383 research outputs found

    Independent sets of some graphs associated to commutative rings

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    Let G=(V,E)G=(V,E) be a simple graph. A set SVS\subseteq V is independent set of GG, if no two vertices of SS are adjacent. The independence number α(G)\alpha(G) is the size of a maximum independent set in the graph. %An independent set with cardinality Let RR be a commutative ring with nonzero identity and II an ideal of RR. The zero-divisor graph of RR, denoted by Γ(R)\Gamma(R), is an undirected graph whose vertices are the nonzero zero-divisors of RR and two distinct vertices xx and yy are adjacent if and only if xy=0xy = 0. Also the ideal-based zero-divisor graph of RR, denoted by ΓI(R)\Gamma_I(R), is the graph which vertices are the set {x\in R\backslash I | xy\in I \quad for some \quad y\in R\backslash I\} and two distinct vertices xx and yy are adjacent if and only if xyIxy \in I. In this paper we study the independent sets and the independence number of Γ(R)\Gamma(R) and ΓI(R)\Gamma_I(R).Comment: 27 pages. 22 figure

    Motion compensation for image compression: pel-recursive motion estimation algorithm

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    In motion pictures there is a certain amount of redundancy between consecutive frames. These redundancies can be exploited by using interframe prediction techniques. To further enhance the efficiency of interframe prediction, motion estimation and compensation, various motion compensation techniques can be used. There are two distinct techniques for motion estimation block matching and pel-recursive block matching has been widely used as it produces a better signal-to-noise ratio or a lower bit rate for transmission than the pel-recursive method. In this thesis, various pel-recursive motion estimation techniques such as steepest descent gradient algorithm have been considered and simulated. [Continues.

    Prevention, inhibition, and degradation effects of melittin alone and in combination with vancomycin and rifampin against strong biofilm producer strains of methicillin-resistant Staphylococcus epidermidis

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    Methicillin-resistant Staphylococcus epidermidis (MRSE) bacteria are being recognized as true pathogens as they are able to resist methicillin and commonly form biofilms. Recent studies have shown that antimicrobial peptides (AMPs) are promising agents against biofilm-associated bacterial infections. In this study, we aimed to explore the antibiofilm activity of melittin, either alone or in combination with vancomycin and rifampin, against biofilm-producing MRSE strains. Minimum biofilm preventive concentration (MBPC), minimum biofilm inhibition concentration (MBIC), and minimum biofilm eradication concentration (MBEC), as well as fractional biofilm preventive-, inhibitory-, and eradication concentrations (FBPCi, FBICi, and FBECi), were determined for the antimicrobial agents tested. Cytotoxicity and hemolytic activity of melittin at its synergistic concentration were examined on human embryonic kidney cells (HEK-293) and Red Blood Cells (RBCs), respectively. The effect of melittin on the downregulation of biofilm-associated genes was explored using Real-Time PCR. MBPC, MBIC, and MBEC values for melittin were in the range of 0.625–20, 0.625–20, and 10–40 μg/μL, respectively. Melittin showed high synergy (FBPCi, FBICi and FBECi < 0.5). The synergism resulted in a 64–512-fold, 2–16 and 2–8-fold reduction in melittin, rifampicin and vancomycin concentrations, respectively. The synergistic melittin concentration found to be effective did not manifest either cytotoxicity on HEK-293 or hemolytic activity on RBCs. Results showed that melittin downregulated the expression of biofilm-associated icaA, aap, and psm genes in all isolates tested, ranging from 0.04-folds to 2.11-folds for icaA and from 0.05 to 3.76-folds for aap and psm. The preventive and therapeutic indexes of melittin were improved 8-fold when combined with vancomycin and rifampin. Based on these findings, the combination of melittin with conventional antibiotics could be proposed for treating or preventing biofilm-associated MRSE infections

    Bacterial biofilm in colorectal cancer: What is the real mechanism of action?

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    Human colorectal cancer is the third most common cancer around the world. Colorectal cancer has various risk factors, but current works have bolded a significant activity for the microbiota of the human colon in the development of this disease. Bacterial biofilm has been mediated to non-malignant pathologies like inflammatory bowel disease but has not been fully documented in the setting of colorectal cancer. The investigation has currently found that bacterial biofilm is mediated to colon cancer in the human and linked to the location of human cancer, with almost all right-sided adenomas of colon cancers possessing bacterial biofilm, whilst left-sided cancer is rarely biofilm positive. The profound comprehension of the changes in colorectal cancer can provide interesting novel concepts for anticancer treatments. In this review, we will summarize and examine the new knowledge about the links between colorectal cancer and bacterial biofilm. © 202

    Prevalence of Enteropathogenic and Shiga Toxin-producing Escherichia coli Among Children With and Without Diarrhoea in Iran

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    The aim of the study was to determine the rates of detection of enteropathogenic Escherichia coli (EPEC) and Shiga toxin-producing E. coli (STEC) strains among children in two randomly-selected populations in Iran. In total, 1,292 randomly-selected faecal samples from children aged less than 10 years were screened for EPEC and STEC. Of the 1,292 cases participated in the study, 184 had diarrhoea, and 1,108 were healthy/asymptomatic children. The conventional culture method and slide agglutination with 12 different commercial EPEC antisera were used for the detection of EPEC. The colony sweep polymyxin- B extraction method, non-sorbitol fermentation (NSF) phenotype, and slide agglutination with O157: H7 antisera were used for the screening and detection of STEC. Of EPEC belonging to 11 different serogroups, O111 and O127 were most commonly found in 36.4% of the diarrhoeal cases and 7.2% of the asymptomatic children. A significant association (p<0.05) was found between isolation of EPEC and diarrhoea. 8.7% of the diarrhoeal cases and 2% of children without diarrhoea were infected with STEC, but none of the isolates belonged to the O157:H7 serotype. A significant association (p<0.05) was found between STEC and diarrhoeal cases. Based on these findings, it can be concluded that different EPEC serogroups may be agents of endemic infantile diarrhoea, and STEC strains are an important enteropathogen among young children
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