Stephan Merl, Der Agrarmarkt und die Neue Ökonomische Politik. Die Anfänge staatlicher Lenkung der Landwirtschaft in der Sowjetunion

Schroder Hans-Henning. Stephan Merl, Der Agrarmarkt und die Neue Ökonomische Politik. Die Anfänge staatlicher Lenkung der Landwirtschaft in der Sowjetunion. In: Revue d'études comparatives Est-Ouest, vol. 14, 1983, n°1. pp. 146-148

Stephan Merl, Der Agrarmarkt und die Neue Ökonomische Politik. Die Anfänge staatlicher Lenkung der Landwirtschaft in der Sowjetunion

Schroder Hans-Henning. Stephan Merl, Der Agrarmarkt und die Neue Ökonomische Politik. Die Anfänge staatlicher Lenkung der Landwirtschaft in der Sowjetunion. In: Revue d'études comparatives Est-Ouest, vol. 14, 1983, n°1. pp. 146-148

Apoptotic cell death in disease-Current understanding of the NCCD 2023

Apoptosis is a form of regulated cell death (RCD) that involves proteases of the caspase family. Pharmacological and genetic strategies that experimentally inhibit or delay apoptosis in mammalian systems have elucidated the key contribution of this process not only to (post-)embryonic development and adult tissue homeostasis, but also to the etiology of multiple human disorders. Consistent with this notion, while defects in the molecular machinery for apoptotic cell death impair organismal development and promote oncogenesis, the unwarranted activation of apoptosis promotes cell loss and tissue damage in the context of various neurological, cardiovascular, renal, hepatic, infectious, neoplastic and inflammatory conditions. Here, the Nomenclature Committee on Cell Death (NCCD) gathered to critically summarize an abundant pre-clinical literature mechanistically linking the core apoptotic apparatus to organismal homeostasis in the context of disease

Looking at the Distant Universe with the MeerKAT Array: Discovery of a luminous OH megamaser at z>0.5z > 0.5

In the local Universe, OH megamasers (OHMs) are detected almost exclusively in infrared-luminous galaxies, with a prevalence that increases with IR luminosity, suggesting that they trace gas-rich galaxy mergers. Given the proximity of the rest frequencies of OH and the hyperfine transition of neutral atomic hydrogen (HI), radio surveys to probe the cosmic evolution of HI in galaxies also offer exciting prospects for exploiting OHMs to probe the cosmic history of gas-rich mergers. Using observations for the Looking At the Distant Universe with the MeerKAT Array (LADUMA) deep HI survey, we report the first untargeted detection of an OHM at $z > 0.5$, LADUMA J033046.20$-$275518.1 (nicknamed "Nkalakatha"). The host system, WISEA J033046.26$-$275518.3, is an infrared-luminous radio galaxy whose optical redshift $z \approx 0.52$ confirms the MeerKAT emission line detection as OH at a redshift $z_{\rm OH} = 0.5225 \pm 0.0001$ rather than HI at lower redshift. The detected spectral line has 18.4$\sigma$ peak significance, a width of $459 \pm 59\,{\rm km\,s^{-1}}$, and an integrated luminosity of $(6.31 \pm 0.18\,{\rm [statistical]}\,\pm 0.31\,{\rm [systematic]}) \times 10^3\,L_\odot$, placing it among the most luminous OHMs known. The galaxy's far-infrared luminosity $L_{\rm FIR} = (1.576 \pm 0.013) \times 10^{12}\,L_\odot$ marks it as an ultra-luminous infrared galaxy; its ratio of OH and infrared luminosities is similar to those for lower-redshift OHMs. A comparison between optical and OH redshifts offers a slight indication of an OH outflow. This detection represents the first step towards a systematic exploitation of OHMs as a tracer of galaxy growth at high redshifts

Apoptotic cell death in disease—current understanding of the NCCD 2023

Apoptosis is a form of regulated cell death (RCD) that involves proteases of the caspase family. Pharmacological and genetic strategies that experimentally inhibit or delay apoptosis in mammalian systems have elucidated the key contribution of this process not only to (post-)embryonic development and adult tissue homeostasis, but also to the etiology of multiple human disorders. Consistent with this notion, while defects in the molecular machinery for apoptotic cell death impair organismal development and promote oncogenesis, the unwarranted activation of apoptosis promotes cell loss and tissue damage in the context of various neurological, cardiovascular, renal, hepatic, infectious, neoplastic and inflammatory conditions. Here, the Nomenclature Committee on Cell Death (NCCD) gathered to critically summarize an abundant pre-clinical literature mechanistically linking the core apoptotic apparatus to organismal homeostasis in the context of disease

Apoptotic cell death in disease-Current understanding of the NCCD 2023.

Apoptosis is a form of regulated cell death (RCD) that involves proteases of the caspase family. Pharmacological and genetic strategies that experimentally inhibit or delay apoptosis in mammalian systems have elucidated the key contribution of this process not only to (post-)embryonic development and adult tissue homeostasis, but also to the etiology of multiple human disorders. Consistent with this notion, while defects in the molecular machinery for apoptotic cell death impair organismal development and promote oncogenesis, the unwarranted activation of apoptosis promotes cell loss and tissue damage in the context of various neurological, cardiovascular, renal, hepatic, infectious, neoplastic and inflammatory conditions. Here, the Nomenclature Committee on Cell Death (NCCD) gathered to critically summarize an abundant pre-clinical literature mechanistically linking the core apoptotic apparatus to organismal homeostasis in the context of disease

Apoptotic cell death in disease—Current understanding of the NCCD 2023

Apoptosis is a form of regulated cell death (RCD) that involves proteases of the caspase family. Pharmacological and genetic strategies that experimentally inhibit or delay apoptosis in mammalian systems have elucidated the key contribution of this process not only to (post-)embryonic development and adult tissue homeostasis, but also to the etiology of multiple human disorders. Consistent with this notion, while defects in the molecular machinery for apoptotic cell death impair organismal development and promote oncogenesis, the unwarranted activation of apoptosis promotes cell loss and tissue damage in the context of various neurological, cardiovascular, renal, hepatic, infectious, neoplastic and inflammatory conditions. Here, the Nomenclature Committee on Cell Death (NCCD) gathered to critically summarize an abundant pre-clinical literature mechanistically linking the core apoptotic apparatus to organismal homeostasis in the context of disease