67 research outputs found
Gene expression profiling of breast cancer survivability by pooled cDNA microarray analysis using logistic regression, artificial neural networks and decision trees
- Author
- Publication venue
- Springer Nature
- Publication date
- 01/01/2013
- Field of study
BACKGROUND: Microarray technology can acquire information about thousands of genes simultaneously. We analyzed published breast cancer microarray databases to predict five-year recurrence and compared the performance of three data mining algorithms of artificial neural networks (ANN), decision trees (DT) and logistic regression (LR) and two composite models of DT-ANN and DT-LR. The collection of microarray datasets from the Gene Expression Omnibus, four breast cancer datasets were pooled for predicting five-year breast cancer relapse. After data compilation, 757 subjects, 5 clinical variables and 13,452 genetic variables were aggregated. The bootstrap method, Mann–Whitney U test and 20-fold cross-validation were performed to investigate candidate genes with 100 most-significant p-values. The predictive powers of DT, LR and ANN models were assessed using accuracy and the area under ROC curve. The associated genes were evaluated using Cox regression. RESULTS: The DT models exhibited the lowest predictive power and the poorest extrapolation when applied to the test samples. The ANN models displayed the best predictive power and showed the best extrapolation. The 21 most-associated genes, as determined by integration of each model, were analyzed using Cox regression with a 3.53-fold (95% CI: 2.24-5.58) increased risk of breast cancer five-year recurrence… CONCLUSIONS: The 21 selected genes can predict breast cancer recurrence. Among these genes, CCNB1, PLK1 and TOP2A are in the cell cycle G2/M DNA damage checkpoint pathway. Oncologists can offer the genetic information for patients when understanding the gene expression profiles on breast cancer recurrence
Review of nanomaterials in dentistry: interactions with the oral microenvironment, clinical applications, hazards, and benefits.
- Author
- Ahn S. J.
- Aitken R. J.
- Al-Hamdan K.
- Al-Hezaimi K.
- Al-Jubory A. R.
- Alexandros Besinis
- Allaker R. P.
- Ashwood P.
- Atai M.
- Attar N.
- Atuma C.
- Aubin-Tam M. E.
- Aydin Sevinc B.
- Berges M. G. M.
- Bernardo M.
- Bertazzo S.
- Besinis A.
- Besinis A.
- Besinis A.
- Besinis A.
- Beyth N.
- Bloom W.
- Bosshardt D. D.
- Bouwmeester H.
- Bradford A.
- Breding K.
- Brune D.
- Bucci-Sabattini V.
- Cai Y.
- Campos M. S.
- Canche-Escamilla G.
- Carvalho S. M.
- Casagrande L.
- Cedervall T.
- Chandra S.
- Chaudhry Q.
- Chen M. H.
- Chen Z.
- Cheng L.
- Chiang Y. C.
- Christopher J. Tredwin
- Chung I. S.
- Coelho P. G.
- Collins L. M.
- Cousins B. G.
- Cox M.
- Cramer N. B.
- Cummins D.
- Cummins D.
- Dabas V. K.
- Dabbagh A.
- Dawes C.
- Diekwisch T. G.
- Dobrovolskaia M. A.
- Donaldson K.
- Donaldson K.
- Earl J. S.
- Earl J. S.
- Edgar W. M.
- Einstein A.
- Elsaka S. E.
- Engeman C. D.
- Ferracane J. L.
- Forsback A. P.
- Fraser T. W.
- Gaikwad R. M.
- Garberoglio R.
- Gardin C.
- Gebauer J. S.
- Gerzina T. M.
- Geurtsen W.
- Gitrowski C.
- Goene R. J.
- Goldberg M.
- Greish K.
- Grine F. E.
- Gupta S.
- Hamlet S.
- Handy R. D.
- Handy R. D.
- Handy R. D.
- Handy R. D.
- Handy R. D.
- Hannig C.
- Hannig C.
- Hannig M.
- Hannig M.
- Hannig M.
- Hara A. T.
- Harris D.
- Hayami T.
- He F.
- Helland A.
- Hellstrand E.
- Hernandez-Sierra J. F.
- Hernández d. l. F.
- Hervas-Garcia A.
- Hook E. R.
- Hoppenbrouwers P. M.
- Hosseinalipour M.
- Huang H.-L.
- Hunt G.
- Iavicoli I.
- Imazato S.
- Jani P. U.
- Jeong S. H.
- Jeong S. H.
- Jiang J.
- Jiang X.
- Ju-Nam Y.
- Juillerat-Jeanneret L.
- Kasraei S.
- Kerebel B.
- Kim J. J.
- Kinney J. H.
- Kishen A.
- Kleinberg I.
- Kodaka T.
- Koeneman B. A.
- Kralj S.
- Kuhlbusch T. A.
- Kumar P. T.
- Kwon S. R.
- Lai S. K.
- Lang B. R.
- Lang N. P.
- Lead J. R.
- Lee H.-H.
- Lees S.
- Lees S.
- Lendenmann U.
- Levine M. J.
- Li L.
- Lin A.
- Llorens A.
- Loberg J.
- Loguercio A. D.
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- Lomer M. C.
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- Lu K. L.
- Lundqvist M.
- Lutz F.
- Lv K. L.
- Ma P.-C.
- Maiorano G.
- Mankani M. H.
- Markowitz K.
- Marshall G. W.
- Meirelles L.
- Melo M. A.
- Mendes V. C.
- Merrifield D. L.
- Minkoff S.
- Mistry S.
- Mitsiadis T. A.
- Mjor I. A.
- Morones J. R.
- Mortazavi V.
- Mota E. G.
- Moy P. K.
- Nakashima S.
- Nam K. Y.
- Namiki Y.
- Nguyen D. H.
- Nguyen T. T.
- Olea N.
- Olyveira G. M.
- O’Sullivan E. A.
- Pablico-Lansigan M. H.
- Pal S.
- Park S.
- Pedersen A. M.
- Petri-Fink A.
- Piesco N. P.
- Piesco N. P.
- Poland C. A.
- Powell J. J.
- Prabhakar P. V.
- Prentice L. H.
- Proctor G. B.
- Quaranta A.
- Radice S.
- Ramsden C. S.
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- Reynolds E. C.
- Rezwan K.
- Richard D. Handy
- Rocks S. A.
- Rolla G.
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- Ruparelia J. P.
- Ruttermann S.
- Sadat-Shojai M.
- Sadeghi M.
- Sadeghi M.
- Salata O.
- Sarrett D. C.
- Sasaki T.
- Sasani N.
- Schenkels L. C.
- Schouten C.
- Schrand A. M.
- Schulte P. A.
- Schulz H.
- Sekiguchi Y.
- Sharma A. K.
- Sharma V.
- Shi J.
- Shi Z.
- Shrestha A.
- Shrivastava R.
- Shvero D. K.
- Silva G. A.
- Smith A. J.
- Song Y.
- Stookey G. K.
- Sun L. M.
- Suwanboon S.
- Svanborg L. M.
- Tay F. R.
- Tay F. R.
- Tayel A. A.
- Tayles N.
- Telleman G.
- Temple D. H.
- Terry D. A.
- Thurnheer T.
- Tiede K.
- Tracy De Peralta
- Travan A.
- Trouiller B.
- Tschoppe P.
- Tye C. E.
- Ubaldini A. L.
- Uchida A.
- van der Weijden F.
- van der Zande M.
- van Meerbeek B.
- Verran J.
- Vidic J.
- Vollenweider M.
- von der Kammer F.
- Wagner A.
- Waltimo T.
- Wang J.
- Wang J.
- Warheit D. B.
- Weatherell J. A.
- Webster T. J.
- Webster T. J.
- Weir A.
- Welch K.
- Wennerberg A.
- Whelton H.
- Windeatt K. M.
- Xu H. H.
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- Yang G. L.
- Yang K.
- Yang L.
- Yao Y.
- Yip K. H.
- Zahradnik R. T.
- Zappa U.
- Zhang F.
- Zhang L.
- Zhang R.
- Publication venue
- 'American Chemical Society (ACS)'
- Publication date
- 24/03/2015
- Field of study
Interest in the use of engineered nanomaterials (ENMs) as either nanomedicines or dental materials/devices in clinical dentistry is growing. This review aims to detail the ultrafine structure, chemical composition, and reactivity of dental tissues in the context of interactions with ENMs, including the saliva, pellicle layer, and oral biofilm; then describes the applications of ENMs in dentistry in context with beneficial clinical outcomes versus potential risks. The flow rate and quality of saliva are likely to influence the behavior of ENMs in the oral cavity, but how the protein corona formed on the ENMs will alter bioavailability, or interact with the structure and proteins of the pellicle layer, as well as microbes in the biofilm, remains unclear. The tooth enamel is a dense crystalline structure that is likely to act as a barrier to ENM penetration, but underlying dentinal tubules are not. Consequently, ENMs may be used to strengthen dentine or regenerate pulp tissue. ENMs have dental applications as antibacterials for infection control, as nanofillers to improve the mechanical and bioactive properties of restoration materials, and as novel coatings on dental implants. Dentifrices and some related personal care products are already available for oral health applications. Overall, the clinical benefits generally outweigh the hazards of using ENMs in the oral cavity, and the latter should not prevent the responsible innovation of nanotechnology in dentistry. However, the clinical safety regulations for dental materials have not been specifically updated for ENMs, and some guidance on occupational health for practitioners is also needed. Knowledge gaps for future research include the formation of protein corona in the oral cavity, ENM diffusion through clinically relevant biofilms, and mechanistic investigations on how ENMs strengthen the tooth structure
Impact of primary kidney disease on the effects of empagliflozin in patients with chronic kidney disease: secondary analyses of the EMPA-KIDNEY trial
- Author
- Abat S.
- Abd Rahman R.
- Abdul Cader R.
- Abdul Hafidz M. I.
- Abdul Wahab M. Z.
- Abdul-Samad T.
- Abdullah N. K.
- Abe M.
- Abraham N.
- Acheampong S.
- Achiri P.
- Acosta J. A.
- Adeleke A.
- Adell V.
- Adewuyi-Dalton R.
- Adnan N.
- Africano A.
- Agharazii M.
- Aguilar F.
- Aguilera A.
- Ahmad M.
- Ahmad M. K.
- Ahmad Miswan N.
- Ahmad N. A.
- Ahmad N. H.
- Ahmad N. I.
- Ahmad Rosdi H.
- Ahmed I.
- Ahmed S.
- Aiello J.
- Aitken A.
- AitSadi R.
- Aker S.
- Akimoto S.
- Akinfolarin A.
- Akram S.
- Al-Rabadi L.
- Al-Zeer B.
- Alberici F.
- Albert C.
- Aldrich L.
- Alegata M.
- Alexander L.
- Alfaress S.
- Alhadj Ali M.
- Ali A.
- Alicic R.
- Aliu A.
- Almaraz R.
- Almasarwah R.
- Almeida J.
- Aloisi A.
- Alscher D.
- Alvarez P.
- Amat M.
- Ambrose C.
- Ammar H.
- An Y.
- Andriaccio L.
- Ansu K.
- Apostolidi A.
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- Arbi A.
- Arechiga O.
- Armstrong S.
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- Arriaga W.
- Arroyo J.
- Arteaga D.
- Asahara S.
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- Awad A.
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- Axler A.
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- Publication venue
- Publication date
- 01/01/2024
- Field of study
Background: The EMPA KIDNEY trial showed that empagliflozin reduced the risk of the primary composite outcome of kidney disease progression or cardiovascular death in patients with chronic kidney disease mainly through slowing progression. We aimed to assess how effects of empagliflozin might differ by primary kidney disease across its broad population. Methods: EMPA-KIDNEY, a randomised, controlled, phase 3 trial, was conducted at 241 centres in eight countries (Canada, China, Germany, Italy, Japan, Malaysia, the UK, and the USA). Patients were eligible if their estimated glomerular filtration rate (eGFR) was 20 to less than 45 mL/min per 1·73 m2, or 45 to less than 90 mL/min per 1·73 m2 with a urinary albumin-to-creatinine ratio (uACR) of 200 mg/g or higher at screening. They were randomly assigned (1:1) to 10 mg oral empagliflozin once daily or matching placebo. Effects on kidney disease progression (defined as a sustained ≥40% eGFR decline from randomisation, end-stage kidney disease, a sustained eGFR below 10 mL/min per 1·73 m2, or death from kidney failure) were assessed using prespecified Cox models, and eGFR slope analyses used shared parameter models. Subgroup comparisons were performed by including relevant interaction terms in models. EMPA-KIDNEY is registered with ClinicalTrials.gov, NCT03594110. Findings: Between May 15, 2019, and April 16, 2021, 6609 participants were randomly assigned and followed up for a median of 2·0 years (IQR 1·5–2·4). Prespecified subgroupings by primary kidney disease included 2057 (31·1%) participants with diabetic kidney disease, 1669 (25·3%) with glomerular disease, 1445 (21·9%) with hypertensive or renovascular disease, and 1438 (21·8%) with other or unknown causes. Kidney disease progression occurred in 384 (11·6%) of 3304 patients in the empagliflozin group and 504 (15·2%) of 3305 patients in the placebo group (hazard ratio 0·71 [95% CI 0·62–0·81]), with no evidence that the relative effect size varied significantly by primary kidney disease (pheterogeneity=0·62). The between-group difference in chronic eGFR slopes (ie, from 2 months to final follow-up) was 1·37 mL/min per 1·73 m2 per year (95% CI 1·16–1·59), representing a 50% (42–58) reduction in the rate of chronic eGFR decline. This relative effect of empagliflozin on chronic eGFR slope was similar in analyses by different primary kidney diseases, including in explorations by type of glomerular disease and diabetes (p values for heterogeneity all >0·1). Interpretation: In a broad range of patients with chronic kidney disease at risk of progression, including a wide range of non-diabetic causes of chronic kidney disease, empagliflozin reduced risk of kidney disease progression. Relative effect sizes were broadly similar irrespective of the cause of primary kidney disease, suggesting that SGLT2 inhibitors should be part of a standard of care to minimise risk of kidney failure in chronic kidney disease. Funding: Boehringer Ingelheim, Eli Lilly, and UK Medical Research Council
ICAR: endoscopic skull‐base surgery
- Author
- Aaron G
- Aaron GP
- Abergel A
- Abouaf L
- Abrishamkar S
- Acioly MA
- Acioly MA
- Acuna RT
- Adams AS
- Adappa ND
- Aferzon M
- Agaimy A
- Agarwal A
- Aggarwal A
- Agrawal D
- Ahmed OH
- Aihara M
- Akagami R
- Alahmadi H
- Albu S
- Albu S
- Albu S
- Alexander A
- Alimohamadi M
- Almefty R
- Almeida JR
- Almeida JR
- Almeida JR
- Almeida JR
- Almeida JR
- Almeida JR
- Almeida JR
- Almond MC
- Alobaid A
- Alobid I
- Alobid I
- Alos L
- AlQahtani A
- Altay T
- Alvarez FL
- Alves MV
- Al‐Mamgani A
- Al‐Mefty O
- Al‐Sheibani S
- Amano K
- Amdur RJ
- Amelot A
- American Academy of Pediatrics Steering Committee on Quality Improvement and Management
- Amichetti M
- Amin SM
- Amit M
- Amit M
- Amit M
- Ammirati M
- Anari S
- Anderson D
- Anderson RL
- Andrade NA
- Andrews JC
- Anik I
- Ansa B
- Ansari M
- Antonelli AR
- Arafah BM
- Arafah BM
- Arafah BM.
- Arai A
- Arat YO
- Arbolay OL
- Ardehali MM
- Armengot M
- Arnautovic KI
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- Wang YY
- Wanibuchi M
- Wannemuehler TJ
- Webb SM
- Weber AL
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- Weidenbecher M
- Weiland DA
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- Wood JW
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- Wormald PJ
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- Zanation AM
- Zhan R
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- Publication venue
- Publication date
- 01/07/2019
- Field of study
n/
Use of anticoagulants and antiplatelet agents in stable outpatients with coronary artery disease and atrial fibrillation. International CLARIFY registry
- Author
- A. L. R. Ng
- Abardia Oliva F. J.
- Abdel Malak S.
- Abdel Wahab H.
- Abdul Kareem B. B.
- Abdul Manap H.
- Abdul Rahim A. A.
- Abdulkader F.
- Abdulla A. H. K.
- AbdulWahab M. Z.
- Abduvalieva V.
- Abele S.
- Aboyans V.
- Abreu P. E.
- Adam-Blanpain M.
- Adamaszek K.
- Adamczyk-Kot D.
- Adamkiewicz-Piejko A.
- Adda M.
- Aftab A.
- Agrawal A.
- Aguiar C.
- Ahuad Guerrero A.
- Ahuja R.
- Ai F.
- Aimouch N.
- Aiyegbayo O.
- Ajani A.
- Akbar M.
- Aksyutina N.
- Al Dhanki M.
- Al Ghool S.
- Al Kandari F.
- Al Lawati A.
- Al Mahmeed W.
- Al Radaideh G.
- Al Rashdan I.
- Al Sousi A.
- Al Suwaidi J.
- Al Tamimi F.
- Al Wahshi Y.
- Al Yazeedi J.
- Al Zaibag M.
- Al-Baker H.
- Al-Faleh H.
- Al-Hersi A.
- Al-Khalidi B.
- Al-Shamiri M.
- Al-Zaibag M.
- Alam A.
- Alanazy M.
- Alanbaei M.
- Albero Martinez V.
- Alberto Ramirez Reyes H.
- Albuquerque A.
- Alcaravela J.
- Alcocer Gamba M.
- Alegret Colomer J. M.
- Alegria Ezquerra E.
- Alekseenko V.
- Alekseeva T.
- Alexanderson E.
- Alexopoulos I.
- Alford K.
- Alitto F.
- Alizon F.
- Allcock A.
- Almeida Fernandez C. A.
- Altevogt B. -M.
- Altmann U.
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- Alvarez Aunon A.
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- Alvarez Gil J.
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- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2015
- Field of study
International Consensus Statement on Rhinology and Allergy: Rhinosinusitis
- Author
- Aanaes K
- Abadie WM
- Abi Najm S
- Abigail P
- Abrahams JJ
- Abrass LJ
- Abreu CB
- Abreu NA
- Abshirini H
- Abuzeid WM
- Abuzeid WM
- Achar P
- Ackerhans M
- Adam P
- Adappa ND
- Adappa ND
- Adappa ND
- Adappa ND
- Adriaensen G
- Agache I
- Aghamohammadi A
- Agius AM
- Agyeman AA
- Ahn CN
- Ahn HJ
- Ahn JC
- Ahn SH
- Ahovuo‐Saloranta A
- Aitken M
- Ajaiyeoba A
- Akbari E
- Akdis CA
- Akira S
- Akiyama K
- Akiyama K
- Akiyama K
- Al Komser MK
- Alam ES
- Alandejani T
- Alanin MC
- Albritton FDT
- Albu S
- Albu S
- Albu S
- Aldini G
- Alexander NS
- Aljebab F
- Alkire BC
- Allen EK
- Alobid I
- Alobid I
- Alobid I
- Aloulah M
- Alqudah M
- Alromaih S
- Alsaleh S
- Alsharif S
- Alt JA
- Alt JA
- Alt JA
- Alt JA
- Alt JA
- Alves MP
- Al‐Qudah M
- Al‐Qudah M.
- Al‐Qudah M.
- Al‐Rawi MM
- Al‐Shaikh S
- Al‐Shemari H
- Al‐Swiahb JN
- Amali A
- Amar YG
- Amble FR
- American Academy of Pediatrics Steering Committee on Quality Improvement and Management (AAP SCQIM)
- Amin MR
- Amine M
- Amodu EJ
- Anamika A
- Anand V
- Anderson DE
- Anderson WC
- Andhale S
- Ankichetty SP
- Annamalai S
- Anon JB
- Anselmo‐Lima WT
- Antisdel JL
- Antisdel JL
- Antunes MB
- Antunes MB
- Anushiravani M
- Anzai Y
- Anzić SA
- Aramani A
- Arden KE
- Arild Danielsen K
- Aring AM
- Arjomandi H
- Armenaka M
- Arnstead N
- Arslan H
- Arslan HH
- Asaka D
- Asero R
- Asero R
- Atighechi S
- Atlas SJ
- Au J
- Aukema AA
- Aurora R
- Autio TJ
- Autio TJ
- Autio TJ
- Autio TJ
- Avdeeva K
- Awad OG
- Awad OGA
- Ayers CM
- Ayoub N
- Ayoub N
- Ayoub N
- Azbell C
- Azila A
- Baba S
- Baba S
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- Babela R
- Babinski D
- Bachert C
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- Bachert C
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- Bakhshaee M
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- Barham HP
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- Baroody FM
- Basharat U
- Bassiouni A
- Bassiouni A
- Bassiouni A
- Batikhan H
- Batra PS
- Batra PS
- Batra PS
- Baudoin T
- Baumann A
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- Bednarski KA
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- Benninger MS
- Benninger MS
- Benninger MS
- Benninger MS
- Benninger MS
- Benoit M
- Bent JP
- Bento LR
- Benítez P
- Berg O
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- Berges‐Gimeno MP
- Berjis N
- Berlucchi M
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- Bernstein JA
- Bernstein JE
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- Berry J
- Beswick DM
- Beswick DM
- Beswick DM
- Beswick DM
- Bettadahalli V
- Beule AG
- Beyea JA
- Bezerra T
- Bezerra TF
- Bhalla RK
- Bhalla V
- Bhandarkar ND
- Bhandarkar ND
- Bhandarkar ND
- Bhatt JM
- Bhattacharjee S
- Bhattacharyya N
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- Bhawana G
- Bidder T
- Biedlingmaier JF
- Biedlingmaier JF
- Biedlingmaier JF.
- Biel MA
- Biel MA
- Bikhazi N
- Bikhazi N
- Bilge T
- Biswas K
- Biswas K
- Biswas K
- Bizaki AJ
- Blackwell DL
- Blackwell KE
- Blair C
- Bleier BS
- Bleier BS
- Bleier BS
- Bleier BS
- Blount A
- Boase S
- Boase S
- Bockmühl U
- Boezaart AP
- Bohman A
- Bohman A
- Bohnhorst I
- Boita MM
- Bolger WE
- Bolger WE
- Bolger WE
- Bomeli SR
- Bondioni MP
- Bonfils P
- Bonfils P
- Bonfils P
- Bonnomet A
- Boonmak P
- Borish L
- Borsetto D
- Bosse Y
- Bosso JV
- Bothra R
- Bousquet J
- Bousquet J
- Brandsted R
- Braun JJ
- Braun JJ
- Braunstahl GJ
- Braverman I
- Brescia G
- Brescia G
- Brietzke SE
- Brietzke SE
- Brodner D
- Brook I
- Brook I
- Brook I
- Brook I
- Brook I.
- Brook I.
- Bross‐Soriano D
- Brot C
- Brown CL
- Brown S
- Brozek JL
- Brozek JL
- Brumund KT
- Brunner JP
- Buckley CE
- Buehring I
- Bugten V
- Bukowy‐Bieryllo Z
- Burgstaller JM
- Burton BN
- Bush A
- Businco LD
- Buysschaert ID
- Byun JY
- Cahill KN
- Cai Y
- Calhoun KH
- Calixto NE
- Callejas CA
- Campbell AP
- Campbell AP
- Campbell RG
- Campbell RG.
- Candoni A
- Cannady SB
- Cannoni M
- Canonica GW
- Cantrell H.
- Cao PP
- Cao P‐P
- Cao Y
- Cao Y
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- Carlton DA
- Carney AS
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- Carr TF.
- Carroll WW
- Carroll WW
- Carroll WW
- Casale M
- Cassandro E
- Castano R
- Castelnuovo P
- Castelnuovo P
- Castro M
- Catalano P
- Catalano PJ
- Catalano PJ
- Caughey RJ
- Caulley L
- Cazzola M
- Cekic B
- Ceri H
- Cervin A
- Cervin A
- Chaaban MR
- Chaaban MR
- Chaaban MR
- Chakrabarti A
- Chakrabarti A
- Chalermwatanachai T
- Chalton R
- Chambers DW
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- Chan KO
- Chan Y
- Chandler JR
- Chandra RK
- Chandra RK
- Chandra RK
- Chandra RK
- Chandy Z
- Chang CC
- Chang CC
- Chang EH
- Chang EH
- Chang EH
- Chang HS
- Chang MT
- Chang MT
- Chang MT
- Chang Y‐S
- Chaudhary N
- Che C
- Chee L
- Chen B
- Chen B
- Chen CF
- Chen F
- Chen FH
- Chen H
- Chen J‐B
- Chen PH
- Chen X
- Chen YT
- Cheng YK
- Cheon YI
- Chester AC
- Chester AC
- Chester AC
- Chiarella SE
- Chiesa‐Estomba CM
- Chin D
- Chinratanapisit S
- Chiu A
- Chiu AG
- Chiu AG
- Chiu AG
- Cho DY
- Cho DY
- Cho DY
- Cho DY
- Cho DY
- Cho DY
- Cho DY
- Cho DY
- Cho DY
- Cho GS
- Cho HJ
- Cho JH
- Cho K
- Cho KS
- Cho KS
- Cho SH
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- Zielinska‐Blizniewska H
- Zou L
- Zuckerman JD
- Zurlo JJ
- Zuskin E
- Čulig J
- Publication venue
- 'Wiley'
- Publication date
- 01/01/2021
- Field of study
Background: The 5 years since the publication of the first International Consensus Statement on Allergy and Rhinology: Rhinosinusitis (ICAR‐RS) has witnessed foundational progress in our understanding and treatment of rhinologic disease. These advances are reflected within the more than 40 new topics covered within the ICAR‐RS‐2021 as well as updates to the original 140 topics. This executive summary consolidates the evidence‐based findings of the document. Methods: ICAR‐RS presents over 180 topics in the forms of evidence‐based reviews with recommendations (EBRRs), evidence‐based reviews, and literature reviews. The highest grade structured recommendations of the EBRR sections are summarized in this executive summary. Results: ICAR‐RS‐2021 covers 22 topics regarding the medical management of RS, which are grade A/B and are presented in the executive summary. Additionally, 4 topics regarding the surgical management of RS are grade A/B and are presented in the executive summary. Finally, a comprehensive evidence‐based management algorithm is provided. Conclusion: This ICAR‐RS‐2021 executive summary provides a compilation of the evidence‐based recommendations for medical and surgical treatment of the most common forms of RS
Tigecycline Therapy for Infections Caused by Extended-Spectrum β-Lactamase-Producing Enterobacteriaceae in Critically Ill Patients
- Publication venue
- 'MDPI AG'
- Publication date
- 05/05/2020
- Field of study
We aimed to evaluate tigecycline on the clinical effectiveness in treating complicated skin and soft tissue infections (cSSTI), complicated intra-abdominal infections (cIAI), and pneumonia, caused by extended-spectrum β-lactamase (ESBL)-producing Enterobacteriaceae, as data are limited. From three medical centers in Taiwan, we retrospectively studied the cSSTI, cIAI, and/or pneumonia caused by ESBL-producing Enterobacteriaceae. Among the 71 patients, including 39 patients infected with Klebsiella pneumoniae, 30 infected with Escherichia coli and others, the clinical success rate of tigecycline-based therapy was 80–90% for pneumonia and cSSTI caused by E. coli and 50–60% for cIAI caused by K. pneumoniae and E. coli. Microbiological and clinical outcome of pneumonia caused by carbapenem-resistant K. pneumoniae was poor. Univariate Cox analysis showed that dyspnea, SOFA score, septic shock, thrombocytopenia, prolonged prothrombin time, and lesser microbiological eradication were significant factors associated with 30-day mortality after the end of therapy. Cox regression proportional hazards model revealed dyspnea and a SOFA score > 8 to be independently associated with time to death. For ESBL producers, tigecycline showed good effects for cSSTI and pneumonia by E. coli, ordinary for cIAI, but ineffective for pneumonia by K. pneumoniae. Dyspnea and a high SOFA score predict a poor outcome
KDBI: Kinetic data of Bio-molecular interactions database
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2003
- Field of study
10.1093/nar/gkg067Nucleic Acids Research311255-257NARH
The role of <it>ALDH2 </it>and <it>ADH1B </it>polymorphism in alcohol consumption and stroke in Han Chinese
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/10/2011
- Field of study
Abstract The genes encoding the enzymes for metabolising alcohol dehydrogenase 1B (ADH1B) and aldehyde dehydrogenase 2 (ALDH2) -- exhibit genetic polymorphism and ethnic variations. Although the ALDH2*2 variant allele has been widely accepted as protecting against the development of alcoholism in Asians, the association of the ADH1B*2 variant allele with drinking behaviour remains inconclusive. The goal of this study was to determine whether the polymorphic ADH1B and ALDH2 genes are associated with stroke in male Han Chinese with high alcohol consumption. Sixty-five stroke patients with a history of heavy drinking (HDS) and 83 stroke patients without such a history (NHDS) were recruited for analysis of the ADH1B and ALDH2 genotypes from the stroke registry in the Tri-Service General Hospital, Taipei, Taiwan, between January 2000 and December 2001. The allelotypes of ADH1B and ALDH2 were determined using the polymerase chain reaction-restriction fragment length polymorphism method. The HDS patients (3 per cent) showed a significantly lower ALDH2*2 allele frequency than NHDS patients (27 per cent) (p p p ALDH2*2 variant allele was an independent variable exhibiting strong protection (odds ratio 0.072; 95 per cent confidence interval 0.02-0.26) against HDS after adjustment for hypertension, diabetes mellitus, smoking status and liver dysfunction. By contrast, allelic variations in ADH1B exerted no significant effect on HDS. The present study indicated that, unlike ALDH2*2, ADH1B*2 appears not to be a significant negative risk factor for high alcohol consumption in male Han Chinese with stroke.</p
Gene Expression Profiling of Colorectal Tumors and Normal Mucosa by Microarrays Meta-Analysis Using Prediction Analysis of Microarray, Artificial Neural Network, Classification, and Regression Trees
- Author
- Publication venue
- Hindawi Limited
- Publication date
- 01/01/2014
- Field of study
Background. Microarray technology shows great potential but previous studies were limited by small number of samples in the colorectal cancer (CRC) research. The aims of this study are to investigate gene expression profile of CRCs by pooling cDNA microarrays using PAM, ANN, and decision trees (CART and C5.0). Methods. Pooled 16 datasets contained 88 normal mucosal tissues and 1186 CRCs. PAM was performed to identify significant expressed genes in CRCs and models of PAM, ANN, CART, and C5.0 were constructed for screening candidate genes via ranking gene order of significances.
Results. The first screening identified 55 genes. The test accuracy of each model was over 0.97 averagely. Less than eight genes achieve excellent classification accuracy. Combining the results of four models, we found the top eight differential genes in CRCs; suppressor genes, CA7, SPIB, GUCA2B, AQP8, IL6R and CWH43; oncogenes, SPP1 and TCN1. Genes of higher significances showed lower variation in rank ordering by different methods. Conclusion. We adopted a two-tier genetic screen, which not only reduced the number of candidate genes but also yielded good accuracy (nearly 100%). This method can be applied to future studies. Among the top eight genes, CA7, TCN1, and CWH43 have not been reported to be related to CRC
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