UHECR Acceleration in Dark Matter Filaments of Cosmological Structure Formation

A mechanism for proton acceleration to ~10^21eV is suggested. It may operate in accretion flows onto thin dark matter filaments of cosmic structure formation. The flow compresses the ambient magnetic field to strongly increase and align it with the filament. Particles begin the acceleration by the ExB drift with the accretion flow. The energy gain in the drift regime is limited by the conservation of the adiabatic invariant p_perp^2/B. Upon approaching the filament, the drift turns into the gyro-motion around the filament so that the particle moves parallel to the azimuthal electric field. In this 'betatron' regime the acceleration speeds up to rapidly reach the electrodynamic limit $cp_{max}=eBR$ for an accelerator with magnetic field $B$ and the orbit radius $R$ (Larmor radius). The periodic orbit becomes unstable and the particle slings out of the filament to the region of a weak (uncompressed) magnetic field, which terminates the acceleration. The mechanism requires pre-acceleration that is likely to occur in structure formation shocks upstream or nearby the filament accretion flow. Previous studies identify such shocks as efficient proton accelerators to a firm upper limit ~10^19.5 eV placed by the catastrophic photo-pion losses. The present mechanism combines explosive energy gain in its final (betatron) phase with prompt particle release from the region of strong magnetic field. It is this combination that allows protons to overcome both the photo-pion and the synchrotron-Compton losses and therefore attain energy 10^21 eV. A requirement on accelerator to reach a given E_max placed by the accelerator energy dissipation \propto E_{max}^{2}/Z_0 due to the finite vacuum impedance Z_0 is circumvented by the cyclic operation of the accelerator.Comment: 34 pages, 10 figures, to be published in JCA

Collective T=0 pairing in N=Z nuclei? Pairing vibrations around 56Ni revisited

We present a new analysis of the pairing vibrations around 56Ni, with emphasis on odd-odd nuclei. This analysis of the experimental excitation energies is based on the subtraction of average properties that include the full symmetry energy together with volume, surface and Coulomb terms. The results clearly indicate a collective behavior of the isovector pairing vibrations and do not support any appreciable collectivity in the isoscalar channel.Comment: RevTeX, two-column, 5 pages, 4 figure

Towards a Simple Model of Compressible Alfvenic Turbulence

A simple model collisionless, dissipative, compressible MHD (Alfvenic) turbulence in a magnetized system is investigated. In contrast to more familiar paradigms of turbulence, dissipation arises from Landau damping, enters via nonlinearity, and is distributed over all scales. The theory predicts that two different regimes or phases of turbulence are possible, depending on the ratio of steepening to damping coefficient (m_1/m_2). For strong damping (|m_1/m_2|<1), a regime of smooth, hydrodynamic turbulence is predicted. For |m_1/m_2|>1, steady state turbulence does not exist in the hydrodynamic limit. Rather, spikey, small scale structure is predicted.Comment: 6 pages, one figure, REVTeX; this version to be published in PRE. For related papers, see http://sdphpd.ucsd.edu/~medvedev/papers.htm

Performing heritage: the use of live 'actors' in heritage presentations

This paper investigates the phenomenon of 'living history' presentations of heritage, using live 'actors' to portray historical characters. Its aim is to discuss these presentations in the context of what may be understood as 'heritage', and of the nature of 'performance'. Four case studies of heritage sites, each important as a tourist attraction, have been selected for detailed study, together with a number of other examples of heritage performance. It is clear from the empirical work that different performance strategies are employed within the heritage industry and by individual 'actors'. Most of the performers take part as a leisure activity, and many do not consider themselves to be 'performing' at all. The greatest concern of participants lies in the degree of authenticity of the performance. Through 'living history', the 'actors' are drawn into an experience of heritage which has real meaning for them, and which may contribute both to a sense of identity and to an enhanced understanding of society, past and present. The popularity of such presentations with visitors also indicates that similar benefits are perceived by the 'audience'

Multibeam Maser Survey of methanol and excited OH in the Magellanic clouds: new detections and maser abundance estimates

‘The definitive version is available at www.blackwell-synergy.com.’ Copyright Blackwell Publishing DOI: 10.1111/j.1365-2966.2008.12888.xPeer reviewe

INSIG1 influences obesity-related hypertriglyceridemia in humans

In our analysis of a quantitative trait locus (QTL) for plasma triglyceride (TG) levels [logarithm of odds (LOD) = 3.7] on human chromosome 7q36, we examined 29 single nucleotide polymorphisms (SNPs) across INSIG1, a biological candidate gene in the region. Insulin-induced genes (INSIGs) are feedback mediators of cholesterol and fatty acid synthesis in animals, but their role in human lipid regulation is unclear. In our cohort, the INSIG1 promoter SNP rs2721 was associated with TG levels (P = 2 × 10−3 in 1,560 individuals of the original linkage cohort, P = 8 × 10−4 in 920 unrelated individuals of the replication cohort, combined P = 9.9 × 10−6). Individuals homozygous for the T allele had 9% higher TG levels and 2-fold lower expression of INSIG1 in surgical liver biopsy samples when compared with individuals homozygous for the G allele. Also, the T allele showed additional binding of nuclear proteins from HepG2 liver cells in gel shift assays. Finally, the variant rs7566605 in INSIG2, the only homolog of INSIG1, enhances the effect of rs2721 (P = 0.00117). The variant rs2721 alone explains 5.4% of the observed linkage in our cohort, suggesting that additional, yet-undiscovered genes and sequence variants in the QTL interval also contribute to alterations in TG levels in humans