89 research outputs found

    Wind braking of magnetars

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    (adapted)Considering recent observations challenging the traditional magnetar model, we explore the wind braking of magnetars. There is evidence for strong multipole magnetic fields in active magnetars, but the dipole field inferred from spin down measurements may be strongly biased by a particle wind. Recent challenging observations of magnetars may be explained naturally in the wind braking scenario: (1) The supernova energies of magnetars are of normal value; (2) The non-detection in Fermi observations of magnetars; (3) The problem posed by the low-magnetic field soft gamma-ray repeaters; (4) The relation between magnetars and high magnetic field pulsars; (5) A decreasing period derivative during magnetar outbursts. Transient magnetars may still be magnetic dipole braking. This may explain why low luminosity magnetars are more likely to have radio emissions. In the wind braking scenario, magnetars are neutron stars with strong multipole field. For some sources, a strong dipole field may be no longer needed. A magnetism-powered pulsar wind nebula and a braking index smaller than three are the two predictions of the wind braking model.Comment: 34 pages, 1 table, 6 figures, accepted by Ap

    EYE MOVEMENT DISORDERS IN BONE MARROW TRANSPLANT PATIENTS ON FK506 (TACROLIMUS) AND GANCICLOVIR

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    7th International Eurasian Hematology Congress -- OCT 13-16, 2016 -- Istanbul, TURKEYWOS: 000391180600067

    The use of port-a-cath in children with hemophilia in Cukurova region

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    WOS: 000457408400176

    Erythrocyte pyruvate kinase activity during chemotherapy in children with leukemia and lymphoma.

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    PubMedID: 12518995OBJECTIVES: To determine both erythrocyte pyruvate kinase activity (ePKA) at the time of diagnosis of patients with acute leukemia or lymphoma and the differences in the ePKA profiles during the malignant disease and its chemotherapy. METHODS: A prospective, longitudinal clinical study was performed involving 57 patients, 10 were the ones with relapse of acute lymphoblastic leukemia, 32 were the ones with acute lymphoblastic leukemia (ALL) and 15 were the ones with lymphoma. None of the subjects in this study group received treatment or blood transfusion before the study, except the ones diagnosed with relapse of ALL. Forty two healthy children were also selected to form the control group. In order to measure ePKA, blood samples were taken for five times, with 1.5 months apart between each other during the study. Statistical analysis were done by using Wilcoxon's signed rank test, Kruskall-Wallis with Mann-Whitney U Test and Spearman rank correlation coefficient test. RESULTS: The ePKA of the patients with relapse of ALL, and ALL, but not the patients with lymphoma, at the time of diagnosis were found to be lower compared to the one's in the control group (respectively p = 0.001, p = 0.003). The comparison between the first ePKA samples and the third ePKA samples of the patients with both ALL and lymphoma showed a significant increase (respectively p = 0.006, and p = 0.047). CONCLUSION: The measurement of ePKA can be considered for follow-up the neoplastic treatment due to the fact that it is detected to be low in leukemia and relapse of ALL and in normal values after chemotherapy. However, more long-term studies, including more number of cases, are required to be carried out in order to prove the accuracy of this hypothesis. (Tab. 2, Fig. 1, Ref. 28.

    NUTRITION, IMMUNITY AND INFECTIONS - T-LYMPHOCYTE SUBPOPULATIONS IN PROTEIN-ENERGY MALNUTRITION

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    Protein energy malnutrition (PEM) is one of the most frequent causes of secondary immune deficiency states. Alterations either in cellular or humoral immune mechanisms increase the susceptibility to infections in the malnourished organism. Infections aggravate the interrelationship of malnutrition to immune deficiency and infections, resulting in future adverse effects of malnutrition on humoral and cellular immune systems, IgG, IgM, IgA, C3, and T lymphocyte subpopulations were identified in 29 patients with PEM and 15 healthy infants serving as the control group, ranging between 3 and 24 months of age. Patients with PEM demonstrated elevated levels of IgG, IgM and IgA when compared to the control group (P<0.01, P<0.01, P<0.01), C3 levels were significantly lower than the values of the control group (P<0.01)
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