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An Analysis of Message Development, Delivery Strategies, Message Topics, Functional Consequences, and Social Responses of Requesting an Accommodation in the Workplace Due to a Chronic Health Condition

By 2020, 157 million Americans will be living with a chronic health condition. Many will continue to work by requesting workplace accommodations such as changes to their work environment or work schedule. In interviews, 38 respondents reported the message development, delivery strategies, message topics, functional consequences, and social responses in requesting a workplace accommodation due to a chronic health condition, as well as provided advice to others who may face a similar situation. The study found that respondents deliberately developed messages, used consistent delivery strategies and demonstrated recurring patterns of topics. Their preparation, delivery, and messages revealed concerns with identity, relationships, and instrumental tasks, as well as concerns with positive and negative face. Overall, respondents reported more positive than negative responses from coworkers and supervisors. Interestingly, respondents' suggestions to others were inconsistent with the report of their experience

A Dyke\u27s Life: Sexual Identity and Gender Performance in Radclyffe Hall\u27s The Well of Loneliness

This thesis studies sexual and gender identity and gender performance in The Well of Loneliness by utilizing postmodern theory. The protagonist in the novel, Stephen Gordon, is not only one example of the many identities of lesbianism, but she is also an example of a multiplicitous identity. This thesis also questions whether we can find the exact moment or reason why an identity is formed. An exploration of not only The Well of Loneliness but also of a character study of Stephen Gordon is important to this dialogue because both studies validate the contradictory and complimentary relationship between sex and gender

Alien Registration- Ellsworth, Wallace L. (Caribou, Aroostook County)

https://digitalmaine.com/alien_docs/26293/thumbnail.jp

Fault structure and mechanics of the Hayward Fault, California, from double-difference earthquake locations

The relationship between small-magnitude seismicity and large-scale crustal faulting along the Hayward Fault, California, is investigated using a double-difference (DD) earthquake location algorithm. We used the DD method to determine high-resolution hypocenter locations of the seismicity that occurred between 1967 and 1998. The DD technique incorporates catalog travel time data and relative P and S wave arrival time measurements from waveform cross correlation to solve for the hypocentral separation between events. The relocated seismicity reveals a narrow, near-vertical fault zone at most locations. This zone follows the Hayward Fault along its northern half and then diverges from it to the east near San Leandro, forming the Mission trend. The relocated seismicity is consistent with the idea that slip from the Calaveras Fault is transferred over the Mission trend onto the northern Hayward Fault. The Mission trend is not clearly associated with any mapped active fault as it continues to the south and joins the Calaveras Fault at Calaveras Reservoir. In some locations, discrete structures adjacent to the main trace are seen, features that were previously hidden in the uncertainty of the network locations. The fine structure of the seismicity suggests that the fault surface on the northern Hayward Fault is curved or that the events occur on several substructures. Near San Leandro, where the more westerly striking trend of the Mission seismicity intersects with the surface trace of the (aseismic) southern Hayward Fault, the seismicity remains diffuse after relocation, with strong variation in focal mechanisms between adjacent events indicating a highly fractured zone of deformation. The seismicity is highly organized in space, especially on the northern Hayward Fault, where it forms horizontal, slip-parallel streaks of hypocenters of only a few tens of meters width, bounded by areas almost absent of seismic activity. During the interval from 1984 to 1998, when digital waveforms are available, we find that fewer than 6.5% of the earthquakes can be classified as repeating earthquakes, events that rupture the same fault patch more than one time. These most commonly are located in the shallow creeping part of the fault, or within the streaks at greater depth. The slow repeat rate of 2–3 times within the 15-year observation period for events with magnitudes around M = 1.5 is indicative of a low slip rate or a high stress drop. The absence of microearthquakes over large, contiguous areas of the northern Hayward Fault plane in the depth interval from ∼5 to 10 km and the concentrations of seismicity at these depths suggest that the aseismic regions are either locked or retarded and are storing strain energy for release in future large-magnitude earthquakes

Seismotectonics of the San Andreas Fault System Between Point Arena and Cape Mendocino in Northern California\u27 Implications for the Development and Evolution of a Young Transform

The northernmost and relatively youthful segment of the San Andreas fault system is situated within a 100+ km wide zone of northwest trending strike-slip faults that includes, from west to east, the San Andreas, Maacama, and Bartlett Springs faults. Although the San Andreas fault is the principal strike-slip fault in this system, it has been virtually aseismic since the 1906 earthquake. Moderate levels of seismicity locate to the east along the Maacama fault and, to a lesser extent, the Bartlett Springs fault at focal depths typical of other strike-slip faults within the San Andreas fault system in central California. North of the San Andreas fault system, within the Cape Mendocino area, earthquakes occur at depths of up to 40 km and primarily reflect internal deformation of the subducting Garda slab, and slip along the Mendocino Fracture Zone. Seismicity along the Maacama and Bartlett Springs faults is dominated by right-lateral to oblique-reverse slip along fault planes that dip 50 °-75 ° to the northeast. The northern extent of seismicity along these faults terminates near the surface projection of the southern edge of the Garda slab. The onset of seismicity along these faults may be related to the abrupt change in the elastic thickness of the North American plate as it enters the asthenaspheric window. The Maacama and Bartlett Springs faults are strike-parallel with active reverse faults within the forearc region of the Cascadia subductian zone. This preexisting structural fabric of northwest trending reverse faults in the forearc area appears to have strongly influenced the initial slip and complexity of these faults. Continuation of the moderately dipping Maacama fault to the southeast along the steeply dipping Healdsburg and Rodgers Creek fault zones and the near-vertical Hayward and Calaveras fault zones in the San Francisco Bay area suggests that these faults evolve toward a more vertical dip to minimize the shear stresses that tend to resist plate motion

Breast Cancer in the Personal Genomics Era

Breast cancer is a heterogeneous disease with a complex etiology that develops from different cellular lineages, progresses along multiple molecular pathways, and demonstrates wide variability in response to treatment. The “standard of care” approach to breast cancer treatment in which all patients receive similar interventions is rapidly being replaced by personalized medicine, based on molecular characteristics of individual patients. Both inherited and somatic genomic variation is providing useful information for customizing treatment regimens for breast cancer to maximize efficacy and minimize adverse side effects. In this article, we review (1) hereditary breast cancer and current use of inherited susceptibility genes in patient management; (2) the potential of newly-identified breast cancer-susceptibility variants for improving risk assessment; (3) advantages and disadvantages of direct-to-consumer testing; (4) molecular characterization of sporadic breast cancer through immunohistochemistry and gene expression profiling and opportunities for personalized prognostics; and (5) pharmacogenomic influences on the effectiveness of current breast cancer treatments. Molecular genomics has the potential to revolutionize clinical practice and improve the lives of women with breast cancer

1964 Grain Sorghum Performance Trials

The 1964 Grain Sorghum Performance Trials were conducted at ten locations in South Dakota. Varieties presently grown by farmers, new varieties not yet widely used and new strains being considered for release were eligible for entry in the 1964 trials. The trials were under the supervision of the Crop Performance Testing Activity of the Agricultural Experiment Station. Grain yields, test weights and other agronomic data are reported

Mechanisms underlying pituitary hypoplasia and failed cell specification in Lhx3-deficient mice

AbstractThe LIM homeodomain transcription factor, LHX3, is essential for pituitary development in mouse and man. Lhx3 engineered null mice have profound pituitary hypoplasia that we find is attributable to an increase in cell death early in pituitary development. Dying cells are localized to regions of TPIT expression indicating that cell death may contribute to the severe reduction in differentiated corticotrope cells and lower expression of the corticotrope transcription factors, TPIT and NEUROD1. Lhx3 deficiency also results in dorsal ectopic expression of transcription factors characteristic of gonadotropes, SF1 and ISL1, but no gonadotropin expression. This apparent disturbance of cell differentiation may be due, in part, to loss of NOTCH2. NOTCH2 is normally expressed in the pituitary at the boundary between dorsal, proliferating cells and ventral, differentiating cells and is important for maintaining dorsal–ventral patterning in other organs. Thus, Lhx3 contributes significantly to pituitary development by maintaining normal dorsal–ventral patterning, cell survival, and normal expression of corticotrope-specific transcription factors, which are necessary for repressing ectopic gonadotrope differentiation