Chemical and microbial leaching of valuable metals from PCBs and tantalum capacitors of spent mobile phones

We compared chemical and microbial leaching for multi-metal extraction from printed circuit boards (PCBs) and tantalum capacitor scrap. A mixed consortium of acidophiles and heterotrophic fungal strains were used in the experiments and compared to chemical leaching using specific acids (sulfuric, citric and oxalic acids). Under optimum conditions, 100% extraction efficiency of Cu, and nearly 85% of Zn, Fe, Al and Ni were achieved from PCB and tantalum capacitor scrap samples using sulfuric acid. The mixed consortium of acidophiles successfully mobilized, Ni and Cu (99% and 96%, respectively) while Fe, Zn, Al and Mn reached an extraction yield of 89, 77, 70 and 43%, respectively, from the PCB samples. For the tantalum capacitor samples, acidophiles mobilized 92% Cu, 88% Ni, 78% Fe, 77% Al, 70% Zn and 57% Mn. Metal mobilization from PCBs and tantalum capacitor scrap by A. niger filtrate showed efficient solubilization of Cu, Fe, Al, Mn, Ni, Pb and Zn at an efficiency of 52, 29, 75, 5, 61, 21 and 35% from PCB samples and 61, 25, 69, 23, 68, 15 and 45% from tantalum capacitor samples, respectively. Microbial leaching proved viable as a method to extract base metals but was less specific for tantalum and precious metals in electronic waste. The implications of these results for further processing of waste electronic and electrical equipment (WEEE) are considered in potential hybrid treatment strategies

Effectiveness of Wildlife Mitigation Treatments Along the Nelsonville Bypass

SJN 135024The Nelsonville Bypass is a 9 mile stretch of U.S. Route 33 that runs through the Wayne National Forest, an area high in species diversity and home to several threatened and endangered species. The motorist safety, economic and conservation values of building effective mitigation features that reduce vehicle-wildlife collisions along the bypass have been nationally recognized. Mitigation features include: high and low fencing to reduce wildlife trespass into the right-of-way (ROW), uni-directional jump outs for wildlife exit from the ROW, underpasses and ecopassages to maintain habitat connectivity across the highway, high-mast lighting to lure bats above traffic flow, and replacement of wetlands and bat roosting habitat. Our two-year study employed road surveys, continuous monitoring of jump outs and wildlife passages, population estimations, detailed mapping of fence structures and breaches, and radio telemetry of an endangered target species. Road surveys of the bypass and control highways revealed that the mitigation structures reduced deer-vehicle collisions, but collisions still occurred on the bypass. Although, generally well-constructed, we identified several ways in which the mitigation features could be made more effective. Placement of fencing near the outer boundary of the ROW made it vulnerable to damage from erosion and tree falls, and isolated high-quality habitats within the ROW. Placement of the fence within 30-50 ft. of the roadway on less rugged terrain away from the forest would likely reduce costs of construction and maintenance while allowing wildlife access to habitat within the ROW. We also recommended regular maintenance inspections and mowing on both sides of the fencing. Jump outs were effective uni-directional exits, but wildlife, particularly deer, were not compelled to exit the expansive area within the ROW fencing. Placement of the fence with jump outs closer to the road would reduce habitat within the fence and combined with traffic noise may increase jump out use. Large wildlife underpasses and crossings were well used by a variety of mammal species. Smaller mammals used the small wildlife ecopassages. Reptiles and amphibians avoided the use of underpasses and road mortality rates of amphibians were high on Ohio State Route 78 (tributary road) near wetlands. Placement and passage design were contributing factors to high amphibian mortality. Radio-tracking of rattlesnakes discovered that snakes easily trespassed the small wildlife fencing and used the habitat within the ROW, likely because it was warmer than the surrounding forested habitat. No road mortality or attempted road crossings by rattlesnakes were detected. Finally, while bats foraged near the lights, most species were detected with equal frequency at different heights under the lighting. Our report details these findings and provides additional recommendations to improve design and construction of wildlife mitigation features both along the Nelsonville Bypass, and for future design of mitigation features for roadways in high-density wildlife areas

Hybrid leaching of tantalum and other valuable metals from tantalum capacitor waste

PeerReviewe

The impact of collection portfolio expansion on key performance indicators of the Dutch recycling system for Post-Consumer Plastic Packaging Waste, a comparison between 2014 and 2017

The recycling network of post-consumer plastic packaging waste (PCPPW) was studied for the Netherlands in 2017 with material flow analysis (MFA) and data reconciliation techniques. In comparison to the previous MFA of the PCPPW recycling network in 2014, the predominant change is the expansion of the collection portfolio from only plastic packages to plastic packages, beverage cartons and metal objects. The analysis shows that the amounts of recycled plastics products (as main washed milled goods) increased from 75 to 103 Gg net and the average polymeric purity of the recycled products remained nearly constant. Furthermore, the rise in the amounts of recycled products was accompanied with a rise in the total amount of rejected materials at cross docking facilities and sorting residues at the sorting facilities. This total amount grew from 19 Gg in 2014 to 70 Gg gross in 2017 and is over-proportional to the rise in recycled products. Hence, there is a clear trade-off between the growth in recycled plastics produced and the growth in rejects and residues. Additionally, since the polymeric purity of the recycled plastics did not significantly improve during the last years, most of the recycled plastics from PCPPW are still only suited for open-loop recycling. Although this recycling system for PCPPW is relatively advanced in Europe, it cannot be considered circular, since the net recycling yield is only 26 ± 2% and the average polymeric purity of the recycled plastics is 90 ± 7%.</p

Nitro-Oleic Acid Prevents Hypoxia- and Asymmetric Dimethylarginine-Induced Pulmonary Endothelial Dysfunction

Pulmonary hypertension (PH) represents a serious health complication accompanied with hypoxic conditions, elevated levels of asymmetric dimethylarginine (ADMA), and overall dysfunction of pulmonary vascular endothelium. Since the prevention strategies for treatment of PH remain largely unknown, our study aimed to explore the effect of nitro-oleic acid (OA-NO2), an exemplary nitro-fatty acid (NO2-FA), in human pulmonary artery endothelial cells (HPAEC) under the influence of hypoxia or ADMA. HPAEC were treated with OA-NO2 in the absence or presence of hypoxia and ADMA. The production of nitric oxide (NO) and interleukin-6 (IL-6) was monitored using the Griess method and ELISA, respectively. The expression or activation of different proteins (signal transducer and activator of transcription 3, STAT3; hypoxia inducible factor 1 alpha, HIF-1 alpha; endothelial nitric oxide synthase, eNOS; intercellular adhesion molecule-1, ICAM-1) was assessed by the Western blot technique. We discovered that OA-NO2 prevents development of endothelial dysfunction induced by either hypoxia or ADMA. OA-NO2 preserves normal cellular functions in HPAEC by increasing NO production and eNOS expression. Additionally, OA-NO2 inhibits IL-6 production as well as ICAM-1 expression, elevated by hypoxia and ADMA. Importantly, the effect of OA-NO2 is accompanied by prevention of STAT3 activation and HIF-1 alpha stabilization. In summary, OA-NO2 eliminates the manifestation of hypoxia- and ADMA-mediated endothelial dysfunction in HPAEC via the STAT3/HIF-1 alpha cascade. Importantly, our study is bringing a new perspective on molecular mechanisms of NO2-FAs action in pulmonary endothelial dysfunction, which represents a causal link in progression of PH

Human venous valve disease caused by mutations in FOXC2 and GJC2

Venous valves (VVs) prevent venous hypertension and ulceration. We report that FOXC2 and GJC2 mutations are associated with reduced VV number and length. In mice, early VV formation is marked by elongation and reorientation (“organization”) of Prox1hi endothelial cells by postnatal day 0. The expression of the transcription factors Foxc2 and Nfatc1 and the gap junction proteins Gjc2, Gja1, and Gja4 were temporospatially regulated during this process. Foxc2 and Nfatc1 were coexpressed at P0, and combined Foxc2 deletion with calcineurin-Nfat inhibition disrupted early Prox1hi endothelial organization, suggesting cooperative Foxc2–Nfatc1 patterning of these events. Genetic deletion of Gjc2, Gja4, or Gja1 also disrupted early VV Prox1hi endothelial organization at postnatal day 0, and this likely underlies the VV defects seen in patients with GJC2 mutations. Knockout of Gja4 or Gjc2 resulted in reduced proliferation of Prox1hi valve-forming cells. At later stages of blood flow, Foxc2 and calcineurin-Nfat signaling are each required for growth of the valve leaflets, whereas Foxc2 is not required for VV maintenance