and middle-income compared with high-income countries. CONCLUSIONS: Poor patient outcomes are common after inpatient surgery. Global initiatives to increase access to surgical treatments should also address the need for safe perioperative care. STUDY REGISTRATION: ISRCTN5181700
Super-resolution:A comprehensive survey
- Author
- A Adler
- A Almansa
- A Chakrabarti
- A Corduneanu
- A Gholipour
- A Giachetti
- A Lorette
- A Marquina
- A Panagiotopoulou
- A Schatzberg
- A Zomet
- AJ Patti
- AJ Patti
- AJ Storkey
- AJ Tatem
- AK Katsaggelos
- ALD Martins
- AWMV Eekeren
- AWMV Eekeren
- B Choi
- B Cohen
- B Huhle
- B Li
- B Li
- B Narayanan
- B Wu
- BC Song
- BGV Kumar
- BK Gunturk
- BK Gunturk
- BK Gunturk
- BK Gunturk
- BR Hunt
- C Jung
- C Liu
- C Liu
- C Miravet
- C Miravet
- C Papathanassiou
- C Pohl
- C Su
- C Wang
- C Wang
- CA Segall
- CA Segall
- CA Segall
- CS Tong
- CV Jiji
- CV Jiji
- CV Jiji
- D Calle
- D Capel
- D Datsenko
- D Lin
- D Pastina
- D Rajan
- D Rajan
- D Rajan
- D Rajan
- D Rajan
- D Rajan
- D Robinson
- D Robinson
- D Yldrm
- D Zhang
- D Zhang
- DO Walsh
- DP Capel
- E Salari
- E Shechtman
- EM Hung
- F Champagnat
- F Humblot
- F Rousseau
- F Sroubek
- F Sroubek
- F Zhou
- FM Candocia
- G Dedeoglu
- G Gilboa
- G Ye
- GH Costa
- GH Costa
- GH Costa
- GK Chantas
- GM Callic
- GM Callico
- H Bouzari
- H Chang
- H Demirel
- H He
- H He
- H Huang
- H Huanga
- H Ji
- H Nasir
- H Shekarforoush
- H Shekarforoush
- H Shekarforoush
- H Stark
- H Su
- H Su
- H Takeda
- H Takeda
- H Yang
- H Zhang
- H Zhang
- H Zhao
- HF Shen
- HK Aghajan
- I Begin
- J Chen
- J Chung
- J Cui
- J Sun
- J Tian
- J Tian
- J Tian
- J Wang
- J Wang
- J Wu
- J Yang
- J Yang
- J Yu
- JA Kennedy
- JD Ouwerkerk van
- JJ Green
- JS Park
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- K Choi
- K Donaldson
- K Jia
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- K Jia
- K Kimura
- K Nasrollahi
- Kamal Nasrollahi
- KD Sauer
- KH Yap
- KI Kim
- KV Suresh
- L Ma
- L Zhang
- LC Pickup
- LC Pickup
- LC Pickup
- LJ Karam
- M Ben-Ezra
- M Ben-Ezra
- M Bertero
- M Carcenac
- M Chappalli
- M Elad
- M Elad
- M Elad
- M Elad
- M Elad
- M Elad
- M Gevrekci
- M Gevrekci
- M Gonzalez-Audcana
- M Irani
- M Irani
- M Jung
- M Protter
- M Protter
- M Shen
- M Shen
- M Shen
- M Singh
- MC Chiang
- MC Hong
- MC Pan
- MD Robinson
- ME Tipping
- ME Tipping
- MH Cheng
- MK Nema
- MK Ng
- MK Ng
- MK Ng
- MM Islam
- MV Joshi
- MV Joshi
- MVW Zibetti
- MVW Zibetti
- MVW Zibetti
- MVW Zibetti
- N Bose
- N Bose
- N Goldberg
- N Kulkarni
- N Nguyen
- N Nguyen
- NA Woods
- NA Yamany
- NK Bose
- NK Bose
- NK Bose
- OA Omer
- OA Omer
- P Chainais
- P Kramer
- P Milanfar
- P Purkait
- P Vandewalle
- P Vandewalle
- P Vandewalle
- PD Santis
- PE Eren
- PP Gajjar
- Q Pan
- Q Yuan
- Q Yuan
- R Fransens
- R He
- R Molina
- R Sasaharay
- R Tsai
- RC Hardie
- RC Hardie
- RC Hardie
- RR Schultz
- RR Schultz
- RR Schultz
- RS Prendergast
- RW Gerchberg
- S Baker
- S Chaudhuri
- S Dai
- S Farsiu
- S Farsiu
- S Farsiu
- S Farsiu
- S Farsiu
- S Kim
- S Liu
- S Lui
- S Mallat
- S Peleg
- S Pelletier
- S Peng
- S Rajaram
- S Yang
- S Zhang
- S Zhao
- SC Park
- SD Babacan
- SH Keller
- SP Belekos
- SW Park
- T Akgun
- T Gotoh
- T Katsuki
- T Komatsu
- T Szydzik
- TA Stephenson
- TC Ho
- TF Gee
- Thomas B. Moeslund
- V Patanavijit
- V Patanavijit
- V Patanavijit
- W Fan
- W Liu
- W Liu
- W Wu
- W Zhang
- W Zhao
- WT Freeman
- WT Freeman
- WWW Zou
- WZ Shao
- X Gao
- X Gao
- X Gao
- X Li
- X Li
- X Li
- X Ma
- X Maa
- X Wang
- X Zeng
- X Zhang
- Y Altunbasak
- Y He
- Y He
- Y Hu
- Y Hu
- Y Huang
- Y Mochizuki
- Y Zhuang
- Y-W Tai
- YJ Ma
- YR Li
- Z Arycan
- Z Bi
- Z Jiang
- Z Lin
- Z Lin
- Z Wang
- Z Wang
- Z Xiong
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 14/06/2014
- Field of study
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Akoumianaki T
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- Alaoui-Jamali MA
- Alberti S
- Alcocer-Gómez E
- Alessandri C
- Ali M
- Alim Al-Bari MA
- Aliwaini S
- Alizadeh J
- Almacellas E
- Almasan A
- Alonso A
- Alonso GD
- Altan-Bonnet N
- Altieri DC
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- Alzaharna MM
- Amadio M
- Amantini C
- Amaral C
- Ambrosio S
- Amer AO
- Ammanathan V
- An Z
- Andersen SU
- Andrabi SA
- Andrade-Silva M
- Andres AM
- Angelini S
- Ann D
- Anozie UC
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- Ashrafizadeh M
- Ashur-Fabian O
- Atanasov AG
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- Baldari CT
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- Čolić M
- Đokić J
- Žerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis.
- Author
- Aadahl M
- Abbas S
- Abecasis G
- Adair LS
- Adams HHH
- Adeyemo AA
- Aguilar-Salinas CA
- Akiyama M
- Al-Mulla F
- Arbeeva L
- Arnett DK
- Asselbergs FW
- Assimes TL
- Attia J
- Banas B
- Bandinelli S
- Baras A
- Bayyana S
- Bechayda SA
- Benjamins JW
- Bennett DA
- Bentley AR
- Beulens JWJ
- Bharadwaj D
- Bhatti KF
- Bielak LF
- Biino G
- Bis JC
- Bjerregaard AA
- Black C
- Boehnke M
- Bollepalli S
- Boomsma DI
- Bork-Jensen J
- Bouchard C
- Bradfield JP
- Bradford Y
- Brandslund I
- Braund PS
- Brody JA
- Brown CD
- Brown M
- Brown MR
- Brumpton BM
- Buring JE
- Burtt NP
- Böger CA
- Cade BE
- Cai Q
- Campbell A
- Campbell H
- Caulfield MJ
- Cañadas-Garre M
- Chai JF
- Chambers JC
- Chandak GR
- Chang K-M
- Chasman DI
- Chaturvedi N
- Chen S
- Chen Y
- Chen Y-DI
- Chen Z
- Cheng C-Y
- Chitrala KN
- Cho K
- Cho YS
- Choi HS
- Choudhury A
- Christensen H
- Christensen K
- Christofidou P
- Chuang L-M
- Ciullo M
- Clarke SL
- Concas MP
- Correa A
- Costanzo MC
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- Cuellar-Partida G
- Cupples L
- Damrauer SM
- Dantoft TM
- Daviglus M
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- de Borst GJ
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- de Silva HJ
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- Dedoussis G
- Delgado GE
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- Demirkan A
- Dorajoo R
- Doumatey AP
- Elders PJM
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- Erdmann J
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- Farmaki A-E
- Faul JD
- Feitosa MF
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- Fernandez-Lopez JC
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- Giardoglou T
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- Giulianini F
- Goel A
- Golightly YM
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- Gu D
- Gudbjartsson DF
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- Gustafsson S
- Haessler J
- Hakonarson H
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- Hattersley AT
- Havulinna AS
- Haworth S
- Hayes MG
- Hayward C
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- Hebbar P
- Heid IM
- Helgadottir A
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- Hewitt AW
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- Hilliard AT
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- Ikeda DD
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- Irvin MR
- Jackson RD
- Jang D
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- Kleber ME
- Koh W-P
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- Zajac GJM
- Zeggini E
- Zeng L
- Zhang J
- Zhang W
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- Zheng W
- Zhou W
- Zhu X
- Zhu X
- Zilhao NR
- Zimmermann ME
- Zmuda JM
- Zonderman AB
- Åsvold BO
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 06/12/2022
- Field of study
BACKGROUND: Genetic variants within nearly 1000 loci are known to contribute to modulation of blood lipid levels. However, the biological pathways underlying these associations are frequently unknown, limiting understanding of these findings and hindering downstream translational efforts such as drug target discovery. RESULTS: To expand our understanding of the underlying biological pathways and mechanisms controlling blood lipid levels, we leverage a large multi-ancestry meta-analysis (N = 1,654,960) of blood lipids to prioritize putative causal genes for 2286 lipid associations using six gene prediction approaches. Using phenome-wide association (PheWAS) scans, we identify relationships of genetically predicted lipid levels to other diseases and conditions. We confirm known pleiotropic associations with cardiovascular phenotypes and determine novel associations, notably with cholelithiasis risk. We perform sex-stratified GWAS meta-analysis of lipid levels and show that 3-5% of autosomal lipid-associated loci demonstrate sex-biased effects. Finally, we report 21 novel lipid loci identified on the X chromosome. Many of the sex-biased autosomal and X chromosome lipid loci show pleiotropic associations with sex hormones, emphasizing the role of hormone regulation in lipid metabolism. CONCLUSIONS: Taken together, our findings provide insights into the biological mechanisms through which associated variants lead to altered lipid levels and potentially cardiovascular disease risk
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