N′-(4-Chloro­benzyl­idene)-2-hydroxy­benzohydrazide

The title mol­ecule, C14H11ClN2O2, adopts a trans configuration with respect to the C=N double bond. An intra­molecular N—H⋯O hydrogen bond contributes to mol­ecular conformation and the two benzene rings form a dihedral angle of 17.9 (8)°. In the crystal structure, inter­molecular O—H⋯O hydrogen bonds link the mol­ecules into chains running along [10]

[2-Hydroxy-N′-(4-oxo-4-phenyl­butan-2-yl­idene)benzohydrazidato(2−)]pyridine­copper(II)

The mononuclear title complex, [Cu(C17H14N2O3)(C5H5N)], was synthesized by the reaction of CuCl2·2H2O with N-(4-oxo-4-phenyl­butan-2-yl­idene)benzohydrazide (H2 L). The central CuII atom exhibits a distorted square-planar coordination geometry, defined by two O atoms, one N atom from the ligand and one pyridine N atom with Cu—N distances of 1.874 (4) and 1.963 (4) Å, while the Cu—O distances are 1.857 (3) and 1.890 (3) Å. An intra­molecular O—H⋯N inter­action occurs

(E)-N′-(3-Fluoro­benzyl­idene)-2-hydroxy­benzohydrazide

The title compound, C14H11FN2O2, adopts an E or trans configuration with respect to the C=N bond. An intra­molecular N—H⋯O hydrogen bond contributes to the relatively planarity of the mol­ecular conformation; the two benzene rings are inclined to one another by 12.5 (2)°. In the crystal structure, inter­molecular O—H⋯O hydrogen bonds link the mol­ecules into chains running parallel to the c axis

2-[4-(4-Methoxy­phen­yl)-5-(2-pyrid­yl)-4H-1,2,4-triazol-3-yl]phenol

In the title compound, C20H16N4O2, the benzene rings of the 2-hydroxy­phenyl and 4-methoxy­lphenyl groups form dihedral angles of 64.02 (8) and 77.39 (7)°, respectively, with the mean plane of the triazole ring. The dihedral angle between the triazole ring mean plane and the pyridyl ring is 9.61 (8)°. In the crystal, inter­molecular N—H⋯O hydrogen bonds link the mol­ecules into zigzag chains propagating in [010]

Epitaxial antiperovskite/perovskite heterostructures for materials design

We demonstrate fabrication of atomically sharp interfaces between nitride antiperovskite Mn$_{3}$GaN and oxide perovskites (La$_{0.3}$Sr$_{0.7}$)(Al$_{0.65}$Ta$_{0.35}$)O$_{3}$ (LSAT) and SrTiO$_{3}$ as paradigms of nitride-antiperovskite/oxide-perovskite heterostructures. Using a combination of scanning transmission electron microscopy (STEM), atomic-resolution spectroscopic techniques, and first-principle calculations, we investigated the atomic-scale structure, composition, and boding at the interface. We show that the epitaxial growth between the antiperovskite and perovskite compounds is mediated by a coherent interfacial monolayer that connects the two anti-structures. We anticipate our results to be a major step for the development of functional antiperovskite/perovskite heterostructures opening to harness a combination of their functional properties including topological properties for ultra low power applications

Symmetry Control of Unconventional Spin–Orbit Torques in IrO\u3csub\u3e2\u3c/sub\u3e

Spin–orbit torques generated by a spin current are key to magnetic switching in spintronic applications. The polarization of the spin current dictates the direction of switching required for energy-efficient devices. Conventionally, the polarizations of these spin currents are restricted to be along a certain direction due to the symmetry of the material allowing only for efficient in-plane magnetic switching. Unconventional spin–orbit torques arising from novel spin current polarizations, however, have the potential to switch other magnetization orientations such as perpendicular magnetic anisotropy, which is desired for higher density spintronic-based memory devices. Here, it is demonstrated that low crystalline symmetry is not required for unconventional spin–orbit torques and can be generated in a nonmagnetic high symmetry material, iridium dioxide (IrO2), using epitaxial design. It is shown that by reducing the relative crystalline symmetry with respect to the growth direction large unconventional spin currents can be generated and hence spin–orbit torques. Furthermore, the spin polarizations detected in (001), (110), and (111) oriented IrO2 thin films are compared to show which crystal symmetries restrict unconventional spin transport. Understanding and tuning unconventional spin transport generation in high symmetry materials can provide a new route towards energy-efficient magnetic switching in spintronic devices

Dynamic prostatic and laser-ablated lesion volume change after transperineal laser ablation in canine: preliminary observation and its clinical significance.

AIM: The purpose of this study is to observe the volume change of prostate and laser-ablated lesions in the canine and to explore the mechanism and clinical significance through histopathology. MATERIALS AND METHODS: Transperineal laser ablation (TPLA) was performed under the guidance of transrectal ultrasound (TRUS) in eight canines. Two canines were sacrificed 1 day and 1 week after TPLA, respectively. The remaining six canines were sacrificed after finishing transrectal contrast-enhanced ultrasound (TR-CEUS) at three phases. RESULTS: The prostatic volumes immediately following TPLA and 1 week later were larger than before TPLA (20.1 ± 3.9 vs 17.1 ± 3.8 ml; 21.7 ± 3.6 vs 17.1 ± 3.8 ml, p \u3c 0.05), but 1 month later, returned to the preoperative level (17.4 ± 3.2 ml). At three time points, the mean volumes of laser-ablated lesions at 3 W/600 J were 0.6 ± 0.2, 1.1 ± 0.4, and 1.7 ± 0.5 ml, respectively, while those of laser-ablated lesions at 3 W/1200 J were 1.2 ± 0.2, 1.6 ± 0.3, and 2.2 ± 0.5 ml, respectively. The mean volumes of laser-ablated lesions increased significantly over time after TPLA (p \u3c 0.050). CONCLUSION: The prostate volume transient enlarges after TPLA, which prompts for clinical application that it should prolong appropriately the duration of urinary catheterization to avoid acute urinary retention. Many inflammatory cells were observed in the laser-ablated lesions and adjacent normal prostate parenchyma through histopathology. It is speculated that the inflammatory response is involved in the progression of tissue damage