14 research outputs found

    Method of magnetic resonance investigation

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    Introduction C’est un grand privilĂšge pour moi d’intervenir aprĂšs des exposĂ©s si documentĂ©s et captivants. C’est aussi une grande difficulté : il m’est demandĂ© aujourd’hui de donner le point de vue d’un ingĂ©nieur des Mines, alors mĂȘme que tant de grands et illustres exemples inviteraient avant tout au silence de l’humilitĂ©. Je me contenterai donc d’essayer de vous faire partager mon Ă©tonnement sur quelques points qui me paraissent porteurs de rĂ©flexions encore d’actualitĂ©. Je grouperai ces po..

    The associations of self-reported salt-intake and spot urine sodium with home blood pressure

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    A limited number of studies have suggested a nonlinear association between spot urine (SU) sodium concentration and office blood pressure (BP). We examined how SU sodium concentration and dietary salt obtained from a food frequency questionnaire associate with more accurately measured home BP in a large, nationwide population sample. We included 1453 participants in cross-sectional and 880 participants in 11-year longitudinal analyses. We investigated associations between baseline salt/sodium variables and (i) baseline and follow-up home BP; and (ii) prevalent and incident hypertension with linear and logistic regression models. We observed positive associations (Îȱstandard error) between salt/sodium variables and BP in unadjusted models. SU sodium concentration associated with baseline systolic (0.04±0.01, P<0.001) and diastolic (0.02±0.01, P<0.001) BP and follow-up systolic (0.03±0.01, P=0.005) and diastolic (0.02±0.01, P<0.001) BP. Dietary salt intake was associated with baseline (0.58±0.20, P=0.003) and follow-up (0.73±0.23, P=0.001) systolic BP. Unadjusted odds of prevalent hypertension were higher in the highest quintile as compared to the lowest quintile of SU sodium concentration (odds ratio 1.66, 95% confidence interval 1.20-2.31). SU or dietary salt intake were not associated with incident hypertension. After adjustments for sex, age, body mass index and alcohol intake, none of the aforementioned associations remained statistically significant. We found no evidence of a J-shaped association between the salt/sodium variables and BP or hypertension. In conclusion, SU sodium concentration and dietary salt intake associated with home BP and hypertension only in some of the unadjusted models. Our results underscore that feasible estimation of sodium intake remains challenging in epidemiology.ErÀÀt tutkimukset ovat ehdottaneet kertavirtsanĂ€ytteen natriumpitoisuuden ja vastaanottoverenpaineiden vĂ€lisen yhteyden olevan epĂ€lineaarinen. TĂ€mĂ€n tutkimuksen tarkoituksena oli tutkia kahden eri suolamuuttujan, kertavirtsanĂ€ytteen natriumpitoisuuden ja ravintofrekvenssikyselyllĂ€ mitatun suolansaannin, ja verenpainetasoa tarkemmin kuvaavan kotiverenpaineen yhteyttĂ€ suuressa vĂ€estöaineistossa. SisĂ€llytimme 1453 osallistujaa poikittaisiin ja 880 osallistujaa pitkittĂ€isiin analyyseihin (11 vuoden seuranta-aika). Suolamuuttujien yhteyttĂ€ tarkasteltiin (i) lĂ€htötilanteen ja seurantatilanteen kotiverenpaineisiin ja (ii) verenpainetaudin vallitsevuuteen ja ilmaantuvuuteen nĂ€hden lineaarisilla ja logistisilla regressiomalleilla. Positiivinen yhteys (Îȱkeskihajonta) havaittiin vakioimattomissa malleissa suolamuuttujien ja verenpainearvojen vĂ€lillĂ€. Kertavirtsan natriumpitoisuus oli yhteydessĂ€ lĂ€htötilanteen systoliseen (0.04±0.01, P<0.001) ja diastoliseen (0.02±0.01, P<0.001) verenpaineeseen, sekĂ€ seurantatilanteen systoliseen (0.03±0.01, P=0.005) ja diastoliseen (0.02±0.01, P<0.001) verenpaineeseen. RavintokyselyllĂ€ mitattu suolansaanti oli yhteydessĂ€ systoliseen verenpaineeseen lĂ€htötilanteessa (0.58±0.20, P=0.003) ja seurantatilanteessa (0.73±0.23, P=0.001). Vakioimattomissa malleissa vallitsevan verenpainetaudin todennĂ€köisyys oli korkeampi korkeimmassa kertavirtsan natriumpitoisuuden kvantiilissa verrattuna alimpaan kvantiiliin (vetosuhde 1.66, 95% luottamusvĂ€li 1.20-2.31). Suolamuuttujat eivĂ€t olleet yhteydessĂ€ verenpainetaudin korkeampaan ilmaantuvuuteen. Kun edellĂ€ mainitut tilastolliset mallit vakioitiin lĂ€htötilanteen iĂ€llĂ€, sukupuolella, painoindeksillĂ€ ja alkoholinkĂ€ytöllĂ€, yhteydet eivĂ€t sĂ€ilyneet tilastollisesti merkitsevinĂ€. J-kĂ€yrĂ€n mallisesta yhteydestĂ€ suolamuuttujien ja verenpaineen vĂ€lillĂ€ ei löytynyt viitteitĂ€. Tulokset alleviivaavat, ettĂ€ kĂ€ytĂ€nnöllinen suolansaannin arviointi on edelleen haastavaa epidemiologiassa

    Vad har svenska företag för syn pÄ sovande data och hur hanterar svenska företag sovande data med avseende pÄ identifiering och lagring?

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    Alltför mÄnga organisationer har datalager innehÄllande stora mÀngder sovande data, det vill sÀga data som sÀllan eller aldrig anvÀnds. Sovande data pÄverkar en organisations datalager negativt eftersom den försÀmrar datalagrets prestanda, kostar pengar i onödan och pÄverkar datalagrets infrastruktur negativt. Enligt Inmon, Glassey och Welch (1997) Àr det en mycket svÄr och komplex process att rensa ut sovande data ur sitt datalager. Administratören mÄste ha kunskap om vilka datatabeller i datalagret som anvÀnds och vilka rader utav data som anvÀnds för att kunna ta bort data frÄn datalagret. Enligt Inmon m.fl. (1997) Àr det nödvÀndigt att anvÀnda nÄgon form av metod för att kunna identifiera vilken data i datalagret som kan klassas som sovande data. Syftet med arbetet Àr att undersöka hur svenska företag hanterar sovande data för att ta reda pÄ vilka metoder de anvÀnder för att identifiera sovande data och vad de gör med den datan som blir klassad som sovande data

    Användning av finansiella rapporter för att slå marknaden : - En utveckling av Piotroskis investeringsstrategi

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    Syftet med denna undersökning är att utveckla den investeringsstrategi som Piotroski (2000) tog fram, grundad på fundamentalanalys, genom att sammanlänka variabler från Lev &amp; Thiagarajan (1993) som visat sig vara värderelevanta indikationer på företags rapporterade resultat. För att genomföra detta utvecklas en modell med Piotroskis (2000) F-score som grund. Antalet signaler i modellen utökas från 9 till 12 stycken. Undersökningen genomförs med data från Stockholmsbörsen under perioden 1998 – 2012. Resultatet visar att både den utvecklade modellen och Piotroskis modell presterar en positiv marknadsjusterad avkastning under hela undersökningsperioden. Samtidigt ökar antalet investeringar i den utvecklade modellen vilket bidrar till en minskad risk och en ökad spridning.

    The associations of self-reported salt-intake and spot urine sodium with home blood pressure

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    Purpose: A limited number of studies have suggested a nonlinear association between spot urine (SU) sodium concentration and office blood pressure (BP). We examined how SU sodium concentration and dietary salt obtained from a food frequency questionnaire are associated with more accurately measured home BP in a large, nationwide population sample. Materials and methods: We included 1398 participants in cross-sectional and 851 participants in 11-year longitudinal analyses. We investigated associations between baseline salt/sodium variables and (i) baseline and follow-up home BP; and (ii) prevalent and incident hypertension with linear and logistic regression models. Results: We observed positive associations (ÎČ ± standard error) between salt/sodium variables and BP in unadjusted models. SU sodium concentration associated with baseline systolic (0.04 ± 0.01, p < 0.001) and diastolic (0.02 ± 0.01, p < 0.001) BP and follow-up systolic (0.03 ± 0.01, p = 0.003) and diastolic (0.02 ± 0.01, p < 0.001) BP. Dietary salt intake was associated with baseline (0.52 ± 0.19, p = 0.008) and follow-up (0.57 ± 0.20, p = 0.006) systolic BP. Compared to the lowest quintile of SU sodium concentration, the highest quintile had greater odds of prevalent hypertension (odds ratio [OR] 1.57, 95% confidence interval [CI] 1.12–2.19) and the second highest quintile with incident hypertension (OR 1.86, 95% CI 1.05–3.34). Unadjusted odds of incident hypertension were higher in the highest as compared to the lowest quintile of dietary salt intake (OR 1.83, 95% CI 1.01–3.35). After adjustments for sex, age, plasma creatinine concentration and alcohol intake, none of the aforementioned associations remained statistically significant. We found no evidence of a J-shaped association between the salt/sodium variables and BP or hypertension. Conclusion: SU sodium concentration and dietary salt intake are associated with home BP and hypertension only in some of the unadjusted models. Our results underscore that feasible estimation of sodium intake remains challenging in epidemiology
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