Probing the BCS-BEC crossover with persistent currents

We study the persistent currents of an attractive Fermi gas confined in a tightly-confining ring trap and subjected to an artificial gauge field all through the BCS-BEC crossover. At weak attractions, on the BCS side, fermions display a parity effect in the persistent currents, ie their response to the gauge field is paramagnetic or diamagnetic depending on the number of pairs on the ring. At resonance and on the BEC side of the crossover we find a doubling of the periodicity of the ground-state energy as a function of the artificial gauge field and disappearance of the parity effect, indicating that persistent currents can be used to infer the formation of tightly-bound bosonic pairs. Our predictions can be accessed in ultracold atoms experiments through noise interferograms.Comment: 7 pages, 5 figure

Un patrimonio culturale e un percorso di valorizzazione

Questo contributo vuole illustrare la collezione di manufatti realizzati dai ricoverati nell’Ospedale Psichiatrico di Collegno (TO) fra XIX e XX secolo e conservati presso il Museo di Antropologia ed Etnografia dell’Università degli Studi di Torino grazie all’opera di due psichiatri torinesi: Giovanni Marro e il padre Antonio. This paper is meant to illustrate the collection of artifacts made by the patients of the Psychiatric Hospital in Collegno, Turin, Italy, in the period between the 19th and the 20th Century. These products are stored at the Museum of Anthropology and Ethnography of University of Turin thanks to the work of two psychiatrists: Giovanni Marro and his father, Antoni

Persistent currents in a strongly interacting multicomponent Bose gas on a ring

We consider a two-component Bose-Bose mixture at strong repulsive interactions in a tightly confining, one-dimensional ring trap and subjected to an artificial gauge field. By employing the Bethe Ansatz exact solution for the many-body wavefunction, we obtain the ground state energy and the persistent currents. For each value of the applied flux, we then determine the symmetry of the state under particles exchange. We find that the ground-state energy and the persistent currents display a reduced periodicity with respect to the case of non-interacting particles, corresponding to reaching states with fractional angular momentum per particle. We relate this effect to the change of symmetry of the ground state under the effect of the artificial gauge field. Our results generalize the ones previously reported for fermionic mixtures with both attractive and repulsive interactions and highlight the role of symmetry in this effectComment: 12 pages, 2 figures, 3 table

Exact solution for SU(2)SU(2)-symmetry breaking bosonic mixtures at strong interactions

We study the equilibrium properties of a one-dimensional mixture of two Tonks-Girardeau gases on a ring geometry in the limit of strongly-repulsive inter-species interactions. We derive the exact many-body wavefunction and compare it to the $SU(2)$ solution where intra- and inter-species interactions are also diverging but equal. We focus on the role of the $SU(2)$-symmetry breaking on the behaviour of the large- and short-distance correlations by studying the zero-momentum occupation number and the Tan's contact from the asymptotic behavior of the momentum distribution. Although the symmetry is only weakly broken, it has important consequences on spin correlations in the system as the reduction by a factor of two of the zero-momentum occupation number with respect to the $SU(2)$ case in the thermodynamic limit and the decrease of the Tan's contact.Comment: 9 pages, 5 figure

MYH9 related disease : a novel missense Ala95Asp mutation of the MYH9 gene

MYH9-related disease (MYH9-RD) is a rare autosomal dominant disorder caused by mutations in MYH9, the gene encoding the heavy chain of non-muscle myosin IIA. Patients present with congenital macrothrombocytopenia and inclusion bodies in neutrophils and might develop sensorineural deafness, presenile cataract, and/or progressive nephritis leading to end-stage renal failure. In a family with eight individuals suffering from macrothrombocytopenia and hearing impairment we identified a novel c.Ala95Asp mutation. Affecting the motor domain of the protein, the mutation is likely to be associated with a severe phenotype. Therefore, this family should be carefully monitored to follow-up the renal status even though the affected members do not seem to be at risk of early kidney disease.Fil: de Rocco, Daniela. Università degli Studi di Trieste; ItaliaFil: Heller, Paula Graciela. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Investigaciones Médicas. Universidad de Buenos Aires. Facultad de Medicina. Instituto de Investigaciones Médicas; ArgentinaFil: Girotto, Giorgia. Università degli Studi di Trieste; ItaliaFil: Pastore, Annalisa. National Institute for Medical Research; Reino UnidoFil: Glembotsky, Ana Claudia. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Investigaciones Médicas. Universidad de Buenos Aires. Facultad de Medicina. Instituto de Investigaciones Médicas; ArgentinaFil: Marta, Rosana Fernanda. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Investigaciones Médicas. Universidad de Buenos Aires. Facultad de Medicina. Instituto de Investigaciones Médicas; ArgentinaFil: Bozzi, Valeria. Universita Degli Studi Di Pavia; ItaliaFil: Pecci, Alessandro. Universita Degli Studi Di Pavia; ItaliaFil: Molinas, Felisa Concepción. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Investigaciones Médicas. Universidad de Buenos Aires. Facultad de Medicina. Instituto de Investigaciones Médicas; ArgentinaFil: Savoia, Anna. Università degli Studi di Trieste; Itali

Dysregulation of oncogenic factors by GFI1B p32: investigation of a novel GFI1B germline mutation

GFI1B is a transcription factor essential for the regulation of erythropoiesis and megakaryopoiesis, and pathogenic variants have been associated with thrombocytopenia and bleeding. Analysing thrombocytopenic families by whole exome sequencing, we identified a novel GFI1B variant (c.648+5G>A), which causes exon 9 skipping and overexpression of a shorter p32 isoform. We report the clinical data of our patients and critically review the phenotype observed in individuals with different GFI1B variants leading to the same effect on the p32 expression. Since p32 is increased in acute and chronic leukemia cells, we tested the expression level of genes playing a role in various type of cancers, including hematological tumors and found that they are significantly dysregulated, suggesting a potential role for GFI1B in carcinogenesis regulation. Increasing the number of individuals with GFI1B variants will allow us to better characterize this rare disease and determine whether it is associated with an increased risk of developing malignancies