E 1 Gap Of Wurtzite Inas Single Nanowires Measured By Means Of Resonant Raman Spectroscopy

Indium arsenide nanowires were synthesized with an intermixing of wurtzite and zincblende structure by chemical beam epitaxy with the vapor-liquid-solid mechanism. Resonant Raman spectroscopy of the transverse optical phonon mode at 215cm -1 reveals an E 1 gap of 2.47 eV which is assigned to the electronic band gap at the A point in the indium arsenide wurtzite phase. © 2011 American Institute of Physics.1399473474 Int. Union Pure Appl. Phys. (IUPAP C8 Comm.),Korean Ministry of Education, Science and Technology,Seoul Metropolitan Government,Office of Naval Research Global,Korea Tourism OrganizationDick, K.A., (2008) Prog. Cryst. Growth Charact. Mater., 54, pp. 138-173Milnes, A.G., Polyakov, A.Y., (1993) Mater. Sci. Eng. B, 18, pp. 237-259Dayeh, S.A., Susac, D., Kavanagh, K.L., Yu, E.T., Wang, D., (2009) Adv. Mater., 19, pp. 2102-2108Cardona, M., (1961) J. Appl. Phys., 32 (SUPPL.), pp. 2151-2155Antoci, S., Reguzzoni, E., Samoggia, G., (1970) Phys. Rev. Lett., 24, pp. 1304-1307Arguello, C.A., Rousseau, D.L., Porto, S.P.S., (1969) Phys. Rev., 181, pp. 1351-136

The structure of superheavy elements newly discovered in the reaction of 86^{86}Kr with 208^{208}Pb

The structure of superheavy elements newly discovered in the $^{208}$Pb($^{86}$Kr,n) reaction at Berkeley is systematically studied in the Relativistic Mean Field (RMF) approach. It is shown that various usually employed RMF forces, which give fair description of normal stable nuclei, give quite different predictions for superheavy elements. Among the effective forces we tested, TM1 is found to be the good candidate to describe superheavy elements. The binding energies of the $^{293}$118 nucleus and its $\alpha-$decay daughter nuclei obtained using TM1 agree with those of FRDM within 2 MeV. Similar conclusion that TM1 is the good interaction is also drawn from the calculated binding energies for Pb isotopes with the Relativistic Continuum Hartree Bogoliubov (RCHB) theory. Using the pairing gaps obtained from RCHB, RMF calculations with pairing and deformation are carried out for the structure of superheavy elements. The binding energy, shape, single particle levels, and the Q values of the $\alpha-$decay $Q_{\alpha}$ are discussed, and it is shown that both pairing correlation and deformation are essential to properly understand the structure of superheavy elements. A good agreement is obtained with experimental data on $Q_{\alpha}$. %Especially, the atomic number %dependence of $Q_{\alpha}$ %seems to match with the experimental observationComment: 19 pages, 5 figure

QCD ghost f(T)-gravity model

Within the framework of modified teleparallel gravity, we reconstruct a f(T) model corresponding to the QCD ghost dark energy scenario. For a spatially flat FRW universe containing only the pressureless matter, we obtain the time evolution of the torsion scalar T (or the Hubble parameter). Then, we calculate the effective torsion equation of state parameter of the QCD ghost f(T)-gravity model as well as the deceleration parameter of the universe. Furthermore, we fit the model parameters by using the latest observational data including SNeIa, CMB and BAO data. We also check the viability of our model using a cosmographic analysis approach. Moreover, we investigate the validity of the generalized second law (GSL) of gravitational thermodynamics for our model. Finally, we point out the growth rate of matter density perturbation. We conclude that in QCD ghost f(T)-gravity model, the universe begins a matter dominated phase and approaches a de Sitter regime at late times, as expected. Also this model is consistent with current data, passes the cosmographic test, satisfies the GSL and fits the data of the growth factor well as the LCDM model.Comment: 19 pages, 9 figures, 2 tables. arXiv admin note: substantial text overlap with arXiv:1111.726

Sociocultural considerations in aging men's health: implications and recommendations for the clinician

http://dx.doi.org/10.1016/j.jomh.2009.07.00

CSF1R inhibitor JNJ-40346527 attenuates microglial proliferation and neurodegeneration in P301S mice

Neuroinflammation and microglial activation are significant processes in Alzheimer's disease pathology. Recent genome-wide association studies have highlighted multiple immune-related genes in association with Alzheimer's disease, and experimental data have demonstrated microglial proliferation as a significant component of the neuropathology. In this study, we tested the efficacy of the selective CSF1R inhibitor JNJ-40346527 (JNJ-527) in the P301S mouse tauopathy model. We first demonstrated the anti-proliferative effects of JNJ-527 on microglia in the ME7 prion model, and its impact on the inflammatory profile, and provided potential CNS biomarkers for clinical investigation with the compound, including pharmacokinetic/pharmacodynamics and efficacy assessment by TSPO autoradiography and CSF proteomics. Then, we showed for the first time that blockade of microglial proliferation and modification of microglial phenotype leads to an attenuation of tau-induced neurodegeneration and results in functional improvement in P301S mice. Overall, this work strongly supports the potential for inhibition of CSF1R as a target for the treatment of Alzheimer's disease and other tau-mediated neurodegenerative diseases

Inflammatory biomarkers in Alzheimer's disease plasma

Introduction: Plasma biomarkers for Alzheimer's disease (AD) diagnosis/stratification are a \u201cHoly Grail\u201d of AD research and intensively sought; however, there are no well-established plasma markers. Methods: A hypothesis-led plasma biomarker search was conducted in the context of international multicenter studies. The discovery phase measured 53 inflammatory proteins in elderly control (CTL; 259), mild cognitive impairment (MCI; 199), and AD (262) subjects from AddNeuroMed. Results: Ten analytes showed significant intergroup differences. Logistic regression identified five (FB, FH, sCR1, MCP-1, eotaxin-1) that, age/APO\u3b54 adjusted, optimally differentiated AD and CTL (AUC: 0.79), and three (sCR1, MCP-1, eotaxin-1) that optimally differentiated AD and MCI (AUC: 0.74). These models replicated in an independent cohort (EMIF; AUC 0.81 and 0.67). Two analytes (FB, FH) plus age predicted MCI progression to AD (AUC: 0.71). Discussion: Plasma markers of inflammation and complement dysregulation support diagnosis and outcome prediction in AD and MCI. Further replication is needed before clinical translation