Chiral symmetry restoration in (2+1)-dimensional QEDQED with a Maxwell-Chern-Simons term at finite temperature

We study the role played by a Chern-Simons contribution to the action in the $QED_3$ formulation of a two-dimensional Heisenberg model of quantum spin systems with a strictly fixed site occupation at finite temperature. We show how this contribution affects the screening of the potential which acts between spinons and contributes to the restoration of chiral symmetry in the spinon sector. The constant which characterizes the Chern-Simons term can be related to the critical temperature $T_c$ above which the dynamical mass goes to zero.Comment: 8 pages, 4 figure

Site occupation constraints in mean-field approaches of quantum spin systems at finite temperature

We study the effect of site occupation on the description of quantum spin systems at finite temperature and mean-field level. We impose each lattice site to be occupied by a single electron. This is realized by means of a specific prescription. The outcome of the prescription is compared to the result obtained by means of a projection procedure which fixes the site occupation to one particle per site on an average. The comparison is performed for different representations of the Hamiltonian in Fock space leading to different types of mean-field solutions. The behaviour of order parameters is analyzed for each choice of the mean-field and constraint which fixes the occupation rate at each site. Sizable quantitative differences between the outcomes obtained with the two different constraints are observed.Comment: 18 pages, 2 figure

Analysis of chronic rejection and obliterative arteriopathy: Possible contributions of donor antigen-presenting cells and lymphatic disruption

Sequential analysis of changes that lead to chronic rejection was undertaken in an animal model of chronic rejection and obliterative arteriopathy. Brown Norway rats are pretreated with a Lewis bone marrow infusion or a Lewis orthotopic liver allograft and a short course of immunosuppression. They are challenged 100 days later with a Lewis heterotopic heart graft without immunosuppression. The heart grafts in both groups undergo a transient acute rejection, but all rats are operationally tolerant; the heart grafts are accepted and remain beating for more than 100 days. Early arterial remodeling, marked by arterial bromodeoxyuridine incorporation, occurred in both groups between 5 and 30 days during the transient acute rejection. It coincided with the presence of interstitial (but not arterial intimal) inflammation and lymphatic disruption and resulted in mild intimal thickening. Significant arterial narrowing occurred only in the bone-marrow-pretreated rats between 60 and 100 days. It was associated with T lymphocyte and macrophage inflammation of the heart graft that accumulated in the endocardium and arterial intima and adventitia near draining lymphatics. There also was loss of passenger leukocytes from the heart graft, up-regulation of cytokine mRNA and major histocompatibility class II on the endothelium, and focal disruption of lymphatics. In contrast, long-surviving heart grafts from the Lewis orthotopic liver allograft pretreated group are near normal and freedom from chronic rejection in this group was associated with persistence of donor major histocompatibility class-II-positive hematolymphoid cells, including OX62+ donor dendritic cells. This study offers insights into two different aspects of chronic rejection: 1) possible mechanisms underlying the persistent immunological injury and 2) the association between immunological injury and the development of obliterative arteriopathy. Based on the findings, it is not unreasonable to raise the testable hypothesis that direct presentation of alloantigen by donor antigen-presenting cells is required for long-term, chronic-rejection-free allograft acceptance. In addition, chronic intermittent lymphatic disruption is implicated as a possible mechanism for the association between chronic interstitial allograft inflammation and the development of obliterative arteriopathy

Kondo effect in Complex Quantum Dots in the presence of an oscillating and fluctuating gate signal

We show how the charge input signal applied to the gate electrode in a double and triple quantum dot may be converted to a pulse in the Kondo cotunneling current being a spin response of a nano-device under a strong Coulomb blockade. The stochastic component of the input signal results in the infrared cutoff of Kondo transmission. The stochastization of the orbital component of the Kondo effect in triple quantum dots results in a noise-induced SU(4) - SU(2) quantum transition.Comment: 16 pages, 12 figure

Corresponding States of Structural Glass Formers

The variation with respect to temperature T of transport properties of 58 fragile structural glass forming liquids (68 data sets in total) are analyzed and shown to exhibit a remarkable degree of universality. In particular, super-Arrhenius behaviors of all super-cooled liquids appear to collapse to one parabola for which there is no singular behavior at any finite temperature. This behavior is bounded by an onset temperature To above which liquid transport has a much weaker temperature dependence. A similar collapse is also demonstrated, over the smaller available range, for existing numerical simulation data.Comment: 6 pages, 2 figures. Updated References, Table Values, Submitted for Publicatio

On the reliability of negative heat capacity measurements

A global protocol for the thermostatistical analysis of hot nuclear sources is discussed. Within our method of minimization of variances we show that the abnormal kinetic energy fluctuation signal recently reported in different experimental data (M.D'Agostino et al.-Phys. Lett. B 473 (2000) 219, N. Le Neindre et al.- contr. to the XXXVIII Bormio Winter Meeting on Nucl. Phys. (2001) 404) is a genuine signal of a first order phase transition in a finite system.Comment: 15 Postscript figures, submitted to NUCL. Phys. A on 24-apr-200

Topologically disordered systems at the glass transition

The thermodynamic approach to the viscosity and fragility of amorphous oxides was used to determine the topological characteristics of the disordered network-forming systems. Instead of the disordered system of atoms we considered the congruent disordered system of interconnecting bonds. The Gibbs free energy of network-breaking defects (configurons) was found based on available viscosity data. Amorphous silica and germania were used as reference disordered systems for which we found an excellent agreement of calculated and measured glass transition temperatures. We reveal that the Hausdorff dimension of the system of bonds changes from Euclidian three-dimensional below to fractal 2.55 ± 0.05-dimensional geometry above the glass transition temperature

Finite size effects and the order of a phase transition in fragmenting nuclear systems

We discuss the implications of finite size effects on the determination of the order of a phase transition which may occur in infinite systems. We introduce a specific model to which we apply different tests. They are aimed to characterise the smoothed transition observed in a finite system. We show that the microcanonical ensemble may be a useful framework for the determination of the nature of such transitions.Comment: LateX, 5 pages, 5 figures; Fig. 1 change

Dynamical heterogeneities in a supercooled Lennard-Jones liquid

We present the results of a large scale molecular dynamics computer simulation study in which we investigate whether a supercooled Lennard-Jones liquid exhibits dynamical heterogeneities. We evaluate the non-Gaussian parameter for the self part of the van Hove correlation function and use it to identify ``mobile'' particles. We find that these particles form clusters whose size grows with decreasing temperature. We also find that the relaxation time of the mobile particles is significantly shorter than that of the bulk, and that this difference increases with decreasing temperature.Comment: 8 pages of RevTex, 4 ps figure

The significance of macrophage phenotype in cancer and biomaterials

Macrophages have long been known to exhibit heterogeneous and plastic phenotypes. They show functional diversity with roles in homeostasis, tissue repair, immunity and disease. There exists a spectrum of macrophage phenotypes with varied effector functions, molecular determinants, cytokine and chemokine profiles, as well as receptor expression. In tumor microenvironments, the subset of macrophages known as tumor-associated macrophages generates byproducts that enhance tumor growth and angiogenesis, making them attractive targets for anti-cancer therapeutics. With respect to wound healing and the foreign body response, there is a necessity for balance between pro-inflammatory, wound healing, and regulatory macrophages in order to achieve successful implantation of a scaffold for tissue engineering. In this review, we discuss the multitude of ways macrophages are known to be important in cancer therapies and implanted biomaterials