海外の保育方法と日本の保育との融合に関する研究 : レッジョ・エミリア・アプローチの日本での融合に着目して<教育科学>

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Observation of B→K*ℓ+ℓ-

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中国的经济増长與就业―构建灵活安全的劳动力市场

2009-03This paper includes four sections. Section one mainly answers the question whether Chinese rapid economic growth has created enough employment or not, and concludes that the economic growth does create as many work opportunities as it should do especially in the early stage of the reform based on the analysis of aggregate data on economic growth and employment. Section two presents a general picture of update trends in labor demand and supply in China, and concludes that unlimited supply of labors seems being departing from China, but this does not necessarily mean that China has entered into the era of labor shortage, but it seems that there is enough supply of labor in next 20 years in China. Section three discusses major problems facing Chinese labor markets, such problems as the structural problems of employment, the trend of informalization of employment, and the problem of distorted primary income distribution have been discussed. Section four concludes with the argument that a good functioned labor market must be able to combine flexibility with security in pursuit of balance between economic growth and employment.本文共分4 部分。运用宏观统计数据、文章首先回答了中国经济増长是否想像就业问题、通货分阶段分析经济増长对就业的拉动效应、认为中国经济増长是大量创造就业的増长、而不是人认为的无就业増长；第二部分分析劳动供求关系的最新变化及其演变趋势、认为劳动供求关系的正在发生根本本性转变、但中国尚没有歩入劳动力短缺时代、在今后20 年左右的时间仍然拥有充足的劳动供给；第三部分讨论当前劳働力市场面临的突出问题、认为劳动供求总量矛盾有所减轻、但存在着多更复杂的矛盾、比较突出的问题包括三个、一是就业结构性矛盾突出、二是就业非正规化严重、三是初次収入分配扭曲；第四部分为结论，认为要寻求经济増长和就业之间的平衡、需要构建既灵活又安全的劳动力市场。departmental bulletin pape

インターネット ニ オケル ジツクウカン ジョウホウ ノ リュウツウ セイギョ ニ カンスル ケンキュウ

奈良先端科学技術大学院大学博士(工学)doctoral thesi

Forskolin enhances PMA-dependent CD86 expression while inhibiting CD11b up-regulation and CaMKKα nuclear localization.

<p>(A and B) U937 cells were pretreated with or without 30 µM forskolin for 1 h prior to activation with 100 nM PMA for 48 h, as indicated. Surface expression of CD11b and CD86 was quantified by flow cytometry using FITC-conjugated CD11b and PE-conjugated CD86 antibodies. Results significantly different from PMA at α = 0.05 are indicated by (*). Results represent an average of three independent experiments±SEM. (C) Cells were treated as in panel A and CaMKKα was quantified by Western analysis of nuclear lysates. Results are representative of three independent experiments. (D) U937 cells were treated as in panel A and ERK1/2 phosphorylation (ERK-p) and mass (ERK) were measured by Western analysis in whole cell lysates. Results are representative of three independent experiments.</p

大山山麓の鹹味ある鉱物について

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Phagocytosis and fluid-phase endocytosis are not increased in PerMΦs from <i>db/db</i> mice.

<p><i>A</i>, <i>Db</i>/+and <i>db/db</i> mice were IP administered 0.21 or 2.6 µm latex microspheres (beads) at 5×10⁹ or 5×10⁷ beads/ml, respectively. Following a 4 h <i>in vivo</i> incubation period, PerMΦs were collected and microsphere uptake was measured by flow cytometry. <i>B, Db</i>/+and <i>db/db</i> mice were IP administered 0.25 mg/ml FITC-dextran. Following a 4 h <i>in vivo</i> incubation period, PerMΦs were collected and dextran uptake was measured by flow cytometry. Results for <i>A</i> and <i>B</i> represent the average of three independent experiments±SEM.</p

Inhibition of CaMKKα by siRNA enhances PMA-dependent up-regulation of CD86 but decreases CD11b expression.

<p>(A and B) Cells were treated with 0.25 µg of either CaMKKα siRNA or scramble siRNA control prior to stimulation with 100 nM PMA for 48 h. Surface expression of CD11b and CD86 was quantified by flow cytometry. Results significantly different from PMA at α = 0.05 are indicated by (*). Results represent an average of three independent experiments±SEM. (C) Cells were treated as in A and CaMKKα protein expression was quantified by intracellular flow. Results significantly different from control at α = 0.05 are indicated by (*). Results represent an average of three independent experiments±SEM.</p

<i>Db/db</i> mice have elevated blood glucose and serum insulin levels.

<p>Fasting blood glucose (FBG), random blood glucose (RBG), fasting serum insulin (FSI), and random serum insulin (RSI). Results represent the average of n = 5 mice±SEM. *: p<0.05.</p

The PKC inhibitor Bisindolylmaleimide inhibits PMA-dependent upregulation of CD86/CD11b expression.

<p>(A & B) U937 cells were pretreated with or without 1 µM bisindolylmaleimide for 15 min prior to activation with 100 nM PMA for 48 h, as indicated. Surface expression of CD11b and CD86 was quantified by flow cytometry using FITC-conjugated CD11b and PE-conjugated CD86 antibodies. Results significantly different from PMA at α = 0.05 are indicated by (*). Results represent an average of three independent experiments±SEM.</p