Generalized Power Attacks against Crypto Hardware using Long-Range Deep Learning

To make cryptographic processors more resilient against side-channel attacks, engineers have developed various countermeasures. However, the effectiveness of these countermeasures is often uncertain, as it depends on the complex interplay between software and hardware. Assessing a countermeasure’s effectiveness using profiling techniques or machine learning so far requires significant expertise and effort to be adapted to new targets which makes those assessments expensive. We argue that including cost-effective automated attacks will help chip design teams to quickly evaluate their countermeasures during the development phase, paving the way to more secure chips. In this paper, we lay the foundations toward such automated system by proposing GPAM, the first deep-learning system for power side-channel analysis that generalizes across multiple cryptographic algorithms, implementations, and side-channel countermeasures without the need for manual tuning or trace preprocessing. We demonstrate GPAM’s capability by successfully attacking four hardened hardware-accelerated elliptic-curve digital-signature implementations. We showcase GPAM’s ability to generalize across multiple algorithms by attacking a protected AES implementation and achieving comparable performance to state-of-the-art attacks, but without manual trace curation and within a limited budget. We release our data and models as an open-source contribution to allow the community to independently replicate our results and build on them

Hybrid Post-Quantum Signatures in Hardware Security Keys

Recent advances in quantum computing are increasingly jeopardizing the security of cryptosystems currently in widespread use, such as RSA or elliptic-curve signatures. To address this threat, researchers and standardization institutes have accelerated the transition to quantum-resistant cryptosystems, collectively known as Post-Quantum Cryptography (PQC). These PQC schemes present new challenges due to their larger memory and computational footprints and their higher chance of latent vulnerabilities. In this work, we address these challenges by introducing a scheme to upgrade the digital signatures used by security keys to PQC. We introduce a hybrid digital signature scheme based on two building blocks: a classically-secure scheme, ECDSA, and a post-quantum secure one, Dilithium. Our hybrid scheme maintains the guarantees of each underlying building block even if the other one is broken, thus being resistant to classical and quantum attacks. We experimentally show that our hybrid signature scheme can successfully execute on current security keys, even though secure PQC schemes are known to require substantial resources. We publish an open-source implementation of our scheme at https://github.com/google/OpenSK/releases/tag/hybrid-pqc so that other researchers can reproduce our results on a nRF52840 development kit

smehner1/scapy: Profinet IO DCP Layer for Scapy

<p>Added Profinet IO DCP layer to craft and read DCP packets.</p&gt

The risk of COVID-19 death is much greater and age dependent with type I IFN autoantibodies

International audienceSignificance There is growing evidence that preexisting autoantibodies neutralizing type I interferons (IFNs) are strong determinants of life-threatening COVID-19 pneumonia. It is important to estimate their quantitative impact on COVID-19 mortality upon SARS-CoV-2 infection, by age and sex, as both the prevalence of these autoantibodies and the risk of COVID-19 death increase with age and are higher in men. Using an unvaccinated sample of 1,261 deceased patients and 34,159 individuals from the general population, we found that autoantibodies against type I IFNs strongly increased the SARS-CoV-2 infection fatality rate at all ages, in both men and women. Autoantibodies against type I IFNs are strong and common predictors of life-threatening COVID-19. Testing for these autoantibodies should be considered in the general population

Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19

BackgroundWe previously reported that impaired type I IFN activity, due to inborn errors of TLR3- and TLR7-dependent type I interferon (IFN) immunity or to autoantibodies against type I IFN, account for 15-20% of cases of life-threatening COVID-19 in unvaccinated patients. Therefore, the determinants of life-threatening COVID-19 remain to be identified in similar to 80% of cases.MethodsWe report here a genome-wide rare variant burden association analysis in 3269 unvaccinated patients with life-threatening COVID-19, and 1373 unvaccinated SARS-CoV-2-infected individuals without pneumonia. Among the 928 patients tested for autoantibodies against type I IFN, a quarter (234) were positive and were excluded.ResultsNo gene reached genome-wide significance. Under a recessive model, the most significant gene with at-risk variants was TLR7, with an OR of 27.68 (95%CI 1.5-528.7, P=1.1x10(-4)) for biochemically loss-of-function (bLOF) variants. We replicated the enrichment in rare predicted LOF (pLOF) variants at 13 influenza susceptibility loci involved in TLR3-dependent type I IFN immunity (OR=3.70[95%CI 1.3-8.2], P=2.1x10(-4)). This enrichment was further strengthened by (1) adding the recently reported TYK2 and TLR7 COVID-19 loci, particularly under a recessive model (OR=19.65[95%CI 2.1-2635.4], P=3.4x10(-3)), and (2) considering as pLOF branchpoint variants with potentially strong impacts on splicing among the 15 loci (OR=4.40[9%CI 2.3-8.4], P=7.7x10(-8)). Finally, the patients with pLOF/bLOF variants at these 15 loci were significantly younger (mean age [SD]=43.3 [20.3] years) than the other patients (56.0 [17.3] years; P=1.68x10(-5)).ConclusionsRare variants of TLR3- and TLR7-dependent type I IFN immunity genes can underlie life-threatening COVID-19, particularly with recessive inheritance, in patients under 60 years old

Correction: Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19

International audienc

The risk of COVID-19 death is much greater and age dependent with type I IFN autoantibodies

International audienceSignificance There is growing evidence that preexisting autoantibodies neutralizing type I interferons (IFNs) are strong determinants of life-threatening COVID-19 pneumonia. It is important to estimate their quantitative impact on COVID-19 mortality upon SARS-CoV-2 infection, by age and sex, as both the prevalence of these autoantibodies and the risk of COVID-19 death increase with age and are higher in men. Using an unvaccinated sample of 1,261 deceased patients and 34,159 individuals from the general population, we found that autoantibodies against type I IFNs strongly increased the SARS-CoV-2 infection fatality rate at all ages, in both men and women. Autoantibodies against type I IFNs are strong and common predictors of life-threatening COVID-19. Testing for these autoantibodies should be considered in the general population