Effect of correlated oxide electrodes on disorder pinning and thermal roughening of ferroelectric domain walls in epitaxial PbZr\u3csub\u3e0.2\u3c/sub\u3eTi\u3csub\u3e0.8\u3c/sub\u3eO\u3csub\u3e3\u3c/sub\u3e thin films

We report the competing effects of disorder pinning and thermal roughening on ferroelectric domain walls as a function of temperature in epitaxial PbZr0.2Ti0.8O3 thin films deposited on (001) SrTiO3 substrates buffered by three types of correlated oxide electrodes, La0.67Sr0.33MnO3, LaNiO3, and SrIrO3. Piezoresponse force microscopy studies show that the 50-nm PbZr0.2Ti0.8O3 films are uniformly polarized in the as-grown states, with the patterned domain structures persisting above 700 °C. For all three types of films, the domain wall roughness is dominated by two-dimensional (2D) random bond disorder at room temperature, and transitions to 1D thermal roughening upon heating. The roughness exponent ζ increases progressively from 0.3 to 0.5 within a temperature window that depends on the bottom conducting oxide type, from which we extracted the distribution of disorder pinning energy. We discuss the possible origins of the disorder pinning and the effect of the correlated oxide electrodes on the energy landscape of DW motion

Bis{μ-4,4′,6,6′-tetra­bromo-2,2′-[o-phenyl­enebis(nitrilo­methyl­idyne)]­diphenol­ato}­bis­[(dimethyl­formamide)cadmium(II)]

The Schiff base ligand derived from the condensation of 3,5-dibromo­salicylaldehyde and 1,2-phenyl­enediamine, in the presence of dimethyl­formamide, forms the centrosymmetric title neutral binuclear distorted complex, [Cd2(C20H10Br4N2O2)2(C3H7NO)2], with the two octa­hedral Cd atoms linked by two O atoms. All bond lengths and angles show normal values

Symmetry Reductions of (2 + 1)-Dimensional CDGKS Equation and Its Reduced Lax Pairs

With the aid of symbolic computation, we obtain the symmetry transformations of the (2 + 1)-dimensional Caudrey-Dodd-Gibbon-Kotera-Sawada (CDGKS) equation by Lou’s direct method which is based on Lax pairs. Moreover, we use the classical Lie group method to seek the symmetry groups of both the CDGKS equation and its Lax pair and then reduce them by the obtained symmetries. In particular, we consider the reductions of the Lax pair completely. As a result, three reduced (1 + 1)-dimensional equations with their new Lax pairs are presented and some group-invariant solutions of the equation are given

HAO2 inhibits malignancy of clear cell renal cell carcinoma by promoting lipid catabolic process.

Hydroxy acid oxidase 2 (HAO2) has been reported to inhibit tumor progression through metabolic pathway. The current study was designed to evaluate the prognostic significance and probable mechanism of HAO2 in patients with clear cell renal cell carcinoma (ccRCC). The study screened The Cancer Genome Atlas Kidney Clear Cell Carcinoma (TCGA-KIRC) database for patients with ccRCC having complete clinical information and HAO2 expression. Low HAO2 was associated with shorter overall survival (OS) and shorter disease-free survival (DFS). Gene set enrichment analysis (GSEA) showed HAO2 was associated with neutral lipid catabolic process, metabolic process, lipid oxidation, epithelial-mesenchymal transition (EMT), and Kirsten rat sarcoma viral oncogene signaling (KRAS). Western blot analysis and immunohistochemistry analysis checked HAO2 expression in ccRCC cancer tissues, normal tissues, and renal cancer cell lines. HAO2 was downregulated in ccRCC cancer tissues and ccRCC cell lines when compared with their control group. Overexpression of HAO2 by plasmid promoted lipid catabolic process, eliminated lipid accumulation, inhibited KRAS expression, controlled the proliferation, migration, and invasion activity of ccRCC tumor cells. Our results indicated that HAO2 inhibits malignancy ccRCC by promoting lipid catabolic process, HAO2 could be an effective molecular marker and treatment for ccRCC

A note on the boundedness in a chemotaxis-growth system with nonlinear sensitivity

This paper deals with a parabolic-elliptic chemotaxis-growth system with nonlinear sensitivity \begin{equation*}\label{1a} \begin{cases} u_t=\Delta u-\chi\nabla\cdot(\psi(u)\nabla v)+f(u), &(x,t)\in \Omega\times (0,\infty), \\ 0=\Delta v-v+g(u), &(x,t)\in \Omega\times (0,\infty), \end{cases} \end{equation*} under homogeneous Neumann boundary conditions in a smooth bounded domain $\Omega\subset \mathbb{R}^{n}$ $(n\geq1)$, where $\chi>0$, the chemotactic sensitivity $\psi(u)\leq(u+1)^{q}$ with $q>0$, $g(u)\leq(u+1)^{l}$ with $l\in \mathbb{R}$ and $f(u)$ is a logistic source. The main goal of this paper is to extend a previous result on global boundedness by Zheng et al. [J. Math. Anal. Appl. 424(2015), 509–522] under the condition that $1\leq q+l<\frac{2}{n}+1$ to the case $q+l<1$