Not So Fun City

This article posits that the destruction of the South Bronx during the 1960s and 1970s was municipally approved across several mayoral administrations, most notably the term of John Lindsay who coined the catch phrase “Fun City,” referring to New York City on the first day of a transit strike. The author outlines several books and articles that detail the destruction caused by fires and city planning decisions that adversely affected White ethnic and neighborhoods of color in the South Bronx. Media portrayals during this period were used to stereotype certain areas of the Bronx which in turn allowed city planners to advocate for their destruction. During this period there was a massive out-migration of Bronxites adding to the urban swirl. In the end the people who live in these maligned areas are ultimately blamed for the destruction of their neighborhoods even though the City of New York and its consultants made decisions that allowed it to happen. The role of the RAND corporation and Fire Commissioner John O’Hagan figure prominently in this narrative

Concourse Dreams: A Bronx Neighborhood And Its Future

This master\u27s thesis focuses on the West Bronx recent past and illustrates what upward mobility meant for the Grand Concourse neighborhood populated with second generation immigrant groups at the beginning of the 20th century, including Jewish, Italian and Irish people. Later, most of the same area became a slum at the hands of city planners, New York City administration, and negative media portrayals. Race and ethnicity became even more talked about when non-white immigrants and migrants began moving to the Grand Concourse in huge numbers after World War Two.Through a celebrated Bronx past before 1950 and eventually the sensational media portrayals of the 1970s, the best residential and civic architecture in America stood sentry over the Grand Concourse. This thesis will also illustrate the remarkable changes and investments being made in the Bronx during the current decade as exemplified by the new Yankee Stadium and Bronx Terminal Market. The future of this neighborhood is also discussed by four informed and involved community residents

From the Classroom to the Concourse: Studying the Grand Concourse to Discover One\u27s Community

Walt Whitman’s poem, “I Hear America Singing” inspired the 13 murals painted on the walls of the Grand Concourse U.S. post office located across the street from Hostos Community College. Visited last fall by Professor Zucker’s Reading class (English 92) these looming murals helped students more fully appreciate how art enhances the Grand Concourse and, by extension, education illuminates everyday life. The 1860 poem itself celebrates America and its diversity, which 150 year later, still holds relevance for students

Opening CUNY: Academic Works at Work

Academic Works, CUNY’s new open access institutional repository, collects and provides public access to the scholarly and creative works produced by CUNY faculty, students and staff. This program will show how opening content to the world impacts CUNY, as each speaker addresses collections at their institution: dissertations at The Graduate Center, Open Educational Resources at Brooklyn College, the “Save Hostos” archival collection at Hostos Community College and faculty research from across CUNY

Donated Records Partnership Project-The Collection Match

Presentation titled, "Donated Records Partnership Project -- The Collection Match" given by Dawn Sherman-Fells, Meghan Ryan Guthorn, William Casari, Beth Harris, and Laura Poll at the MARAC Spring 2014 conference, S4 - April 25, 2014, Rochester, N

Giosuè Carducci prosatore

Questo volume su Giosuè Carducci prosatore raccoglie i contributi presentati al XVII Convegno internazionale di Letteratura italiana "Gennaro Barbarisi", tenutosi a Palazzo Feltrinelli (Gargnano del Garda) dal 29 settembre al 1° ottobre 2016. Si è trattato di una proficua occasione di incontro, di studio e di approfondimento su un tema forse poco frequentato, soprattutto in tempi recenti, ma ricco di sollecitazioni per una più articolata e storicamente fondata definizione della personalità di un autore così significativo nel panorama della cultura italiana fra Otto e primo Novecento; non soltanto sul versante della poesia (un primato sancito dal premio Nobel nel 1906) ma anche, e forse ancora di più, su quello della prosa saggistica, degli scritti di polemica, delle curatele editoriali, delle ricerche erudite, fino alle prove di alta oratoria e all'epistolografia

Whole-Exome Sequencing Identifies Homozygous AFG3L2 Mutations in a Spastic Ataxia-Neuropathy Syndrome Linked to Mitochondrial m-AAA Proteases

We report an early onset spastic ataxia-neuropathy syndrome in two brothers of a consanguineous family characterized clinically by lower extremity spasticity, peripheral neuropathy, ptosis, oculomotor apraxia, dystonia, cerebellar atrophy, and progressive myoclonic epilepsy. Whole-exome sequencing identified a homozygous missense mutation (c.1847G>A; p.Y616C) in AFG3L2, encoding a subunit of an m-AAA protease. m-AAA proteases reside in the mitochondrial inner membrane and are responsible for removal of damaged or misfolded proteins and proteolytic activation of essential mitochondrial proteins. AFG3L2 forms either a homo-oligomeric isoenzyme or a hetero-oligomeric complex with paraplegin, a homologous protein mutated in hereditary spastic paraplegia type 7 (SPG7). Heterozygous loss-of-function mutations in AFG3L2 cause autosomal-dominant spinocerebellar ataxia type 28 (SCA28), a disorder whose phenotype is strikingly different from that of our patients. As defined in yeast complementation assays, the AFG3L2Y616C gene product is a hypomorphic variant that exhibited oligomerization defects in yeast as well as in patient fibroblasts. Specifically, the formation of AFG3L2Y616C complexes was impaired, both with itself and to a greater extent with paraplegin. This produced an early-onset clinical syndrome that combines the severe phenotypes of SPG7 and SCA28, in additional to other “mitochondrial” features such as oculomotor apraxia, extrapyramidal dysfunction, and myoclonic epilepsy. These findings expand the phenotype associated with AFG3L2 mutations and suggest that AFG3L2-related disease should be considered in the differential diagnosis of spastic ataxias