Food Waste in Public Food Service Sector-Surplus and Leftovers

Food waste occurs at various stages of the food supply chain, starting from the production stage on farms to processing, distribution, and consumption. It is an important global problem that has social, environmental, ethical, and economic costs. The present work aims to discuss the differences in waste generated from surpluses or leftovers, the monitoring systems of food waste in public food services, and the good practices to reduce food waste. Besides the accessible knowledge about reducing waste, tonnes of food were daily discarded and wasted. To achieve a sustainable food system, food waste must be reduced significantly. Collaborative efforts are necessary to achieve significant reductions in food waste for achieving SDG 12.3 in public food services. Thus, policies that support or enforce larger implementation of best practices are needed. Knowledge about food waste is important to change attitudes and behaviors toward surplus food and leftovers. Some best practices can include promoting awareness campaigns to educate staff about the impact of food waste and the best practices to reduce it, including information on proper storage, portion control, and meal planning; implementing inventory management systems; donating surplus food via collaboration with food banks and charities; optimizing portion sizes, ensuring they are adequate and manageable for customers, reducing the likelihood of leftover food on the plate; allowing customers to order smaller quantities or share a meal; providing packaging to allow customers take their leftovers home for later consumption. The procedures to be adopted by public food services should provide guidance for the selection of products that can be donated. However, specific temperature requirements depending on the food category should be complied with. This paper enlarges the understanding of surpluses and leftovers and the best practices that could be implemented in public food services to minimize food waste.The participation of CG is supported by FCT/UIDB/04033/2020. The participation of FN is supported by FCT UIDB/00616/2020 and UIDP/00616/2020. The participation of SS and CS were supported by the projects UIDB/CVT/00772/2020 and LA/P/0059/2020 funded by the Portuguese Foundation for Science and Technology (FCT). CG, FN and CS would like to thank to project I & D AgriFood XXI, operation NORTE-01-0145-FEDER43 000041, co-funded by European Regional Development Fund (FEDER) through NORTE 2020 (Programa Operacional Regional do Norte 2014/2020)

The Expression of Chemokines Is Downregulated in a Pre-Clinical Model of TTR V30M Amyloidosis

Inflammation is a hallmark of several neurodegenerative disorders including hereditary amyloidogenic transthyretin amyloidosis (ATTRv). ATTRv is an autosomal dominant neurodegenerative disorder with extracellular deposition of mutant transthyretin (TTR) aggregates and fibrils, particularly in nerves and ganglia of the peripheral nervous system. Nerve biopsies from ATTRv patients show increased cytokine production, but interestingly no immune inflammatory cellular infiltrate is observed around TTR aggregates. Here we show that as compared to Wild Type (WT) animals, the expression of several chemokines is highly downregulated in the peripheral nervous system of a mouse model of the disease. Interestingly, we found that stimulation of mouse Schwann cells (SCs) with WT TTR results in the secretion of several chemokines, a process that is mediated by toll-like receptor 4 (TLR4). In contrast, the secretion of all tested chemokines is compromised upon stimulation of SCs with mutant TTR (V30M), suggesting that V30M TTR fails to activate TLR4 signaling. Altogether, our data shed light into a previously unappreciated mechanism linking TTR activation of SCs and possibly underlying the lack of inflammatory response observed in the peripheral nervous system of ATTRv patients.The work was funded by the project Norte-01-0145-FEDER-000008 - Porto Neurosciences and Neurologic Disease Research Initiative at I3S, supported by Norte Portugal Regional Operational Programme (NORTE 2020), under the PORTUGAL 2020 Partnership Agreement, through the European Regional Development Fund (FEDER). JM was supported by FCT with a PhD fellowship SFRH/BD/129345/2017. MS is funded by FCT through Estı́mulo Individual ao Emprego Cientı́fico

Thermal inactivation of Byssochlamys nivea in pineapple nectar combined with preliminary high pressure treatments

Byssochlamys nivea is a thermal resistant filamentous fungi and potential micotoxin producer. Recent studies have verified the presence of ascospores of such microorganism in samples of pineapple nectars. Although the majority of filamentous fungi have limited heat resistance and are easily destroyed by heat, Byssochlamys nivea ascospores have shown high thermal resistance. The aim of this work was to evaluate the application of linear and Weibull models on thermal inactivation (70, 80 and 90ºC) of Byssochlamys nivea ascospores in pineapple nectar after pretreatment with high pressure (550MPa or 650MPa during 15min). Following the treatments, survival curves were built up for each processing temperature and adjusted for both models. It was observed that survival curves at 90°C after high pressure pretreatment at 550 MPa/15 min did not fit well to linear and Weibull models. For all the other treatments, the Weibull model presented a better fit. At 90ºC without pressure treatment, the Weibull model also showed a better adjustment, having a larger R2 and a smaller RMSE. Regarding the process effectiveness, a 5-log reduction (t5), as recommended for pasteurization, was only achieved for Byssochlamys nivea ascospores presented in pineapple nectar at 90ºC/10.7 min with previous high pressure treatment of 650 MPa for 15 min. Considering the high intensity and energy demanding process with possibly product damage, other preventive and alternative treatments are being investigated

Heteroscedastic latent variable modelling with applications to multivariate statistical process control

We present an approach for conducting multivariate statistical process control (MSPC) in noisy environments, i.e., when the signal to noise ratio is low, and, furthermore, noise standard deviation (uncertainty) affecting each collected value can vary over time, and is assumingly known. This approach is based upon a latent variable model structure, HLV (standing for heteroscedastic latent variable model), that explicitly integrates information regarding data uncertainty. Moderate amounts of missing data can also be handled in a coherent and fully integrated way through HLV. Several examples show the added value achieved under noisy conditions by adopting such an approach and a case study illustrates its application to a real industrial context of pulp and paper product quality data analysis.http://www.sciencedirect.com/science/article/B6TFP-4GX1HVW-2/1/c5e6b0a181b2fb4ffd7803ff38c9dac

Multiscale statistical process control with multiresolution data

An approach is presented for conducting multiscale statistical process control that adequately integrates data at different resolutions (multiresolution data), called MR-MSSPC. Its general structure is based on Bakshi's MSSPC framework designed to handle data at a single resolution. Significant modifications were introduced in order to process multiresolution information. The main MR-MSSPC features are presented and illustrated through three examples. Issues related to real world implementations and with the interpretation of the multiscale covariance structure are addressed in a fourth example, where a CSTR system under feedback control is simulated. Our approach proved to be able to provide a clearer definition of the regions where significant events occur and a more sensitive response when the process is brought back to normal operation, when it is compared with previous approaches based on single resolution data. © 2006 American Institute of Chemical Engineers AIChE J, 200

High Efficacy of Two Artemisinin-Based Combinations (Artesunate + Amodiaquine and Artemether + Lumefantrine) in Caala, Central Angola.

In April 2004, 137 children 6-59 months of age with uncomplicated Plasmodium falciparum (Pf) malaria (Caala, Central Angola) were randomized to receive either artemether-lumefantrine (Coartem) or artesunate + amodiaquine (ASAQ). After 28 days of follow-up, there were 2/61 (3.2%) recurrent parasitemias in the Coartem group and 4/64 (6.2%) in the ASAQ group (P = 0.72), all classified as re-infections after PCR genotyping (cure rate = 100% [95%CI: 94-100] in both groups). Only one patient (ASAQ group) had gametocytes on day 28 versus five (Coartem) and three (ASAQ) at baseline. Compared with baseline, anemia was significantly improved after 28 days of follow-up in both groups (Coartem: from 54.1% to 13.4%; ASAQ: from 53.1% to 15.9%). Our findings are in favor of a high efficacy of both combinations in Caala. Now that Coartem has been chosen as the new first-line anti-malarial, the challenge is to insure that this drug is available and adequately used

Membrane Type 1 Matrix Metalloproteinase Regulates Monocyte Migration and Collagen Destruction in Tuberculosis

Tuberculosis (TB) remains a global pandemic and drug resistance is rising. Multicellular granuloma formation is the pathological hallmark of Mycobacterium tuberculosis infection. The membrane type 1 matrix metalloproteinase (MT1-MMP or MMP-14) is a collagenase that is key in leukocyte migration and collagen destruction. In patients with TB, induced sputum MT1-MMP mRNA levels were increased 5.1-fold compared with matched controls and correlated positively with extent of lung infiltration on chest radiographs (r = 0.483; p < 0.05). M. tuberculosis infection of primary human monocytes increased MT1-MMP surface expression 31.7-fold and gene expression 24.5-fold. M. tuberculosis-infected monocytes degraded collagen matrix in an MT1-MMP-dependent manner, and MT1-MMP neutralization decreased collagen degradation by 73%. In human TB granulomas, MT1-MMP immunoreactivity was observed in macrophages throughout the granuloma. Monocyte-monocyte networks caused a 17.5-fold increase in MT1-MMP surface expression dependent on p38 MAPK and G protein-coupled receptor-dependent signaling. Monocytes migrating toward agarose beads impregnated with conditioned media from M. tuberculosis-infected monocytes expressed MT1-MMP. Neutralization of MT1-MMP activity decreased this M. tuberculosis network-dependent monocyte migration by 44%. Taken together, we demonstrate that MT1-MMP is central to two key elements of TB pathogenesis, causing collagen degradation and regulating monocyte migration

Quasi-one-dimensional system as a high-temperature superconductor

It is well-known that quasi-one-dimensional superconductors suffer from the pairing fluctuations that significantly reduce the superconducting temperature or even completely suppress any coherent behavior. Here we demonstrate that a coupling to a robust pair condensate changes the situation dramatically. In this case the quasi-one-dimensional system can be a high temperature superconductor governed by the proximity to the Lifshitz transition at which the Fermi level approaches the lower edge of the single-particle spectrum.Comment: 5 pages, 1 figur