164 research outputs found
Optimal conclusive teleportation of quantum states
Quantum teleportation of qudits is revisited. In particular, we analyze the
case where the quantum channel corresponds to a non-maximally entangled state
and show that the success of the protocol is directly related to the problem of
distinguishing non-orthogonal quantum states. The teleportation channel can be
seen as a coherent superposition of two channels, one of them being a maximally
entangled state thus, leading to perfect teleportation and the other,
corresponding to a non-maximally entangled state living in a subspace of the
d-dimensional Hilbert space. The second channel leads to a teleported state
with reduced fidelity. We calculate the average fidelity of the process and
show its optimality.Comment: 8 pages, revtex, no figure
Professor - pesquisador de sua prática : dificuldades e motivações
Neste trabalho analisamos três casos de professores de ciências que pesquisaram sua prática docente. Focalizamos os processos de elaboração de suas pesquisas, que resultaram na descoberta de falhas e acertos na condução docente e numa correspondente mudança subjetiva em relação à atividade de ensinar ciências. Os dados empíricos deste trabalho são constituídos pelos diários de pesquisa, memoriais de qualificação e pelas dissertações destes professores, além das entrevistas e conversas informais com os mesmos. Destacamos a insatisfação inicial em relação a sua prática docente, a elaboração do diário de bordo, ocasião de reflexão inicial sobre sua prática e também registro fundamental de pesquisa, a adoção de referenciais para elaborar e analisar os dados e o auxílio de um grupo de interlocutores na academia, formado por colegas e orientadores
Holonomic quantum computation in the presence of decoherence
We present a scheme to study non-abelian adiabatic holonomies for open
Markovian systems. As an application of our framework, we analyze the
robustness of holonomic quantum computation against decoherence. We pinpoint
the sources of error that must be corrected to achieve a geometric
implementation of quantum computation completely resilient to Markovian
decoherence.Comment: I. F-G. Now publishes under name I. Fuentes-Schuller Published
versio
Spin-1/2 geometric phase driven by decohering quantum fields
We calculate the geometric phase of a spin-1/2 system driven by a one and two
mode quantum field subject to decoherence. Using the quantum jump approach, we
show that the corrections to the phase in the no-jump trajectory are different
when considering an adiabatic and non-adiabatic evolution. We discuss the
implications of our results from both the fundamental as well as quantum
computational perspective.Comment: 4 page
Vacuum induced Spin-1/2 Berry phase
We calculate the Berry phase of a spin-1/2 particle in a magnetic field considering the quantum nature of the field. The phase reduces to the standard Berry phase in the semiclassical limit. An eigenstate of the particle acquires a phase in the vacuum which is undetectable. We show how to generate a vacuum induced Berry phase which can be detected considering two quantized modes of the field
Antioxidant peroxiredoxin 6 protein rescues toxicity due to oxidative stress and cellular hypoxia in vitro, and attenuates prion-related pathology in vivo
Protein misfolding, mitochondrial dysfunction and oxidative stress are common pathomechanisms that underlie neurodegenerative diseases. In prion disease, central to these processes is the post-translational transformation of cellular prion protein (PrPc) to the aberrant conformationally altered isoform; PrPSc. This can trigger oxidative reactions and impair mitochondrial function by increasing levels of peroxynitrite, causing damage through formation of hydroxyl radicals or via nitration of tyrosine residues on proteins. The 6 member Peroxiredoxin (Prdx) family of redox proteins are thought to be critical protectors against oxidative stress via reduction of H2O2, hydroperoxides and peroxynitrite. In our in vitro studies cellular metabolism of SK-N-SH human neuroblastoma cells was significantly decreased in the presence of H2O2 (oxidative stressor) or CoCl2 (cellular hypoxia), but was rescued by treatment with exogenous Prdx6, suggesting that its protective action is in part mediated through a direct action. We also show that CoCl2-induced apoptosis was significantly decreased by treatment with exogenous Prdx6. We proposed a redox regulator role for Prdx6 in regulating and maintaining cellular homeostasis via its ability to control ROS levels that could otherwise accelerate the emergence of prion-related neuropathology. To confirm this, we established prion disease in mice with and without astrocyte-specific antioxidant protein Prdx6, and demonstrated that expression of Prdx6 protein in Prdx6 Tg ME7-animals reduced severity of the behavioural deficit, decreased neuropathology and increased survival time compared to Prdx6 KO ME7-animals. We conclude that antioxidant Prdx6 attenuates prion-related neuropathology, and propose that augmentation of endogenous Prdx6 protein represents an attractive adjunct therapeutic approach for neurodegenerative diseases
Alterations to mTORC1 signaling in the skeletal muscle differentially affect whole-body metabolism
The mammalian target of rapamycin complex 1 (mTORC1) is a central node in a network of signaling pathways controlling cell growth and survival. This multiprotein complex integrates external signals and affects different nutrient pathways in various organs. However, it is not clear how alterations of mTORC1 signaling in skeletal muscle affect whole-body metabolism.; We characterized the metabolic phenotype of young and old raptor muscle knock-out (RAmKO) and TSC1 muscle knock-out (TSCmKO) mice, where mTORC1 activity in skeletal muscle is inhibited or constitutively activated, respectively. Ten-week-old RAmKO mice are lean and insulin resistant with increased energy expenditure, and they are resistant to a high-fat diet (HFD). This correlates with an increased expression of histone deacetylases (HDACs) and a downregulation of genes involved in glucose and fatty acid metabolism. Ten-week-old TSCmKO mice are also lean, glucose intolerant with a decreased activation of protein kinase B (Akt/PKB) targets that regulate glucose transporters in the muscle. The mice are resistant to a HFD and show reduced accumulation of glycogen and lipids in the liver. Both mouse models suffer from a myopathy with age, with reduced fat and lean mass, and both RAmKO and TSCmKO mice develop insulin resistance and increased intramyocellular lipid content.; Our study shows that alterations of mTORC1 signaling in the skeletal muscle differentially affect whole-body metabolism. While both inhibition and constitutive activation of mTORC1 induce leanness and resistance to obesity, changes in the metabolism of muscle and peripheral organs are distinct. These results indicate that a balanced mTORC1 signaling in the muscle is required for proper metabolic homeostasis
Amaya
De cada obra s'ha digitalitzat un programa sencer. De la resta s'han digitalitzat les parts que són diferents.Empresa Juan A. PamiasÒpera de Jeús Guridi amb llibret de José María Arroita Jáuregu
Geometric phase in open systems
We calculate the geometric phase associated to the evolution of a system
subjected to decoherence through a quantum-jump approach. The method is general
and can be applied to many different physical systems. As examples, two main
source of decoherence are considered: dephasing and spontaneous decay. We show
that the geometric phase is completely insensitive to the former, i.e. it is
independent of the number of jumps determined by the dephasing operator.Comment: 4 pages, 2 figures, RevTe
Topological Features in Ion Trap Holonomic Computation
Topological features in quantum computing provide controllability and noise
error avoidance in the performance of logical gates. While such resilience is
favored in the manipulation of quantum systems, it is very hard to identify
topological features in nature. This paper proposes a scheme where holonomic
quantum gates have intrinsic topological features. An ion trap is employed
where the vibrational modes of the ions are coherently manipulated with lasers
in an adiabatic cyclic way producing geometrical holonomic gates. A crucial
ingredient of the manipulation procedures is squeezing of the vibrational
modes, which effectively suppresses exponentially any undesired fluctuations of
the laser amplitudes, thus making the gates resilient to control errors.Comment: 9 pages, 4 figures, REVTE
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