Zener transitions between dissipative Bloch bands. II: Current Response at Finite Temperature

We extend, to include the effects of finite temperature, our earlier study of the interband dynamics of electrons with Markoffian dephasing under the influence of uniform static electric fields. We use a simple two-band tight-binding model and study the electric current response as a function of field strength and the model parameters. In addition to the Esaki-Tsu peak, near where the Bloch frequency equals the damping rate, we find current peaks near the Zener resonances, at equally spaced values of the inverse electric field. These become more prominenent and numerous with increasing bandwidth (in units of the temperature, with other parameters fixed). As expected, they broaden with increasing damping (dephasing).Comment: 5 pages, LateX, plus 5 postscript figure

Time Periodic Behavior of Multiband Superlattices in Static Electric Fields

We use an analytic perturbation expansion for the two-band system of tight binding electrons to discuss Bloch oscillations and Zener tunneling within this model. We make comparison with recent numerical results and predict analytically the frequency of radiation expected from Zener tunneling, including its disappearance, as a function of the system parameters.Comment: 12 pages, no figure include

ac-Field-Controlled Anderson Localization in Disordered Semiconductor Superlattices

An ac field, tuned exactly to resonance with the Stark ladder in an ideal tight binding lattice under strong dc bias, counteracts Wannier-Stark localization and leads to the emergence of extended Floquet states. If there is random disorder, these states localize. The localization lengths depend non-monotonically on the ac field amplitude and become essentially zero at certain parameters. This effect is of possible relevance for characterizing the quality of superlattice samples, and for performing experiments on Anderson localization in systems with well-defined disorder.Comment: 10 pages, Latex; figures available on request from [email protected]

Localization of interacting electrons in quantum dot arrays driven by an ac-field

We investigate the dynamics of two interacting electrons moving in a one-dimensional array of quantum dots under the influence of an ac-field. We show that the system exhibits two distinct regimes of behavior, depending on the ratio of the strength of the driving field to the inter-electron Coulomb repulsion. When the ac-field dominates, an effect termed coherent destruction of tunneling occurs at certain frequencies, in which transport along the array is suppressed. In the other, weak-driving, regime we find the surprising result that the two electrons can bind into a single composite particle -- despite the strong Coulomb repulsion between them -- which can then be controlled by the ac-field in an analogous way. We show how calculation of the Floquet quasienergies of the system explains these results, and thus how ac-fields can be used to control the localization of interacting electron systems.Comment: 7 pages, 6 eps figures V2. Minor changes, this version to be published in Phys. Rev.

Rickettsia parkeri in Amblyomma americanum Ticks, Tennessee and Georgia, USA

To determine the geographic distribution of the newly recognized human pathogen Rickettsia parkeri, we looked for this organism in ticks from Tennessee and Georgia, USA. Using PCR and sequence analysis, we identified R. parkeri in 2 Amblyomma americanum ticks. This rickettsiosis may be underdiagnosed in the eastern United States

Clinical and Research Considerations for Patients with Hypertensive Acute Heart Failure

Management approaches for patients in the emergency department (ED) who present with acute heart failure (AHF) have largely focused on intravenous diuretics. Yet, the primary pathophysiologic derangement underlying AHF in many patients is not solely volume overload. Patients with hypertensive AHF (H-AHF) represent a clinical phenotype with distinct pathophysiologic mechanisms that result in elevated ventricular filling pressures. To optimize treatment response and minimize adverse events in this subgroup, we propose that clinical management be tailored to a conceptual model of disease based on these mechanisms. This consensus statement reviews the relevant pathophysiology, clinical characteristics, approach to therapy, and considerations for clinical trials in ED patients with H-AHF

Validation and Potential Mechanisms of Red Cell Distribution Width as a Prognostic Marker in Heart Failure

Adverse outcomes have recently been linked to elevated red cell distribution width (RDW) in heart failure. Our study sought to validate the prognostic value of RDW in heart failure and to explore the potential mechanisms underlying this association

REVEILLE8 and PSEUDO-REPONSE REGULATOR5 Form a Negative Feedback Loop within the Arabidopsis Circadian Clock

Circadian rhythms provide organisms with an adaptive advantage, allowing them to regulate physiological and developmental events so that they occur at the most appropriate time of day. In plants, as in other eukaryotes, multiple transcriptional feedback loops are central to clock function. In one such feedback loop, the Myb-like transcription factors CCA1 and LHY directly repress expression of the pseudoresponse regulator TOC1 by binding to an evening element (EE) in the TOC1 promoter. Another key regulatory circuit involves CCA1 and LHY and the TOC1 homologs PRR5, PRR7, and PRR9. Purification of EE–binding proteins from plant extracts followed by mass spectrometry led to the identification of RVE8, a homolog of CCA1 and LHY. Similar to these well-known clock genes, expression of RVE8 is circadian-regulated with a dawn phase of expression, and RVE8 binds specifically to the EE. However, whereas cca1 and lhy mutants have short period phenotypes and overexpression of either gene causes arrhythmia, rve8 mutants have long-period and RVE8-OX plants have short-period phenotypes. Light input to the clock is normal in rve8, but temperature compensation (a hallmark of circadian rhythms) is perturbed. RVE8 binds to the promoters of both TOC1 and PRR5 in the subjective afternoon, but surprisingly only PRR5 expression is perturbed by overexpression of RVE8. Together, our data indicate that RVE8 promotes expression of a subset of EE–containing clock genes towards the end of the subjective day and forms a negative feedback loop with PRR5. Thus RVE8 and its homologs CCA1 and LHY function close to the circadian oscillator but act via distinct molecular mechanisms