The impact of EU enlargement on migration flows

This document is a report commissioned by the Home Office in order to assess the magnitude of potential migration flows to the UK after the enlargement of the European Union (EU). The countries which are expected to join the EU on 1 May 2004 are Cyprus, the Czech Republic, Estonia, Hungary, Latvia, Lithuania, Malta, Poland, Slovakia and Slovenia. The aim of the report is to provide extensive information that will be helpful in evaluating the migration potential from these countries to the UK. This is done by (i) describing the socio-economic situation of the accession countries nowadays and since the early 1990s and comparing it to that of the UK and Germany, (ii) critically reviewing the existing literature that attempts to predict the effects of the current EU enlargement on migration flows, (iii) analysing the so-called Southern enlargement (the accessions of Greece, Portugal and Spain) and comparing it to the current one, and (iv) presenting a quantitative analysis of the effect of the current enlargement on migration to the UK

Unitary expansion of the time evolution operator

We propose an expansion of the unitary evolution operator, associated to a given Schr\"odinger equation, in terms of a finite product of explicit unitary operators. In this manner, this unitary expansion can be truncated at the desired level of approximation, as shown in the given examples.Comment: 6 pages, 7 figures. Updated version, minor final change

HESS J1826−-130: A Very Hard γ\gamma-Ray Spectrum Source in the Galactic Plane

HESS J1826$-$130 is an unidentified hard spectrum source discovered by H.E.S.S. along the Galactic plane, the spectral index being $\Gamma$ = 1.6 with an exponential cut-off at about 12 TeV. While the source does not have a clear counterpart at longer wavelengths, the very hard spectrum emission at TeV energies implies that electrons or protons accelerated up to several hundreds of TeV are responsible for the emission. In the hadronic case, the VHE emission can be produced by runaway cosmic-rays colliding with the dense molecular clouds spatially coincident with the H.E.S.S. source.Comment: 6 pages, 3 figures, Proceedings of the 6th International Symposium on High Energy Gamma-Ray Astronomy (Gamma2016), Heidelberg, German

Pushing 1D CCSNe to explosions: model and SN 1987A

We report on a method, PUSH, for triggering core-collapse supernova explosions of massive stars in spherical symmetry. We explore basic explosion properties and calibrate PUSH such that the observables of SN1987A are reproduced. Our simulations are based on the general relativistic hydrodynamics code AGILE combined with the detailed neutrino transport scheme IDSA for electron neutrinos and ALS for the muon and tau neutrinos. To trigger explosions in the otherwise non-exploding simulations, we rely on the neutrino-driven mechanism. The PUSH method locally increases the energy deposition in the gain region through energy deposition by the heavy neutrino flavors. Our setup allows us to model the explosion for several seconds after core bounce. We explore the progenitor range 18-21M$_{\odot}$. Our studies reveal a distinction between high compactness (HC) and low compactness (LC) progenitor models, where LC models tend to explore earlier, with a lower explosion energy, and with a lower remnant mass. HC models are needed to obtain explosion energies around 1 Bethe, as observed for SN1987A. However, all the models with sufficiently high explosion energy overproduce $^{56}$Ni. We conclude that fallback is needed to reproduce the observed nucleosynthesis yields. The nucleosynthesis yields of $^{57-58}$Ni depend sensitively on the electron fraction and on the location of the mass cut with respect to the initial shell structure of the progenitor star. We identify a progenitor and a suitable set of PUSH parameters that fit the explosion properties of SN1987A when assuming 0.1M$_{\odot}$ of fallback. We predict a neutron star with a gravitational mass of 1.50M$_{\odot}$. We find correlations between explosion properties and the compactness of the progenitor model in the explored progenitors. However, a more complete analysis will require the exploration of a larger set of progenitors with PUSH.Comment: revised version as accepted by ApJ (results unchanged, text modified for clarification, a few references added); 26 pages, 20 figure

Klein tunneling and Dirac potentials in trapped ions

We propose the quantum simulation of the Dirac equation with potentials, allowing the study of relativistic scaterring and the Klein tunneling. This quantum relativistic effect permits a positive-energy Dirac particle to propagate through a repulsive potential via the population transfer to negative-energy components. We show how to engineer scalar, pseudoscalar, and other potentials in the 1+1 Dirac equation by manipulating two trapped ions. The Dirac spinor is represented by the internal states of one ion, while its position and momentum are described by those of a collective motional mode. The second ion is used to build the desired potentials with high spatial resolution.Comment: 4 pages, 3 figures, minor change

Report, Holly Springs Branch Experiment Station, 1927

Mode of access: Internet

Activation of ARF6 by ARNO stimulates epithelial cell migration through downstream activation of both Rac1 and phospholipase D

Migration of epithelial cells is essential for tissue morphogenesis, wound healing, and metastasis of epithelial tumors. Here we show that ARNO, a guanine nucleotide exchange factor for ADP-ribosylation factor (ARF) GTPases, induces Madin-Darby canine kidney epithelial cells to develop broad lamellipodia, to separate from neighboring cells, and to exhibit a dramatic increase in migratory behavior. This transition requires ARNO catalytic activity, which we show leads to enhanced activation of endogenous ARF6, but not ARF1, using a novel pulldown assay. We further demonstrate that expression of ARNO leads to increased activation of endogenous Rac1, and that Rac activation is required for ARNO-induced cell motility. Finally, ARNO-induced activation of ARF6 also results in increased activation of phospholipase D (PLD), and inhibition of PLD activity also inhibits motility. However, inhibition of PLD does not prevent activation of Rac. Together, these data suggest that ARF6 activation stimulates two distinct signaling pathways, one leading to Rac activation, the other to changes in membrane phospholipid composition, and that both pathways are required for cell motility