Foreign Object Damage to Fan Rotor Blades of Aeroengine Part I: Experimental Study of Bird Impact

AbstractThe conditions of experiment for bird impact to blades have been improved. The experiment of bird impact to the fan rotor blades of an aeroengine is carried out. Through analyzing the transient state response of blades impacted by bird and the change of blade profile before and after the impact, the anti-bird impact performance of blades in the first fan rotor is verified. The basis of anti-foreign object damage design for the fan rotor blades of an aeroengine is provided

Foreign Object Damage to Fan Rotor Blades of Aeroengine Part II: Numerical Simulation of Bird Impact

AbstractBird impact is one of the most dangerous threats to flight safety. The consequences of bird impact can be severe and, therefore, the aircraft components have to be certified for a proven level of bird impact resistance before being put into service. The fan rotor blades of aeroengine are the components being easily impacted by birds. It is necessary to ensure that the fan rotor blades should have adequate resistance against the bird impact, to reduce the flying accidents caused by bird impacts. Using the contacting-impacting algorithm, the numerical simulation is carried out to simulate bird impact. A three-blade computational model is set up for the fan rotor blade having shrouds. The transient response curves of the points corresponding to measured points in experiments, displacements and equivalent stresses on the blades are obtained during the simulation. From the comparison of the transient response curves obtained from numerical simulation with that obtained from experiments, it can be found that the variations in measured points and the corresponding points of simulation are basically the same. The deforming process, the maximum displacements and the maximum equivalent stresses on blades are analyzed. The numerical simulation verifies and complements the experiment results

Are Hylobates lar Extirpated from China?

The Nangunhe Nature Reserve in Southwest Yunnan (PRC) has long been presumed to be the last stronghold of lar (or white-handed) gibbons (Hylobates lar) in China and the likely last place of occurrence of Hylobates lar yunnanensis. We conducted a comprehensive survey to assess the status of lar gibbons at Nangunhe. We found no visual or auditory evidence of them still residing at the reserve and therefore tentatively conclude that lar gibbons have become extinct in China. It appears that large-scale destruction of primary forests in the 1960s and 1970s brought about an initial decline in their numbers, and subsequent uncontrolled hunting has resulted in their extirpation. The situation for the six Chinese ape taxa is nothing less than disastrous, with 1 taxon assumed to have become extinct during the last few years, 1 taxon not having been confirmed since the 1980s, and 2 species at the very brink of extinction with only tens of individuals remaining in Chin

Query Twice: Dual Mixture Attention Meta Learning for Video Summarization

Video summarization aims to select representative frames to retain high-level information, which is usually solved by predicting the segment-wise importance score via a softmax function. However, softmax function suffers in retaining high-rank representations for complex visual or sequential information, which is known as the Softmax Bottleneck problem. In this paper, we propose a novel framework named Dual Mixture Attention (DMASum) model with Meta Learning for video summarization that tackles the softmax bottleneck problem, where the Mixture of Attention layer (MoA) effectively increases the model capacity by employing twice self-query attention that can capture the second-order changes in addition to the initial query-key attention, and a novel Single Frame Meta Learning rule is then introduced to achieve more generalization to small datasets with limited training sources. Furthermore, the DMASum significantly exploits both visual and sequential attention that connects local key-frame and global attention in an accumulative way. We adopt the new evaluation protocol on two public datasets, SumMe, and TVSum. Both qualitative and quantitative experiments manifest significant improvements over the state-of-the-art methods.Comment: This manuscript has been accepted at ACM MM 202

4-CPA (4-chlorophenoxyacetic acid) induces the formation and development of defective “fenghou” (vitis vinifera × v. labrusca) grape seeds

For some horticultural plants, auxins can not only induce normal fruit setting but also form fake seeds in the induced fruits. This phenomenon is relatively rare, and, so far, the underlying mechanism remains unclear. In this study, “Fenghou” (Vitis vinifera × V. labrusca) grapes were artificially emasculated before flowering and then sprayed with 4-CPA (4-chlorophenoxyacetic acid) to analyze its effect on seed formation. The results show that 4-CPA can induce normal fruit setting in “Fenghou” grapes. Although more seeds were detected in the fruits of the 4-CPA-treated grapevine, most seeds were immature. There was no significant difference in the seed shape; namely, both fruit seeds of the grapevines with and without 4-CPA treatment contained a hard seed coat. However, the immature seeds lacked embryo and endosperm tissue and could not germinate successfully; these were considered defective seeds. Tissue structure observation of defective seeds revealed that a lot of tissue redifferentiation occurred at the top of the ovule, which increased the number of cell layers of the outer integument; some even differentiated into new ovule primordia. The qRT-PCR results demonstrated that 4-CPA application regulated the expression of the genes VvARF2 and VvAP2, which are associated with integument development in “Fenghou” grape ovules. Together, this study evokes the regulatory role of 4-CPA in the division and continuous redifferentiation of integument cells, which eventually develop into defective seeds with thick seed coats in grapes

Mechanism Enhancing Arabidopsis Resistance to Cadmium: The Role of NRT1.5 and Proton Pump

Aim: Heavy metal pollution is serious in China, and abscisic acid (ABA) is an important stress hormone. How it regulates plant tolerance to cadmium remains unclear, so we aimed to explore the molecular mechanism responsible for enhanced cadmium resistance in Arabidopsis wild-type and mutant plants and Brassica napus seedlings.Methods: Arabidopsis/B. napus were cultured hydroponically for 28/15 days and then treated with 20/10 μM Cd/Cd+ABA (5 μM) for 3/4 days. Chlorophyll degradation rate, SPAD values, proline, MDA, ABA, NO3−, and Cd concentrations were measured in root vacuoles and protoplasts; root to shoot NO3− and Cd concentration ratios were determined and NRT1.5-, NRT1.8-, BnNRT1.5-, and BnNRT1.8-related gene expression was studied.Results: Cytoplasmic ABA levels in root cells of bglu10 and bglu18 Arabidopsis mutants were significantly lower than those in the wild-type, apparently making the latter more resistant to Cd. NO3− long-distance transporter NRT1.5 responded to ABA signaling by downregulating its own expression, while NRT1.8 did not respond. Concomitantly, proton pump activity in wild-type plants was higher than in the bglu10 and bglu18 mutants; thus, more NO3− and Cd accumulated in the vacuoles of wild-type root cells. ABA application inhibited Cd absorption by B. napus. BnNRT1.5 responded to exogenous ABA signal by downregulating its own expression, while the lack of response by BnNRT1.8 resulted in increased amount of NO3− accumulating in the roots to participate in the anti-cadmium reaction.Conclusion:NRT1.5 responds to the ABA signal to inhibit its own expression, whereas unresponsiveness of NRT1.8 causes accumulation of NO3− in the roots; thus, enhancing Cd resistance. In Arabidopsis, because of proton pump action, more NO3− and Cd accumulate in the vacuoles of Arabidopsis root cells, thereby reducing damage by Cd toxicity. However, in B. napus, the addition of exogenous ABA inhibited Cd absorption. Our data provide a sound basis to the theoretical molecular mechanism involved in hormone signaling during response of plants to heavy metal stress

Turicibacter fermentation enhances the inhibitory effects of Antrodia camphorata supplementation on tumorigenic serotonin and Wnt pathways and promotes ROS-mediated apoptosis of Caco-2 cells

Introduction: Diet-induced obesity has been shown to decrease the abundance of Turicibacter, a genus known to play a role in the serotonin signaling system, which is associated with colorectal tumorigenesis, making the presence of Turicibacter potentially influential in the protection of intestinal tumorigenesis. Recently, Antrodia camphorata (AC), a medicinal fungus native to Taiwan, has emerged as a promising candidate for complementary and alternative cancer therapy. Small molecules and polysaccharides derived from AC have been reported to possess health-promoting effects, including anti-cancer properties.Methods: Bacterial culture followed with cell culture were used in this study to determine the role of Turicibacter in colorectal tumorigenesis and to explore the anti-cancer mechanism of AC with Turicibacter fermentation.Results:Turicibacter fermentation and the addition of AC polysaccharide led to a significant increase in the production of nutrients and metabolites, including α-ketoglutaric acid and lactic acid (p < 0.05). Treatment of Turicibacter fermented AC polysaccharide was more effective in inhibiting serotonin signaling-related genes, including Tph1, Htr1d, Htr2a, Htr2b, and Htr2c (p < 0.05), and Wnt-signaling related protein and downstream gene expressions, such as phospho-GSK-3β, active β-catenin, c-Myc, Ccnd1, and Axin2 (p < 0.05). Additionally, it triggered the highest generation of reactive oxygen species (ROS), which activated PI3K/Akt and MAPK/Erk signaling and resulted in cleaved caspase-3 expression. In comparison, the treatment of AC polysaccharide without Turicibacter fermentation displayed a lesser effect.Discussion: Our findings suggest that AC polysaccharide effectively suppresses the tumorigenic serotonin and Wnt-signaling pathways, and promotes ROS-mediated apoptosis in Caco-2 cells. These processes are further enhanced by Turicibacter fermentation

NRT1.1 Regulates Nitrate Allocation and Cadmium Tolerance in Arabidopsis

Abiotic stress induces nitrate (NO3-) allocation to roots, which increases stress tolerance in plants. NRT1.1 is broadly involved in abiotic stress tolerance in plants, but the relationship between NRT1.1 and NO3- allocation under stress conditions is unclear. In this study, we found that Arabidopsis wild-type Col-0 was more cadmium (Cd2+)-tolerant than the nrt1.1 mutant at 20 μM CdCl2. Cd2+ exposure repressed NRT1.5 but upregulated NRT1.8 in roots of Col-0 plants, resulting in increased NO3- allocation to roots and higher [NO3-] root-to-shoot (R:S) ratios. Interestingly, NITRATE REGULATORY GENE2 (NRG2) was upregulated by Cd2+ stress in Col-0 but not in nrt1.1. Under Cd2+ stress, nrg2 and nrg2-3chl1-13 mutants exhibited similar phenotypes and NO3- allocation patterns as observed in the nrt1.1 mutant, but overexpression of NRG2 in Col-0 and nrt1.1 increased the [NO3-] R:S ratio and restored Cd2+ stress tolerance. Our results indicated that NRT1.1 and NRG2 regulated Cd2+ stress-induced NO3- allocation to roots and that NRG2 functioned downstream of NRT1.1. Cd2+ uptake did not differ between Col-0 and nrt1.1, but Cd2+ allocation to roots was higher in Col-0 than in nrt1.1. Stressed Col-0 plants increased Cd2+ and NO3- allocation to root vacuoles, which reduced their cytosolic allocation and transport to the shoots. Our results suggest that NRT1.1 regulates NO3- allocation to roots by coordinating Cd2+ accumulation in root vacuoles, which facilitates Cd2+ detoxification