337 research outputs found

    Why don't clumps of cirrus dust gravitationally collapse?

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    We consider the Herschel-Planck infrared observations of presumed condensations of interstellar material at a measured temperature of approximately 14 K (Juvela et al., 2012), the triple point temperature of hydrogen. The standard picture is challenged that the material is cirrus-like clouds of ceramic dust responsible for Halo extinction of cosmological sources (Finkbeiner, Davis, and Schlegel 1999). Why would such dust clouds not collapse gravitationally to a point on a gravitational free-fall time scale of 10810^8 years? Why do the particles not collide and stick together, as is fundamental to the theory of planet formation (Blum 2004; Blum and Wurm, 2008) in pre-solar accretion discs? Evidence from 3.3 μ\mum and UIB emissions as well as ERE (extended red emission) data point to the dominance of PAH-type macromolecules for cirrus dust, but such fractal dust will not spin in the manner of rigid grains (Draine & Lazarian, 1998). IRAS dust clouds examined by Herschel-Planck are easily understood as dark matter Proto-Globular-star-Cluster (PGC) clumps of primordial gas planets, as predicted by Gibson (1996) and observed by Schild (1996).Comment: 8 pages, 2 figures, Conference FQMT'1

    Evolution of primordial planets in relation to the cosmological origin of life

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    We explore the conditions prevailing in primordial planets in the framework of the HGD cosmologies as discussed by Gibson and Schild. The initial stages of condensation of planet-mass H-4He gas clouds in trillion-planet clumps is set at 300,000 yr (0.3My) following the onset of plasma instabilities when ambient temperatures were >1000K. Eventual collapse of the planet-cloud into a solid structure takes place against the background of an expanding universe with declining ambient temperatures. Stars form from planet mergers within the clumps and die by supernovae on overeating of planets. For planets produced by stars, isothermal free fall collapse occurs initially via quasi equilibrium polytropes until opacity sets in due to molecule and dust formation. The contracting cooling cloud is a venue for molecule formation and the sequential condensation of solid particles, starting from mineral grains at high temperatures to ice particles at lower temperatures, water-ice becomes thermodynamically stable between 7 and 15 My after the initial onset of collapse, and contraction to form a solid icy core begins shortly thereafter. Primordial-clump-planets are separated by ~ 1000 AU, reflecting the high density of the universe at 30,000 yr. Exchanges of materials, organic molecules and evolving templates readily occur, providing optimal conditions for an initial origin of life in hot primordial gas planet water cores when adequately fertilized by stardust. The condensation of solid molecular hydrogen as an extended outer crust takes place much later in the collapse history of the protoplanet. When the object has shrunk to several times the radius of Jupiter, the hydrogen partial pressure exceeds the saturation vapour pressure of solid hydrogen at the ambient temperature and condensation occurs.Comment: 14 pages 7 figures SPIE Conference 7819 Instruments, Methods, and Missions for Astrobiology XIII Proceedings, Aug 3-5, 2010, San Diego, Ed. Richard B. Hoove

    Detecting and Mitigating Denial-of-Service Attacks on Voice over IP Networks

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    Voice over IP (VoIP) is more susceptible to Denial of Service attacks than traditional data traffic, due to the former's low tolerance to delay and jitter. We describe the design of our VoIP Vulnerability Assessment Tool (VVAT) with which we demonstrate vulnerabilities to DoS attacks inherent in many of the popular VoIP applications available today. In our threat model we assume an adversary who is not a network administrator, nor has direct control of the channel and key VoIP elements. His aim is to degrade his victim's QoS without giving away his presence by making his attack look like a normal network degradation. Even black-boxed, applications like Skype that use proprietary protocols show poor performance under specially crafted DoS attacks to its media stream. Finally we show how securing Skype relays not only preserves many of its useful features such as seamless traversal of firewalls but also protects its users from DoS attacks such as recording of conversations and disruption of voice quality. We also present our experiences using virtualization to protect VoIP applications from 'insider attacks'. Our contribution is two fold we: 1) Outline a threat model for VoIP, incorporating our attack models in an open-source network simulator/emulator allowing VoIP vendors to check their software for vulnerabilities in a controlled environment before releasing it. 2) We present two promising approaches for protecting the confidentiality, availability and authentication of VoIP Services

    Concept for a Low-Cost, High-Efficiency Precipitation Radar System Based on Ferroelectric Reflectarray Antenna

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    This work proposes a concept on a novel scanning phased array, based on thin film ferroelectric phase shifters, for an X-band precipitation monostatic radar

    Autoreactivity to Glucose Regulated Protein 78 Links Emphysema and Osteoporosis in Smokers

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    Rationale: Emphysema and osteoporosis are epidemiologically associated diseases of cigarette smokers. The causal mechanism(s) linking these illnesses is unknown. We hypothesized autoimmune responses may be involved in both disorders. Objectives: To discover an antigen-specific autoimmune response associated with both emphysema and osteoporosis among smokers. Methods: Replicate nonbiased discovery assays indicated that autoimmunity to glucose regulated protein 78 (GRP78), an endoplasmic reticulum chaperone and cell surface signaling receptor, is present in many smokers. Subject assessments included spirometry, chest CT scans, dual x-ray absorptiometry, and immunoblots for anti-GRP78 IgG. Anti-GRP78 autoantibodies were isolated from patient plasma by affinity chromatography, leukocyte functions assessed by flow cytometry, and soluble metabolites and mediators measured by immunoassays. Measurements and Main Results Circulating anti-GRP78 IgG autoantibodies were detected in plasma specimens from 86 (32%) of the 265 smoking subjects. Anti-GRP78 autoantibodies were singularly prevalent among subjects with radiographic emphysema (OR 3.1, 95%CI 1.7–5.7, p = 0.003). Anti-GRP78 autoantibodies were also associated with osteoporosis (OR 4.7, 95%CI 1.7–13.3, p = 0.002), and increased circulating bone metabolites (p = 0.006). Among emphysematous subjects, GRP78 protein was an autoantigen of CD4 T-cells, stimulating lymphocyte proliferation (p = 0.0002) and IFN-gamma production (p = 0.03). Patient-derived anti-GRP78 autoantibodies had avidities for osteoclasts and macrophages, and increased macrophage NFkB phosphorylation (p = 0.005) and productions of IL-8, CCL-2, and MMP9 (p = 0.005, 0.007, 0.03, respectively). Conclusions: Humoral and cellular GRP78 autoimmune responses in smokers have numerous biologically-relevant pro-inflammatory and other deleterious actions, and are associated with emphysema and osteoporosis. These findings may have relevance for the pathogenesis of smoking-associated diseases, and development of biomarker immunoassays and/or novel treatments for these disorders
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