6,903 research outputs found
SPIDER X - Environmental effects in central and satellite early-type galaxies through the stellar fossil record
A detailed analysis of how environment affects the star formation history of
early-type galaxies (ETGs) is undertaken via high signal to noise ratio stacked
spectra obtained from a sample of 20,977 ETGs (morphologically selected) from
the SDSS-based SPIDER survey. Two major parameters are considered for the
study: the central velocity dispersion (sigma), which relates to local drivers
of star formation, and the mass of the host halo, which relates to
environment-related effects. In addition, we separate the sample between
centrals (the most massive galaxy in a halo) and satellites. We derive trends
of age, metallicity, and [alpha/Fe] enhancement, with sigma. We confirm that
the major driver of stellar population properties in ETGs is velocity
dispersion, with a second-order effect associated to the central/satellite
nature of the galaxy. No environmental dependence is detected for satellite
ETGs, except at low sigma - where satellites in groups or in the outskirts of
clusters tend to be younger than those in the central regions of clusters. In
contrast, the trends for centrals show a significant dependence on halo mass.
Central ETGs in groups (i.e. with a halo mass >10^12.5 M_Sun) have younger
ages, lower [alpha/Fe], and higher internal reddening, than "isolated" systems
(i.e. centrals residing in low-mass, <10^12.5 M_Sun, halos). Our findings imply
that central ETGs in groups formed their stellar component over longer time
scales than "isolated" centrals, mainly because of gas-rich interactions with
their companion galaxies.Comment: 22 pages, 19 figures, accepted for publication in MNRA
Reductive titration of CoQ-depleted Complex III from Baker's yeast Evidence for an exchange-coupled complex between QH· and low-spin ferricytochrome b
Growth of a sinkhole in a seismic zone of the northern Apennines (Italy)
Sinkhole collapse is a major hazard causing substantial social and economic losses. However, the surface deformations and sinkhole evolution are rarely recorded, as these sites are known mainly after a collapse, making the assessment of sinkhole-related hazard challenging. Furthermore, more than 40% of the sinkholes of Italy are in seismically hazardous zones; it remains unclear whether seismicity may trigger sinkhole collapse. Here we use a multidisciplinary data set of InSAR, surface mapping and historical records of sinkhole activity to show that the Prà di Lama lake is a long-lived sinkhole that was formed in an active fault zone and grew through several events of unrest characterized by episodic subsidence and lake-level changes. Moreover, InSAR shows that continuous aseismic subsidence at rates of up to 7.1mmyr-1occurred during 2003-2008, between events of unrest. Earthquakes on the major faults near the sinkhole do not trigger sinkhole activity but low-magnitude earthquakes at 4-12 km depth occurred during sinkhole unrest in 1996 and 2016. We interpret our observations as evidence of seismic creep at depth causing fracturing and ultimately leading to the formation and growth of the Prà di Lama sinkhole
On the Nature of Fossil Galaxy Groups: Are they really fossils ?
We use SDSS-DR4 photometric and spectroscopic data out to redshift z~0.1
combined with ROSAT All Sky Survey X-ray data to produce a sample of
twenty-five fossil groups (FGs), defined as bound systems dominated by a
single, luminous elliptical galaxy with extended X-ray emission. We examine
possible biases introduced by varying the parameters used to define the sample
and the main pitfalls are discussed. The spatial density of FGs, estimated via
the V/V_ MAX} test, is 2.83 x 10^{-6} h_{75}^3 Mpc^{-3} for L_x > 0.89 x 10^42
h_{75}^-2 erg/s consistent with Vikhlinin et al. (1999), who examined an X-ray
overluminous elliptical galaxy sample (OLEG). We compare the general properties
of FGs identified here with a sample of bright field ellipticals generated from
the same dataset. These two samples show no differences in the distribution of
neighboring faint galaxy density excess, distance from the red sequence in the
color-magnitude diagram, and structural parameters such as a and internal
color gradients. Furthermore, examination of stellar populations shows that our
twenty-five FGs have similar ages, metallicities, and -enhancement as
the bright field ellipticals, undermining the idea that these systems represent
fossils of a physical mechanism that occurred at high redshift. Our study
reveals no difference between FGs and field ellipticals, suggesting that FGs
might not be a distinct family of true fossils, but rather the final stage of
mass assembly in the Universe.Comment: 18 pages, Accepted to A
RNA microarray analysis in prenatal mouse cochlea reveals novel IGF-I target genes: implication of MEF2 and FOXM1 transcription factors
Background: Insulin-like growth factor-I (IGF-I) provides pivotal cell survival and differentiation signals during inner ear development throughout evolution. Homozygous mutations of human IGF1 cause syndromic sensorineural deafness, decreased intrauterine and postnatal growth rates, and mental retardation. In the mouse, deficits in IGF-I result in profound hearing loss associated with reduced survival, differentiation and maturation of auditory neurons. Nevertheless, little is known about the molecular basis of IGF-I activity in hearing and deafness.
Methodology/Principal Findings: A combination of quantitative RT-PCR, subcellular fractionation and Western blotting, along with in situ hybridization studies show IGF-I and its high affinity receptor to be strongly expressed in the embryonic and postnatal mouse cochlea. The expression of both proteins decreases after birth and in the cochlea of E18.5 embryonic Igf1(-/-) null mice, the balance of the main IGF related signalling pathways is altered, with lower activation of Akt and ERK1/2 and stronger activation of p38 kinase. By comparing the Igf1(-/-) and Igf1(+/+) transcriptomes in E18.5 mouse cochleae using RNA microchips and validating their results, we demonstrate the up-regulation of the FoxM1 transcription factor and the misexpression of the neural progenitor transcription factors Six6 and Mash1 associated with the loss of IGF-I. Parallel, in silico promoter analysis of the genes modulated in conjunction with the loss of IGF-I revealed the possible involvement of MEF2 in cochlear development. E18.5 Igf1(+/+) mouse auditory ganglion neurons showed intense MEF2A and MEF2D nuclear staining and MEF2A was also evident in the organ of Corti. At P15, MEF2A and MEF2D expression were shown in neurons and sensory cells. In the absence of IGF-I, nuclear levels of MEF2 were diminished, indicating less transcriptional MEF2 activity. By contrast, there was an increase in the nuclear accumulation of FoxM1 and a corresponding decrease in the nuclear cyclin-dependent kinase inhibitor p27(Kip1).
Conclusions/Significance: We have defined the spatiotemporal expression of elements involved in IGF signalling during inner ear development and reveal novel regulatory mechanisms that are modulated by IGF-I in promoting sensory cell and neural survival and differentiation. These data will help us to understand the molecular bases of human sensorineural deafness associated to deficits in IGF-I
- …