Renormalization Group Improvement and Dynamical Breaking of Symmetry in a Supersymmetric Chern-Simons-matter Model

In this work, we investigate the consequences of the Renormalization Group Equation (RGE) in the determination of the effective superpotential and the study of Dynamical Symmetry Breaking (DSB) in an N = 1 supersymmetric theory including an Abelian Chern-Simons superfield coupled to N scalar superfields in (2+1) dimensional spacetime. The classical Lagrangian presents scale invariance, which is broken by radiative corrections to the effective superpotential. We calculate the effective superpotential up to two-loops by using the RGE and the beta functions and anomalous dimensions known in the literature. We then show how the RGE can be used to improve this calculation, by summing up properly defined series of leading logs (LL), next-to-leading logs (NLL) contributions, and so on... We conclude that even if the RGE improvement procedure can indeed be applied in a supersymmetric model, the effects of the consideration of the RGE are not so dramatic as it happens in the non-supersymmetric case.Comment: v4: 11 pages, 1 figure. Version accepted for publication in NP

Non-perturbative fixed points and renormalization group improved effective potential

The stability conditions of a renormalization group improved effective potential have been discussed in the case of scalar QED and QCD with a colorless scalar. We calculate the same potential in these models assuming the existence of non-perturbative fixed points associated to a conformal phase. In the case of scalar QED the barrier of instability found previously is barely displaced as we approach the fixed point, and in the case of QCD with a colorless scalar not only the barrier is changed but the local minimum of the potential is also changed.Comment: 6 pages, 8 figures, References added. Matching the journal versio

Bachelor of Arts in Communication Students Asked: “What is my Contextual Communication Competence Level?”

In general, students need to be able to interact and communicate with one another from all walks of life, whether they are in personal or professional settings. Therefore, it is essential to examine students’ communication skills to ascertain whether they have adequate competence or not. In this regard, this research endeavored to determine the contextual communication competence of 266 Bachelor of Arts in Communication (BA Comm) students in one prime state university in Cordillera Administrative Region, Philippines. Through a quantitative-descriptive research design, one result revealed that BA Comm students are able to use their understanding of successful and appropriate communication patterns in public, meeting, group, and dyad situations with an average level of contextual communication competence. On the same level, BA Comm majors are committed to the field because they have the necessary communication abilities to adapt and improve various communication tactics when conversing with friends, acquaintances, and strangers. Lastly, compared to female BA Comm students, male students are more self-assured and open to communicating in various communication contexts, such as in public, meeting, group, or dyad contexts, as well as with strangers, friends, and acquaintances. Further, BA Comm students see a growth in their level of contextual communication competence as they move through their year levels because of their communication classes and social involvement. Recommendations are also included for future research directions

Regulation of NFKB through the nuclear processing of p105 (NFKB1) in Epstein-Barr Virus immortalized B cell lines.

Transcription factors of the NF-κB/Rel family are retained in the cytoplasm as inactive complexes through association with IκB inhibitory proteins. Several NF-κB activators induce the proteolysis of IκB proteins, which results in the nuclear translocation and DNA binding of NF-κB complexes. Here, we report a novel mechanism of NF-κB regulation mediated by p105 (NF-κB1) precursor of p50 directly at the nuclear level. In Epstein- Barr virus-immortalized B cells, p105 was found in the nucleus, where it was complexed with p65. In concomitance with NF-κB activation, mitomycin C induced the processing of p105 to p50 in the nucleus, while it did not affect the steady-state protein levels of IκBα and p105 in the cytoplasm. Differently, phorbol 12-myristate 13-acetate induced a significant proteolysis of both IκBα and p105 in the cytoplasm, while it did not affect the protein level of p105 in the nucleus. These results suggest that in Epstein-Barr virus-positive B cell lines the nuclear processing of p105 can contribute to NF-κB activation in response to specific signaling molecules, such as DNA-damaging agents

Bioclimatology, structure, and conservation perspectives of Quercus pyrenaica, Acer opalus subsp. Granatensis, and Corylus avellana deciduous forests on Mediterranean bioclimate in the South-Central part of the Iberian Peninsula

The plant variability in the southern Iberian Peninsula consists of around 3500 different taxa due to its high bioclimatic, geographic, and geological diversity. The deciduous forests in the southern Iberian Peninsula are located in regions with topographies and specific bioclimatic conditions that allow for the survival of taxa that are typical of cooler and wetter bioclimatic regions and therefore represent the relict evidence of colder and more humid paleoclimatic conditions. The floristic composition of 421 samples of deciduous forests in the south-central part of the Iberian Peninsula were analyzed. The ecological importance index (IVI) was calculated, where the most important tree species were Quercuspyrenaica, Aceropalus subsp. Granatensis, and Corylusavellana. These species are uncommon in the south-central part of the Iberian Peninsula, forming forests of little extension. An analysis of the vertical distribution of the species (stratum) shows that the majority of the species of stratum 3 (hemicriptophics, camephytes, geophites, and nanophanerophytes) are characteristic of deciduous forests, and their presence is positively correlated with high values of bioclimatic variables related to humidity and presence of water in the soil (nemoral environments), while they are negatively correlated with high values of bioclimatic variables related to high temperatures, evapotranspiration, and aridity. This work demonstrates that several characteristic deciduous forest taxa are more vulnerable to disappearance due to the loss of their nemoral conditions caused by gaps in the tree or shrub canopy. These gaps lead to an increase in evapotranspiration, excess insolation, and a consequent loss of water and humidity in the microclimatic conditions.info:eu-repo/semantics/publishedVersio

An NF-kB site in the 5'-untraslated leader region of the Human Immunodeficiency virus type 1 enhances the viral expression in response to NF-kB-activating stimuli.

The 5'-untranslated leader region of human immunodeficiency virus, type 1 (HIV-1), includes a complex array of putative regulatory elements whose role in the viral expression is not completely understood. Here we demonstrate the presence of an NF-κB-responsive element in the trans- activation response (TAR) region of HIV-1 that confers the full induction of HIV-1 long terminal repeat (LTR) in response to NF-κB-activating stimuli, such as DNA alkylating agents, phorbol 12-myristate 13-acetate, and tumor necrosis factor-α. The TAR NF-κB site GGGAGCTCTC spans from positions +31 to +40 and cooperates with the NF-κB enhancer upstream of the TATA box in the NF-κB-mediated induction of HIV-1 LTR. The conclusion stems from the following observations: (i) deletion of the two NF-κB sites upstream of the TATA box reduces, but does not abolish, the HIV-1 LTR activation by NF-κB inducers; (ii) deletion or base pair substitutions of the TAR NF-κB site significantly reduce the HIV-1 LTR activation by NF-κB inducers; (iii) deletions of both the NF-κB sites upstream of the TATA box and the TAR NF- κB site abolish the activation of HIV-1 LTR in response to NF-κB inducers. Moreover, the p50·p65 NF-κB complex binds to the TAR NF-κB sequence and trans-activates the TAR NF-κB-directed expression. The identification of an additional NF-κB site in the HIV-1 LTR points to the relevance of NF-κB factors in the HIV-1 life cycle

Partial purification and MALDI-TOF MS analysis of UN1, a tumor antigen membrane glycoprotein.

UN1 is a membrane glycoprotein that is expressed in immature human thymocytes, a subpopulation of peripheral T lymphocytes, the HPB acute lymphoblastic leukemia (ALL) T-cell line and fetal thymus. We previously reported the isolation of a monoclonal antibody (UN1 mAb) recognizing the UN1 protein that was classified as "unclustered" at the 5th and 6th International Workshop and Conference on Human Leukocyte Differentiation Antigens. UN1 was highly expressed in breast cancer tissues and was undetected in non-proliferative lesions and in normal breast tissues, indicating a role for UN1 in the development of a tumorigenic phenotype of breast cancer cells. In this study, we report a partial purification of the UN1 protein from HPB-ALL T cells by anion-exchange chromatography followed by immunoprecipitation with the UN1 mAb and MALDI-TOF MS analysis. This analysis should assist in identifying the amino acid sequence of UN