Rupture Process of the 1999 M_w 7.1 Duzce Earthquake from Joint Analysis of SPOT, GPS, InSAR, Strong-Motion, and Teleseismic Data: A Supershear Rupture with Variable Rupture Velocity

We analyze the rupture process of the 1999 M_w 7.1 Duzce earthquake using seismological, remote sensing, and geodetic data. Ground deformation measured from the subpixel cross correlation of Satellite Pour l'Observation de la Terre (SPOT) images reveals a 55 km long fault trace and smooth surface-slip distribution peaking at 3.5–4 m. The westernmost segment overlaps for over 10 km with ruptures from the M_w 7.4 Izmit earthquake. The 15 km long easternmost segment, which cuts across mountainous topography, had not been reported previously. We determine a well-constrained source model using a four-segment fault geometry using constraints on surface fault slip and inverting Global Positioning System and Interferometric Synthetic Aperture Radar data along with strong-motion records. Our results show that some variability of the rupture velocity and an eastward supershear velocity are required to fit the strong-motion data. The rise time, up to 6 sec, correlates with cumulative slip, suggesting a sliding velocity of about 1 m/sec. The source model predicts teleseismic waveforms well, although early by 2 sec. This time shift is probably due to the weak beginning of the earthquake that is not observable at teleseismic distances. Strong-motion records are relatively well predicted from a source model derived from the teleseismic data using the fault geometry derived from the satellite images. This study demonstrates the benefit of using accurate fault geometries to determine finite-fault source models

Slip distribution of the 2017 M(w)6.6 Bodrum-Kos earthquake: resolving the ambiguity of fault geometry

SUMMARY The 2017 July 20, Mw6.6 Bodrum–Kos earthquake occurred in the Gulf of Gökova in the SE Aegean, a region characterized by N–S extension in the backarc of the easternmost Hellenic Trench. The dip direction of the fault that ruptured during the earthquake has been a matter of controversy where both north- and south-dipping fault planes were used to model the coseismic slip in previous studies. Here, we use seismic (seismicity, main shock modelling, aftershock relocations and aftershock mechanisms using regional body and surface waves), geodetic (GPS, InSAR) and structural observations to estimate the location, and the dip direction of the fault that ruptured during the 2017 earthquake, and the relationship of this event to regional tectonics. We consider both dip directions and systematically search for the best-fitting locations for the north- and south-dipping fault planes. Comparing the best-fitting planes for both dip directions in terms of their misfit to the geodetic data, proximity to the hypocenter location and Coulomb stress changes at the aftershock locations, we conclude that the 2017 earthquake ruptured a north-dipping fault. We find that the earthquake occurred on a 20–25 km long, ∼E–W striking, 40° north-dipping, pure normal fault with slip primarily confined between 3 and 15 km depth, and the largest slip exceeding 2 m between depths of 4 and 10 km. The coseismic fault, not mapped previously, projects to the surface within the western Gulf, and partly serves both to widen the Gulf and separate Kos Island from the Bodrum Peninsula of SW Anatolia. The coseismic fault may be an extension of a mapped, north-dipping normal fault along the south side of the Gulf of Gökova. While all of the larger aftershocks are consistent with N–S extension, their spatially dispersed pattern attests to the high degree of crustal fracturing within the basin, due to rapid trenchward extension and anticlockwise rotation within the southeastern Aegean

The effect of humic acid substances on the thyroid function and structure in lead poisoning

Lead (Pb) is a heavy metal, which adversely affects thyroid gland function and structure. Due to its high molecular weight and abundant functional groups, humic acid substances (HAS) can form chelates with heavy metals. The experiment was conducted to evaluate the prophylactic effect of HAS on thyroid hormone levels and histopathological lesions of laying hens exposed to lead (Pb) poisoning. After a week of adaptation, 192 Lohmann White laying hens (25 weeks of age) were fed one of four diets: a basal diet (BD) or the BD with HAS (0.15%), with Pb (0.3 g/kg), or with both. Experimental groups were replicated in 12 cages, with four hens each. Pb poisoning did not alter triiodothyronine (FT3; 3.22 ± 0.20 ng/dL) or thyroxine (FT4; 0.71 ± 0.08 ng/dL) concentrations, but caused a 167% increase in thyroid stimulating hormone (TSH) concentration. HAS supplementation returned the high TSH levels of hens exposed to Pb poisoning to normal values. Degenerative changes in the epithelial cells of the thyroid gland of the hens exposed to Pb poisoning were evidenced. Connective tissue cells in the interfollicular area and total amount of colloids with partially atrophic follicles were observed. These histopathological findings were less severe when HAS was added to the diet. In conclusion, HAS alleviates the effects of Pb poisoning on thyroid gland function and structure, possibly preventing its internalization by the tissue by forming chelates and exerting anti-inflammatory effects

Source model of the 2007 M_w 8.0 Pisco, Peru earthquake: Implications for seismogenic behavior of subduction megathrusts

We use Interferometric Synthetic Aperture Radar, teleseismic body waves, tsunami waveforms recorded by tsunameters, field observations of coastal uplift, subsidence, and runup to develop and test a refined model of the spatiotemporal history of slip during the M_w 8.0 Pisco earthquake of 15 August 2007. Our preferred solution shows two distinct patches of high slip. One patch is located near the epicenter while another larger patch ruptured 60 km further south, at the latitude of the Paracas peninsula. Slip on the second patch started 60 s after slip initiated on the first patch. We observed a remarkable anticorrelation between the coseismic slip distribution and the aftershock distribution determined from the Peruvian seismic network. The proposed source model is compatible with regional runup measurements and open ocean tsunami records. From the latter data set, we identified the 12 min timing error of the tsunami forecast system as being due to a mislocation of the source, caused by the use of only one tsunameter located in a nonoptimal azimuth. The comparison of our source model with the tsunami observations validate that the rupture did not extend to the trench and confirms that the Pisco event is not a tsunami earthquake despite its low apparent rupture velocity (<1.5 km/s). We favor the interpretation that the earthquake consists of two subevents, each with a conventional rupture velocity (2–4 km/s). The delay between the two subevents might reflect the time for the second shock to nucleate or, alternatively, the time it took for afterslip to increase the stress level on the second asperity to a level necessary for static triggering. The source model predicts uplift offshore and subsidence on land with the pivot line following closely the coastline. This pattern is consistent with our observation of very small vertical displacement along the shoreline when we visited the epicentral area in the days following the event. This earthquake represents, to our knowledge, one of the best examples of a link between the geomorphology of the coastline and the pattern of surface deformation induced by large interplate ruptures

Rupture Kinematics of the 2005 M_w 8.6 Nias–Simeulue Earthquake from the Joint Inversion of Seismic and Geodetic Data

The 2005 M_w 8.6 Nias–Simeulue earthquake was caused by rupture of a portion of the Sunda megathrust offshore northern Sumatra. This event occurred within an array of continuous Global Positioning System (GPS) stations and produced measurable vertical displacement of the fringing coral reefs above the fault rupture. Thus, this earthquake provides a unique opportunity to assess the source characteristics of a megathrust event from the joint analysis of seismic data and near-field static co-seismic displacements. Based on the excitation of the normal mode data and geodetic data we put relatively tight constraints on the seismic moment and the fault dip, where the dip is determined to be 8° to 10° with corresponding moments of 1.24 x 10^(22) to 1.00 x 10^(22) N m, respectively. The geodetic constraints on slip distribution help to eliminate the trade-off between rupture velocity and slip kinematics. Source models obtained from the inversion of various combinations of the teleseismic body waves and geodetic data are evaluated by comparing predicted and observed long-period seismic waveforms (100–500 sec). Our results indicate a relatively slow average rupture velocity of 1.5 to 2.5 km/sec and long average rise time of up to 20 sec. The earthquake nucleated between two separate slip patches, one beneath Nias and the other beneath Simeulue Island. The gap between the two patches and the hypocentral location appears to be coincident with a local geological disruption of the forearc. Coseismic slip clearly tapers to zero before it reaches the trench probably because the rupture propagation was inhibited when it reached the accretionary prism. Using the models from joint inversions, we estimate the peak ground velocity on Nias Island to be about 30 cm/sec, an order of magnitude slower than for thrust events in continental areas. This study emphasizes the importance of utilizing multiple datasets in imaging seismic ruptures

Partial rupture of a locked patch of the Sumatra megathrust during the 2007 earthquake sequence

The great Sumatra–Andaman earthquake and tsunami of 2004 was a dramatic reminder of the importance of understanding the seismic and tsunami hazards of subduction zones [1,2,3,4]. In March 2005, the Sunda megathrust ruptured again, producing an event [5] of moment magnitude (Mw) 8.6 south of the 2004 rupture area, which was the site of a similar event in 1861 (ref. 6). Concern was then focused on the Mentawai area, where large earthquakes had occurred in 1797 (Mw = 8.8) and 1833 (Mw = 9.0) [6,7]. Two earthquakes, one of Mw = 8.4 and, twelve hours later, one of Mw = 7.9, indeed occurred there on 12 September 2007. Here we show that these earthquakes ruptured only a fraction of the area ruptured in 1833 and consist of distinct asperities within a patch of the megathrust that had remained locked in the interseismic period. This indicates that the same portion of a megathrust can rupture in different patterns depending on whether asperities break as isolated seismic events or cooperate to produce a larger rupture. This variability probably arises from the influence of non-permanent barriers, zones with locally lower pre-stress due to the past earthquakes. The stress state of the portion of the Sunda megathrust that had ruptured in 1833 and 1797 was probably not adequate for the development of a single large rupture in 2007. The moment released in 2007 amounts to only a fraction both of that released in 1833 and of the deficit of moment that had accumulated as a result of interseismic strain since 1833. The potential for a large megathrust event in the Mentawai area thus remains large

Neonatal tetanus in Turkey; what has changed in the last decade?

dikici, bunyamin/0000-0001-7572-6525WOS: 000259222800001PubMed: 18713452Background: Neonatal tetanus (NT) is still considered as one of the major causes of neonatal death in many developing countries. The aim of the present study was to assess the characteristics of sixty-seven infants with the diagnosis of neonatal tetanus followed-up in the Pediatric Infectious Diseases Ward of Dicle University Hospital, Diyarbakir, between 1991 and 2006, and to draw attention to factors that may contribute (or may have contributed) to the elimination of the disease in Diyarbakir. Methods: The data of sixty-seven infants whose epidemiological and clinical findings were compatible with neonatal tetanus were reviewed. Patients were stratified into two groups according to whether they survived or not to assess the effect of certain factors in the prognosis. Factors having a contribution to the higher rate of tetanus among newborn infants were discussed. Results: A total of 55 cases of NT had been hospitalized between 1991 and 1996 whereas only 12 patients admitted in the last decade. All of the infants had been delivered at home by untrained traditional birth attendants (TBA), and none of the mothers had been immunized with tetanus toxoid during her pregnancy. Twenty-eight (41.8%) of the infants died during their follow-up. Lower birth weight, younger age at onset of symptoms and at the time admission, the presence of opisthotonus, risus sardonicus and were associated with a higher mortality rate. Conclusion: Although the number of neonatal tetanus cases admitted to our clinic in recent years is lower than in the last decade efforts including appropriate health education of the masses, ensurement of access to antenatal sevices and increasing the rate of tetanus immunization among mothers still should be made in our region to achieve the goal of neonatal tetanus elimination