Leveraging Cataloging and Collection Development Expertise to Improve OER Discovery

While there is ongoing improvement in some of the larger open educational resources (OER) search engines, librarians sending emails to listservs asking “anyone know of OER on this topic?” and keeping old-fashioned reading lists of valuable OER are common occurrences. Compared to searching for books in a library catalog or scholarly articles in a research database, finding OER wherever they may be is challenging even for librarians, not to mention instructional faculty. The reason is technical: subpar and variable metadata in OER search engines leads to difficulties searching, capturing, and sharing data across repositories. In other words, the current lack of robust, descriptive metadata for OER results in fewer access points. Thus, OER are comparatively hard to find. Bibliographic control for purposes of information storage and retrieval is something librarians are experts in, but we have not shared our methods with the Open Education community yet. So far, the majority of library advocates for OER have been reference and instruction librarians, as well as library directors. This is great, and we need them to continue to champion OER creation and adoption, but the Open Education movement needs technical services librarians to step forward and apply their cataloging and systems administration expertise to streamline access to the sprawling landscape of OER content; our profession would do well to share our collection development expertise as well. To this end, Clare Sobotka, Reference Assistant at Linn-Benton Community College (LBCC), Holly Wheeler, Library Cataloging and Metadata Specialist at Mt. Hood Community College (MHCC), and Heather White, Library Technical Services & OER Coordinator, along with their colleagues, have started to experiment with creating collection development policies and MARC records for OER. Ultimately, they hope for the development of a metadata schema that is shared between the Open Education and library communities and is mapped to MARC and RDA, to improve both catalog records and OER search engines across the web

Esophageal Adenocarcinoma: Opportunities for Targeted Prevention in Ohio

Objective: The incidence of esophageal adenocarcinoma, one of the most lethal gastroenterological diseases, has been increasing since the 1960s. Prevention of esophageal adenocarcinoma is important because no early detection screening programs have been shown to reduce mortality. Obesity, gastroesophageal reflux disease, and tobacco smoking are risk factors for esophageal adenocarcinoma. Due to the high prevalence in Ohio of obesity (32.6%) and cigarette smoking (21.0%), this study sought to identify trends and patterns of these risk factors and esophageal adenocarcinoma in Ohio as compared with the United States. Methods: Data from the Ohio Cancer Incidence Surveillance System, Surveillance Epidemiology and End Results Program (SEER), and Behavioral Risk Factor Surveillance System were used. Incidence rates overall, by demographics and by county, as well as trends in incidence of esophageal adenocarcinoma and the percent of esophageal adenocarcinoma among esophageal cancers were examined. Trends in obesity and cigarette smoking in Ohio, and the prevalence of each by county, were reported. Results: There was an increasing trend in esophageal adenocarcinoma incidence in Ohio. Ohio’s average annual esophageal adenocarcinoma incidence rate was higher than the SEER rate overall and for each sex, race, and age group in 2009 to 2013. There was also an increasing prevalence of obesity in Ohio. Although the prevalence of cigarette smoking has been stable, it was high in Ohio compared with the United States. Conclusions: Health care providers and researchers should be aware of the esophageal adenocarcinoma incidence rates and risk factor patterns and tailor interventions for areas and populations at higher risk

Alpha-Synuclein Pathology in Sensory Nerve Terminals of the Upper Aerodigestive Tract of Parkinson’s Disease Patients

Dysphagia is common in Parkinson’s disease (PD) and causes significant morbidity and mortality. PD dysphagia has usually been explained as dysfunction of central motor control, much like other motor symptoms that are characteristic of the disease. However, PD dysphagia does not correlate with severity of motor symptoms nor does it respond to motor therapies. It is known that PD patients have sensory deficits in the pharynx, and that impaired sensation may contribute to dysphagia. However, the underlying cause of the pharyngeal sensory deficits in PD is not known. We hypothesized that PD dysphagia with sensory deficits may be due to degeneration of the sensory nerve terminals in the upper aerodigestive tract (UAT). We have previously shown that Lewy-type synucleinopathy (LTS) is present in the main pharyngeal sensory nerves of PD patients, but not in controls. In this study, the sensory terminals in UAT mucosa were studied to discern the presence and distribution of LTS. Whole-mount specimens (tongue–pharynx–larynx–upper esophagus) were obtained from 10 deceased human subjects with clinically diagnosed and neuropathologically confirmed PD (five with dysphagia and five without) and four age-matched healthy controls. Samples were taken from six sites and immunostained for phosphorylated α-synuclein (PAS). The results showed the presence of PAS-immunoreactive (PAS-ir) axons in all the PD subjects and in none of the controls. Notably, PD patients with dysphagia had more PAS-ir axons in the regions that are critical for initiating the swallowing reflex. These findings suggest that Lewy pathology affects mucosal sensory axons in specific regions of the UAT and may be related to PD dysphagia