179 research outputs found
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Soil conditions and the iron chlorosis of mature vine
Iron-deficiency chlorosis is a usual routine problem on calcareous carbonated soils of Crimea. Different reasons cause vine chlorosis: soil properties, physiological status of plants and others. It was shown that chlorosis spot in the vineyard has constant location. Chlorosis can be identified visually and instrumentally. In this study, an attempt was made to find the relationship between soil electrical resistance and the spread of vine chlorosis
First Order Phase Transition in a Reaction-Diffusion Model With Open Boundary: The Yang-Lee Theory Approach
A coagulation-decoagulation model is introduced on a chain of length L with
open boundary. The model consists of one species of particles which diffuse,
coagulate and decoagulate preferentially in the leftward direction. They are
also injected and extracted from the left boundary with different rates. We
will show that on a specific plane in the space of parameters, the steady state
weights can be calculated exactly using a matrix product method. The model
exhibits a first-order phase transition between a low-density and a
high-density phase. The density profile of the particles in each phase is
obtained both analytically and using the Monte Carlo Simulation. The two-point
density-density correlation function in each phase has also been calculated. By
applying the Yang-Lee theory we can predict the same phase diagram for the
model. This model is further evidence for the applicability of the Yang-Lee
theory in the non-equilibrium statistical mechanics context.Comment: 10 Pages, 3 Figures, To appear in Journal of Physics A: Mathematical
and Genera
Spreading with immunization in high dimensions
We investigate a model of epidemic spreading with partial immunization which
is controlled by two probabilities, namely, for first infections, , and
reinfections, . When the two probabilities are equal, the model reduces to
directed percolation, while for perfect immunization one obtains the general
epidemic process belonging to the universality class of dynamical percolation.
We focus on the critical behavior in the vicinity of the directed percolation
point, especially in high dimensions . It is argued that the clusters of
immune sites are compact for . This observation implies that a
recently introduced scaling argument, suggesting a stretched exponential decay
of the survival probability for , in one spatial dimension,
where denotes the critical threshold for directed percolation, should
apply in any dimension and maybe for as well. Moreover, we
show that the phase transition line, connecting the critical points of directed
percolation and of dynamical percolation, terminates in the critical point of
directed percolation with vanishing slope for and with finite slope for
. Furthermore, an exponent is identified for the temporal correlation
length for the case of and , , which
is different from the exponent of directed percolation. We also
improve numerical estimates of several critical parameters and exponents,
especially for dynamical percolation in .Comment: LaTeX, IOP-style, 18 pages, 9 eps figures, minor changes, additional
reference
Dyck Paths, Motzkin Paths and Traffic Jams
It has recently been observed that the normalization of a one-dimensional
out-of-equilibrium model, the Asymmetric Exclusion Process (ASEP) with random
sequential dynamics, is exactly equivalent to the partition function of a
two-dimensional lattice path model of one-transit walks, or equivalently Dyck
paths. This explains the applicability of the Lee-Yang theory of partition
function zeros to the ASEP normalization.
In this paper we consider the exact solution of the parallel-update ASEP, a
special case of the Nagel-Schreckenberg model for traffic flow, in which the
ASEP phase transitions can be intepreted as jamming transitions, and find that
Lee-Yang theory still applies. We show that the parallel-update ASEP
normalization can be expressed as one of several equivalent two-dimensional
lattice path problems involving weighted Dyck or Motzkin paths. We introduce
the notion of thermodynamic equivalence for such paths and show that the
robustness of the general form of the ASEP phase diagram under various update
dynamics is a consequence of this thermodynamic equivalence.Comment: Version accepted for publicatio
Nonequilibrium Steady States of Matrix Product Form: A Solver's Guide
We consider the general problem of determining the steady state of stochastic
nonequilibrium systems such as those that have been used to model (among other
things) biological transport and traffic flow. We begin with a broad overview
of this class of driven diffusive systems - which includes exclusion processes
- focusing on interesting physical properties, such as shocks and phase
transitions. We then turn our attention specifically to those models for which
the exact distribution of microstates in the steady state can be expressed in a
matrix product form. In addition to a gentle introduction to this matrix
product approach, how it works and how it relates to similar constructions that
arise in other physical contexts, we present a unified, pedagogical account of
the various means by which the statistical mechanical calculations of
macroscopic physical quantities are actually performed. We also review a number
of more advanced topics, including nonequilibrium free energy functionals, the
classification of exclusion processes involving multiple particle species,
existence proofs of a matrix product state for a given model and more
complicated variants of the matrix product state that allow various types of
parallel dynamics to be handled. We conclude with a brief discussion of open
problems for future research.Comment: 127 pages, 31 figures, invited topical review for J. Phys. A (uses
IOP class file
Single-molecule experiments in biological physics: methods and applications
I review single-molecule experiments (SME) in biological physics. Recent
technological developments have provided the tools to design and build
scientific instruments of high enough sensitivity and precision to manipulate
and visualize individual molecules and measure microscopic forces. Using SME it
is possible to: manipulate molecules one at a time and measure distributions
describing molecular properties; characterize the kinetics of biomolecular
reactions and; detect molecular intermediates. SME provide the additional
information about thermodynamics and kinetics of biomolecular processes. This
complements information obtained in traditional bulk assays. In SME it is also
possible to measure small energies and detect large Brownian deviations in
biomolecular reactions, thereby offering new methods and systems to scrutinize
the basic foundations of statistical mechanics. This review is written at a
very introductory level emphasizing the importance of SME to scientists
interested in knowing the common playground of ideas and the interdisciplinary
topics accessible by these techniques. The review discusses SME from an
experimental perspective, first exposing the most common experimental
methodologies and later presenting various molecular systems where such
techniques have been applied. I briefly discuss experimental techniques such as
atomic-force microscopy (AFM), laser optical tweezers (LOT), magnetic tweezers
(MT), biomembrane force probe (BFP) and single-molecule fluorescence (SMF). I
then present several applications of SME to the study of nucleic acids (DNA,
RNA and DNA condensation), proteins (protein-protein interactions, protein
folding and molecular motors). Finally, I discuss applications of SME to the
study of the nonequilibrium thermodynamics of small systems and the
experimental verification of fluctuation theorems. I conclude with a discussion
of open questions and future perspectives.Comment: Latex, 60 pages, 12 figures, Topical Review for J. Phys. C (Cond.
Matt
Local Persistence in the Directed Percolation Universality Class
We revisit the problem of local persistence in directed percolation,
reporting improved estimates of the persistence exponent in 1+1 dimensions,
discovering strong corrections to scaling in higher dimensions, and
investigating the mean field limit. Moreover, we introduce a graded persistence
probability that a site does not flip more than n times and demonstrate how
local persistence can be studied in seed simulations. Finally, the problem of
spatial (as opposed to temporal) persistence is investigated.Comment: LaTeX, 24 pages, 12 figures; references added and corrected, section
4.3 rewritte
Demagnetization via Nucleation of the Nonequilibrium Metastable Phase in a Model of Disorder
We study both analytically and numerically metastability and nucleation in a
two-dimensional nonequilibrium Ising ferromagnet. Canonical equilibrium is
dynamically impeded by a weak random perturbation which models homogeneous
disorder of undetermined source. We present a simple theoretical description,
in perfect agreement with Monte Carlo simulations, assuming that the decay of
the nonequilibrium metastable state is due, as in equilibrium, to the
competition between the surface and the bulk. This suggests one to accept a
nonequilibrium "free-energy" at a mesoscopic/cluster level, and it ensues a
nonequilibrium "surface tension" with some peculiar low-T behavior. We
illustrate the occurrence of intriguing nonequilibrium phenomena, including:
(i) Noise-enhanced stabilization of nonequilibrium metastable states; (ii)
reentrance of the limit of metastability under strong nonequilibrium
conditions; and (iii) resonant propagation of domain walls. The cooperative
behavior of our system may also be understood in terms of a Langevin equation
with additive and multiplicative noises. We also studied metastability in the
case of open boundaries as it may correspond to a magnetic nanoparticle. We
then observe burst-like relaxation at low T, triggered by the additional
surface randomness, with scale-free avalanches which closely resemble the type
of relaxation reported for many complex systems. We show that this results from
the superposition of many demagnetization events, each with a well- defined
scale which is determined by the curvature of the domain wall at which it
originates. This is an example of (apparent) scale invariance in a
nonequilibrium setting which is not to be associated with any familiar kind of
criticality.Comment: 26 pages, 22 figure
Coaggregation of RNA-Binding Proteins in a Model of TDP-43 Proteinopathy with Selective RGG Motif Methylation and a Role for RRM1 Ubiquitination
TAR DNA-binding protein 43 (TDP-43) is a major component within ubiquitin-positive inclusions of a number of neurodegenerative diseases that increasingly are considered as TDP-43 proteinopathies. Identities of other inclusion proteins associated with TDP-43 aggregation remain poorly defined. In this study, we identify and quantitate 35 co-aggregating proteins in the detergent-resistant fraction of HEK-293 cells in which TDP-43 or a particularly aggregate prone variant, TDP-S6, were enriched following overexpression, using stable isotope-labeled (SILAC) internal standards and liquid chromatography coupled to tandem mass spectrometry (LC-MS/MS). We also searched for differential post-translational modification (PTM) sites of ubiquitination. Four sites of ubiquitin conjugation to TDP-43 or TDP-S6 were confirmed by dialkylated GST-TDP-43 external reference peptides, occurring on or near RNA binding motif (RRM) 1. RRM-containing proteins co-enriched in cytoplasmic granular structures in HEK-293 cells and primary motor neurons with insoluble TDP-S6, including cytoplasmic stress granule associated proteins G3BP, PABPC1, and eIF4A1. Proteomic evidence for TDP-43 co-aggregation with paraspeckle markers RBM14, PSF and NonO was also validated by western blot and by immunocytochemistry in HEK-293 cells. An increase in peptides from methylated arginine-glycine-glycine (RGG) RNA-binding motifs of FUS/TLS and hnRNPs was found in the detergent-insoluble fraction of TDP-overexpressing cells. Finally, TDP-43 and TDP-S6 detergent-insoluble species were reduced by mutagenesis of the identified ubiquitination sites, even following oxidative or proteolytic stress. Together, these findings define some of the aggregation partners of TDP-43, and suggest that TDP-43 ubiquitination influences TDP-43 oligomerization
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