Poster:Discovering requirements of behaviour change software systems from negative user experience

Behavior Change Software systems (BCSSs) have shown promising outcomes in terms of promoting healthy behaviors. However, a negative User Experience (UX) can be induced by BCSS if designers do not have clear understanding of the requirements that factually help in changing the user behavior that achieves a sustainability goal. In order to get insights into how to discover such sustainability requirements, we propose a discovery approach, whose emphasis is placed on negative UX assessed through attitudes and behaviors expressed by users due to the lack of fulfillment of actual user needs. The approach is tested on existing software systems designed for preventing or reducing Repetitive Strain Injury as a particular category of BCSS. Twelve requirements that contribute to social sustainability were discovered

Towards a Non-Functional Requirements Discovery Approach for Persuasive Systems

[Abstract] A number of software systems that attempt to help people achieve behavior change have been proposed in various domains such as health and wellness. However, sometimes, such systems have failed to provide a satisfactory or sustainable User Experience (UX), as it is observed when users may be reluctant to respond to the activation of the systems' changing demands. Moreover, a negative User Experience (UX) can be exposed by Behavior Change Support Systems (BCSS) if designers do not have clear understanding of the requirements that factually help changing the user behavior that accomplishes a sustainability goal. We first explored the Persuasive System Design (PSD) model that should be considered in UX assessment of BCSSs. Then, we propose a requirements discovery process that can be considered to redesign a software interactive system based on negative UX.This work has received partial support by the projects FEDER-UE CSI ED431G/01, ED43C 2018/29, accreditation 2016-2019 ED431G/08, KUSISQA supported by N° 014-2019-FONDECYT-BM-INC.INV, NOVA LINCS UID/CEC/04516/2019, MICINN IJC2018-037522-I, RTI2018-099646-B-I00Xunta de Galicia; ED431G/01Xunta de Galicia; ED43C 2018/29Xunta de Galicia; ED431G/08Gobierno de Chile; 014-2019-FONDECYTBM-INC.INVRepública Portuguesa. Fundação para a Ciência e a Tecnologia; UID/CEC/04516/201

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Comparing motivational, self-regulatory and habitual processes in a computer-tailored physical activity intervention in hospital employees - Protocol for the PATHS randomised controlled trial

Background: Most people do not engage in sufficient physical activity to confer health benefits and to reduce risk of chronic disease. Healthcare professionals frequently provide guidance on physical activity, but often do not meet guideline levels of physical activity themselves. The main objective of this study is to develop and test the efficacy of a tailored intervention to increase healthcare professionals' physical activity participation and quality of life, and to reduce work-related stress and absenteeism. This is the first study to compare the additive effects of three forms of a tailored intervention using different techniques from behavioural theory, which differ according to their focus on motivational, self-regulatory and/or habitual processes. Methods/Design: Healthcare professionals (N = 192) will be recruited from four hospitals in Perth, Western Australia, via email lists, leaflets, and posters to participate in the four group randomised controlled trial. Participants will be randomised to one of four conditions: (1) education only (non-tailored information only), (2) education plus intervention components to enhance motivation, (3) education plus components to enhance motivation and self-regulation, and (4) education plus components to enhance motivation, self-regulation and habit formation. All intervention groups will receive a computer-tailored intervention administered via a web-based platform and will receive supporting text-messages containing tailored information, prompts and feedback relevant to each condition. All outcomes will be assessed at baseline, and at 3-month follow-up. The primary outcome assessed in this study is physical activity measured using activity monitors. Secondary outcomes include: quality of life, stress, anxiety, sleep, and absenteeism. Website engagement, retention, preferences and intervention fidelity will also be evaluated as well as potential mediators and moderators of intervention effect. Discussion: This is the first study to examine a tailored, technology-supported intervention aiming to increase physical activity in healthcare professionals. The study will evaluate whether including additional theory-based behaviour change techniques aimed at promoting motivation, self-regulation and habit will lead to increased physical activity participation relative to information alone. The online platform developed in this study has potential to deliver efficient, scalable and personally-relevant intervention that can be translated to other occupational settings. Trial registration: Australian New-Zealand Clinical Trial Registry: ACTRN12616000462482, submitted 29/03/2016, prospectively registered 8/04/2016

Segregation of myoblast fusion and muscle-specific gene expression by distinct ligand-dependent inactivation of GSK-3β

Myogenic differentiation involves myoblast fusion and induction of muscle-specific gene expression, which are both stimulated by pharmacological (LiCl), genetic, or IGF-I-mediated GSK-3β inactivation. To assess whether stimulation of myogenic differentiation is common to ligand-mediated GSK-3β inactivation, myoblast fusion and muscle-specific gene expression were investigated in response to Wnt-3a. Moreover, crosstalk between IGF-I/GSK-3β/NFATc3 and Wnt/GSK-3β/β-catenin signaling was assessed. While both Wnt-3a and LiCl promoted myoblast fusion, muscle-specific gene expression was increased by LiCl, but not by Wnt-3a or β-catenin over-expression. Furthermore, LiCl and IGF-I, but not Wnt-3a, increased NFATc3 transcriptional activity. In contrast, β-catenin-dependent transcriptional activity was increased by Wnt-3a and LiCl, but not IGF-I. These results for the first time reveal a segregated regulation of myoblast fusion and muscle-specific gene expression following stimulation of myogenic differentiation in response to distinct ligand-specific signaling routes of GSK-3β inactivation