Seven zero nine zero computer program for determination of precision lattice constants

Computer program for calculation of lattice parameters for cubic, tetragonal, and hexagonal crystals - magnesium oxide and aluminum oxid

Arthrogryposis in piglets

This report documents experimental reproduction of tetramelic arthrogryposis in purebred Yorkshire swine by breeding trials. Data from the trials indigated arthrogryposis may be due to homozygosity of a simple autosomal recessive gene. No affected pig was observed to be alive at parturition, although most had been alive in utero just prior to delivery. Other than dystocia observed in the sows, no other significant clinical findings were noted. The condition was noted in five litters from two sows which were bred to the same boar. Approximately 25% of the pigs exhibited the clinical signs of arthrogryposis which included malformed rigidly extended legs.; Swine Day, Manhattan, KS, November 9, 197

Solar sail capture trajectories at Mercury

Mercury is an ideal environment for future planetary exploration by solar sail since it has proved difficult to reach with conventional propulsion and hence remains largely unexplored. In addition, its proximity to the Sun provides a solar sail acceleration of order ten times the sail characteristic acceleration at 1 AU. Conventional capture techniques are shown to be unsuitable for solar sails and a new method is presented. It is shown that capture is bound by upper and lower limits on the orbital elements of the approach orbit and that failure to be within limits results in a catastrophic collision with the planet. These limits are presented for a range of capture inclinations and sail characteristic accelerations. It is found that sail hyperbolic excess velocity is a critical parameter during capture at Mercury, with only a narrow allowed band in order to avoid collision with the planet. The new capture methodis demonstrated for a Mercury sample return mission

Albinism and heterochromia irides in the Hereford

Call number: LD2668 .T4 1967 L4Master of Scienc

Measurement of a 2D fast-ion velocity distribution function by tomographic inversion of fast-ion D-alpha spectra

We present the first measurement of a local fast-ion 2D velocity distribution function f(v||, v⊥). To this end, we heated a plasma in ASDEX Upgrade by neutral beam injection and measured spectra of fast-ion Dα (FIDA) light from the plasma centre in three views simultaneously. The measured spectra agree very well with synthetic spectra calculated from a TRANSP/NUBEAM simulation. Based on the measured FIDA spectra alone, we infer f(v||, v⊥) by tomographic inversion. Salient features of our measurement of f(v||, v⊥) agree reasonably well with the simulation: the measured as well as the simulated f(v||, v⊥) are lopsided towards negative velocities parallel to the magnetic field, and they have similar shapes. Further, the peaks in the simulation of f(v||, v⊥) at full and half injection energies of the neutral beam also appear in the measurement at similar velocity-space locations. We expect that we can measure spectra in up to seven views simultaneously in the next ASDEX Upgrade campaign which would further improve measurements of f(v||, v⊥) by tomographic inversion

A MITF Mutation Associated with a Dominant White Phenotype and Bilateral Deafness in German Fleckvieh Cattle

A dominantly inherited syndrome associated with hypopigmentation, heterochromia irides, colobomatous eyes and bilateral hearing loss has been ascertained in Fleckvieh cattle (German White Fleckvieh syndrome). This syndrome has been mapped to bovine chromosome (BTA) 22 using a genome-wide association study with the bovine high density single nucleotide polymorphism array. An R210I missense mutation has been identified within microphthalmia-associated transcription factor (MITF) as responsible for this syndrome. The mutation is located in the highly conserved basic region of the protein and causes a negative-dominant effect. SOX10 and PAX3 promoter binding site mutations in MITF could be ruled out as causative for the German White Fleckvieh syndrome. Molecular characterization of this newly detected bovine syndrome means a large animal model is now available for the Tietz syndrome in humans