82 research outputs found

    Supersymmetry of the Schrodinger and PP Wave Solutions in Einstein-Weyl Supergravities

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    We obtain the Schrodinger and general pp-wave solutions with or without the massive vector in Einstein-Weyl supergravity. The vector is an auxiliary field in the off-shell supermultiplet and it acquires a kinetic term in the Weyl-squared super invariant. We study the supersymmetry of these solutions and find that turning on the massive vector has a consequence of breaking all the supersymmetry. The Schrodinger and also the pp-wave solutions with the massive vector turned off on the other hand preserve 1/4 of the supersymmetry.Comment: 13 pages, no figur

    Nuclear Physics without High-Momentum Potentials: Constructing the Nuclear Effective Interaction Directly from Scattering Observables

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    The traditional approach to nuclear physics encodes phase shift information in a nucleon-nucleon (NN) potential, producing a nucleon-level interaction that captures the sub-GeV consequences of QCD. A further reduction to the nuclear scale is needed to produce an effective interaction for soft Hilbert spaces, such as those employed in the shell model. Here we describe an alternative construction of this effective interaction, from QCD directly to the nuclear scale, that is direct and precise. This eliminates the need for constructing and renormalizing the high-momentum NN potential. Instead, continuum phase shifts and mixing angles are used directly at the nuclear scale. The method exploits the analytic continuity in energy of HOBET (Harmonic-Oscillator-Based Effective Theory) to connect bound states to continuum solutions at specific energies. The procedure is systematic, cutoff independent, and convergent, yielding keV accuracy at NNLO or N3^3LO, depending on the channel. Lepage plots are provided.Comment: 9 page

    SO(2,1) conformal anomaly: Beyond contact interactions

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    The existence of anomalous symmetry-breaking solutions of the SO(2,1) commutator algebra is explicitly extended beyond the case of scale-invariant contact interactions. In particular, the failure of the conservation laws of the dilation and special conformal charges is displayed for the two-dimensional inverse square potential. As a consequence, this anomaly appears to be a generic feature of conformal quantum mechanics and not merely an artifact of contact interactions. Moreover, a renormalization procedure traces the emergence of this conformal anomaly to the ultraviolet sector of the theory, within which lies the apparent singularity.Comment: 11 pages. A few typos corrected in the final versio

    Genome-wide analysis of 102,084 migraine cases identifies 123 risk loci and subtype-specific risk alleles

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    Genome-wide association analyses identify 123 susceptibility loci for migraine and implicate neurovascular mechanisms in its pathophysiology. Subtype analyses highlight risk loci specific for migraine with or without aura in addition to shared risk variants.Migraine affects over a billion individuals worldwide but its genetic underpinning remains largely unknown. Here, we performed a genome-wide association study of 102,084 migraine cases and 771,257 controls and identified 123 loci, of which 86 are previously unknown. These loci provide an opportunity to evaluate shared and distinct genetic components in the two main migraine subtypes: migraine with aura and migraine without aura. Stratification of the risk loci using 29,679 cases with subtype information indicated three risk variants that seem specific for migraine with aura (in HMOX2, CACNA1A and MPPED2), two that seem specific for migraine without aura (near SPINK2 and near FECH) and nine that increase susceptibility for migraine regardless of subtype. The new risk loci include genes encoding recent migraine-specific drug targets, namely calcitonin gene-related peptide (CALCA/CALCB) and serotonin 1F receptor (HTR1F). Overall, genomic annotations among migraine-associated variants were enriched in both vascular and central nervous system tissue/cell types, supporting unequivocally that neurovascular mechanisms underlie migraine pathophysiology.Clinical epidemiolog
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