Changing a League Structure: Marketing Implications for Rugby League Clubs in the UK

AIM The sport of rugby league in the UK underwent a Policy Review in 2013 that saw the sport move from a licensing system which was first introduced in 2009 to restore a promotion and relegation league system to be implemented in 2015 (RFL, 2015). In its 2015-2021 strategic plan, the Rugby Football League (RFL) outlined its rationale and vision to increase the visibility and profile of the sport in the UK (RFL, 2015). The resulting 2015 season new competition format included a 12 team elite Super League, a 12 team Championship, and a new 14 team League One. The integration of all league competitions through promotion and relegation presented a particular opportunity to increase the interest and attractiveness for rugby league as a sport product in the Championship division. The aim was to explore the impact of the new league structure on the Championship clubs marketing activities to reinvigorate fan interest ahead of the 2015 season

Competitive intensity, fans' expectations and match day tickets sold in the Italian football Serie A, 2012-2015

This article investigates the impact of the competitive intensity on the stadium attendance for Italian soccer in three Serie A seasons (2012-13 to 2014-15). The central element of novelty concerning the existing literature is that fans expectations are also included among the explanatory variables of the Tobit model. Our results show that competitive intensity has a significant impact on match-day attendance in relation to all the sporting prizes but Europa League qualification. Moreover, we find evidence supporting the existence of reference- dependent preferences, where the attendance increases when the home team rank is higher than the pre-season expectations

Altered tumor formation and evolutionary selection of genetic variants in the human MDM4 oncogene

A large body of evidence strongly suggests that the p53 tumor suppressor pathway is central in reducing cancer frequency in vertebrates. The protein product of the haploinsufficient mouse double minute 2 (MDM2) oncogene binds to and inhibits the p53 protein. Recent studies of human genetic variants in p53 and MDM2 have shown that single nucleotide polymorphisms (SNPs) can affect p53 signaling, confer cancer risk, and suggest that the pathway is under evolutionary selective pressure (1–4). In this report, we analyze the haplotype structure of MDM4, a structural homolog of MDM2, in several different human populations. Unusual patterns of linkage disequilibrium (LD) in the haplotype distribution of MDM4 indicate the presence of candidate SNPs that may also modify the efficacy of the p53 pathway. Association studies in 5 different patient populations reveal that these SNPs in MDM4 confer an increased risk for, or early onset of, human breast and ovarian cancers in Ashkenazi Jewish and European cohorts, respectively. This report not only implicates MDM4 as a key regulator of tumorigenesis in the human breast and ovary, but also exploits for the first time evolutionary driven linkage disequilibrium as a means to select SNPs of p53 pathway genes that might be clinically relevant