I Cannot Teach Because I Am Not Smart : Working Class Mothers’ Support for Their Children\u27s Education in Japan

Social class is a powerful element which predicts mothers’ support for their children’s academic development in Japan. Middle class mothers tend to hold higher educational expectations, invest in their children’s educational opportunities, and interact with the teachers more frequently than working class mothers (Stevenson & Stigler, 1992; Yamamoto, 2006). While ample evidence shows social class differences in parents’ academic support, few have examined why working class mothers are not as involved in their children’s education as middle class mothers. In order to understand the mechanisms of social class reproduction and mobility, it is critical to investigate the experiences and elements that encourage and discourage working class mothers’ involvement in their children’s education. The present study is based on analyses of in-depth interviews with eight working class mothers of young children in Japan. All mothers did not have a college degree and lived in a working class neighborhood. The findings demonstrated that a lack of confidence due to limited education or negative school experiences inhibit these mothers’ involvement in their children’s education. The mothers tended to believe that they lacked ability to effectively support their children’s education. Further analyses showed that social capital, a form of capital that existed in human relationships, was a critical element that helped working class mothers redevelop and renegotiate their parenting models and increase their confidence in navigating their children’s schooling

Factors Affecting Clinical Nursing Competency: A Cross Sectional Study

[Background] The purpose of this study was to elucidate the factors which affect the achievement of clinical nursing competency. [Methods] A survey was conducted on 717 nurses using an anonymous self-administered questionnaire. Their clinical nursing competency was assessed using the Clinical Nursing Competence Self-Assessment Scale (CNCSS). This study examined the factors affecting clinical nursing competency using regression analyses. A simple regression analysis was performed with the CNCSS as the objective variable. A multiple regression analysis was performed using the items for which the relationship was clarified as explanatory variables. [Results] The factors affecting the “basic nursing competency” were age, ease of taking time off, workplace with a clear vision, and good interpersonal relationships. The factors affecting the “competency in providing assistance commensurate with the patient’s health status” were total years of experience, workplace with a clear vision, ease of taking time off, and use of acquired certifications. The factors affecting the “coordinating care environment and teamwork” were total years of experience, workplace with a clear vision, use of acquired certifications, and ease of taking time off. The factors affecting the “ability for professional growth in nursing practice” were use of acquired certifications, workplace with a clear vision, total years of experience, and ease of taking time off. [Conclusion] For improvement of clinical nursing competency, the factors elucidated to be necessary were accumulation of experience as a nurse, a clear vision of goals, and a work environment with good interpersonal relationships and ease of getting days off. The way nurses make their nursing practice experience meaningful contributed toward their growth as nurses. It is important to train nurses through basic education and continued education with awareness of achievement and improvement of clinical nursing competency. Basic education should promote the ability to make clinical training experience meaningful and continuing education should enable nurses to continue to grow independently through reflection

Meiotic gene silencing complex MTREC/NURS recruits the nuclear exosome to YTH-RNA-binding protein Mmi1.

Accurate target recognition in transcript degradation is crucial for regulation of gene expression. In the fission yeast Schizosaccharomyces pombe, a number of meiotic transcripts are recognized by a YTH-family RNA-binding protein, Mmi1, and selectively degraded by the nuclear exosome during mitotic growth. Mmi1 forms nuclear foci in mitotically growing cells, and the nuclear exosome colocalizes to such foci. However, it remains elusive how Mmi1 and the nuclear exosome are connected. Here, we show that a complex called MTREC (Mtl1-Red1 core) or NURS (nuclear RNA silencing) that consists of a zinc-finger protein, Red1, and an RNA helicase, Mtl1, is required for the recruitment of the nuclear exosome to Mmi1 foci. Physical interaction between Mmi1 and the nuclear exosome depends on Red1. Furthermore, a chimeric protein involving Mmi1 and Rrp6, which is a nuclear-specific component of the exosome, suppresses the ectopic expression phenotype of meiotic transcripts in red1Δ cells and mtl1 mutant cells. These data indicate that the primary function of MTREC/NURS in meiotic transcript elimination is to link Mmi1 to the nuclear exosome physically

The Co-evolution of Disk and Star in Embedded Stages: The Case of the Very Low-mass Protostar

We have observed the CCH (N=3-2, J=7/2-5/2, F=4-3 and 3-2) and SO (6_7-5_6) emission at a 0"2 angular resolution toward the low-mass Class 0 protostellar source IRAS 15398-3359 with ALMA. The CCH emission traces the infalling-rotating envelope near the protostar with the outflow cavity extended along the northeast-southwest axis. On the other hand, the SO emission has a compact distribution around the protostar. The CCH emission is relatively weak at the continuum peak position, while the SO emission has a sharp peak there. Although the maximum velocity shift of the CCH emission is about 1 km s^-1 from the systemic velocity, a velocity shift higher than 2 km s^{-1} is seen for the SO emission. This high velocity component is most likely associated with the Keplerian rotation around the protostar. The protostellar mass is estimated to be 0.007^{+0.004}_{-0.003} from the velocity profile of the SO emission. With this protostellar mass, the velocity structure of the CCH emission can be explained by the model of the infalling-rotating envelope, where the radius of the centrifugal barrier is estimated to be 40 au from the comparison with the model. The disk mass evaluated from the dust continuum emission by assuming the dust temperature of 20 K-100 K is 0.1-0.9 times the stellar mass, resulting in the Toomre Q parameter of 0.4-5. Hence, the disk structure may be partly unstable. All these results suggest that a rotationally-supported disk can be formed in the earliest stages of the protostellar evolution

Infalling-Rotating Motion and Associated Chemical Change in the Envelope of IRAS 16293-2422 Source A Studied with ALMA

We have analyzed rotational spectral line emission of OCS, CH3OH, HCOOCH3, and H2CS observed toward the low-mass Class 0 protostellar source IRAS 16293-2422 Source A at a sub-arcsecond resolution (~0".6 x 0".5) with ALMA. Significant chemical differentiation is found at a 50 AU scale. The OCS line is found to well trace the infalling-rotating envelope in this source. On the other hand, the CH3OH and HCOOCH3 distributions are found to be concentrated around the inner part of the infalling-rotating envelope. With a simple ballistic model of the infalling-rotating envelope, the radius of the centrifugal barrier (a half of the centrifugal radius) and the protostellar mass are evaluated from the OCS data to be from 40 to 60 AU and from 0.5 to 1.0 Msun, respectively, assuming the inclination angle of the envelope/disk structure to be 60 degrees (90 degrees for the edge-on configuration). Although the protostellar mass is correlated with the inclination angle, the radius of the centrifugal barrier is not. This is the first indication of the centrifugal barrier of the infalling-rotating envelope in a hot corino source. CH3OH and HCOOCH3 may be liberated from ice mantles due to weak accretion shocks around the centrifugal barrier, and/or due to protostellar heating. The H2CS emission seems to come from the disk component inside the centrifugal barrier in addition to the envelope component. The centrifugal barrier plays a central role not only in the formation of a rotationally-supported disk but also in the chemical evolution from the envelope to the protoplanetary disk

タバコ懸濁培養細胞におけるアルミニウムの二価鉄、銅およびカドミウム毒性に対する影響

The effects of aluminum (Al) on the cytotoxicity of ferrous iron (Fe(Ⅱ)), copper(Cu) and cadmium(Cd) were studied. Log-phase cells were treated with either FeSO4,CuSO4, or CdCl2 in the presence or absence of AlCl3(120μM) for 18h at pH 4.0. After the treatment, the viability was determined as relative growth of the metal-treated cells to the untreated control cells during the post-treated culture. A single treated with either Al, Fe(Ⅱ) or Cd did not inhibit the growth at the metal concerntrations up to 300 μM, 200 μM and 500 μM, respectively, whereas the growth was markedly inhibited at 15 μM Cu. Thus,the cells were relatively insensitive to Al, Fe(Ⅱ) and Cd and sensitive to Cu. When cells were treated with both Fe(Ⅱ)(120 μM)and Al(120μM), the growth was significantly inhibited and the cellular contents of both Al and Fe increased synergistically. After the treatment with Cu(0 to 10 μM) with or without Al, the cells grew more vigorously when they were treated in the presence of Al, althrouh the Cu content of the cells were not alterd by Al. The presence of Al during the treatmemt with Cd(0 to 2 μM) had no effect on the degree of growth inhibition by Cd. Thus, Al interacts with the toxicity of Fe(Ⅱ), Cu and Cd in different manners; synergistic with Fe(Ⅱ), antagonistic with Cu and apparently no effeco on Cd.二価鉄[Fe(Ⅱ)]、銅（Cu)およびカドミウム（Cd）の細胞毒性に対するアルミニウム（Al）の効果について検討した。120μM AlCl3の存在もしくは非存在下において、対数増殖期の細胞をFeSO4,CuSO4,CdCl2で各々18時間、pH4.0で処理した。処理後の生存率は、処理後の細胞を増殖させたのちに、未処理細胞（コントロール）の増殖に対する金属で処理した細胞の相対増殖率で求めた。Al,Fe(Ⅱ),Cdの単独処理による増殖阻害は、各々300μM、200μM、500μMの濃度まで観察されなかったが、Cuでは15μMで大きく阻害された。このように、タバコ細胞は相対的にAl、Fe（Ⅱ）、Cdには感受性が低く、Cuには感受性が高かった。細胞をFe（Ⅱ）およびAlの両方で処理すると、増殖は著しく阻害され、AlおよびFeの細胞内含量も相乗的に増加した。Cuで処理した場合、Alを加えることにより逆に増殖率が増加した。しかし、細胞内Cu含量はAlの影響を受けなかった。Cd処理の場合、Alを加えてもCdによる増殖阻害の程度は変わらなかった。このように、AlはFe（Ⅱ）、Cu、Cd毒性に対して各々異なる相互作用を示し、Fe（Ⅱ）に対し、相乗効果を、Cuに対しては拮抗的な阻害を示したが、Cdに対してはみかけ上効果がなかった