Optimal conclusive teleportation of quantum states

Quantum teleportation of qudits is revisited. In particular, we analyze the case where the quantum channel corresponds to a non-maximally entangled state and show that the success of the protocol is directly related to the problem of distinguishing non-orthogonal quantum states. The teleportation channel can be seen as a coherent superposition of two channels, one of them being a maximally entangled state thus, leading to perfect teleportation and the other, corresponding to a non-maximally entangled state living in a subspace of the d-dimensional Hilbert space. The second channel leads to a teleported state with reduced fidelity. We calculate the average fidelity of the process and show its optimality.Comment: 8 pages, revtex, no figure

Professor - pesquisador de sua prática : dificuldades e motivações

Neste trabalho analisamos três casos de professores de ciências que pesquisaram sua prática docente. Focalizamos os processos de elaboração de suas pesquisas, que resultaram na descoberta de falhas e acertos na condução docente e numa correspondente mudança subjetiva em relação à atividade de ensinar ciências. Os dados empíricos deste trabalho são constituídos pelos diários de pesquisa, memoriais de qualificação e pelas dissertações destes professores, além das entrevistas e conversas informais com os mesmos. Destacamos a insatisfação inicial em relação a sua prática docente, a elaboração do diário de bordo, ocasião de reflexão inicial sobre sua prática e também registro fundamental de pesquisa, a adoção de referenciais para elaborar e analisar os dados e o auxílio de um grupo de interlocutores na academia, formado por colegas e orientadores

Holonomic quantum computation in the presence of decoherence

We present a scheme to study non-abelian adiabatic holonomies for open Markovian systems. As an application of our framework, we analyze the robustness of holonomic quantum computation against decoherence. We pinpoint the sources of error that must be corrected to achieve a geometric implementation of quantum computation completely resilient to Markovian decoherence.Comment: I. F-G. Now publishes under name I. Fuentes-Schuller Published versio

Spin-1/2 geometric phase driven by decohering quantum fields

We calculate the geometric phase of a spin-1/2 system driven by a one and two mode quantum field subject to decoherence. Using the quantum jump approach, we show that the corrections to the phase in the no-jump trajectory are different when considering an adiabatic and non-adiabatic evolution. We discuss the implications of our results from both the fundamental as well as quantum computational perspective.Comment: 4 page

Vacuum induced Spin-1/2 Berry phase

We calculate the Berry phase of a spin-1/2 particle in a magnetic field considering the quantum nature of the field. The phase reduces to the standard Berry phase in the semiclassical limit. An eigenstate of the particle acquires a phase in the vacuum which is undetectable. We show how to generate a vacuum induced Berry phase which can be detected considering two quantized modes of the field

Antioxidant peroxiredoxin 6 protein rescues toxicity due to oxidative stress and cellular hypoxia in vitro, and attenuates prion-related pathology in vivo

Protein misfolding, mitochondrial dysfunction and oxidative stress are common pathomechanisms that underlie neurodegenerative diseases. In prion disease, central to these processes is the post-translational transformation of cellular prion protein (PrPc) to the aberrant conformationally altered isoform; PrPSc. This can trigger oxidative reactions and impair mitochondrial function by increasing levels of peroxynitrite, causing damage through formation of hydroxyl radicals or via nitration of tyrosine residues on proteins. The 6 member Peroxiredoxin (Prdx) family of redox proteins are thought to be critical protectors against oxidative stress via reduction of H2O2, hydroperoxides and peroxynitrite. In our in vitro studies cellular metabolism of SK-N-SH human neuroblastoma cells was significantly decreased in the presence of H2O2 (oxidative stressor) or CoCl2 (cellular hypoxia), but was rescued by treatment with exogenous Prdx6, suggesting that its protective action is in part mediated through a direct action. We also show that CoCl2-induced apoptosis was significantly decreased by treatment with exogenous Prdx6. We proposed a redox regulator role for Prdx6 in regulating and maintaining cellular homeostasis via its ability to control ROS levels that could otherwise accelerate the emergence of prion-related neuropathology. To confirm this, we established prion disease in mice with and without astrocyte-specific antioxidant protein Prdx6, and demonstrated that expression of Prdx6 protein in Prdx6 Tg ME7-animals reduced severity of the behavioural deficit, decreased neuropathology and increased survival time compared to Prdx6 KO ME7-animals. We conclude that antioxidant Prdx6 attenuates prion-related neuropathology, and propose that augmentation of endogenous Prdx6 protein represents an attractive adjunct therapeutic approach for neurodegenerative diseases

Alterations to mTORC1 signaling in the skeletal muscle differentially affect whole-body metabolism

The mammalian target of rapamycin complex 1 (mTORC1) is a central node in a network of signaling pathways controlling cell growth and survival. This multiprotein complex integrates external signals and affects different nutrient pathways in various organs. However, it is not clear how alterations of mTORC1 signaling in skeletal muscle affect whole-body metabolism.; We characterized the metabolic phenotype of young and old raptor muscle knock-out (RAmKO) and TSC1 muscle knock-out (TSCmKO) mice, where mTORC1 activity in skeletal muscle is inhibited or constitutively activated, respectively. Ten-week-old RAmKO mice are lean and insulin resistant with increased energy expenditure, and they are resistant to a high-fat diet (HFD). This correlates with an increased expression of histone deacetylases (HDACs) and a downregulation of genes involved in glucose and fatty acid metabolism. Ten-week-old TSCmKO mice are also lean, glucose intolerant with a decreased activation of protein kinase B (Akt/PKB) targets that regulate glucose transporters in the muscle. The mice are resistant to a HFD and show reduced accumulation of glycogen and lipids in the liver. Both mouse models suffer from a myopathy with age, with reduced fat and lean mass, and both RAmKO and TSCmKO mice develop insulin resistance and increased intramyocellular lipid content.; Our study shows that alterations of mTORC1 signaling in the skeletal muscle differentially affect whole-body metabolism. While both inhibition and constitutive activation of mTORC1 induce leanness and resistance to obesity, changes in the metabolism of muscle and peripheral organs are distinct. These results indicate that a balanced mTORC1 signaling in the muscle is required for proper metabolic homeostasis

Amaya

De cada obra s'ha digitalitzat un programa sencer. De la resta s'han digitalitzat les parts que són diferents.Empresa Juan A. PamiasÒpera de Jeús Guridi amb llibret de José María Arroita Jáuregu

Geometric phase in open systems

We calculate the geometric phase associated to the evolution of a system subjected to decoherence through a quantum-jump approach. The method is general and can be applied to many different physical systems. As examples, two main source of decoherence are considered: dephasing and spontaneous decay. We show that the geometric phase is completely insensitive to the former, i.e. it is independent of the number of jumps determined by the dephasing operator.Comment: 4 pages, 2 figures, RevTe

Topological Features in Ion Trap Holonomic Computation

Topological features in quantum computing provide controllability and noise error avoidance in the performance of logical gates. While such resilience is favored in the manipulation of quantum systems, it is very hard to identify topological features in nature. This paper proposes a scheme where holonomic quantum gates have intrinsic topological features. An ion trap is employed where the vibrational modes of the ions are coherently manipulated with lasers in an adiabatic cyclic way producing geometrical holonomic gates. A crucial ingredient of the manipulation procedures is squeezing of the vibrational modes, which effectively suppresses exponentially any undesired fluctuations of the laser amplitudes, thus making the gates resilient to control errors.Comment: 9 pages, 4 figures, REVTE