374 research outputs found
Cardiac magnetic resonance imaging parameters as surrogate endpoints in clinical trials of acute myocardial infarction
- Author
- A Abbate
- A Kastrati
- A Kribben
- A Schmidt
- A Schomig
- A Wagner
- AH Aletras
- AH Aletras
- AN Mather
- AS Flett
- AT Yan
- CM Kramer
- D Atar
- DJ Pennell
- DP O'Regan
- DS Fieno
- E Heiberg
- E Larose
- E Wu
- EA Geiser
- F Van de Werf
- G Ambrosio
- G Ndrepepa
- Georg Fuernau
- Gerhard Schuler
- H Abdel-Aty
- H Abdel-Aty
- H Engblom
- H Thiele
- H Thiele
- H Thiele
- H Thiele
- H Thiele
- HD White
- HJ Kang
- Holger Thiele
- HW Kim
- HW Kim
- I Eitel
- Ingo Eitel
- J Ganame
- J Lonborg
- J Schulz-Menger
- J Vicente
- J Wohrle
- JC Nilsson
- JD Haeck
- JE Lowe
- JJ Milavetz
- JY Hahn
- KA Reimer
- KC Wollert
- KC Wu
- KH Darasz
- KM Choi
- LC Amado
- LY Hsu
- LY Hsu
- M Carlsson
- M Tendera
- Matthias Gutberlet
- MG Engelmann
- MG Friedrich
- MR Patel
- NG Bellenger
- O Bondarenko
- P Kellman
- P Kellman
- Philipp Lurz
- R Nijveldt
- RA Weir
- RJ Gibbons
- RJ Kim
- RS Ripa
- RS Ripa
- S de Waha
- S Desch
- S Giri
- S Janssens
- SC Bekkers
- Steffen Desch
- Suzanne de Waha
- T Dill
- T Ibrahim
- T Ibrahim
- T Ibrahim
- TD Miller
- V Hombach
- YB Song
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
Cardiac magnetic resonance (CMR) offers a variety of parameters potentially suited as surrogate endpoints in clinical trials of acute myocardial infarction such as infarct size, myocardial salvage, microvascular obstruction or left ventricular volumes and ejection fraction. The present article reviews each of these parameters with regard to the pathophysiological basis, practical aspects, validity, reliability and its relative value (strengths and limitations) as compared to competitive modalities. Randomized controlled trials of acute myocardial infarction which have used CMR parameters as a primary endpoint are presented
Cleavage of von Willebrand Factor by Granzyme M Destroys Its Factor VIII Binding Capacity
- Author
- B Bade
- B Goldstein
- C Weeterings
- Christopher B. Doering
- CJ Froelich
- CM Chamberlain
- CR De
- D Chowdhury
- D Kaiserman
- E Groot
- E Groot
- EH Spaeny-Dekking
- F Banno
- GG Levy
- H Ulrichts
- HJ Weiss
- HM Tsai
- HM Tsai
- JB van Woensel
- JC Choy
- JE Sadler
- JF Dong
- JJ Hulstein
- JM Sodetz
- Ka Wai Lai
- KC Desch
- L Hilbert
- M Furlan
- M Furlan
- Marcel van Deuren
- Martine J. Hollestelle
- MJ Hollestelle
- MS Buzza
- MS Buzza
- N Bovenschen
- N Bovenschen
- Niels Bovenschen
- Peter J. Lenting
- Philip G. de Groot
- PJ de Koning
- PM Mannucci
- RM van der Plas
- S Jorieux
- S Mahrus
- SP Cullen
- SS Metkar
- TJ Raife
- Tom Sprong
- W Gao
- ZM Ruggeri
- ZM Ruggeri
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Von Willebrand factor (VWF) is a pro-hemostatic multimeric plasma protein that promotes platelet aggregation and stabilizes coagulation factor VIII (FVIII) in plasma. The metalloproteinase ADAMTS13 regulates the platelet aggregation function of VWF via proteolysis. Severe deficiency of ADAMTS13 is associated with thrombotic thrombocytopenic purpura, but does not always correlate with its clinical course. Therefore, other proteases could also be important in regulating VWF activity. In the present study, we demonstrate that VWF is cleaved by the cytotoxic lymphocyte granule component granzyme M (GrM). GrM cleaved both denaturated and soluble plasma-derived VWF after Leu at position 276 in the D3 domain. GrM is unique in that it did not affect the multimeric size and pro-hemostatic platelet aggregation ability of VWF, but instead destroyed the binding of VWF to FVIII in vitro. In meningococcal sepsis patients, we found increased plasma GrM levels that positively correlated with an increased plasma VWF/FVIII ratio in vivo. We conclude that, next to its intracellular role in triggering apoptosis, GrM also exists extracellularly in plasma where it could play a physiological role in controlling blood coagulation by determining plasma FVIII levels via proteolytic processing of its carrier VWF
Illustrated State-of-the-Art Capsules of the ISTH 2023 Congress
- Author
- Publication venue
- Elsevier BV
- Publication date
- 01/05/2023
- Field of study
This year’s Congress of the International Society of Thrombosis and Haemostasis (ISTH) took place in person in Montréal, Canada, from June 24-28, 2023. The conference, held annually, highlighted cutting-edge advances in basic, translational, population and clinical sciences relevant to the Society. As for all ISTH congresses, we offered a special, congress-specific scientific theme; this year, the special theme was immunothrombosis. Certainly, over the last few years, COVID-19 infection and its related thrombotic and other complications have renewed interest in the concepts of thromboinflammation and immunothrombosis; namely, the relationship between inflammation, infection and clotting. Other main scientific themes of the Congress included Arterial Thromboembolism, Coagulation and Natural Anticoagulants, Diagnostics and Omics, Fibrinolysis and Proteolysis, Hemophilia and Rare Bleeding Disorders, Hemostatic System in Cancer, Inflammation and Immunity, Pediatrics, Platelet Disorders, von Willebrand Disease and Thrombotic Microangiopathies, Platelets and Megakaryocytes, Vascular Biology, Venous Thromboembolism and Women’s Health. Among other sessions, the program included 28 State-of-the-Art (SOA) sessions with a total of 84 talks given by internationally recognized leaders in the field. SOA speakers were invited to prepare brief illustrated reviews of their talks that were peer reviewed and are included in this article. These illustrated capsules highlight the major scientific advances with potential to impact clinical practice. Readers are invited to take advantage of the excellent educational resource provided by these illustrated capsules. They are also encouraged to use the image in social media to draw attention to the high quality and impact of the science presented at the Congress
Genome-wide association trans-ethnic meta-analyses identifies novel associations regulating coagulation Factor VIII and von Willebrand Factor plasma levels
- Author
- Bartz TM
- Becker DM
- Becker LC
- Boerwinkle E
- Brody JA
- Campbell H
- Chen M-H
- Chong MR
- Cushman M
- Davies NM
- de Haan HG
- de Maat MPM
- de Vries PS
- Dehghan A
- Delgado G
- Desch KC
- Eicher JD
- Fernandez-Cadenas I
- Fornage M
- Franco OH
- Germain M
- Ginsburg D
- Gudnason V
- Guo X
- Harris TB
- Hayward C
- Hofman A
- Huffman JE
- INVENT Consortium
- Jenny NS
- Johnson AD
- Joshi PK
- Jukema JW
- Kleber ME
- Kolcic I
- Launer LJ
- Li JZ
- Lindgren A
- Lowenstein CJ
- Marten J
- Martinez-Perez A
- Mastrangelo MA
- McKnight B
- MEGASTROKE consortium of the International Stroke Genetics Corpo
- Morange P-E
- Morrison AC
- März W
- O'Donnell CJ
- Ozel AB
- Pankratz N
- Polasek O
- Psaty BM
- Reiner AP
- Rivadeneira F
- Rosand J
- Rosendaal FR
- Rotter JI
- Rudan I
- Sabater-Lleal M
- Smith AV
- Smith NL
- Song C
- Soria JM
- Souto JC
- Stott DJ
- Strachan DP
- Suchon P
- Tang W
- Tofler GH
- Trompet S
- Trégounët D-A
- Uitterlinden AG
- Vaidya D
- van Hylckama Vlieg A
- Ward-Caviness CK
- Welsh P
- Wilson JW
- Yanek LR
- Publication venue
- 'Ovid Technologies (Wolters Kluwer Health)'
- Publication date
- 20/11/2018
- Field of study
BACKGROUND: Factor VIII (FVIII) and its carrier protein von Willebrand factor (VWF) are associated with risk of arterial and venous thrombosis and with hemorrhagic disorders. We aimed to identify and functionally test novel genetic associations regulating plasma FVIII and VWF. METHODS: We meta-analyzed genome-wide association results from 46 354 individuals of European, African, East Asian, and Hispanic ancestry. All studies performed linear regression analysis using an additive genetic model and associated ≈35 million imputed variants with natural log-transformed phenotype levels. In vitro gene silencing in cultured endothelial cells was performed for candidate genes to provide additional evidence on association and function. Two-sample Mendelian randomization analyses were applied to test the causal role of FVIII and VWF plasma levels on the risk of arterial and venous thrombotic events. RESULTS: We identified 13 novel genome-wide significant ( P≤2.5×10-8) associations, 7 with FVIII levels ( FCHO2/TMEM171/TNPO1, HLA, SOX17/RP1, LINC00583/NFIB, RAB5C-KAT2A, RPL3/TAB1/SYNGR1, and ARSA) and 11 with VWF levels ( PDHB/PXK/KCTD6, SLC39A8, FCHO2/TMEM171/TNPO1, HLA, GIMAP7/GIMAP4, OR13C5/NIPSNAP, DAB2IP, C2CD4B, RAB5C-KAT2A, TAB1/SYNGR1, and ARSA), beyond 10 previously reported associations with these phenotypes. Functional validation provided further evidence of association for all loci on VWF except ARSA and DAB2IP. Mendelian randomization suggested causal effects of plasma FVIII activity levels on venous thrombosis and coronary artery disease risk and plasma VWF levels on ischemic stroke risk. CONCLUSIONS: The meta-analysis identified 13 novel genetic loci regulating FVIII and VWF plasma levels, 10 of which we validated functionally. We provide some evidence for a causal role of these proteins in thrombotic events
The Irish DNA Atlas: Revealing Fine-Scale Population Structure and History within Ireland
- Author
- AL Price
- B McEvoy
- B McEvoy
- B McEvoy
- B McEvoy
- B Weir
- B Winney
- C Capelli
- C Dolan
- C McGuigan
- CT O’Dushlaine
- D Petkova
- EW Hill
- G Fiorito
- G Hellenthal
- GW Dawson
- J Yang
- JF Wilson
- JH Relethford
- KC Desch
- KP Coss
- LM Cassidy
- LT Moore
- M Karakachoff
- M Murphy
- M Mylotte
- O Delaneau
- O Lao
- PM Farrell
- S Cronin
- S Leslie
- S Purcell
- WE Hackett
- WTCCC2 IMSGC
- Y Itan
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
The extent of population structure within Ireland is largely unknown, as is the impact of historical migrations. Here we illustrate fine-scale genetic structure across Ireland that follows geographic boundaries and present evidence of admixture events into Ireland. Utilising the ‘Irish DNA Atlas’, a cohort (n = 194) of Irish individuals with four generations of ancestry linked to specific regions in Ireland, in combination with 2,039 individuals from the Peoples of the British Isles dataset, we show that the Irish population can be divided in 10 distinct geographically stratified genetic clusters; seven of ‘Gaelic’ Irish ancestry, and three of shared Irish-British ancestry. In addition we observe a major genetic barrier to the north of Ireland in Ulster. Using a reference of 6,760 European individuals and two ancient Irish genomes, we demonstrate high levels of North-West French-like and West Norwegian-like ancestry within Ireland. We show that that our ‘Gaelic’ Irish clusters present homogenous levels of ancient Irish ancestries. We additionally detect admixture events that provide evidence of Norse-Viking gene flow into Ireland, and reflect the Ulster Plantations. Our work informs both on Irish history, as well as the study of Mendelian and complex disease genetics involving populations of Irish ancestry
The effector T cell response to influenza infection
- Author
- A Ballesteros-Tato
- A Bot
- A Kallies
- A Kotelkin
- A Kupfer
- A Mullbacher
- AE Lukacher
- AI Wolf
- AK Heer
- AN Desch
- AW Ho
- B Lowin
- B Manicassamy
- B Moltedo
- BN Lambrecht
- BS Bender
- BT Edelson
- C Zhong
- CA Benedict
- CH GeurtsvanKessel
- CM Walsh
- CW Lawrence
- D Damjanovic
- D Kagi
- D Kaiserman
- D Masopust
- DH Dockrell
- DJ Topham
- DJ Topham
- DJ Topham
- DJ Topham
- DM Brown
- DM Brown
- DN Cook
- DV Dolfi
- E Ishikawa
- E Rouvier
- EL Brincks
- EL Brincks
- F Mattei
- G Karupiah
- G Schreibelt
- GT Belz
- H Nakano
- H Shin
- H Yoon
- I Voskoboinik
- IK Pang
- IK Pang
- IK Pang
- J Banchereau
- J Helft
- J Helft
- J McGill
- J McGill
- J McGill
- J Sun
- J Sun
- J Sun
- J Thatte
- JA Lopez
- JA Villadangos
- JA Wiley
- JB Roths
- JE Kohlmeier
- JH Li
- JH Russell
- JJ Lum
- JJ O’Shea
- JK Yoo
- JR Aldridge Jr
- K Man
- K Neyt
- K Obaru
- K Sun
- K Sun
- K Yadava
- KC Verbist
- KG Anderson
- KJ Zee Van
- KK McKinstry
- KL Legge
- KL Lin
- KM Hargadon
- KN Couper
- KP Gisbergen van
- L Hua
- L Ottonello
- LT Joeckel
- M Eichelberger
- M Faroudi
- M Gereke
- M Grell
- M Huse
- M Imai
- M Kandasamy
- M Lettau
- MA Purbhoo
- MA Wells
- MA Williams
- MB Graham
- MB Graham
- ML Dustin
- ML Rio del
- MM Hufford
- MM Hufford
- MS Buzza
- N Baumgarth
- N Kayagaki
- N Lee
- N Parameswaran
- N Said-Sadier
- N Zhang
- NJ Waterhouse
- O Susanto
- P Allavena
- P Menten
- P Schneider
- PB Stranges
- PG Thomas
- R Blumenthal
- R Goffic Le
- RA Langlois
- RB Fulton
- RB Fulton
- RL Peper
- RM Siegel
- RW Walters
- S Allen
- S Crotty
- S Herold
- S Hou
- S Isaaz
- S Koyama
- S Valitutti
- S Yao
- S Zelenay
- SE Belisle
- SJ Turner
- SP Cullen
- SS Metkar
- SS Sung
- SY Tsai
- T Hussell
- T Ichinohe
- T Zhao
- TD Kanneganti
- TJ Braciale
- TM Wilkinson
- TS Kim
- WJ Karpus
- X Hao
- Y Guo
- Z Mikhak
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Influenza virus infection induces a potent initial innate immune response, which serves to limit the extent of viral replication and virus spread. However, efficient (and eventual) viral clearance within the respiratory tract requires the subsequent activation, rapid proliferation, recruitment, and expression of effector activities by the adaptive immune system, consisting of antibody producing B cells and influenza-specific T lymphocytes with diverse functions. The ensuing effector activities of these T lymphocytes ultimately determine (along with antibodies) the capacity of the host to eliminate the viruses and the extent of tissue damage. In this review, we describe this effector T cell response to influenza virus infection. Based on information largely obtained in experimental settings (i.e., murine models), we will illustrate the factors regulating the induction of adaptive immune T cell responses to influenza, the effector activities displayed by these activated T cells, the mechanisms underlying the expression of these effector mechanisms, and the control of the activation/differentiation of these T cells, in situ, in the infected lungs
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adam Bourdarios C
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Aikot A
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
- Akimov AV
- Akiyama D
- Akolkar NN
- Aktas S
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Albouy GL
- Alderweireldt S
- Alegria ZL
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi F
- Algren M
- Alhroob M
- Ali B
- Ali HMJ
- Ali S
- Alibocus SW
- Aliev M
- Alimonti G
- Alkakhi W
- Allaire C
- Allbrooke BMM
- Allen JF
- Allendes Flores CA
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alvarez Fernandez A
- Alves Cardoso M
- Alviggi MG
- Aly M
- Amaral Coutinho Y
- Ambler A
- Amelung C
- Amerl M
- Ames CG
- Amidei D
- Amirie KJ
- Amor Dos Santos SP
- Amos KR
- An S
- Ananiev V
- Anastopoulos C
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Anulli F
- Aoki M
- Aoki T
- Aparisi Pozo JA
- Aparo MA
- Aperio Bella L
- Appelt C
- Apyan A
- Arbiol Val SJ
- Arcangeletti C
- Arce ATH
- Arena E
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Arnaez O
- Arnold H
- Artoni G
- Asada H
- Asai K
- Asai S
- Asbah NA
- Assamagan K
- Astalos R
- Astrand KSV
- Atashi S
- Atkin RJ
- Atkinson M
- Atmani H
- Atmasiddha PA
- Augsten K
- Auricchio S
- Auriol AD
- Austrup VA
- Avolio G
- Axiotis K
- Azuelos G
- Babal D
- Bachacou H
- Bachas K
- Bachiu A
- Backman F
- Badea A
- Baer TM
- Bagnaia P
- Bahmani M
- Bahner D
- Bai K
- Bailey AJ
- Baines JT
- Baines L
- Baker OK
- Bakos E
- Bakshi Gupta D
- Balakrishnan V
- Balasubramanian R
- Baldin EM
- Balek P
- Ballabene E
- Balli F
- Baltes LM
- Balunas WK
- Balz J
- Banas E
- Bandieramonte M
- Bandyopadhyay A
- Bansal S
- Barak L
- Barakat M
- Barberio EL
- Barberis D
- Barbero M
- Barel MZ
- Barends KN
- Barillari T
- Barisits M-S
- Barklow T
- Baron Moreno DA
- Baron P
- Baroncelli A
- Barone G
- Barr AJ
- Barr JD
- Barreiro Guimarães da Costa J
- Barreiro F
- Barron U
- Barros Teixeira MG
- Barsov S
- Bartels F
- Bartoldus R
- Barton AE
- Bartos P
- Basan A
- Baselga M
- Bassalat A
- Basso MJ
- Bates RL
- Batlamous S
- Batool B
- Battaglia M
- Battulga D
- Bauce M
- Bauer M
- Bauer P
- Bazzano Hurrell LT
- Beacham JB
- Beau T
- Beaucamp JY
- Beauchemin PH
- Bechtle P
- Beck HP
- Becker K
- Beddall AJ
- Bednyakov VA
- Bee CP
- Beemster LJ
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- Beirer JF
- Beisiegel F
- Belfkir M
- Bella G
- Bellagamba L
- Bellerive A
- Bellos P
- Beloborodov K
- Benchekroun D
- Bendebba F
- Benhammou Y
- Benkendorfer KC
- Beresford L
- Beretta M
- Bergeaas Kuutmann E
- Berger N
- Bergmann B
- Beringer J
- Bernardi G
- Bernius C
- Bernlochner FU
- Bernon F
- Berrocal Guardia A
- Berry T
- Berta P
- Berthold A
- Bethke S
- Betti A
- Bevan AJ
- Bhalla NK
- Bhamjee M
- Bhatta S
- Bhattacharya DS
- Bhattarai P
- Bhide KD
- Bhopatkar VS
- Bianchi RM
- Bianco G
- Biebel O
- Bielski R
- Biglietti M
- Billingsley CS
- Bindi M
- Bingul A
- Bini C
- Biondini A
- Birch-sykes CJ
- Bird GA
- Birman M
- Biros M
- Biryukov S
- Bisanz T
- Bisceglie E
- Biswal JP
- Biswas D
- Bjørke K
- Bloch I
- Blue A
- Blumenschein U
- Blumenthal J
- Bobrovnikov VS
- Boehler M
- Boehm B
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bold T
- Bomben M
- Bona M
- Boonekamp M
- Booth CD
- Borbély AG
- Bordulev IS
- Borecka-Bielska HM
- Borissov G
- Bortoletto D
- Boscherini D
- Bosman M
- Bossio Sola JD
- Bouaouda K
- Bouchhar N
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bouquet R
- Boveia A
- Boyd J
- Boye D
- Boyko IR
- Bracinik J
- Brahimi N
- Brandt G
- Brandt O
- Braren F
- Brau B
- Brau JE
- Brener R
- Brenner L
- Brenner R
- Bressler S
- Britton D
- Britzger D
- Brock I
- Brooijmans G
- Brost E
- Brown LM
- Bruce LE
- Bruckler TL
- Bruckman de Renstrom PA
- Bruni A
- Bruni G
- Bruschi M
- Bruscino N
- Brüers B
- Buanes T
- Buat Q
- Buchin D
- Buckley AG
- Bulekov O
- Bullard BA
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burlayenko O
- Burr JTP
- Burton CD
- Burzynski JC
- Busch EL
- Bussey PJ
- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
- Buxo Vazquez CJ
- Buzykaev AR
- Büscher V
- Cabrera Urbán S
- Cadamuro L
- Caforio D
- Cai H
- Cai Y
- Cai Y
- Cairo VMM
- Cakir O
- Calace N
- Calafiura P
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Calvet D
- Calvet S
- Calvetti M
- Camacho Toro R
- Camarda S
- Camarero Munoz D
- Camarri P
- Camerlingo MT
- Cameron D
- Camincher C
- Campanelli M
- Camplani A
- Canale V
- Canbay AC
- Canonero E
- Cantero J
- Cao Y
- Capocasa F
- Capua M
- Carbone A
- Cardarelli R
- Cardenas JCJ
- Cardillo F
- Carducci G
- Carli T
- Carlino G
- Carlotto JI
- Carlson BT
- Carlson EM
- Carminati L
- Carnelli A
- Carnesale M
- Caron S
- Carquin E
- Carratta G
- Carroll AM
- Carrá S
- Carter TM
- Casado MP
- Caspar M
- Castillo Garcia L
- Castillo Gimenez V
- Castillo FL
- Castro NF
- Catinaccio A
- Catmore JR
- Cavaliere T
- Cavaliere V
- Cavalli N
- Cekmecelioglu YC
- Celebi E
- Cella S
- Celli F
- Centonze MS
- Cepaitis V
- Cerny K
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cervato B
- Cervelli A
- Cesarini G
- Cetin SA
- Chakraborty D
- Chan J
- Chan WY
- Chapman JD
- Chapon E
- Chargeishvili B
- Charlton DG
- Chatterjee M
- Chauhan C
- Che Y
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chen A
- Chen B
- Chen B
- Chen H
- Chen H
- Chen J
- Chen J
- Chen M
- Chen S
- Chen SJ
- Chen X
- Chen X
- Chen Y
- Cheng CL
- Cheng HC
- Cheong S
- Cheplakov A
- Cheremushkina E
- Cherepanova E
- Cherkaoui El Moursli R
- Cheu E
- Cheung K
- Chevalier L
- Chiarella V
- Chiarelli G
- Chiedde N
- Chiodini G
- Chisholm AS
- Chitan A
- Chitishvili M
- Chizhov MV
- Choi K
- Chou Y
- Chow EYS
- Chu KL
- Chu MC
- Chu X
- Chudoba J
- Chwastowski JJ
- Cieri D
- Ciesla KM
- Cindro V
- Ciocio A
- Cirotto F
- Citron ZH
- Citterio M
- Ciubotaru DA
- Clark A
- Clark PJ
- Clarry C
- Clavijo Columbie JM
- Clawson SE
- Clement C
- Clercx J
- Coadou Y
- Cobal M
- Coccaro A
- Coelho Barrue RF
- Coelho Lopes De Sa R
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- Yao W-M
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- Yin P
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- Young CJS
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- Yuan R
- Yue L
- Zaazoua M
- Zabinski B
- Zaid E
- Zak ZK
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zamora Saa JA
- Zang J
- Zanzi D
- Zaplatilek O
- Zeitnitz C
- Zeng H
- Zeng JC
- Zenger DT
- Zenin O
- Zenz S
- Zerradi S
- Zerwas D
- Zhai M
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- Zhang X
- Zhang X
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- Zhang Y
- Zhang Y
- Zhang Z
- Zhang Z
- Zhao H
- Zhao T
- Zhao Y
- Zhao Z
- Zhao Z
- Zhemchugov A
- Zheng J
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
- Zhou B
- Zhou H
- Zhou N
- Zhou Y
- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Measurement of the tt¯tt¯ production cross section in pp collisions at √s=13 TeV with the ATLAS detector
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- Zeißner SV
- Zemaityte G
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhu Y
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- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Ziolkowski M
- Zoccoli A
- Zoch K
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 16/11/2021
- Field of study
A measurement of four-top-quark production using proton-proton collision data at a centre-of-mass energy of 13 TeV collected by the ATLAS detector at the Large Hadron Collider corresponding to an integrated luminosity of 139 fb−1 is presented. Events are selected if they contain a single lepton (electron or muon) or an opposite-sign lepton pair, in association with multiple jets. The events are categorised according to the number of jets and how likely these are to contain b-hadrons. A multivariate technique is then used to discriminate between signal and background events. The measured four-top-quark production cross section is found to be 26+17−15 fb, with a corresponding observed (expected) significance of 1.9 (1.0) standard deviations over the background-only hypothesis. The result is combined with the previous measurement performed by the ATLAS Collaboration in the multilepton final state. The combined four-top-quark production cross section is measured to be 24+7−6 fb, with a corresponding observed (expected) signal significance of 4.7 (2.6) standard deviations over the background-only predictions. It is consistent within 2.0 standard deviations with the Standard Model expectation of 12.0 ± 2.4 fb
Measurements of Higgs bosons decaying to bottom quarks from vector boson fusion production with the ATLAS experiment at √=13TeV
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- Zemaityte G
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- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
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- Zhu C
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- Zhu H
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- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 23/06/2021
- Field of study
The paper presents a measurement of the Standard Model Higgs Boson decaying to b-quark pairs in the vector boson fusion (VBF) production mode. A sample corresponding to 126 fb−1 of s√=13TeV proton–proton collision data, collected with the ATLAS experiment at the Large Hadron Collider, is analyzed utilizing an adversarial neural network for event classification. The signal strength, defined as the ratio of the measured signal yield to that predicted by the Standard Model for VBF Higgs production, is measured to be 0.95+0.38−0.36 , corresponding to an observed (expected) significance of 2.6 (2.8) standard deviations from the background only hypothesis. The results are additionally combined with an analysis of Higgs bosons decaying to b-quarks, produced via VBF in association with a photon
Muon reconstruction and identification efficiency in ATLAS using the full Run 2 pp collision data set at \sqrt{s}=13 TeV
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Agullo PM
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahnen JV
- Ai X
- Aielli G
- Akatsuka S
- Akbiyik M
- Akesson TPA
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Ali S
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
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- Alves FLL
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amoroso S
- Amrouche CS
- An F
- Anastopoulos C
- Andari N
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angerami A
- Anisenkov AV
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- Bobbink GJ
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- Bogavac D
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- Burghgrave B
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- Buschmann E
- Bussey PJ
- Butler JM
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- Butterworth JM
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- Buttinger W
- Buzatu A
- Buzykaev AR
- Bylund OB
- Cabras G
- Caforio D
- Cai H
- Cairo VMM
- Cakir IT
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- Calace N
- Calafiura P
- Calderini G
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- Caloba LP
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- Calvet D
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- Camerlingo MT
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- Campana S
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- Canale V
- Canesse A
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- Cao T
- Cao Y
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- Casado MP
- Casha AF
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- Castillo FL
- Castillo LRF
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- Catmore JR
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- Cheu E
- Cheung K
- Chevalerias TJA
- Chevalier L
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- Chisholm AS
- Chitan A
- Chiu I
- Chiu YH
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- Chou Y
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- Christopher LD
- Chu MC
- Chu X
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- Cioara IA
- Ciocio A
- Cirotto F
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- de Renstrom PAB
- De Sa RCL
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- Klimentov A
- Klimpel F
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- Knue A
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- Kondo T
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- Kotwal A
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- Zhemchugov A
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- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 01/01/2021
- Field of study
This article documents the muon reconstruction and identification efficiency obtained by the ATLAS experiment for 139 \hbox {fb}^{-1} of pp collision data at \sqrt{s}=13 TeV collected between 2015 and 2018 during Run 2 of the LHC. The increased instantaneous luminosity delivered by the LHC over this period required a reoptimisation of the criteria for the identification of prompt muons. Improved and newly developed algorithms were deployed to preserve high muon identification efficiency with a low misidentification rate and good momentum resolution. The availability of large samples of Z\rightarrow \mu \mu and J/\psi \rightarrow \mu \mu decays, and the minimisation of systematic uncertainties, allows the efficiencies of criteria for muon identification, primary vertex association, and isolation to be measured with an accuracy at the per-mille level in the bulk of the phase space, and up to the percent level in complex kinematic configurations. Excellent performance is achieved over a range of transverse momenta from 3 GeV to several hundred GeV, and across the full muon detector acceptance of |\eta |<2.7
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