Analyticity constraints for hadron amplitudes : going high to heal low energy issues

Analyticity constitutes a rigid constraint on hadron scattering amplitudes. This property is used to relate models in different energy regimes. Using meson photoproduction as a benchmark, we show how to test contemporary low-energy models directly against high-energy data. This method pinpoints deficiencies of the models and treads a path to further improvement. The implementation of this technique enables one to produce more stable and reliable partial waves for future use in hadron spectroscopy and new physics searches

Determination of the pole position of the lightest hybrid meson candidate

Mapping states with explicit gluonic degrees of freedom in the light sector is a challenge, and has led to controversies in the past. In particular, the experiments have reported two different hybrid candidates with spin-exotic signature, pi1(1400) and pi1(1600), which couple separately to eta pi and eta' pi. This picture is not compatible with recent Lattice QCD estimates for hybrid states, nor with most phenomenological models. We consider the recent partial wave analysis of the eta(') pi system by the COMPASS collaboration. We fit the extracted intensities and phases with a coupled-channel amplitude that enforces the unitarity and analyticity of the S-matrix. We provide a robust extraction of a single exotic pi1 resonant pole, with mass and width 1564 +- 24 +- 86 MeV and 492 +- 54 +- 102 MeV, which couples to both eta(') pi channels. We find no evidence for a second exotic state. We also provide the resonance parameters of the a2(1320) and a2'(1700).Comment: 6 pages + 3 pages of supplemental material. Version to appear on Phys.Rev.Let

RING-Type E3 Ubiqitin Ligase BarleyGenes(HvYrg1–2) Control Characteristics of Both Vegetative Organs and Seeds as Yield Components

Previously, studies on RING-type E3 ubiquitin ligases in cereals were preferentially focused on GW2 genes primarily controlling seed parameters in rice and wheat. Here we report cloning two HvYrg genes from barley that share significant homology with rice GW2 gene. In antisense genotypes efficiency of gene silencing varied between genes and transgenic lines: ASHvYrg1: 30-50% and ASHvYrg2: 20-27%. Reduced activity of both genes altered shoot system with increasing number of side shoots. Changes in leaf width, weight, or plant weight and height reached significant levels in some transgenic lines. Lowering expression of the two barley HvYrg genes caused opposite responses in spike development. Plants with ASHvYrg1 gene construct showed earlier heading time and prolonged grain-filling period, while plants from ASHvYrg2 genotype flowered in delay. Digital imaging of root development revealed that down-regulation of HvYrg1 gene variant stimulated root growth, while ASHvYrg2 plants developed reduced root system. Comparison of seed parameters indicated an increase in thousand grain weight accompanied with longer and wider seed morphology. In summary we conclude that in contrast to inhibition of GW2 genes in rice and wheat plants, down-regulation of the barely HvYrg genes caused substantial changes in vegetative organs in addition to alteration of seed parameters

The hadronic vacuum polarization contribution to the muon g − 2 from lattice QCD

We present a calculation of the hadronic vacuum polarization contribution to the muon anomalous magnetic moment, $a_\mu^{\mathrm hvp}$, in lattice QCD employing dynamical up and down quarks. We focus on controlling the infrared regime of the vacuum polarization function. To this end we employ several complementary approaches, including Pad\'e fits, time moments and the time-momentum representation. We correct our results for finite-volume effects by combining the Gounaris-Sakurai parameterization of the timelike pion form factor with the L\"uscher formalism. On a subset of our ensembles we have derived an upper bound on the magnitude of quark-disconnected diagrams and found that they decrease the estimate for $a_\mu^{\mathrm hvp}$ by at most 2%. Our final result is $a_\mu^{\mathrm hvp}=(654\pm32\,{}^{+21}_{-23})\cdot 10^{-10}$, where the first error is statistical, and the second denotes the combined systematic uncertainty. Based on our findings we discuss the prospects for determining $a_\mu^{\mathrm hvp}$ with sub-percent precision.Comment: 42 pages, 7 figures, version published in JHE

Impact of HIV Infection and Kaposi Sarcoma on Human Herpesvirus-8 Mucosal Replication and Dissemination in Uganda

Kaposi sarcoma (KS) is the leading cause of cancer in Uganda and occurs in people with and without HIV. Human herpesvirus-8 (HHV-8) replication is important both in transmission of HHV-8 and progression to KS. We characterized the sites and frequency of HHV-8 detection in Ugandans with and without HIV and KS.Participants were enrolled into one of four groups on the basis of HIV and KS status (HIV negative/KS negative, HIV positive/KS negative, HIV negative/KS positive, and HIV positive/KS positive). Participants collected oral swabs daily and clinicians collected oral swabs, anogenital swabs, and plasma samples weekly over 4 weeks. HHV-8 DNA at each site was quantified by polymerase chain reaction (PCR).78 participants collected a total of 2063 orals swabs and 358 plasma samples. Of these, 428 (21%) oral swabs and 96 (27%) plasma samples had detectable HHV-8 DNA. HHV-8 was detected more frequently in both the oropharynx of persons with KS (24 (57%) of 42 persons with KS vs. 8 (22%) of 36 persons without, p = 0.002) and the peripheral blood (30 (71%) of 42 persons with KS vs. 8 (22%) of 36 persons without, p<0.001). In a multivariate model, HHV-8 viremia was more frequent among men (IRR = 3.3, 95% CI = 1.7-6.2, p<0.001), persons with KS (IRR = 3.9, 95% CI = 1.7-9.0, p = 0.001) and persons with HIV infection (IRR = 1.7, 95% CI = 1.0-2.7, p = 0.03). Importantly, oral HHV-8 detection predicted the subsequent HHV-8 viremia. HHV-8 viremia was significantly more common when HHV-8 DNA was detected from the oropharynx during the week prior than when oral HHV-8 was not detected (RR = 3.3, 95% CI = 1.8-5.9 p<0.001). Genital HHV-8 detection was rare (9 (3%) of 272 swabs).HHV-8 detection is frequent in the oropharynx and peripheral blood of Ugandans with endemic and epidemic KS. Replication at these sites is highly correlated, and viremia is increased in men and those with HIV. The high incidence of HHV-8 replication at multiple anatomic sites may be an important factor leading to and sustaining the high prevalence of KS in Uganda