Can the South Atlantic Opening Model be Applied to the India Margins?

The presence of SDRs (seawrd dipping reflectors) on the regional lines around the Indian continent strongly suggest the breakup of the lithosphere and the onset of the sea-floor spreading were similar to those proposed and described for the South Atlantic, which, in fact, is quite similar to the opening of the North Atlantic

Can the South Atlantic Opening Model be Applied to the India Margins?

The presence of SDRs (seawrd dipping reflectors) on the regional lines around the Indian continent strongly suggest the breakup of the lithosphere and the onset of the sea-floor spreading were similar to those proposed and described for the South Atlantic, which, in fact, is quite similar to the opening of the North Atlantic

Validation of tectonic models for an intraplate seismic zone, Charleston, South Carolina, with GPS geodetic data

Although the average strain rate in intraplate settings is 2--3 orders of magnitude lower than at plate boundaries, there are pockets of high strain rates within intraplate regions. The results of a Global Positioning System survey near the location of current seismicity (and the inferred location of the destructive 1886 Charleston, South Carolina earthquake) suggest that there is anomalous strain build-up occurring there. By reoccupying 1930 triangulation and 1980 GPS sites with six Trimble SST dual frequency receivers, a strain rate of 0.4 {times} 10{sup {minus}7} yr{sup {minus}1} was observed. At the 95% confidence level, this value is not significant; however, at a lower level of confidence ({approximately} 85%) it is about two orders of magnitude greater than the background of 10{sup {minus}9} to 10{sup {minus}10} yr{sup {minus}1}. The direction of contraction inferred from the GPS survey 66{degree} {+-} 11{degree} is in excellent agreement with the direction of the maximum horizontal stress (N 60{degree} E) in the area, suggesting that the observed strain rate is also real. 66 refs

Multiple effects of toxins isolated from Crotalus durissus terrificus on the hepatitis C virus life cycle

Hepatitis C virus (HCV) is one of the main causes of liver disease and transplantation worldwide. Current therapy is expensive, presents additional side effects and viral resistance has been described. Therefore, studies for developing more efficient antivirals against HCV are needed. Compounds isolated from animal venoms have shown antiviral activity against some viruses such as Dengue virus, Yellow fever virus and Measles virus. In this study, we evaluated the effect of the complex crotoxin (CX) and its subunits crotapotin (CP) and phospholipase A2 (PLA2-CB) isolated from the venom of Crotalus durissus terrificus on HCV life cycle. Huh 7.5 cells were infected with HCVcc JFH-1 strain in the presence or absence of these toxins and virus was titrated by focus formation units assay or by qPCR. Toxins were added to the cells at different time points depending on the stage of virus life cycle to be evaluated. The results showed that treatment with PLA2-CB inhibited HCV entry and replication but no effect on HCV release was observed. CX reduced virus entry and release but not replication. By treating cells with CP, an antiviral effect was observed on HCV release, the only stage inhibited by this compound. Our data demonstrated the multiple antiviral effects of toxins from animal venoms on HCV life cycle